You can attack me for my rep status, which is fine, but that doesn't actually mean you know what you are talking about, nor does it enhance your argument. Here's the truth bigsmall, you don't care about finding the truth, you only care about what you want to believe, even if it is in stark contrast to the evidence. You will dismiss the following simply because it doesn't fit what you think or you'll dismiss it because you don't understand the differences between supplementation of something, and that same thing being released under completely different conditions. So i say it again, if you don't care enough to find the truth, then at least care enough to stop trying to scare people with lies and untruths..
https://www.ncbi.nlm.nih.gov/pubmed/9507233
AA supplementation caused significant increases in the in vitro secretion of LTB4, and PGE2, but it did not alter the in vitro secretion of tumor necrosis factor alpha; interleukins 1 beta, 2, 6;
Hmm funny that. AA supplementation increased the same eiconsaoids as training does, but did not alter the secretion of the markers of inflammation we avoid.
Surely thats a fluke.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2217562/
[On IL-6] In fact, one of the initial theories related to the potential ergogenic value of AA for athletes was that dietary supplementation of AA during training would promote greater localized inflammation thereby stimulating subsequent intramuscular protein synthesis during resting/repair phases between workouts. Nonetheless, present findings do not support this hypothesis. Rather, results suggest that AA supplementation during training may reduce chronic levels of IL-6 thereby reducing associated inflammation that may occur during high volume training. Theoretically, this may allow athletes to tolerate high levels of training to a better degree.
And also:
Beyond these potential limitations, our results suggest that prolonged AA supplementation may increase cycling anaerobic capacity while
reducing circulating IL-6 levels in resistance-trained males. Moreover, that 1.0 g·day-1 of AA administration over a 50 days period significantly increased dietary AA intake and appears to be well-tolerated and exerts no adverse side effects in young, resistance-trained males.
So no, i'd go ahead and say that you have no idea the differences between supplementation of AA vs. its release during diseased states and the effect on the release of eiconsaoids.
Maybe you should just skip working out, after-all it increase the exact same markers.