Witch’s Log

Just added 10mcg T3 in the AM and will adjust as needed, likely will be AM and some few hours later both 10-15mcg.
 
Old Witch said:
Just added 10mcg T3 in the AM and will adjust as needed, likely will be AM and some few hours later both 10-15mcg.
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Why so little? I thought a healthy thyroid put out about 30mcg. Ive gotten the impression there wasnt much point in going much less than 50.
 
jrock645 said:
Why so little? I thought a healthy thyroid put out about 30mcg. Ive gotten the impression there wasnt much point in going much less than 50.
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You are correct, a healthy thyroid produces 25-30mcg of t3 daily. I personally go 75-100mcg if you actually want to loose fat, anything less than 50 is mostly maintenance imo. If you're on anabolics you won't loose muscle especially if you keep protein intake up. At 25mcg he will maintain thyroid levels if that's his objective.
 
jrock645 said:
Why so little? I thought a healthy thyroid put out about 30mcg. Ive gotten the impression there wasnt much point in going much less than 50.
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Yes. I have an under active thyroid. And it’s going up slowly. First two weeks, it’s 20-30mcg.
 
mixedup said:
It all depends on the cook. I use to home brew 100mg/ml and no pip
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The stuff I have is good. Very minimal pip if it’s close to a nerve or blood vessel, no pip otherwise. 100mg/ml. Came in 20ml vials too so it all equals out vs other more concentrated brewers. Smart move I think.

I will be using this same source for as long as I can, for sure.
 
Old Witch said:
The stuff I have is good. Very minimal pip if it’s close to a nerve or blood vessel, no pip otherwise. 100mg/ml. Came in 20ml vials too so it all equals out vs other more concentrated brewers. Smart move I think.

I will be using this same source for as long as I can, for sure.
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His 300mg/ml test has zero pip practically.
Pinned it with 125mg/ml NPP and was pretty baffled how smooth two high potency oils were
 
Matthersby said:
His 300mg/ml test has zero pip practically.
Pinned it with 125mg/ml NPP and was pretty baffled how smooth two high potency oils were
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Yeah I am pretty impressed with the quality of it. I get no pip whatsoever not even the welt from that test E. It absorbs fast, for me.

Better than pharma IMO.
 
And, yeah, I’m not really cutting. I was just kind of joking a bit, but I am cleaning things up and did add the T3 to speed up my metabolic rate and cheat off some fat cells with the test and dhb while I keep gaining weight, while also in a caloric deficit.

I know, it sounds like I just broke reality with that statement but that’s how my body works when it’s tuned into the drugs, diet , and training.

It will burn fat for fuel and begin to build muscle out of all consumed calories.

So really it’s a huge caloric surplus, because in that state 0kcal per day is maintenance for me. Technically.
 
Ended up taking two extra rest days because of some car trouble. Busses don’t run at 1AM.

Will be on chest and upper back tonight.

Dumbbell Incline Press 4x12
Barbell Incline Press 4x8
Machine chest press 3x12
Cable Crossover 3x20
Pulldown 4x12
Single Hand Seated Cable Row 4x8
Chest Supported Barbell Row 4x12
Pullover 4x12
Cable Reverse Flye 3x20

Pinned my mix 1 ml test E 300, 2ml DHB Cyp 100. Last night (3AM) and took 0.5mg PRO liquid Adex directly after.

Slept til about an hour ago. Taking my morning pills and liquids now.

600mg Swanson Nac
120mg Swanson CoQ10
50mg ugl Proviron
20mg ugl Cardarine
1mg Jarrow Methylcobalamin
100mcg NOW Menaquinone 7
20mcg PRO T3
 
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...while I keep gaining weight, while also in a caloric deficit...
....It will burn fat for fuel and begin to build muscle out of all consumed calories. ...

:) Hehehe! Chasing the holy grail, are we?

Sounds like magic.

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hairygrandpa said:
...while I keep gaining weight, while also in a caloric deficit...
....It will burn fat for fuel and begin to build muscle out of all consumed calories. ...

:) Hehehe! Chasing the holy grail, are we?

Sounds like magic.

Invalid Link Removed
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I’m using my sensitivity to the compounds while I still have it.

Being in a deficit mobilizes fat stores, being in high muscular demand spares eaten calories and the delta 1 steroids command the process recruit satellite cells for assimilation as muscle tissue rapidly.

It’s all very scientific. 😁
 
Old Witch said:
I’m using my sensitivity to the compounds while I still have it.

Being in a deficit mobilizes fat stores, being in high muscular demand spares eaten calories and the delta 1 steroids command the process recruit satellite cells for assimilation as muscle tissue rapidly.

It’s all very scientific. 😁
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All very cool my friend. I’m in a deficit too. However I take in enough carbs to run and enough protein and roids to maintain and build some muscle........my fat stores take care of the rest 🙄🙄🙄🙄
 
Old Witch said:
I’m using my sensitivity to the compounds while I still have it.

Being in a deficit mobilizes fat stores, being in high muscular demand spares eaten calories and the delta 1 steroids command the process recruit satellite cells for assimilation as muscle tissue rapidly.

It’s all very scientific.
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App up vote +.
 
Blood is VERY bright red today so that means HGB is going up. Clotting is happening very fast so RBC totals must be going up.
 
So, the water from M1A fell off and I’m sitting at 226lbs and slightly leaner than I was.

So I gained 15 lbs of muscle, 11lbs of water, and the “5lbs fat gain” was really “1lb fat loss”

The “you got fat” was just pure insanity. My traps are apparently not a muscle, but a fatty neck roll... until she actually touched it.

I’m not getting fat. I didn’t get fat. I did get bloated from mk677 though.

Well, I was already fat.
 
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Oh, and I’m up to 40mcg T3. I decided not to ramp up so slowly. Will max out at 60 most likely.
 
Also, hey, did anyone know that T2 and T3 will cause cardiac hypertrophy after only 4 weeks?

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183150
 
Cardiac hypertrophy unless accompanied by exercises isn’t a good thing. Thinking of the heart is generally bad unless it’s thicker because of exercise (as this generates more mitochondria etc and better aerobic and anaerobic pathway metabolisms). Not sure exactly why but a hypertrophied heart from exercise is different than a hypertrophied heart via steroids etc. possibly due to the different type of tissue build up. Exercise is more gradual so there may be less collagen build up. Steroids have been known to cause the heart to build up more collagen which leads to a bunch of issues down the road.

Steroids often cause inward hypertrophy where the heart DEcreases LV chamber size where as exercise tends to promote a more outward thickening. Possibly because the heart has to deal with increased blood volume associated with EPO production in bone marrow as well as increased fluid retention.


Quick google search yielded this

“Pathological cardiac hypertrophy is associated with poor prognosis and is a hallmark of heart failure (72, 73, 103). In contrast, exercise training-induced physiological cardiac hypertrophy presents cardioprotective effects and is not related to heart failure”

Same article
“LV remodeling induced by pathological stress leads to progressive declines in cardiac output, myocardial rarefaction, increased apoptosis, cardiomyocyte metabolism switch from fatty acid to glucose use, and increased fibrosis (35, 49).”

Got the above from here
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ChocolateClen said:
Cardiac hypertrophy unless accompanied by exercises isn’t a good thing. Thinking of the heart is generally bad unless it’s thicker because of exercise (as this generates more mitochondria etc and better aerobic and anaerobic pathway metabolisms). Not sure exactly why but a hypertrophied heart from exercise is different than a hypertrophied heart via steroids etc. possibly due to the different type of tissue build up. Exercise is more gradual so there may be less collagen build up. Steroids have been known to cause the heart to build up more collagen which leads to a bunch of issues down the road.

Steroids often cause inward hypertrophy where the heart DEcreases LV chamber size where as exercise tends to promote a more outward thickening. Possibly because the heart has to deal with increased blood volume associated with EPO production in bone marrow as well as increased fluid retention.


Quick google search yielded this

“Pathological cardiac hypertrophy is associated with poor prognosis and is a hallmark of heart failure (72, 73, 103). In contrast, exercise training-induced physiological cardiac hypertrophy presents cardioprotective effects and is not related to heart failure”

Same article
“LV remodeling induced by pathological stress leads to progressive declines in cardiac output, myocardial rarefaction, increased apoptosis, cardiomyocyte metabolism switch from fatty acid to glucose use, and increased fibrosis (35, 49).”

Got the above from here
Invalid Link Removed
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Yeah the cholesterol and heart effects of these compounds worry me more than other sides..
 
Interesting...

I might have another infection. This time there’s nothing that could have done to contaminate a vial, only my bad technique. I’m certain of that.

Might not be an infection. Will see tomorrow for sure though.
 
ChocolateClen said:
Cardiac hypertrophy unless accompanied by exercises isn’t a good thing. Thinking of the heart is generally bad unless it’s thicker because of exercise (as this generates more mitochondria etc and better aerobic and anaerobic pathway metabolisms). Not sure exactly why but a hypertrophied heart from exercise is different than a hypertrophied heart via steroids etc. possibly due to the different type of tissue build up. Exercise is more gradual so there may be less collagen build up. Steroids have been known to cause the heart to build up more collagen which leads to a bunch of issues down the road.

Steroids often cause inward hypertrophy where the heart DEcreases LV chamber size where as exercise tends to promote a more outward thickening. Possibly because the heart has to deal with increased blood volume associated with EPO production in bone marrow as well as increased fluid retention.


Quick google search yielded this

“Pathological cardiac hypertrophy is associated with poor prognosis and is a hallmark of heart failure (72, 73, 103). In contrast, exercise training-induced physiological cardiac hypertrophy presents cardioprotective effects and is not related to heart failure”

Same article
“LV remodeling induced by pathological stress leads to progressive declines in cardiac output, myocardial rarefaction, increased apoptosis, cardiomyocyte metabolism switch from fatty acid to glucose use, and increased fibrosis (35, 49).”

Got the above from here
Invalid Link Removed
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Right. I meant it as a bad thing.
 
ChocolateClen said:
Ahhh my bad, I thought you were like “big heart? AWESOME! Can’t wait to get swole everywhere” haha. My apologies bro. It was a late night when I typed that up
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Nope, I want smooth and unremarkable cardiac tissue when I’m on the coroner’s block. I’m doing the math and it seems like it’s a LOT of T3 it takes to do this kind of permanent damage, they were saying the metabolism increase was permanent as well at that high a dose.

Crazy stuff.
 
Old Witch said:
Nope, I want smooth and unremarkable cardiac tissue when I’m on the coroner’s block. I’m doing the math and it seems like it’s a LOT of T3 it takes to do this kind of permanent damage, they were saying the metabolism increase was permanent as well at that high a dose.

Crazy stuff.
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Perm cardiac damage would cause a metabolic increase, probably a lot of cellular changes through the body though. Depends on how high the dose needed is but I assume it’s either toxicity related or it’s dose dependent for some other reason. If it’s not toxicity then I think it would be safe to say that at any dose you’re going to have some changes regardless
 
Buuuuut... it could be halotestin or anavar or superdrol or winstrol or injectable MSten or...
 
ChocolateClen said:
I wouldn’t touch it with a 20 foot stick personally
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Wouldn’t touch what? T3? T2 they showed in that study was for certain the much more harmful chemical. More negative feedback per mcg and less activity at the thyroid hormone receptors.

And, I’m only on it for about a month, come off a month, on another month. Then this blast part of the cycle will be over and I’ll be cruising on 100mg test U a week and hitting some mod grf and hexarelin preworkout and keeping in the mk677 and proviron etc. for a couple of months before I hit the next big blast. By the time the Test U builds up to a blast dose I’ll be back on enanthate and DHB cyp, hopefully proviron still and primobolan as well this time... and phera.

I’ve got everything but the peptides. Got the mk, need IGF, GH, Hex, Mod GRF, cjc dac...
 
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Best 2 roids I did in the past were pheraplex and epistane. Did superdrol, s-drol, and it did not compare to pheraplex or epi.
 
ValiantThor08 said:
Best 2 roids I did in the past were pheraplex and epistane. Did superdrol, s-drol, and it did not compare to pheraplex or epi.
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Winstrol knocks epistane right out of the running. I have winstrol. And anavar. Real anavar.

Phera was great. I have some. Planning to use it fairly soon.
 
Old Witch said:
Winstrol knocks epistane right out of the running. I have winstrol. And anavar. Real anavar.

Phera was great. I have some. Planning to use it fairly soon.
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If you have already used a bulking compound, it would be best to take the mild steroid for 4 to 6 weeks, then hit 5he phera, so that it is more pronounced. That is a feel good steroid, and the pumps are insane.
 
ValiantThor08 said:
If you have already used a bulking compound, it would be best to take the mild steroid for 4 to 6 weeks, then hit 5he phera, so that it is more pronounced. That is a feel good steroid, and the pumps are insane.
Click to expand...

I have anavar and winstrol for the end of this blast. Unless I’m not all that lean. In which case I’ll be using superdrol for that.

What comes in 3 weeks will be strong, it will be a bulking steroid, it won’t be phera. The phera is coming into use in about five-six months from now after my break.

I have a big stash for a bit of a long crazy pair of blasts complete with kickstarts, finishers, and surprise additions.
 
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