My joints are fine, but I have never had any joint issues. My left elbow has never liked curls, so I do them by sort of rowing the bar vertically up my body along with hammer curls. I have good core stability and hip/shoulder mobility which (a lack thereof) is the cause of most joint issues anyway
Regarding the previous discussion as well, it's really starting to push the limits of my knowledge and ability to articulate how I feel
So I'll take it down a notch and go back to a more high level view of things
If I play devils advocate and say that consuming a large amount of protein will cause more gluconeogenesis and take me out of ketosis then so what?
What is the impact of this happening
Studies have shown that infusing alanine and other amino acids cause a rise in blood sugar, but no rise in serum insulin - in fact, glucagon rose which would suggest a catabolic state
[video=youtube;z3fO5aTD6JU]https://www.youtube.com/watch?v=z3fO5aTD6JU[/video]
At 9 minutes explains this better than I can!
I've only listened to about 20 minutes of this so far, and it's a good talk for sure. Thanks for that.
I am guessing you are talking about the statement he makes that arginine only spikes insulin in the presence of blood glucose - but this makes perfect sense. As he says in the video, arginine is the prototypical gluconeogenic amino acid. The key here is, it's gluconeogenic - not directly insulinogenic.
Basically, arginine upregulates glucose production - which could in some instances elevate blood sugar, which will thus increase insulin release - so it's indirectly insulogenic.
With that understanding, it would make sense if you already have elevated blood sugar and thus elevated insulin, and then arginine comes along and triggers gluconeogenesis - your body is going to basicallly react with, "Oh Sh1t! Just got word from production that they're about to dump a boatload of glucose in our blood in a minute!!! Pump out more insulin!! Hurry!" And your insulin will shoot up. Now, if you have low blood sugar already - the fact you are about to make some glucose isn't so demanding, so it doesn't require so much insulin, etc.
I could be misinterpreting...would more discussion.
Regardless, if you start creating glucose - regardless of your insulin levels - you have glucose which is much preferred for energy to lipids/ketones. Enough gluconeogenesis and you will have enough glucose that you don't need to be in ketosis anymore and your body will use the glucose.
Having said that - your question, "What does it matter" is really the key. In the grand scheme, it probably does not, unless you're trying to follow a ketogenic diet.
But what most people don't realize is that one of the things that happen in obesity is that carb use is down regulated and we become very good at burning fat stores. If you go into a deficit or even worse - a combo of low carbs and a deficit - you will increase the enzymes that cause this shift from carbs to fat burning - which basically just took one of the issues with obese and made it worse. (this is not just theory)
And then people wonder why they rebound and they think their metabolism magically slowed - but I theorize it has more to do with the fact that we are using fat instead of carbs for energy, which slows weight loss. But this is my own theory and thoughts...
I think gluconeogenesis can be your friend, but it can also knock you out of ketosis.
