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DCP 2.0

dsade said:
I wish I could give a better timeframe, but these ingredients are a huge pain to work with and we get one shot to make the tableting work. If we screw anything up, we have $35k worth of crappy goop.
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If it is that hard why not capsules? You can use 00 or 000 even if it means you don't end up with a $35K loss. I don't know the specifics but that is the first thing that comes to mind to me when I read what you said above.
 
oufinny said:
If it is that hard why not capsules? You can use 00 or 000 even if it means you don't end up with a $35K loss. I don't know the specifics but that is the first thing that comes to mind to me when I read what you said above.
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There is too much heat involved in encapsulation. The product turns into a goo and clogs up the holes in the machine every 200 caps or so.
 
dsade said:
There is too much heat involved in encapsulation. The product turns into a goo and clogs up the holes in the machine every 200 caps or so.
Click to expand...

If those machines are like some of the girls I know, they don't like goo in the holes.
 
jwa254 said:
If those machines are like some of the girls I know, they don't like goo in the holes.
Click to expand...

You know the wrong kind of girls.
 
I remember back in the day, DCP was some large horse capsules that had black liquid in them. When the tablets came, I much preferred them.
 
testosteronet said:
Didn't know heat was involved in encapsulation. Why would this be? I thought capsules were just pressed together?
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It's the actual filling of the capsule.
 
'Scuse me while I whip dis out
 
MidwestBeast said:
Are you suggesting that the DCP 2.0 bottles should include funny fortunes in them? Anabolic fortune cookies?!?!
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That would be hilariously awesome to open DCP and get a fortune. "Confucius say: Get swole or GTFO!"
 
Resolve said:
Wasn't directed at you, that was another 'fortune.' Probably the one I'd get, too, knowing my luck. :D
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Oh I know it wasn't (though now I'm having second thoughts...uhhh....*runs away*). I just meant in general I know such a thing is mean, but I still laughed in the office when I read it. lol
 
ETA?
 
Going on 10 weeks now...
 
::insert cliche here:::
 
First ingredient released is....High Potency Mangiferin

Invalid Link Removed

Invalid Link Removed 2011 Dec;55(12):1809-18. doi: 10.1002/mnfr.201100392. Epub 2011 Oct 31.
[h=1]Beneficial effects of mangiferin on hyperlipidemia in high-fat-fed hamsters.[/h]Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed.
[h=3]Source[/h]Department of Nutrition and Food Hygiene, Public Health College, Harbin Medical University, Harbin, PR China.

[h=3]Abstract[/h][h=4]SCOPE:[/h]Mangiferin, a natural polyphenol, has been shown to have hypolipidemic effect in rat and mouse. However, the mechanism of action is not well understood. This study was conducted to determine the effect and mechanism of action of mangiferin on hyperlipidemia induced in hamsters by a high-fat diet.
[h=4]METHODS AND RESULTS:[/h]Forty male hamsters were randomly assigned to normal control, high-fat control, and high fat with mangiferin (50 and 150 mg/kg BW) groups. Mangiferin treatment significantly decreased final body weight, liver weight and visceral fat-pad weight, serum triglyceride (TG) and total free fatty acid (FFA) concentrations, hepatic TG levels and hepatic and muscle total FFA contents. Mangiferin upregulated mRNA expression of peroxisome proliferator-activated receptor-α (PPAR-α), fatty acid translocase (CD36) and carnitine palmitoyltransferase 1 (CPT-1), but downregulated mRNA expression of sterol regulatory element-binding protein 1c (SREBP-1c), acetyl CoA carboxylase (ACC), acyl-CoA:diacylglycerol acyltransferase 2 (DGAT-2) and microsomal triglyceride transfer protein (MTP) in liver. Mangiferin also stimulated mRNA expression of PPAR-α, CD36, CPT-1 and lipoprotein lipase (LPL) in muscle.
[h=4]CONCLUSIONS:[/h]The results suggest that mangiferin may ameliorate hypertriglyceridemia partly by modulating the expression levels of genes involved in lipid oxidation and lipogenesis.
Copyright © 2011 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim.


PMID:22038976 [PubMed - indexed for MEDLINE]
 
dsade said:
First ingredient released is....High Potency Mangiferin

Invalid Link Removed

Invalid Link Removed 2011 Dec;55(12):1809-18. doi: 10.1002/mnfr.201100392. Epub 2011 Oct 31.
[h=1]Beneficial effects of mangiferin on hyperlipidemia in high-fat-fed hamsters.[/h]Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed.
[h=3]Source[/h]Department of Nutrition and Food Hygiene, Public Health College, Harbin Medical University, Harbin, PR China.

[h=3]Abstract[/h][h=4]SCOPE:[/h]Mangiferin, a natural polyphenol, has been shown to have hypolipidemic effect in rat and mouse. However, the mechanism of action is not well understood. This study was conducted to determine the effect and mechanism of action of mangiferin on hyperlipidemia induced in hamsters by a high-fat diet.
[h=4]METHODS AND RESULTS:[/h]Forty male hamsters were randomly assigned to normal control, high-fat control, and high fat with mangiferin (50 and 150 mg/kg BW) groups. Mangiferin treatment significantly decreased final body weight, liver weight and visceral fat-pad weight, serum triglyceride (TG) and total free fatty acid (FFA) concentrations, hepatic TG levels and hepatic and muscle total FFA contents. Mangiferin upregulated mRNA expression of peroxisome proliferator-activated receptor-α (PPAR-α), fatty acid translocase (CD36) and carnitine palmitoyltransferase 1 (CPT-1), but downregulated mRNA expression of sterol regulatory element-binding protein 1c (SREBP-1c), acetyl CoA carboxylase (ACC), acyl-CoA:diacylglycerol acyltransferase 2 (DGAT-2) and microsomal triglyceride transfer protein (MTP) in liver. Mangiferin also stimulated mRNA expression of PPAR-α, CD36, CPT-1 and lipoprotein lipase (LPL) in muscle.
[h=4]CONCLUSIONS:[/h]The results suggest that mangiferin may ameliorate hypertriglyceridemia partly by modulating the expression levels of genes involved in lipid oxidation and lipogenesis.
Copyright © 2011 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim.


PMID:22038976 [PubMed - indexed for MEDLINE]
Click to expand...


And so it begins!
 
This looks very good. I cannot wait to see how the people respond to this. Almost looks like a tri-monthly staple, taken for 12 weeks ;).
 
Carried over from original DCP...

Invalid Link Removed 2012 Mar;35(3):481-6. doi: 10.1007/s12272-012-0311-8. Epub 2012 Apr 5.
[h=1]Bioassay-guided isolation of fatty acid synthase inhibitory diterpenoids from the roots of Salvia miltiorrhiza Bunge.[/h]Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed.
[h=3]Source[/h]College of Medicine, CHA University, Seoul 135-080, Korea.

[h=3]Abstract[/h]Fatty acid synthase (FAS) is considered as a novel drug target for the development of anticancer and anti-obesity agents. Bioassay-guided fractionation of a n-hexane-soluble extract prepared from the roots of Salvia miltiorrhiza Bunge (Labiatae), using an in vitro enzyme assay, led to the isolation of five abietane diterpenoids: 15,16-dihydrotanshinone I (1), cryptotanshinone (2), tanshinone I (3), tanshinone IIA (4), and dansenspiroketallactone (5). Compounds 1-5 were tested for their in vitro FAS inhibitory activity and, except for compound 5 (IC(50) > 100 μM), compounds 1-4 inhibited the enzyme activity with IC(50) values ranging from 12.0 to 30.3 μM. Our findings may be partially related to the anticancer activity of abietane diterpenoids from the plant, suggesting a further study on the anticancer potential of tanshinone derivatives.


PMID:22477195 [PubMed - indexed for MEDLINE]
 
Invalid Link Removed 2002 Aug;25(4):446-8.
[h=1]Inhibitory activity of diacylglycerol acyltransferase by tanshinones from the root of Salvia miltiorrhiza.[/h]Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed.
[h=3]Source[/h]Cardiovascular Research Laboratory, Korea Research Institute of Bioscience and Biotechnology, Yusong, Taejon.

[h=3]Abstract[/h]The inhibitory activity of tanshinones from Salvia miltiorrhiza was tested on rat liver diacylglycerol acyltransferase (DGAT). Cryptotanshinone (1) and 15,16-dihydrotanshinone I (3) exhibited potent DGAT inhibitory activities dose-dependently with IC50 values of 10.5 microg/ml and 11.1 microg/ml. However, tanshinone IIA (2) and tanshinone I (4) showed very weak inhibition (IC50 value: > 250 microg/ml). A dihydrofuran moiety was seemed to be responsible for the stronger inhibitory activity.


PMID:12214853 [PubMed - indexed for MEDLINE]
 
That's all for now....We still have around 3 weeks production time, and last thing we need is to be copied. Though, to be honest, we are taking a pretty small profit margin on this product to make sure you have the most badass, and affordable, Recomposition Ammunition we can make.

Most other companies wouldn't waste the time.
 
dsade said:
That's all for now....We still have around 3 weeks production time, and last thing we need is to be copied. Though, to be honest, we are taking a pretty small profit margin on this product to make sure you have the most badass, and affordable, Recomposition Ammunition we can make.

Most other companies wouldn't waste the time.
Click to expand...

Good stuff as always, Matt! I am a huge fan of your work and the original, so I am really looking forward to trying the new formula. I guess you could say I'm a fan:


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