Jan's BloodTest April13/2007

JanSz

JanSz

Well-known member
Awards
1
  • Established
The Private MD difference:
Confidential: No doctor visit needed; we provide the lab order
Convenient: Choose the lab location best for you.....near home, near work or one convenient during travel
Affordable: No hidden charges, taxes or draw fees for blood or other lab tests

Online blood testing lab tests, STD testing blood tests lab test by Private MD

Estradiol $47.99
Estradiol, Sensitive $79.99

----------------------------------------------------
Estradiol, Sensitive

Description: This sensitive estradiol assay is designed for the investigation of infertility, particularly in situations where low estradiol levels can be expected. The analytic range of the assay is appropriate for the assessment of the low levels of estradiol typically observed in men, prepubertal girls, and postmenopausal women.
--------------------------------
http://www.privatemdlabs.com/lab_tests.php?view=all&show=1246&category=12&search=#1246
Dehydroepiandrosterone (DHEA) -------- $68.99
Dehydroepiandrosterone (DHEA) Sulfate -------- $68.99
Dihydrotestosterone (DHT) -------- $119.99
Estradiol, Sensitive -------- $68.49
Growth Hormone, Urine 24 Hour -------- $199.49
Hemochromatosis Screen (Comprehensive) -------- $349.99
Insulin-Like Growth Factor I (IGF-I) -------- $69.49
Male Ultimate Anti-Aging Panel -------- $299.99
Pregnenolone -------- $98.49
Progesterone -------- $44.49
Sex Hormone binding Globulin, Serum -------- $49.49
Testosterone, Free and Weakly Bound -------- $89.49
Thyroid TSH w/ Free T4 & Free T3 -------- $78.49

--------------------------------
Holistic Doctor Gynecologist Thyroid NYC New York - Weight Loss & First Line Therapy

Patients Medical, PC.
800 Second Avenue, Suite 900
New York, NY 10017
Phone: 212-679-9667
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Life-flo - Progesta-Care Men's Formulaâ„¢ 3oz

Progesta-Care Men's Formula™ 3oz



Life-flo - Pregnenoloneâ„¢
Pregnenolone™
Pregnenolone Natural Pregnenolone Cream

================================================================================
Inhouse Pharmacy Prometrium, Micronized progesterone, Micronized progesterone capsules, natural progesterone
Microgest 100mg 50 Capsules US $40.00 0.008
Microgest 100mg 100 Capsules US $70.00 0.007
Microgest 200mg 50 Capsules US $48.00 0.0048
Microgest 200mg 100 Capsules US $80.00 0.004


Prometrium 100mg 30 Capsules (ITL) US $36.00 0.012
Prometrium 100mg 60 Capsules (ITL) US $59.00 0.009833333
Prometrium 100mg 90 Capsules (ITL) US $81.00 0.009


Nutrient Search – NutritionData.com
=========================================
Progesta-Care Men's Formula™ 3oz
949695 Being Processed Wed, Oct 1, 2008 $42.94 $42.94*




============================================
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
A4M :: Conference Library

Thierry Hertoghe, MD

PC01d - Adult Growth Deficiences Treatments
$20.00 Purchase Conference: A4M Orlando 2007
Speaker: Thierry Hertoghe, MD
Date/Time: April 26, 2007 6:00 pm - 7:00 pm
Length: 40m 25s - 124 Slides



PC01d - Cortisone
$20.00 Purchase Conference: A4M Las Vegas 2006
Speaker: Thierry Hertoghe, MD
Date/Time: December 7, 2006 6:00 pm - 7:00 pm
Length: 55m 31s - 175 Slides



PC01g - Cortisone
$20.00 Purchase Conference: A4M Orlando 2007
Speaker: Thierry Hertoghe, MD
Date/Time: April 26, 2007 6:00 pm - 7:00 pm
Length: 48m 58s - 261 Slides



PC01g - Cortisone
$20.00 Purchase Conference: A4M Chicago 2007
Speaker: Thierry Hertoghe, MD
Date/Time: August 2, 2007 6:00 pm - 5:30 pm
Length: 57m 20s - 175 Slides



PC01c - DHEA - The Mother Hormone
$20.00 Purchase Conference: A4M Chicago 2007
Speaker: Thierry Hertoghe, MD
Date/Time: August 2, 2007 6:00 pm - 5:30 pm
Length: 32m 01s - 87 Slides



GS02a - Eye Opener: Progesterone Therapy in Men
$20.00 Purchase Conference: A4M Las Vegas 2006
Speaker: Thierry Hertoghe, MD
Date/Time: December 9, 2006 6:00 pm - 4:10 pm
Length: 38m 22s - 158 Slides



PC04d - Female Hormones
$20.00 Purchase Conference: A4M Chicago 2006
Speaker: Thierry Hertoghe, MD
Date/Time: July 14, 2006 6:00 pm - 5:30 pm
Length: 01h 00m 18s - 224 Slides



PC05Bb - Hormone Balance to Intimacy Health and Quality of Life
$16.00 Purchase Conference: A4M Las Vegas 2006
Speaker: Thierry Hertoghe, MD
Date/Time: December 7, 2006 6:00 pm - 5:30 pm
Length: 22m 07s - 1 Slide



GS01c - How Efficient are Melatonin, Growth Hormone, Thyroid, DHEA and Estrogen Corrective Therapies to Prevent or Reverse Coronary Insufficienty: The Data
$16.00 Purchase Conference: A4M Chicago 2006
Speaker: Thierry Hertoghe, MD
Date/Time: July 15, 2006 6:00 pm - 7:30 pm
Length: 32m 02s - 1 Slide



GS01h - How to Improve Hormone Levels by Positive Psychological Attitudes: The Scientific Data
$20.00 Purchase Conference: A4M Chicago 2007
Speaker: Thierry Hertoghe, MD
Date/Time: August 3, 2007 6:00 pm - 4:00 pm
Length: 44m 28s - 184 Slides



PC01a - Introduction to Treating Adult Hormone Deficiency
$20.00 Purchase Conference: A4M Chicago 2007
Speaker: Thierry Hertoghe, MD
Date/Time: August 2, 2007 6:00 pm - 5:30 pm
Length: 01h 00m 14s - 308 Slides



PC01a - Introduction to Treating Adult Hormone Deficiency
$20.00 Purchase Conference: A4M Las Vegas 2007
Speaker: Thierry Hertoghe, MD
Date/Time: December 12, 2007 6:00 pm - 6:00 pm
Length: 57m 18s - 309 Slides



PC01a - Introduction to Treating Hormone Deficiency
$16.00 Purchase Conference: A4M Las Vegas 2006
Speaker: Thierry Hertoghe, MD
Date/Time: December 7, 2006 6:00 pm - 7:00 pm
Length: 01h 07m 51s - 1 Slide



PC01a - Introduction to Treating Hormone Deficiency
$20.00 Purchase Conference: A4M Orlando 2007
Speaker: Thierry Hertoghe, MD
Date/Time: April 26, 2007 6:00 pm - 7:00 pm
Length: 01h 04m 24s - 364 Slides



PC04a - Introduction to Treating Hormone Deficiency: The ABC's with Tips on How to Boost their Safety and Efficacy
$20.00 Purchase Conference: A4M Chicago 2006
Speaker: Thierry Hertoghe, MD
Date/Time: July 14, 2006 6:00 pm - 5:30 pm
Length: 01h 03m 28s - 469 Slides



EW03 - Live Consultation
$20.00 Purchase Conference: A4M Orlando 2007
Speaker: Thierry Hertoghe, MD
Date/Time: April 26, 2007 6:00 pm - 8:00 pm
Length: 02h 08m 16s - 11 Slides



PC02B - Office Microscopy: Basics in Office Methods of Blood and Saliva Microscopic Diagnostics
$20.00 Purchase Conference: A4M Chicago 2006
Speakers: Nick Delgado, PhD , Thierry Hertoghe, MD
Date/Time: July 14, 2006 6:00 pm - 5:00 pm
Length: 01h 50m 02s - 66 Slides



EW05a - Questions & Answers - Hormone Therapy Problems
$16.00 Purchase Conference: A4M Chicago 2007
Speaker: Thierry Hertoghe, MD
Date/Time: August 2, 2007 6:00 pm - 7:00 pm
Length: 01h 21m 24s - 1 Slide



PC01k - Questions and Answers
$20.00 Purchase Conference: A4M Las Vegas 2006
Speaker: Thierry Hertoghe, MD
Date/Time: December 7, 2006 6:00 pm - 7:00 pm
Length: 01h 08m 04s - 11 Slides



PC01b - Testosterone - Andropause and Sexual Health: DHEA - The Mother Hormone
$20.00 Purchase Conference: A4M Chicago 2007
Speaker: Thierry Hertoghe, MD
Date/Time: August 2, 2007 6:00 pm - 5:30 pm
Length: 57m 03s - 114 Slides



PC01b - Testosterone: For Men and Women and Sexual Health
$20.00 Purchase Conference: A4M Las Vegas 2007
Speaker: Thierry Hertoghe, MD
Date/Time: December 12, 2007 6:00 pm - 6:00 pm
Length: 59m 59s - 195 Slides



GS03b - The Psychological Attitudes of Centenarians and their Hormonal Roots
$20.00 Purchase Conference: A4M Las Vegas 2005
Speaker: Thierry Hertoghe, MD
Date/Time: December 12, 2005 6:00 pm - 12:00 am
Length: 55m 16s - 155 Slides



PC01g - Thyroid Hormone
$20.00 Purchase Conference: A4M Las Vegas 2006
Speaker: Thierry Hertoghe, MD
Date/Time: December 7, 2006 6:00 pm - 7:00 pm
Length: 35m 41s - 272 Slides



PC04g - Thyroid Hormones
$20.00 Purchase Conference: A4M Chicago 2006
Speaker: Thierry Hertoghe, MD
Date/Time: July 14, 2006 6:00 pm - 5:30 pm
Length: 56m 05s - 272 Slides



PC01i - Weight Loss: The Answer
$16.00 Purchase Conference: A4M Orlando 2007
Speakers: Thierry Hertoghe, MD , Pamela Smith, M.D., MPH
Date/Time: April 26, 2007 6:00 pm - 7:00 pm
Length: 88 Slides
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://muscle chat room.com/forum/showthread.php?p=21934#post21934

My pregnenolone and progesterone levels bother me to no end.
My lattest known 3/19/08
Progesterone, LC/MS/MS =0.1(<1.4)ng/mL
Pregnenolone---------- =11(13-208)ng/dL

Reading Tierry Herthoge book, he wants Pregnenolone> 100ng/dL
Now I came across his Dec 2006 speach in Las Vegas.
I posted below summary of the info included in previev.
I have also bought the course ($20)
Will post additional info as I listen to presentation.
I guess that there is no doubts that dr Herhoge is one of the giants in anti-aging, can't wait to listen to his opinion.

I know that dr John is against supplying progesterone to men,
if it is ofensive, I will store my notes elsevere.
=====================================================


A4M :: Conference Library

GS02a - Eye Opener: Progesterone Therapy in Men
Conference: A4M Las Vegas 2006
Speaker: Thierry Hertoghe, MD
Date/Time: December 9, 2006 7:00 am - 4:10 pm
Length: 38m 22s - 158 Slides
===================================================

Progesterone theraphy in men.
By Thierry Herthoge, MD

About giving progesterone to men.

Progesterone amount in men is
7x lower than testosterone
more than DHT
more that estrodial and estrone
10x more than melatonin
many more times more than FreeT4

quantity of progesterone than men have is the same as in women during half of their reproductive life (same as during folicular phase, first phase of cycle). During that (folicular phase) progesterone in men and women comes from adrenal glands only. In second part of woman's cycle additional progesterone is made by ovary.
.
......................................................................................
http://www.musc lechatroom.com/forum/showthread.php?4785-What-does-Dr-Thierry-Hertoghe-take-on-a-daily-basis&

What does Dr Thierry Hertoghe take on a daily basis?
This is what he takes on a daily basis:

testosterone gel 10%: 1 dose/day
pregnenolone: 50 mg/day
melatonin: 0.5 mg sublingual/day
thyroid armour: 1.5 grain a day
hydrocortison: 30 mg/day
9 alfa fludrocortison: 100 ug/day
dhea: 40 mg/day
finasteride: 2.5 mg a day
progesterone: 100 mg/day
gh: 0.01 iu/day
vitamin e: 400 mg/day
coq10: 100 mg/day
multivitamin: 1/day
carnitine: 4g/day
fish oil: 3/day
b-complex: 1/day

.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://muscle chatroom.com/forum/showpost.php?p=22363&postcount=9
Right on.

But may I add that even with all other things in order I still had high SHBG 57-65 (Quest 18-47) long before I started TRT and even 3-4 months into it. Then Doc Shippen suggested low dose danazol (20mg/day) and my SHBG plummeted to the 22-28 range ever since (almost 12 months now). That pill alone almost doubled my free test (per Shippen's chart).

Funny thing is, if you Google "danazol" you won't find any mention of SHBG. Must have been a secondary effect thing.
You're a star.

After reading your note I found this incredible abstract on Pubmed:

Changes in the SHBG concentration during danazol t...[Acta Obstet Gynecol Scand Suppl. 1984] - PubMed Result

This incredible abstract actually spells out all the juicy bits instead of trying to get you to buy the original document (only available on paper)

####

Changes in the SHBG concentration during danazol treatment.
Gershagen S, Döberl A, Rannevik G.

Serum levels of sex hormone binding globulin (SHBG) were measured by radioelectro-immunoassay before and during administration of danazol in daily doses varying between 200 and 800 mg for a period of 1-6 months. The patients consisted of different groups of regularly menstruating women (n = 76) and of postmenopausal women (n = 12). A rapid proportional decrease in SHBG was seen at all dose levels in both pre- and postmenopausal women, starting within the first 24 hours and reaching statistical significance by 48 hours. The fractional rate of fall appeared to be determined by the metabolic half-life of the protein itself. Plotting log concentrations of SHBG versus time and using the slope of the linear regression for calculations, the half-life of SHBG appeared to be 15 +/- 5.7 (SD) days. After approximately one month of treatment, the SHBG concentrations began to approach a new steady state at a level of approximately 20-30% of the original concentration, depending on the dose of danazol. The proportional suppression of SHBG was significantly greater following 600 or 800 mg of danazol daily than following 200 or 400 mg. However, with all doses the SHBG levels after one month of treatment were well below the levels normally found in healthy males. The mean proportional reduction following a certain dose was almost identical in premenopausal and postmenopausal women. The findings of the present investigation suggest that danazol exerts a direct inhibitory effect on the hepatic synthesis of SHBG, which is dependent of the dose of danazol, independent of estrogen concentration, but possibly accentuated by endogenous androgens.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established

edit 12/12/2012

Dr Hertoghe

The Hormone Handbook 2Ed 2010

------------
important, just found looking thru his book

page 69

GH Growth Hormone
Excess, lowers SHBG


////////////////////////////////////////

For our friends (man & women)
with high SHBG

I have not heard of this connection before today.




///
page 58

IGF-1 men
220-300 optimal
0-180 GH probably deficient
reference (21-30years) (114-492)ug/L

IGF-BP3
3000 optimal
>4000 GH probably deficient
reference (21-30years) (2000-4000)ug/L



....



pppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppp

ScottHart
On GH my fasting insulin has gone from 3 to 15.
I see my doc next week. Hopefully he has a trick up his sleeve.


cpeil2
I think the usual "trick" is to cut your hGH dose.

pppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppppp

http://www.beckmancoulter.com/CustomerSupport/IFU/ivdd/6600.pdf

IGFBP-3
ng/mL X 0.035 = nmol/L
1000 ng/mL = 1 mg/L
1000 mcg= 1mg
4mg/L=4000(ng/mL) x 0.035=140(nmol/L)

--------------------------------------------------
Growth Hormone
per Tierry Hertoghe MD Hormone Solution
supplementation required when
IGF-1<150mcg/L
IGFBP-3>4000mcg/L=4mg/L=140(nmol/L)
-------------
dose 0.02 - 0.4mg/day
1 mg. of powder GH = 3 iu
0.02-0.4mg/day=0.06-1.2iu/day
-------
Dr Braveman in his presentation shows (0.54, 0.52, 0.48, 0.47, 0.46)mg/day in consecutive years
There are also values for 10 years study.
Increasing IGF-1 Levels to the Upper Range of Normal to Fight Disease - A4M Orlando 2009 - American Academy of Anti-Aging Medicine :: PROLibraries.com - Online Professional Education - Online Conferences - Professional Lectures - Conference Education
--------
Suzan Somers uses 0.08mg=0.24iu/day (because of her breast cancer)(page 66 of her book Breakthru)
Hertoghe uses 0.02iu/day(page 16) =0.007mg/day

body produces 0.25 - 0.5mg/day
body produces 0.75 - 1.5iu/day

Note.
Hertoghe, top p267 (attached) advices HGH at bed time,
Suzan is takig her's in the morning.


Signs of overdosage:
swolen feet, pins and needles in the fingers
carpral tunnel syndrome
pain in the joints (real serious)
----------------------------------------------------------------------------------------

Arginine(7-12)g/day at bed time or one hr befor exercise (from table p268), 2-3x/day (text p267), work best in young(20-35) and lean
Glutamine 2g/day at bed time, works in older adults (32-64)---------4x500mg pills
Lysine (1-3)g/day at bed time, work in young(15-35) w/arginine
Ornithine (2.5-5)g/day works in young(20-35) and lean
Glycine (5-7)g/day-------------------------------------2x1000mg pills
Tryptophan (5-10)g/day small GH response, best add B6 & C
GHB(furanone) (0.5-1)g/day
Niacin (0.2-1)g
=====================================================================
NutrEval June 8/08(uncompleted)

Arginine=9.3(7.5-13)(33%)
Glutamine=53(50-70)(15%)
Lysine=15.3(15.5-27.5)(-1.7%)-------low
Ornithine=6.33(4.25-11.5)(29%)
Glycine=21(19-43)(8%)
Tryptophan=3.68(3.3-6.5)(12%)
GHB(furanone)
Niacin

ONE 11/28/07
Arginine=25(10-64)
Glutamine=186(153-483)
Lysine=59(34-226)
Ornithine=25(3-16)----high
Glycine=439(434-1688)
Tryptophan=46(23-88)
=================================

DHEA article by James South.

DHEA --> INSULIN -- glucose
DHEA --> IGF-1 -- GH
=================================
-----http://muscle--------chatroom.com/forum/showpost.php?p=35600&postcount=13



1 IU per day. 6 days on, 1 day off. 4 weeks on 1 week off. For 3 months, then stop.
Sleep improved in the "on" weeks, to the point where I didn't need melatonin. But I wasn't stressing at the time because my job had hit a cruisey stage, and I wasn't chasing variety (ie: stress).
This concept of a "constant" pulse is for body builders and hollywood types who don't know better.

The smart ones amongst us dose up "just right" (using urinary GH labs and symptoms, not IGF-1 or IGFBP-3) and we get a nice gradual rise and fall overnight - a 12 hour pulse.

When you actually use recombinant GH, the theories stop and the practical starts.
 

Attachments

JanSz

JanSz

Well-known member
Awards
1
  • Established
Fatty Acid Equivalent Names
Type Isomer Systematic Name Common Name

Saturated Fats

4:0 butanoic acid butyric acid
6:0 hexanoic acid caproic acid
8:0 octanoic acid caprylic acid
10:0 decanoic acid capric acid
12:0 dodecanoic acid lauric acid
13:0 tridecanoic acid
14:0 tetradecanoic acid myristic acid
15:0 pentadecanoic acid
16:0 hexadecanoic acid palmitic acid
17:0 heptadecanoic acid margaric acid
18:0 octadecanoic acid stearic acid
19:0 nonadecanoic acid
20:0 eicosanoic acid arachidic acid
22:0 docosanoic acid behenic acid
24:0 tetracosanoic acid lignoceric acid

Type Isomer Systematic Name Common Name

Monounsaturated Fats

14:1 tetradecenoic acid myristoleic acid
15:1 pentadecenoic acid
16:1 undifferentiated hexadecenoic acid palmitoleic acid
16:1 c
16:1 t
17:1 heptadecenoic acid
18:1 undifferentiated octadecenoic acid oleic acid
18:1 c
18:1 t
20:1 eicosenoic acid gadoleic acid
22:1 undifferentiated docosenoic acid erucic acid
22:1 c
22:1 t
24:1 c cis-tetracosenoic acid nervonic acid

Type Isomer Systematic Name Common Name

Polyunsaturated Fats

16:2 undifferentiated hexadecadienoic acid
18:2 undifferentiated octadecadienoic acid linoleic acid
18:2 n-6 c,c
18:2 c,t
18:2 t,c
18:2 t,t
18:2 i
18:2 t not further defined
18:3 undifferentiated octadecatrienoic acid linolenic acid
18:3 n-3 c,c,c alpha-linolenic acid
18:3 n-6 c,c,c gamma-linolenic acid
18:4 undifferentiated octadecatetraenoic acid parinaric acid
20:2 n-6 c,c eicosadienoic acid
20:3 undifferentiated eicosatrienoic acid
20:3 n-3
20:3 n-6
20:4 undifferentiated eicosatetraenoic acid arachidonic acid
20:4 n-3
20:4 n-6
20:5 n-3 eicosapentaenoic acid (EPA) timnodonic acid
22:2 docosadienoic acid brassic acid
22:5 n-3 docosapentaenoic acid (DPA) clupanodonic acid
22:6 n-3 docosahexaenoic acid (DHA)

Click on any link to see foods highest in that nutrient.

Fats & Fatty Acids – NutritionData.com
.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
This post is a place maker, please do not respond, thank you.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
This post is a place maker, please do not respond, thank you.
 

sarcazmo

New member
Awards
0
Question re T-dose chart

hi JanSz,

You state below that "in the study endogenous testosterone was blocked, testis were not producing T during study". Does this imply that if one's natural baseline TT is, say 350ng/Dl, then for any given x-y value on the table, one should add 350 to the TT value ( i.e, 200mg TE/wk = ( 1058 + 350 ), or does the 212.23 value represent such a baseline measurement?

Cheers,

srczm

Using my study above using RoidCalculator, I note that on weekly injection test blood level are half on minimum of what they are at the max.
------
Using this study, I know T levels at the minimum. Blood drawn on the day of weekly shot right before the shot.

AJP - Endocrinology and Metabolism -- Bhasin et al. 281 (6): E1172 Table 2

Testosterone dose-response relationships in healthy young men -- Bhasin et al. 281 (6): E1172 -- AJP - Endocrinology and Metabolism

Testosterone dose-response relationships in healthy young men -- Bhasin et al. 281 (6): E1172 -- AJP - Endocrinology and Metabolism

------------------------
My analysis is shown on the attached chart, also bottom line results are below. Variation= ±75 should be added when reading the table.
Also remember that in the study endogenous testosterone was blocked, testis were not producing T during study.

DepoT TotalT SHBG FreeT SHBG FreeT
25 353 xxxx 300 xxxx 250
30 381 xxxx 300 xxxx 250
35 410 xxxx 300 xxxx 250
40 438 xxxx 300 xxxx 250
45 466 xxxx 300 xxxx 250
50 494 xxxx 300 xxxx 250
55 522 xxxx 300 xxxx 250
60 551 xxxx 300 xxxx 250
65 579 xxxx 300 xxxx 250
70 607 xxxx 300 xxxx 250
75 635 xxxx 300 xxxx 250
80 663 xxxx 300 xxxx 250
85 692 xxxx 300 xxxx 250
90 720 xxxx 300 xxxx 250
95 748 xxxx 300 xxxx 250
100 776 xx5 300 14.4 250
105 805 xx7 300 16.5 250
110 833 xx9 300 18.6 250
115 861 10.8 300 20.7 250
120 889 xx13 300 xx23 250
125 917 xx15 300 xx25 250
130 946 16.7 300 x27.3 250
135 974 18.7 300 x29.4 250
140 1002 xx21 300 x31.5 250
145 1030 22.6 300 x33.5 250
150 1058 24.5 300 x35.6 250
155 1087 26.4 300 xx38 250
160 1115 28.4 300 xx40 250
165 1143 30.4 300 x42.2 250
170 1171 32.3 300 x44.4 250
175 1199 34.3 300 x46.3 250
180 1228 36.3 300 x48.5 250
185 1256 38.2 300 x50.7 250
190 1284 xx40 300 x52.8 250
195 1312 xxxx 300 xx55 250
200 1340 xx44 300 xx57 250
205 1369 xxxx 300 x59.3 250
210 1397 xx48 300 x61.5 250
215 1425 xxxx 300 x63.4 250
220 1453 51.7 300 xxxx 250
225 1481 xxxx 300 xxxx 250
230 1510 55.6 300 xxxx 250
235 1538 xxxx 300 xxxx 250
240 1566 xxxx 300 xxxx 250
245 1594 xxxx 300 xxxx 250
250 1622 63.5 300 xxxx 250
255 1651 xxxx 300 xxxx 250
260 1679 xxxx 300 xxxx 250
265 1707 xxxx 300 xxxx 250
270 1735 xxxx 300 xxxx 250
275 1763 xxxx 300 xxxx 250
280 1792 xxxx 300 xxxx 250
285 1820 xxxx 300 xxxx 250
290 1848 xxxx 300 xxxx 250
295 1876 xxxx 300 xxxx 250
300 1904 83.0 300 99.9 250
=======================
Free & Bioavailable Testosterone calculator
------------------------------
Unit conversion
Conventional units - SI units

Chemistry Conversion

Unit Prefix Conversion Calculator
------------------------------
Adrenal Labs - How to Interpret them ***
Stop The Thyroid Madness :: View topic - *** Adrenal Labs - How to Interpret them ***

Stop The Thyroid Madness » ADRENALS FAQ–the most frequently asked questions
------------------------------
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
candida Remove Replace Reinoculate Repair

==========================================================
SCIO - Bio-Energetic Wellness Centre



Treatment of leaky gut syndrome
The aim of the treatment protocol is to remove obstructions to metabolic processes, and to encourage the body to self-regulate these vital processes. The four pillars of treatment are: remove, replace, reinoculate and repair.

1. Remove

1.1 Parasites, bacteria and fungi: Removal of the latter can be effected with herbal products such as Warburgia,
Taheebo or Pau D’Arco, Citricidal, Para 90 etc., or an essential oil product such as
Origanum capsules (Pranarom), or Rife Resonator therapy. Usually about 2 weeks’ treatment is needed.

1.2 Dietary toxins, additives, chemicals and foods to which the patient may be sensitive can be avoided by placing
the patient on a diet eliminating foods commonly found to be sensitising, such as wheat, dairy, sugar
and additives. Occasionally one needs to go further and eliminate soya, gluten and corn as well.

1.3 Colon Cleansing: Removal of colonic plaque

2. Replace

2.1 Insufficient stomach acid and digestive enzymes can be supplemented with products containing Betaine HCl,
and digestive enzymes such as DigestCo (Mediherb), Digestizyme (Amipro), etc.

2.2 Nutritional deficiencies, arising from poor digestion, can be corrected with a good functional food (see below).

3. Re-inoculate

3.1 Friendly bacteria must be replenished as soon as the parasite cleanse (above) has been completed. This should
be ongoing for several months. Use a reliable product such as Reuteri, Intestiflora or Probiflora.

4. Repair

4.1 Gut lining can be repaired with L-Glutamine, MSM and good-quality, pharmaceutical-grade Omega 3 oils.


A good-quality functional food forms the cornerstone of the above regimen, by providing a complete range of nutrients, vitamins, minerals, pre-biotics such as FOS (Fructo-Oligo-Saccharide) and insulin to support the bacteria, L-Glutamine and MSM. The locally produced Target Candida is effective and less expensive, but is based on soya, which may disagree with patients if they are intolerant to it. The imported UltraClear Sustain is excellent and rice-based, but more expensive. UltraInflamX is used when there is inflammation in the body, and UltraClear Plus is for promoting Phase 1 and 2 detoxification pathways in the liver. These products are best used under the supervision of a knowledgeable integrative practitioner.


Liver support and detox
It is important to support the liver through the above programme with herbal products such as Milk Thistle, LivCo (Mediherb), Livotibb, etc. UltraClear Plus and Chlorella both improve detoxification processes in the liver very effectively.
===============================================
============================================================================================
===============================================
http://www.genovadiagnostics.com/files/profile_assets/interpretation_guides/Intestinal Permeability-InterpGuide.pdf
Intestinal Permeability Assessment

Consider
Consider “4 R” approach to GI health:

1) Remove mucosal irritants such as allergenic foods, alcohol, gluten (if sensitive), NSAIDS:
• Consider elimination diet
• Remove possible pathogens (bacteria, yeast, parasites)
• Consider Comprehensive Digestive Stool Analysis (CDSA) or Comprehensive Parasitology, Bacterial Overgrowth of the Small Intestine Breath Test
• Reduce sugar, refined carbohydrates, saturated fat, red meat (meat can induce bacterial enzyme activity)
• Restore proper transit time
–Increase dietary fiber (esp. insoluble) and water

2) Replace agents for digestive support:
• Consider pancreatic or plant enzymes, bile salts, betaine HCl, digestive herbs, or disaccharidases (e.g. lactase) where needed
• Consider CDSA, Lactose Intolerance Breath Test (or other disaccharide) to rule out disaccharidase deficiency

3) Reinoculate with friendly bacteria, if low:
• Consider CDSA, Microbiology, or Comprehensive Parasitology to rule out gut flora insufficiencies
• Consider probiotic supplementation, including Lactobacilli and Bifidobacteria
• Consider fructooligosaccharides and inulin to enhance growth of friendly flora

4) Repair mucosal lining:
• Consider L-glutamine, EFAs, zinc, pantothenic acid, vitamins C, E, and A, beta carotene, N-acetyl glucosamine, gamma oryzanol, glycerrhiza, aloe vera
• Consider antioxidants such as vitamins C, E and A, selenium, carotenoids, glutathione, N-acetyl cysteine, pycnogenol and flavonoids
• Consider Saccharomyces boulardii, whey globulin concentrate, or bovine colostrum to improve local immunity
• Consider ginkgo biloba to enhance circulation to intestinal epithelium
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Lots of information about hoe to test and how to treat.

NATURE'S INTENTIONS NATUROPATHIC CLINIC
Toronto Naturopathic Clinic - Toronto Naturopath Sushma Shah ND (Yonge & Davisville)

TORONTO NATUROPATHIC MEDICAL TESTING CENTRE
MEDICAL LABORATORY TESTING (BLOOD, SALIVA, URINE, STOOL)
FROM GAMMA-DYNACARE MEDICAL LABORATORIES
Medical Laboratory Testing (Blood, Saliva, Urine, Stool) -- Nature's Intentions Naturopathic Clinic

BLOOD TESTS TESTS TO MEASURE VARIOUS HORMONES
Thyroid Panel

Anti Micrsomal Globulin (Thyroid)
Antithyroglobulin
Antithyroperoxidase
Free T3
Free T4
Parathyroid
Thyroid stimulating hormone

Male And Female Hormones

Bioavailable Testosterone
DHEAS
Estradiol
Luteinizing hormone
Follicle stimulating hormone
Progesterone
Prolactin

Standard Blood Testing

CBC

Lipid Panel

Cholesterol, HDL / LDL

Mineral / Vitamin / Nutrient Status In Blood

Beta Carotene
Calcium - Phosphatase
CO2
Copper Cyanide
Copper Serum
Glucose (Random & Fasting)
Glycated HGB
Ionized Calcium
Iron / Ferritin / Total Iron Binding Capacity
Lead
Lithium
Magnesium
Mercury
Phosphate
Phosphorous
Plasma Amino Acid
Serum Folate
Silver Cyanide
Vitamin A, B12, E

Liver And Pancreatic Enzymes

Amylase
Gamma glutamyl transferase
Homocysteine (plasma)
Insulin
TP, ALB, ALKP, ALT, TBIL, DBIL

Food And Environmental Allergy Testing

93 IgG Food - Antibody Screen
Cold Agglutins
Endomysial Abs
Food Allergy (Ig E) Group sensitivity
Food Allergy (Ig E) Individual Foods
Gliadin Igg / Ige (Celiac disease)
IgE Antibody Screening for foods and environment allergens (list of substances tested available from the clinic)

Enzymes, Cancer Markers And Other Miscellaneous Tests

Alkaline Phosphatase isoenzymes (bone specific)
CA 19-9
CA 27-29
CA 15-3 (Breast)
CA 125 (Ovary)
Cardiac Enzymes: CK-MB, CK, Total, Myoglobin, CPK
Ceruloplasmin
CEA
IGF – 1
Immunocyte (for bladder CA)
Prostatic Specific Antigen (PSA Ratio - Free PSA)

Inflammatory Markers

Anti-Nuclear Antibody
ANA
C-Peptide (serum)
C-Reactive Protein
Creatinine
ESR
PT (INR)
PTT

For Infections

H Pylori Breath Test
Ova and Parasites (stool)
Pinworm
Stool Culture
Stool Parasites
Swab (General)
Throat Swab

Pregnancy & Fertility Tests

Pregancy - HCG (Serum)
Pregnancy Test (Urine)
Sperm Count - Fertility Test
Blood typing: - ABO - Rh



URINE TESTS TESTS TO MEASURE VARIOUS HORMONES
Chemical Status Of Urine

Sodium, Potassium, Creatnine, Uric Acid
Cortisol
Deoxypyridinoline (urine) "DPD"
Methylmelonic Acid (random urine)
Gram Stain
Urea / BUN
Urine Culture
Urine (random)
Electrophoresis
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Hormone Testing / Endocrinology -- Nature's Intentions Naturopathic Clinic

HORMONE TESTING / ENDOCRINOLOGY
FROM GREAT SMOKIES DIAGNOSTIC LABS
Women's Hormonal Health Assessment provides a focused overview of hormonal balance in both pre- and post-menopausal women, using a single serum sample to evaluate dynamics of sex steroid metabolism that can profoundly affect a woman's health throughout her lifetime.

Estrogen Metabolism Assessments, Urine or Serum evaluate how estrogen is being processed in the body. The tests yield clinical insight into many estrogen-dependent conditions and provide important tools for monitoring dietary, lifestyle and hormone therapies.

Comprehensive Thyroid Assessment is a comprehensive analysis of thyroid hormone secretion and metabolism, including central thyroid regulation and activity, peripheral thyroid function, and thyroid autoimmunity. This serum test allows the practitioner to pinpoint commonly occurring imbalances that underlie a broad spectrum of chronic illness.

Bone Resorption Assessment is a simple, direct urinary assay of pyridinium crosslinks and deoxypyridinoline, useful in identifying current rate of bone loss, lytic bone disease, and efficacy of bone support therapies.

Adrenocortex Stress Profile is a salivary assay of cortisol and DHEA, imbalances of which are associated with ailments ranging from obesity and menstrual disorders to immune deficiency and increased risk of cardiovascular disease.

Male Hormone Profile analyzes four saliva samples over a 24-hour period for levels of testosterone. Elevated levels suggest androgen resistance, while decreased levels can result from such causes as hypogonadism, hepatic cirrhosis, lipid abnormalities and aging. The comprehensive profile includes the Adrenocortex Stress Profile and the Comprehensive Melatonin Profile to reveal how testosterone is influenced by cortisol, DHEA, and melatonin.

Female Hormone Profile analyzes eleven saliva samples over a 28-day period for the levels of ß-estradiol, progesterone, and testosterone, providing clues about menstrual irregularities, infertility, endometriosis, breast cancer, and osteoporosis. The comprehensive profile includes the Adrenocortex Stress Profile and the Comprehensive Melatonin Profile to reveal how the sex hormones are influenced by cortisol, DHEA, and melatonin.

Menopause Profile examines three salivary samples over a 5-day period to determine levels of ß-estradiol, estriol, estrone, progesterone, and testosterone for women who are menopausal. The comprehensive profile includes the Adrenocortex Stress Profile and the Comprehensive Melatonin Profile to reveal how the sex hormones are affected by the influences of cortisol, DHEA, and melatonin.

Comprehensive Melatonin Profile analyzes three saliva samples for the secretion pattern of this important hormone. Melatonin imbalance has been associated with Seasonal Affective Disorder, infertility, sleep disorders, and compromised immune function.

FROM ZRT LABS

Hormones Available for Testing (Saliva Collection):
AM Cortisol (C) is recommended with situational stress and fatigue; indicator of adrenal imbalance.

AM/PM Cortisol is recommended with excess stress, morning and evening fatigue, difficulty sleeping etc.; indicator of adrenal dysfunction.

Adrenal Function Test (AFT) is the best comprehensive assessment of adrenal gland function; recommended with excessive stress, chronic fatigue, sleep disorders and allergies; indicator of immune function. Tests; DHEA's and four cortisols: morning, noon, evening and night.

DHEA-S is recommended with androgen deficiency or excess symptoms; indicator of stress level, mental performance and insulin resistance.

Estradiol (E2) is the most predominant of the estrogens. Testing is recommended with symptoms of estrogen deficiency or excess; best tested in concert with progesterone, as an imbalance of these two hormones is associated with major symptoms of menopause and disorders of the reproductive system).

Estriol (E3) it is recommended that Estriol levels be measured when supplementing with a compounded preparation of bioidentical estrogens containing Estriol (e.g. "Biest" preparations contain estriol and estradiol OR "Triest" preparations containing estriol, estradiol and estrone).

Estrone (E1) it is recommended that Estrone levels be measured when supplementing with a compounded preparation of bioidentical estrogens containing Estrone (e.g. "Triest" preparations contain estrone, estradiol and estriol).

Hormone Profile I (5 Tests) is a good basic assessment of overall steroid hormone balance or imbalance and association with specific symptoms; indicator of hypothyroidism, bone loss and insulin resistance. Five Tests: Estradiol (E2), Progesterone (Pg), Testosterone (T), DHEA's, and morning Cortisol (C).

Hormone Profile II + PM Cortisol (6 Tests) is the best value for money; most comprehensive evaluation of overall hormone balance or imbalance; indicator of thyroid and adrenal function; breast cancer profile. Six Tests: Hormone Profile I plus pm Cortisol (C4)

Hormone Profile III + AFT (8 Tests) tests provides the most complete and in-depth evaluation of hormone balance, imbalance and interrelationship of specific hormone levels as related to symptoms of menopause, andropause and adrenal function. Eight Tests: Estradiol (E2), Progesterone (Pg), Testosterone (T), DHEA's, and 'Diurnal Cortisol' (All four cortisols: morning (C1), noon (C2), evening (C3) and night (C4).

Progesterone (Pg) is recommended with symptoms of deficiency or excess; best tested in concert with estradiol as an imbalance of these two hormones is associated with major symptoms of menopause and disorders of the reproductive system.

Testosterone (T) is recommmended with deficiency or excess symptoms; indicator of low sex drive, hair loss/excess, muscle and bone status.

BloodSpot Test Products
Fasting Insulin - Fasting Insulin in blood spot for the detection of insulin resistance is collected in the morning after a 12 hour fast and before eating or drinking. The home test kit facilitates optimal collection time for a fasting sample. Process involves a finger prick with lancet, dripping of blood spots and drying on filter paper. Kit is self-contained with step-by-step instructions, filter paper, 2 lancets, antiseptic wipes, etc.

Fasting Insulin in blood spot is collected in the morning with our home test kit, after a 12 hour fast and before eating or drinking.

Follicle Stimulating Hormone - (FSH) Marker of Menopause or Andropause onset.

Free Thyroxine (fT4) - Thyroxine is the main thyroid hormone produced by the thyroid. A well- regulated process causes thyroxine to generate the much more potent thyroid hormone T3 (Triiodothyronine).

Free Triiodothyronine (fT3) measures the level of active thyroid hormone T3. This level must remain within optimal range to keep the body functioning properly and is crucial for maintaining physical and mental health.

Lutenizing Hormone (LH) - Marker of Menopause or Andropause onset.

Male Profile I (T, SHBG, PSA) is an overall assessment of male vitality, performance and prostate health; indicator of prostate enlargement or cancer risk.

Male Profile II (IGF-1, T, SHBG, PSA) is the best comprehensive assessment of overall male health, vitality, and prostate health; indicator of Adult Growth Hormone deficiency, rapid aging and increased prostate cancer risk.

Prostate Specific Antigen (PSA) - PSA a protein produced by the prostate gland is an important indicator of prostatic enlargement or increased risk of prostate cancer. High PSA levels are a warning sign of prostate health risks. A normal PSA reading is prerequisite to initiating testosterone therapy.

Sex Hormone Binding Globulin (SHBG) - SHBG (sex hormone binding globulin) measurement is used as a relative index of overall exposure to all forms of estrogens, as an indirect index of estrogen interaction with the liver and as an indicator of bioavailable testosterone. High levels indicate excess exposure to estrogens and lower bio-availability of testosterone to tissues, It is recommended that SHBG be tested along with testosterone in order to determine an imbalance between testosterone and estrogen. Such an imbalance is an important indicator of Andropause onset and / or premature aging in men.

Somatomedin C (IGF-1) - IGF-1 is important because it is a reliable indicator of human growth hormone. IGF-1 needs to be within the expected range for this reason: Low IGF-1 levels indicate Adult Growth Hormone Deficiency associated with rapid aging, decreased muscle and bone mass, slowing cognition, low libido and poor quality of life.

Testosterone (T) is recommended with deficiency or excess symptoms; indicator of low sex drive, hair loss, muscle mass and bone status. It is recommended that Testosterone and SHBG be tested together to determine imbalances of testosterone and estrogen; an important indicator of Andropause onset and/or premature aging in men.

Thyroid Comprehensive Profile is an in-depth comprehensive assessment of overall thyroid function checking TSH, freeT4, freeT3, and TPO antibodies (Hashimotos). More than 10 million Americans have been diagnosed with thyroid disease, and another 13 million people are estimated to have undiagnosed thyroid problems in the U.S. alone. Women are at greatest risk, developing thyroid problems seven times more often than men.

Thyroid hormones are transported in the blood "bound" to Thyroid Binding Globulin (TBG), which temporarily holds them inactive. This protein may be manipulated by many illnesses and medications. Therefore, measurement of the "unbound," free levels of T3 and T4 thyroid hormones provides a more accurate interpretation. When TSH, free T3 and T4 are found to be within normal range, a functional thyroid deficiency may be suspected. This can be clarified by testing TPO antibodies (Thyroid Peroxidase Antibody) as part of the comprehensive thyroid panel.

Thyroid Peroxidase Antibody (TPO) - In 90% of patients with Hashimoto’s (autoimmune thyroiditis) TPO levels are elevated; indicator of other autoimmune disorders and polycystic ovaries.

Thyroid Stimulating Hormone (TSH) measures the amount of thyroid hormone manufactured by the thyroid gland. General indication of thyroid activity.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Diagnostic Tests for Addison's Disease - WrongDiagnosis.com


Diagnostic Test list for Addison's Disease:

The list of diagnostic tests mentioned in various sources as used in the diagnosis of Addison's Disease includes:
Physical examination - particularly for hyperpigmentation of the skin
Cortisol levels blood test
X-ray adrenal glands - detect calcium deposits from TB-related Addison's.
X-ray pituitary glands
CT scan pituitary glands
ACTH level blood test
ACTH Stimulation Test
Insulin-Induced Hypoglycemia Test

===========================================
Diseases Center - WrongDiagnosis.com
Asthma
Allergies
Alzheimer's Disease
Breast Cancer
Cancer
Cholesterol
Common Cold
Depression
Diabetes
Eye Disorders
Flu
Headache
Heart Disease
Heart Attack
Hepatitis
Herpes
HIV
Hypertension
Infertility
Kidney Disease
Liver disease
Meningococcal Disease
Menopause
Metabolic Syndrome
Migraine
Multiple Sclerosis
Obesity
Osteoporosis
Parkinsons Disease
Pregnancy
Reflux
Stroke
Thyroid disease
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Kimball's Biology Pages

Kimball's Biology Pages

====================================================
Hormones of the Pituitary


Index to this page
The Anterior Lobe
Thyroid Stimulating Hormone (TSH)
Gonadotropins
Follicle Stimulating Hormone (FSH)
FSH in females
FSH in males
Luteinizing Hormone (LH)
LH in females
LH in males
Prolactin (PRL)
Growth Hormone (GH)
Hormone-replacement therapy
ACTH
Alpha Melanocyte-Stimulating Hormone (α-MSH)
The Posterior Lobe
Vasopressin
Oxytocin

Hormones of the Pituitary
The pituitary gland is pea-sized structure located at the base of the brain. In humans, it consists of two lobes:
the Anterior Lobe and
the Posterior Lobe
Link to graphic showing the location
of the pituitary and other endocrine
glands (92K).

The Anterior Lobe
The anterior lobe contains six types of secretory cells, all but one of which (#2 above) are specialized to secrete only one of the anterior lobe hormones. All of them secrete their hormone in response to hormones reaching them from the hypothalamus of the brain.
Thyroid Stimulating Hormone (TSH)
TSH (also known as thyrotropin) is a glycoprotein consisting of:
a beta chain of 112 amino acids and
an alpha chain of 89 amino acids. The alpha chain is identical to that found in two other pituitary hormones, FSH and LH as well as in the hormone chorionic gonadotropin. Thus it is its beta chain that gives TSH its unique properties.
The secretion of TSH is
stimulated by the arrival of thyrotropin releasing hormone (TRH) from the hypothalamus.
inhibited by the arrival of somatostatin from the hypothalamus.
As its name suggests, TSH stimulates the thyroid gland to secrete its hormone thyroxine (T4). It does this by binding to transmembrane G-protein-coupled receptors (GPCRs) on the surface of the cells of the thyroid.
Some people develop antibodies against their own TSH receptors. When these bind the receptors, they "fool" the cell into making more T4 causing hyperthyroidism. The condition is called thyrotoxicosis or Graves' disease.

Hormone deficiencies
A deficiency of TSH causes hypothyroidism: inadequate levels of T4 (and thus of T3 [Link]). Recombinant human TSH (Thyrogen®) is now available to treat patients with TSH deficiency.

Some people inherit mutant TSH receptors. This, too, results in hypothyroidism.

A deficiency of TSH, or mutant TSH receptors, have also been implicated as a cause of osteoporosis. Mice, whose TSH receptors have been knocked out, develop increased numbers of bone-reabsorbing osteoclasts.

Follicle-Stimulating Hormone (FSH)
FSH is a heterodimeric glycoprotein consisting of
the same alpha chain found in TSH (and LH)
a beta chain of 115 amino acids, which gives it its unique properties.
Synthesis and release of FSH is triggered by the arrival from the hypothalamus of gonadotropin-releasing hormone (GnRH). The effect of FSH depends on one's sex
FSH in females
In sexually-mature females, FSH (assisted by LH) acts on the follicle to stimulate it to release estrogens.

FSH produced by recombinant DNA technology (Gonal-f®) is available to promote ovulation in women planning to undergo in vitro fertilization (IVF) and other forms of assisted reproductive technology.
FSH in males
In sexually-mature males, FSH acts on spermatogonia stimulating (with the aid of testosterone) the production of sperm.
Luteinizing Hormone (LH)
LH is synthesized within the same pituitary cells as FSH and under the same stimulus (GnRH). It is also a heterodimeric glycoprotein consisting of
the same 89-amino acid alpha subunit found in FSH and TSH (as well as in chorionic gonadotropin);
a beta chain of 115 amino acids that is responsible for its properties.
The effects of LH also depend on sex.
LH in females
In sexually-mature females,
a surge of LH triggers the completion of meiosis I of the egg and its release (ovulation) in the middle of the cycle;
stimulates the now-empty follicle to develop into the corpus luteum, which secretes progesterone during the latter half of the menstrual cycle.
Women with a severe LH deficiency can now be treated with human LH (Luveris®) produced by recombinant DNA technology.
LH in males
LH acts on the interstitial cells (also known as Leydig cells) of the testes stimulating them to synthesize and secrete the male sex hormone, testosterone.
LH in males is also known as interstitial cell stimulating hormone (ICSH).

Discussion of the negative-feedback loops that control the levels of estrogen, progesterone, and testosterone.

Prolactin (PRL)
Prolactin is a protein of 198 amino acids. During pregnancy it helps in the preparation of the breasts for future milk production.
After birth, prolactin promotes the synthesis of milk.

Prolactin secretion is
stimulated by TRH
repressed by estrogens and dopamine.
In pregnant mice, prolactin stimulates the growth of new neurons in the olfactory center of the brain.

Growth Hormone (GH)
Human growth hormone (HGH; also called somatotropin) is a protein of 191 amino acids. The GH-secreting cells are stimulated to synthesize and release GH by the intermittent arrival of growth hormone releasing hormone (GHRH) from the hypothalamus. GH promotes body growth by:
binding to receptors on the surface of liver cells
this stimulates them to release insulin-like growth factor-1 (IGF-1; also known as somatomedin)
IGF-1 acts directly on the ends of the long bones promoting their growth
Things that can go wrong.

In childhood,
hyposecretion of GH produces the stunted — but normally well-proportioned — growth of a midget.
Growth retardation can also result from an inability to respond to GH. This can be caused by inheriting two mutant genes encoding the receptors for
GHRH or
GH
or homozygosity for a disabling mutation in STAT5b, which is part of the "downstream" signaling process after GH binds its receptor.
hypersecretion leads to gigantism
In adults, a hypersecretion of GH or GHRH leads to acromegaly.
Hormone-replacement therapy
GH from domestic mammals like cows and pigs does not work in humans. So for many years, the only source of GH for therapy was that extracted from the glands of human cadavers. But this supply was shut off when several patients died from a rare neurological disease attributed to contaminated glands. Now, thanks to recombinant DNA technology, recombinant human GH (rHGH) is available. While a great benefit to patients suffering from GH deficiency, there has also been pressure to use it to stimulate growth in youngsters who have no deficiency but whose parents want them to grow up tall. And so, in the summer of 2003, the U.S. FDA approved the use of human growth hormone (HGH) for
boys predicted to grow no taller than 5′3″ and
for girls, 4′11″
even though otherwise perfectly healthy.
ACTH — the adrenocorticotropic hormone
ACTH is a peptide of 39 amino acids. It is cut from a larger precursor proopiomelanocortin (POMC).

ACTH acts on the cells of the adrenal cortex, stimulating them to produce
glucocorticoids, like cortisol
mineralocorticoids, like aldosterone
androgens (male sex hormones, like testosterone
in the fetus, ACTH stimulates the adrenal cortex to synthesize a precursor of estrogen called dehydroepiandrosterone sulfate (DHEA-S) which helps prepare the mother for giving birth.
Production of ACTH depends on the intermittent arrival of corticotropin-releasing hormone (CRH) from the hypothalamus.

Hypersecretion of ACTH is a frequent cause of Cushing's disease.

Alpha Melanocyte-Stimulating Hormone (α-MSH)
Alpha MSH is also a cleavage product of proopiomelanocortin (POMC). In fact, α-MSH is identical to the first 13 amino acids at the amino terminal of ACTH.
MSH is discussed in a separate page. Link to it.

The Posterior Lobe
The posterior lobe of the pituitary releases two hormones, both synthesized in the hypothalamus, into the circulation.

Vasopressin
Vasopressin is a peptide of 9 amino acids (Cys-Tyr-Phe-Gln-Asn-Cys-Pro-Arg-Gly). It is also known as arginine vasopressin (AVP) and the antidiuretic hormone (ADH).
Vasopressin acts on the collecting ducts of the kidney to facilitate the reabsorption of water into the blood. This it acts to reduce the volume of urine formed (giving it its name of antidiuretic hormone).

Link to discussion of kidney physiology.


A deficiency of vasopressin or
inheritance of mutant genes for its receptor (called V2)
leads to excessive loss of urine, a condition known as diabetes insipidus. The most severely-afflicted patients may urinate as much as 30 liters (almost 8 gallons!) of urine each day. The disease is accompanied by terrible thirst, and patients must continually drink water to avoid dangerous dehydration.
Another type of receptor for vasopressin (designated V1a) is found in the brain, e.g., in voles and mice (rodents) and in primates like monkeys and humans.

Male prairie voles (Microtus pinetorum) and marmoset monkeys
have high levels of the V1a receptor in their brains,
tend to be monogamous, and
help with care of their young.
Male meadow voles (Microtus montanus) and rhesus monkeys
have lower levels of the V1a receptor in their brains,
are promiscuous, and
give little or no help with the care of their young.
Meadow voles whose brains have been injected with a vector causing increased expression of the V1a receptor become more like prairie voles in their behavior. (See Lim, M. M. et al., Nature, 17 June 2004.)

The level of expression of the V1a receptor gene is controlled by a "microsatellite" region upstream (5') of the ORF. This region contains from 178 to 190 copies of a repeated tetranucleotide (e.g., CAGA). Prairie voles have more copies of the repeat than meadow voles, and they express higher levels of the receptor in the parts of the brain associated with these behaviors. A similar microsatellite region is present in the pygmy chimpanzee or bonobo (Pan paniscus) but is much shorter in the less-affectionate common chimpanzee (Pan troglodytes).

Link to a discussion of some human diseases caused by trinucleotide repeats.

Changes in the regulatory region of the human gene for the V1a receptor have been linked to autism.

Oxytocin
Oxytocin is a peptide of 9 amino acids (Cys-Tyr-Ile-Gln-Asn-Cys-Pro-Leu-Gly).
It acts on certain smooth muscles:
stimulating contractions of the uterus at the time of birth;
stimulating release of milk when the baby begins to suckle.
Oxytocin is often given to prospective mothers to hasten birth.

Oxytocin also acts on the nucleus accumbens and amygdala in the brain where it enhances:
bonding between males and females after they have mated;
bonding between a mother and her newborn;
and, in humans, increases the level of one's trust in other people.
Welcome&Next Search

--------------------------------------------------------------------------------
18 November 2008
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://mus clechatroom.com/forum/showpost.php?p=30730&postcount=21


I will never recommend 5-AR Inhibitors.

I would instead switch them from TD to IM TRT, in order to reduce androgenic conversion.
This is a mile stone statement.

Where do you draw the line? (DHT>??)

Anything else?

/
/
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Only parts of article is posted.


http://www.t-nation.com/free_online_article/sports_body_training_performance_steroids_drugs/the_steroid_interviews



The Steroid Interviews
by Chris Street

As part of an agreement with Testosterone Magazine, excerpts from my new book will be published in the coming months. These excerpts detail the use of performance enhancing drugs by elite athletes as well as those in non-athletic populations. The book looks at these drugs individually in a scientific manner, something not done to date by popular books on the subject or those less read books published for members of the scientific community.

-----------------------------
During the period after the injury, this athlete briefly increased his dosage of Oxandrin to 50 mg/d and experienced complete recovery in approximately 14 days.
-----------------------------
The following protocol was used 14 weeks prior to the opening of the 2000 MLB season:

Week(s) 1-3

20 mg/d Winstrol tablets

200 mg/wk testosterone cypionate (generic)

20 mg/d Nolvadex

1 mg/d Arimidex


Week(s) 4-7

25 mg/d Anadrol

300 mg/wk testosterone cypionate (generic)

20 mg/d Nolvadex

1 mg/d Arimidex

4 IU/d rHGH

500 mg metformin taken with meals


Week(s) 8-9

10 mg/d Oxandrin

20 mg/d Nolvadex

1 mg/d Arimidex

2500 IU HCG every other day


Week(s) 10-11

20 mg/d Nolvadex


Week(s) 12-14

No drugs used

His in season drug protocol consists of non-stop use of rHGH, Testosterone cypionate, or a Testosterone ester blend (Sostenon), with oxandrolone. Drugs are taken on a continuous basis with dosages periodically shifting if excessively tired, or in cases of injury. The details of the in season cycle will be available when the book is published.

Immediately after the season he takes an 8-week lay off from both training and drugs. This time is used to rejuvenate his body and mind. After the season he is mentally and physically drained and needs a break. The time away from training is more mental than a physiologic need for recovery. The toll a season of Major League Baseball takes on your life is considerable. In addition to having to perform up to the parameters of a high dollar salary, players must simultaneously deal with family issues, wives, girlfriends (sometimes both), and various matters of business. The lifestyle of professional sports affects some players more than others, but for this athlete a post season break in the action is an indispensable part his program.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Links to Merck Manual

Male Hypogonadism
http://www.merck.com/mmpe/sec17/ch227/ch227b.html

Infertility
http://www.merck.com/mmpe/sec18/ch256/ch256a.html

Primary Hemochromatosis
http://www.merck.com/mmpe/sec11/ch145/ch145b.html


Testosterone therapy stimulates erythropoietin secretion, increasing the Hct.
The polycythemia that may occur with testosterone therapy is independent of the baseline erythropoietin level and is neither dose-dependent nor duration-dependent.
http://www.merck.com/mkgr/mmg/sec14/ch115/ch115c.jsp
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://muscle-------chatroom.com/forum/showpost.php?p=35106&postcount=30

I'm putting this in here for the benefit of others searching. Hopefully it gets picked up by Google too.

Los Angeles is a big city. Scouring the internet and making perhaps 30 phone calls found many people who had not heard of a Therapeutic Phlebotomy or who didn't provide them. This includes Red Cross, Blood Banks, etc.

Finally, I found two providers. They are both hospitals and I shudder to think what it costs. i.e. I know it's not $60. However, when it comes to health, you gotta do what you gotta do.

Therapeutic Phlebotomy Southern California

Therapeutic Phlebotomy Los Angeles

Cedars Sinai
Blood Donor Facility
Cedars Sinai Medical Center
8700 Gracie Allen
South Tower
Los Angeles, CA 90048
(310)423-5346
(310)423-0174
http://www.cedars-sinai.edu/blood

I also found Long Beach Memorial Hospital phone# (562)933-0808.

Cedars can also do a "double red cell procedure" which is what it sounds like. 2x the normal pull is made, but plasma is replaced. This allows you to drop Hemoglobin/Hematocrit twice as fast. They wanted my doc to write the script as "double red cell procedure until hematocrit at 45"

I hope this helps someone else.

Mark
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://forum.mesomorphosis.com/mens-health-forum/thyroid-question-dr-m-134269499.html

http://muscle---------chatroom.com/forum/showthread.php?p=35167#post35167

==========================================================
An incredible well written post from Dr M. It might deserve a stick

When interpreting thyroid function, it is very important to obtain a Total T4.
T4 is about 98 percent of circulating thyroid hormone.

If one is treated with T3 (Liothyronine, Triiodotyronine), then Total T3 will also be important to determine what is occurring.

Thus a more complete thyroid panel would include:
Free T4, TSH
Free T3
Total T3
Total T4

One reason total values are important is that the free levels are influenced by the availability of the various thyroid binding proteins - such as albumin, thyroid binding globulin, and transthyretin.

These binding proteins are influenced by other factors, such as:
Albumin - hydration, general nutrition
Thyroid binding globulin - estrogen signaling strength
Transthyretin - vitamin A signaling strength (since it not only binds T3 preferentially but also viltamin A)

The binding protein levels are not accounted for by the free levels of T3 or T4. Thus when other factors come into play, they will directly interfere with or complicate interpretation.

Additionally, you have weakly bound versus strongly bound interactions with the binding proteins - just as Albumin vs. SHBG have weak vs. strong binding to testosterone. (This is why total testosterone is the best measure overall of testosterone signaling strength.)

Total T4 can be used as a ceiling for how much T4 can be given. Similarly with T3.

Free T4 is not a sensitive indicator of total thyroid signaling strength.

Free T3 is one indicator of total thyroid signaling strength, but I would also take into account Total T4 since thyroid can also be converted within certain cells to T3 prior to use.

Using Free T3 without a total T4 (and Total T3 if needed) to determine thyroid hormone dosing is like flying blind in fog. There is no indication of the endpoint. It would be like using Free Testosterone to determine how much testosterone to give.

In addition to lab tests, it would be important to also try to establish physical markers as targets when doing thyroid replacement therapy. This would include reduction or correction of signs of thyroid hormone deficiency. When one can establish physical markers/signs to determine thyroid dosing, it can be as sensitive or as good as lab tests. This is how physicians did it prior to the development of lab tests.

Combining both physical exam and lab testing would be ideal though patients may not have the means for frequent lab testing. Thus the choice of labs needs to be tailored to the patient and their circumstances.

Winter is a particularly stressful time. One factor is colder weather which forces an increase in sympathetic nervous system activity. This may lead to a reduction in serotonin signaling. This then may result in a reduction in thyroid hormone production. Additionally, the stress resulting from cold weather may result in adrenal fatigue, which would result in a reduction in T4 to T3 conversion. Lower vitamin D levels - as it is used up from fat stores in darker light - also may result in a reduction in serotonin signaling, resulting in a reduction in thyroid hormone production. Stress may also result in zinc loss, impairing thyroid hormone production. Stress also increase insulin resistance, leading to a renal loss of iodine, possibly impairing thyroid hormone production.
Assuming the nervous system is working well enough (a huge assumption) to:
1. monitor thyroid hormone signaling well
2. produce TSH well
then certain doses of thyroid hormone replacement do not necessarily result in a lower TSH.

There are many factors involved. For example:

1. Does the additional exogenous thyroid hormone lead to suppression of thyroid hormone production such that there is more or equivalent loss of thyroid hormone than addition of thyroid hormone? This lead to TSH remaining the same or going higher.

2. Does the additional thyroid hormone lead to stress on the adrenal glands and adrenal fatigue? Or is there already adrenal fatigue, which can be worsened by the addition of thyroid hormone? If so, then thyroid hormone activation from T4 to T3 is impaired. This would increase TSH or break even and keep TSH the same.

3. Does the additional thyroid hormone trigger metabolic signaling pathways such that thyroid binding hormones are increased? This would lead to the same or lower free thyroid levels. TSH would then either remain the same or increase.

4. etc. etc.

Note that one alternative way to do thyroid hormone replacement is to deal directly with the hormone levels, forgetting about TSH. TSH varies much between people and actual thyroid hormone levels and it makes a huge assumption that the nervous system is functioning well enough to appropriately monitor thyroid levels and appropriately produce TSH (despite aging, for example). Thus TSH in many people (such as those with metabolic illnesses, heart disease, diabetes, mental illness, etc.) is not a good measure of thyroid function. This method is analogous to doing testosterone replacement. Who determines testosterone level based on LH or FSH? Hardly any one. It is easier to dose testosterone based on total level than by monitoring LH and FSH levels. With thyroid, Free T3 is actually a useful tool, unlike Free Testosterone (which reflects SHBG which is determined by a multitude of hormone signals). When combined with Total T4, Total T3, and Free T4, and the patient's signs and symptoms, this is all that is needed. TSH in this case is not needed.
.
 
crazycrew

crazycrew

New member
Awards
0
This thread is like a shot gun. It pelts one with info and some actually sticks. Just gotta keep pulling the trigger. Talk about info overload.. Two years in the making? I'm sure I'll keep coming back for study.

Good job JanSz
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://allnutri.com/chrysin.aspx
This link seemed pretty informative...
There is a sinister problem with Chrysin which is debilitating for males. This debilitation outweighs the positive effect of Chrysin's aromatase inhibition.

Chrysin is far less than optimum for male health because it also blocks the action of a high percentage of human 17β-hydroxysteroid dehydrogenase type 5.

17β-hydroxysteroid dehydrogenase type 5 is the enzyme responsible for the following conversions:
a) androstenedione <---> testosterone
b) androstenediol <--> androsterone

Since we males need these conversions to take place at their uninhibited levels, therefore we don't want chrysin in our systems.

reference:
"Phytoestrogens inhibit human 17β-hydroxysteroid dehydrogenase type 5"

http://gbic.biol.rug.nl/~rbreitling/publications/HSD5Inhibition.pdf

==========================
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Copied from:
http://www.cuttingedgemuscle.com/Forum/showthread.php?s=&threadid=174

Formula for Cycling and Dosing T3
One subject that keeps coming up that many people have difficulty is with properly dosing and tapering T3.

Now while THERE IS NO EVIDENCE WHATSOEVER for the persistent rumor that improper T3 use will shut down your thyroid forever, it is also not something to take lightly and like all AAS, should be respected. Ive come up with a formula based on the research Ive done, and both theory as well as practical experience point that it should work well for your fat burning goals as well as give you a proper taper so that the thyroid is able to recover its normal function as quickly as possible. The key to this is having a long enough taper coming off of it. Since origionally designing this formula some will note that I have taken 5% off of the ramp period and placed it toward the back taper insted - this is because I have become convinced that in the presences of exogeneous supplementation, the thyroid shuts down fairly quickly and so the better to spend that time on the taper down.

NOTE 1: If you have never used T3 before, it is suggested that you lessen your constant time and increase your ramp up period to determin your reaction to T3 before heavy use.

NOTE 2: Synthroid (t4) may also be used to good effect with this formula but of course the maximums are diffferent -usually t4 convers to T3 at around a 4.5:1 ratio.

________________________________________
CYCLEONS T3 CYCLE FORMULA

Its pretty simple really – 5/40/55 is a time-based formula whereby X% of the time of the entire cycle should be spent in one of 3 periods – up/constant/down:

RULE – 1
5% of the time is spent ramping up to your maximum
40% of the time is at your maximum
55% of the time is spent ramping down to cessation (nothing)

RULE – 2
Each up/down period is further broken down into equal segments for each dosing level with the emphasis being the dosing level toward the end of the period.

RULE – 3
I dont ever recommend taking more than 125mcg per day and 100mcg will do for most. Above this amount is quite catabolic without hefty concurrent doses of AAS. There are those who advocate higher doses and it is feasible to do so but IMO the effectiveness gains above 100mcg are not worth it.

________________________________________
Example 1
An example for a 20-day cycle with a max of 100mcg ED using 25mcg pills. Calculate the number of days of each period first (Notice that where the up/down period is unable to be broken into 3 exactly equal parts, the extra is put on the dose level at the last part of the period. (.5) means 1/2 a pill or 12.5mcg

UP CONST DOWN
4 days 6days 10days
5% 40% 55%
2 44444444 33222111.5.5.5

______________________________
Example 2
An example for a 60-day cycle with a max of 125mcg ED using 25mcg pills. Notice that where the up/down period is unable to be broken into 3 exactly equal parts, the extra is put on the dose level at the last part of the period.


UP CONST DOWN
12 days 18days 30days
5% 40% 55%
234 555555555555555555555555 44444433333322222221111111.5.5.5.5.5.5.5

Hope this helps someone!
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://musc lechatroom.com/forum/showpost.php?p=40867&postcount=9


300 IU every third day amounts to 100 IU per day.

When I'm not supplementing with GH, I supplement with only ovidrel HCG predominantly, and some T gel and a little arimidex, ie:

220IU of Ovidrel daily
80mg compounded T gel daily (plus extra 40mg on some nights I may get lucky)
0.2mg arimidex daily

On that dose my total and bioavail T hovers close to the top of the reference range (rarely over it, usually under it).

On that dose my SHBG hovers around mid-way in the reference range.

On that dose my E2 hovers around the top of the male ultrasensitive reference range.

On that schedule I don't get spontaneous erections during the day (I find them annoying) and I occasionally get morning wood, but I can get an erection on demand, and hold it for around 30 minutes, around 9 nights out of 10.

###

Now anecdotally 6000IU of Ovidrel has the same efficacy as around 10000 IU of urinary HCG.

Ie: My 220 IU of Ovidrel daily has the same efficacy as around 350 IU of urinary derived HCG daily.

Even if your testicles respond better to HCG better than do mine, I still doubt that you would be able to maintain high total T (per the reference range) on your current schedule.

###

On an E3D schedule, your T will be highest the day after your injection, so if you measure your T on the day after your injection, then you should measure your highest T.

On an E3D schedule, if you measure T on the day of your HCG injection, or 2 days after your injection, then you should measure your lowest T.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://musc lechatroom.com/forum/showthread.php?p=41095&posted=1#post41095

Bitter melon for blood sugar control

So, as a pre-diabetic/metabolic syndrome dude, I've tried a LOT of different supplements which purportedly lower blood sugar. Never noticed any significant effect from any of them.

Saw a couple of studies supporting bitter melon's action. So wotthehell I picked up some bitter melon tea from a local Chinese grocery store. Drank 3 cups (turns out that's way too much) before going to bed the other night.
Woke up with a fasting glucose of 64 mg/dl!?! That's friggin' crazy. I've never seen a number that low in almost 3 years of monitoring. Drank some more the next day, consumed my usual foodstuffs during the day (nuts, nut butters, legumes, fruit, flourless bread and 85% dark chocolate) and came home to a reading of 80mg/dl. Never seen a number that low that time of day before either.

So, for me at least, this is a very powerful hypoglycemic agent. I'll keep using it but I'm gonna have to be a lot more careful with the dosage.


So, for me at least, this is a very powerful hypoglycemic agent. I'll keep using it but I'm gonna have to be a lot more careful with the dosage.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Goal:
DHEAs(500-640)mcg/dL(13.55-17.34)µmol/L------------------major player, 95% time overlooked
============================================================
Does anyone else not tolerate DHEA?

http://musc lechatroom.com/forum/showthread.php?t=3397


I know how important DHEA is... and mine drops to <200 if I don't take it, but it makes me very anxious, achy, shaky. My doctor says this is because it antagonizes already low cortisol and stimulates the immune system. That's exactly what it feels like...an overactive immune response with almost flu-like symptoms. Every time I try to get my DHEA up this happens, even with 15mg of hydrocortisone. Kind of a bummer, I was just wondering if anyone else has the same experience.
I don't necessarily believe that is true. Because it balances high corticosteroids does not automatically mean it antagonizes low cortisol.

More than likely it is dramatically increasing T3 levels, by stimulating enzyme D1, potentially causing what can be a very uncomfortable hyperthyroid situation.
The ensuing stimulation usually resolves in a few days.

Having said that, ANYTHING is possible where hormones are concerned.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://musc lechatroom.com/forum/showpost.php?p=40976&postcount=5

Actually, I don't think there is much difference between the two (extract vs recombinant) at least not enough to worry about. Here is a study that has it the other way around JanSz.

"Recombinant hCG at a dose of 250 mg (6500 IU) gives the same results as 5000 IU extractive hCG."

Evaluation of endocrine testing of Leydig cell function using
extractive and recombinant human chorionic gonadotropin and
different doses of recombinant human LH in normal men


From: European Journal of Endocrinology (2008) 159 171–178

(6500iu)rHCG=(5000iu)HCG

------------
Evaluation of endocrine testing of Leydig cell function using extractive and recombinant human chorionic gonadotropin and different doses of recombinant human LH in normal men

Results: ehCG induced dose-dependent increases in plasma estradiol and testosterone levels. They respectively peaked at 24 and 72 h after the injection. The most potent dose of ehCG (5000 IU) induced results similar to those observed with 250 µg (6500 IU) rhCG. By comparison with placebo, rhLH induced a significant and dose-dependent increase in plasma testosterone levels 4 h after the injection. Peak response of testosterone to rhLH and rhCG was significantly correlated. rhLH did not induce significant change in plasma estradiol level.

Conclusions: In normal men, a single i.v. injection of 150 IU rhLH induces a 25% rise in plasma testosterone levels by comparison with placebo. At the moment, the dynamic evaluation using hCG remains the gold standard test to explore the Leydig cell function. The use of 250 µg rhCG avoiding any contamination should be recommended.

====================================================================
http://www.emdserono.com/cmg.emdserono_us/en/images/ovidrel_prefilled_syringe_tcm115_19352.pdf

HOW SUPPLIED
Ovidrel® PreFilled Syringe (choriogonadotropin alfa injection) is
supplied in a sterile, liquid single dose pre-filled 1 mL syringe. Each
Ovidrel® PreFilled Syringe is fi lled with 0.515 mL containing 257.5
μg of chorio-gonadotropin alfa, 28.1 mg mannitol, 505 μg 85%
O-phosphoric acid, 103 μg L-methionine, 51.5 μg Poloxamer 188,
Sodium Hydroxide (for pH adjustment), and Water for Injection to
deliver 250 μg of chorio-gonadotropin alfa in 0.5 mL.
The following package combination is available:
• 1 pre-filled syringe containing 250 μg Ovidrel® PreFilled Syringe
NDC 44087-1150-1
====================================================================
Mixing instructions.

250 µg rhCG =(6500iu)rHCG=(5000iu)HCG=0.515 mL=51.5units(on insuline syringe)

Lets make solution having total volume=2.5 mL=250units
Lets use units of "regular/natural" HCG

That solution will have density of

(5000iu)/(2.5mL)=2000iu/mL=(2000iu)/(100units)=20iu/unit

To empty sterile vial add

content of Ovidrel syringe=0.515mL
and
2.5mL - 0.515mL=~2mL bacteriostatic water

250iu =250/20=12.5units
300iu =300/20=15units
400iu =400/20=20units
500iu =500/20=25units
600iu =600/20=30units

all above can be handled by the smallest syringe:

BD Ultrafine II U-100 Insulin Syringe 31 Gauge 3/10cc 5/16inch Short Needle--1/2 Unit Markings
===================================================================
http://musc lechatroom.com/forum/showpost.php?p=48255&postcount=11

July/31/2009
I mix my 0.5 mL (250 mcg) Ovidrel with 2.5 mL water.

Each day I inject 0.08 mL (6.7 mcg) of the combined mixture. It lasts around 37 days.

Plus I also apply a little transdermal T (testosterone) gel / cream daily.

I get most of my T boost from HCG, but not all. Like Dr Crisler, I also believe that we do need daily variability in our T levels.
===========================================================
Mirësevini në ccTLD .al lthingsmale.com/pdfs/instructions/USE_OVIDREL.pdf

Ovidrel mixing instructions from Dr. John's forum.

You need the Ovidrel injection, which is a .5ml injection equivalent to
10000iu of HCG. You need bacteriostatic water and a sterile vial.
Draw out 9.5 ml of the BC water and inject into the sterile vial. Inject the
Ovidrel into the sterile vial with the 9.5ml BC water and mix GENTLY to end
up with a total of 10 ml. Then 25 units on an insulin syringe will be the
equivalent of 250iu of HCG. This will last 20 weeks at the 250iu 2x a week
dosing. Prescribe one injection at a time, potency lasts longer.
BD Ultrafine II U-100 Insulin Syringe 31 Gauge 3/10cc 5/16inch Short
Needle--1/2 Unit Markings
The advice for script is to simply say "use as directed".

-------------
Buy Ovidrel / Ovitrelle (Choriogonadotropin Alfa)
Ovitrelle 250 mcg Serono Syringes $79.00

Generic Name: Choriogonadotropin Alfa
In order to buy Ovidrel online you will require a valid prescription
===================
===================
http://www.genericcancerdrugs.com/drug_details/Ovitrelle_3746.html
Ovitrelle syringe 250mcg



=================
=================
http://www.musc lechatroom.com/forum/showthread.php?14401-Clomid-instead-of-HCG&p=144967#post144967
That's only because your HCG dose was too low.

The response to HCG varies very widely amongst males.

You and your medical professional adviser know your HCG does is close to optimum when your testicle size is restored back to where they were before you started supplementing with exogenous T. The concept of a fixed dose of HCG (eg: 500IU) being optimal is not reliable, and has definitely not been validated among a wide sample of males.

eg: in order for my testicle size to be close to where they were before I started hormone modulation therapy, I have to supplement with around 34 micrograms of HCG (as ovidrel) per day, which amounts to either:
a) 800 IU per day (if you believe 250 mcg of Ovidrel HCG = 6,000 IU urinary derived HCG)
or:
b) 1350 IU per day (if you believe 250 mcg of Ovidrel HCG = 10,000 IU urinary derived HCG).

I am a well known hypermetabolizer of testosterone, but that's not relevant to testicular size, because most of our testicular size is due to sertoli cells, which are triggered by FSH. Ie: only a much smaller fraction of our testicular size is due to the leydig cells which are tirggered by LH in order to make testosterone.

.


There is research paper correlating HCG dose with 17-hydroxy-progesterone (healty individuals tested)

do you
accept those findings
measure your 17-hydroxy-progesterone

Other than testicle size
what would you miss in significant way, if you reverted to 350iu/EOD (as supported by above research?


=============================
5/14/2011
http://www.mus clechatroom.com/forum/showthread.php?17926-HCG-amp-Ovidrel-protocol&p=145668#post145668

Re: HCG & Ovidrel protocol
Originally Posted by JanSz
There is research paper correlating HCG dose with 17-hydroxy-progesterone (healty individuals tested)
They obviously didn't encounter any hypermetabolizer males amongst their group.

Hypermetabolizers need far more HCG than "average" in order to stay optimum.

Originally Posted by JanSz
Other than testicle size
what would you miss in significant way, if you reverted to 350iu/EOD (as supported by above research)?
35mcg of Ovidrel per day (900 IU per day) is for me a baseline minimum. I take 17 mcg (450 IU) at 7am, and 17 mcg (450 IU) at 7pm.

Some days I need to increase HCG dose above 35mcg (900 IU) per day because I've been extra energetic during the week. I always notice the symptoms of too low T around the middle of the day. On such days I have to increase my 7pm dose of HCG to be even greater than 17 mcg (450 IU) to compensate. My medical professional adviser allows me to adjust my dosage in this way.

=================================================
=================================================
http://www.mus clechatroom.com/forum/showthread.php?17988-Help-Ups-and-Down-of-HCG-amp-TRT&p=147133#post147133

Actually I now apply 800 IU of HCG daily, every day.

I had to switch to 800 IU daily PLUS 1.6 grams of testosterone applied transdermally daily.

I had to make this switch once my thyroid hormone levels increased to youthful, because I increased the rate I excrete testosterone, and I was finding that on "transdermal-only" days it was too messy trying to apply 3.2 grams of testosterone applied transdermally, as 16mL of 20% testosterone. (JanSz comment, this must be typo, 1.6mL)

As it is, my body completely absorbs 4mL of testosterone in lipoderm within 12 hours, so when I apply my second dose of transdermal T around 12 hours after my first dose, then the original area is almost completely dry.

Note that before I started boosting my resting metabolic rate, my skin was still a little grasy 24 hours later, on the few occasions when I had to apply large doses of T (eg: when I was ill).

In other words, absorption is entirely dependent on resting metabolic rate.

.
My own benchmark when using TD testosterone, 10grams/day Androgel(1%)-->100mg/day testosterone
TT=1100, SHBG~18, BAT~575, Albumin=4.3
no HCG at that time, completely shrunken testicles
---------------------------------------------------------------------------------------
Assume 1gram=1mL (often it is closer to 1mL=0.9gram)

1.6grams of 20% TD testosterone contains 1.6*1000*0.2=320mg testosterone
This are huge amounts of testosterone.
I am concerned that lots of test does not enter the system.

It would help me in understanding if you could post TotalTest in serum on such a protocol.
If that is not asking for too much, please
TT, SHBG, Albumin
For privacy, PM would be ok too.
===============================================

I would appreciate if you could discuss thyroid hormones numerically.

I am still working on assumption that for 99% of us TSH provides resonable indication.
That is, while supplementing with any combination of T4 and or T3, from pov of TSH the goal is
TSH(0.5-1)




As it is, my body completely absorbs 4mL of testosterone in lipoderm within 12 hours, so when I apply my second dose of transdermal T around 12 hours after my first dose, then the original area is almost completely dry.
Note that before I started boosting my resting metabolic rate, my skin was still a little grasy 24 hours later, on the few occasions when I had to apply large doses of T (eg: when I was ill).
Where are you applying testosterone? I am concerned, if skin stays moist for almost 12 hrs (or sometimes 24hrs), it must be exposed skin (face, neck) otherwise most of it will rub off clothes.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://www.matrixnutritionandfitness.com/forum/showthread.php?p=41405#post41405

Sarapin

Pitcher Plant

http://www.sarapin.com/inj_clinical.html#Dosage

///
CLINICAL OBSERVATIONS
Following extensive clinical experience with SARAPIN, Dr. Bernard D. Judovich issued the first report of his findings in an article entitled "For the Relief of Pain, a Preliminary Report on a New Therapy."6 Largely through his efforts and those of Dr. William Bates, of Philadelphia, the injection technique necessary for satisfactory results have become established. In a rapidly growing number of pain clinics, in industrial plants and in private practice as well, SARAPIN® is giving welcome relief to sufferers.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
ferritin
hemochromatosis
marianco

Low ferritin low thyroid - DR M
detailed description provided by dr marianco on this thread:
http://forum.mesomorphosis.com/mens-health-forum/low-ferritin-low-thyroid-134273298.html

Without iron, the metabolic actions triggered by thyroid hormone grind to a halt. No ATP, not cellular metabolism. Thyroid hormone replacement does not work well without adequate iron levels - best measured by serum Ferritin levels.
Note that excessive iron is highly oxidizing and destructive. Thus I prefer the midrange when it comes to an "optimal" level.
---------------------------------------------------------------------
QuestDiagnostics
Ferritin(20-380)ng/dL
------------------------
For people with confirmed hemohromatosis
Iron Overload Diseases Association, INC.
confirmed by all three tests as shown here:
http://www.ironoverload.org/diagnosis.html
if positive for hemochromatosis then use ferritin range as discussed here:
http://www.ironoverload.org/anemia.htm

Ferritin: 5 to probably 50.
-------------------------------------
Dr marianco
Thank you for your opinion.
Your statement above is giving nice
desired ferritin boundary (75-150)ng/dL

with preference toward higher end.
---------------------------------------
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
HUMAN ENGINE SPECS

In ideal world, where doctors know what they are doing, we would have few problems.

When I disassembled engines and put them back together, I had to start somewhere, otherwise the engine would not even turn.

Engines have specs, then there is fine tunning.
=========================================================
I worked out this set of specs:


My own Goals
DHEAs(500-640)mcg/dL(13.55-17.34)µmol/L------------------major player, 95% time overlooked
Progesterone(0.9-1.2)ng/mL
Pregnenolone(> 100ng/dL)
Estradiol, Ultrasensitive(25-29)pg/mL
Estrone, LC/MS/MS (23244X)
do not use Anastrozole if possible or minimize its use
BATest(342, 460-575)ng/dL------------stay around 342 if you need more than 1.5mg/week Anastrozole to control E2
DHT(60-90)ng/dL (I am active when it gets over or under this range)
RT3 in lower half of range
TotalT3 in upper 1/3 range
FreeT3~400pg/dL or higher if TotalT3 goal not reached
TotalT4>bottom of range
Body temperature (36.25 - 36.80)C = (97.25 - 98.24)F
Ferritin(100-150)

###

============================================================
You do not (usually) influence SHBG directly (Danazol)
SHBG(15-25) is very good
SHBG(10-30) is goo too
outside this ranges not so good
============================================================
Prolactin, if high, repeat the test first
if persist, investigate (heavily) before taking medicine (cabergoline)
============================================================
DO NOT do direct FreeT testing
DO NOT worry much about TotalTestosterone, always look at it in the context of BAT & FreeT
---
If you can't do Quest's
Testosterone, Free, Bio/Total (LC/MS/MS) Code: 14966X
do
TotalT, SHBG and Albumin
and
read your FreeT from chart

Androgen deficiency in the adult ... - Google Books

FreeT(160-300) is desirable range (insist on it if IGF-1 is low or IGFBP-3 high)
FreeT(230-300) is best, (300-350) even better
FreeT>100 will start engine turning
=====================================================================
Do not waste money measuring LH and FSH while taking external testosterone, they are suppessed.
=====================================================================
When supplying external testosterone do it at least EOD(EveryOtherDay)
Transdemals-Every day
Injectable, T-cypionate, enanthate, Sustanon 250,---EOD
Nebido-15 days
=====================================================================
E2
One of major reasons for frequent T injections is to keep TT levels at certain (tolerable) levels.
High (TTmax-TTmin) will result in high variations in E2 levels.
Among other problems, high variations of E2 makes it impossible to figure out its average levels using one time blood draw.
Information about average E2 levels is actionable.
We want E2 to be in certain range (see above)
Without good measurements we are not able to decide on dose of Arimidex or if we have to withdraw AI completely.

On attached chart you can see that E2 can be 44 one day and 5 just 24 hours latter.
====================================================================
.
 

Attachments

JanSz

JanSz

Well-known member
Awards
1
  • Established
Yeah - the 12 week interval will cause potential difficulties in adjustment of dose. I found a presentation (click on "view presentation" in this page - agingmale2006.com/abstracts/abs_sag_long-acting_vs_standard_testosterone.asp (sorry can't post links yet - not enough posts!)) that implies that it is quite stable. Note though that the presentation is sponsored by Schering!

It is a very expensive form of test. Luckily in the UK the Health Service will bear the brunt of this otherwise it would be pretty hard to afford!

I guess my life's not too bad at the moment. The ED comes and goes and I'm liable to mood swings and short episodes of depression. Libido's not great either. I think I need to think long and hard about going on test. The thing is with my levels being borderline they're not going to be getting better. I'll probably find that my symptoms get gradually worse over the next few years unless I treat it. I'm hoping that they might find some way of increasing my test without supplementing. My running's also important to me and I'm a bit worried it may be detrimentally affected by mucking about with my endocrine system! I guess some may say that the running would only get better with higher levels of test and the associated increase in hematocrit!
I was not able to figure out the link that you have given me.
But instead, (fishing around your info) I found this:

Clinical experience with a new long-acting injectable testosterone undecanoate (Nebido)

I looked only at:

Comparison of kinetics, efficacy and safety of the long-acting testosterone undecanoate formulation with standard testosterone enanthate

Found that Nebido comes in 4mL ampoules.
So it may be difficult to divide doses, but I am sure it can be done if one really want.

Second, I have found a chart showing kinetics of both
types of testosterone.
I am not commenting on them assuming once every 3 weeks T-enanthate shots (stupid).
But I noted two items:
#1, Nebido's TT drops after 2.5-3 weeks
#2, study aims to achieve TT~12nmol/L=350ng/dL (or there about, vey low levels)

We here aim at BAT(BioAvailableTestosterone)
BAT~(460-575)

that (on average, depending on SHBG) ends with TT~(900-1100)ng/dL

350ng/dL is better than nothing, is something ment for 69yo men (like me),
but I am not buying it.

So possibly my previous assumptions was too optimistic,
better would be to suck the Nebido from the 4mL ampoule into 3 equal syringes, 4/3=1 1/3 cc
and inject every

1000/3/22=15 days


That would be on average:

=1000/3/15*7=155.6mg/week

Or put it into one larger syringe and replace needles every shot,
store in refrigerator unused portion.

With Nebido, slow acting, I would not test my BAT levels sooner than after 3 months.
But if you can afford it, the more tests the better.
Hematocrit, DHEAs, E2 & DHT tests you may want to do more often.
----------------------------------------

Dependind on ones starting point, assuming it is you with current low TT,
it may be a good idea to start with 2 doses at the first shot and then follow normal routine.
----------------------------------------
You being in UK, do not have access to BAT
do
TotalT
SHBG
Albumin
and use chart to figure out FreeT
desirable FreeT(250-300)
do not make FreeT direct tests, useless, worst-confusing.
.
 

Attachments

JanSz

JanSz

Well-known member
Awards
1
  • Established
http://library.umsmed.edu/Medicare/medi-ser-iron.html

SUBJECT: SERUM IRON STUDIES

¨ Iron

¨ Total Iron Binding Capacity (TIBC)

¨ Transferrin

¨ Ferritin

ICD-9 CODES THAT SUPPORT MEDICAL NECESSITY: ©

250.01-250.91 Insulin dependent diabetes mellitus

275.0 Disorders of iron metabolism

280.0 Iron deficiency anemia secondary to blood loss (chronic)

280.1 Iron deficiency anemia secondary to inadequate dietary intake

280.8 Other specified iron deficiency anemias

280.9 Iron deficiency anemia, unspecified

282.4 Thalassemia

282.60 Sickle cell anemia, unspecified

282.63 Sickle cell/hb-c disease

282.69 Other sickle cell anemia

282.7 Other hemoglobinopathies

307.52 Pica

425.4 Other primary cardiomyopathies

536.0 Achlorhydria

579.0 Celiac disease

579.2 Blind loop syndrome

579.8 Other and unspecified postsurgical nonabsorption

579.9 Unspecified intestinal malabsorption

585 Chronic renal failure

608.3 Atrophy of testis

648.2 Anemia

713.0 Arthropathy associated with other endocrine and metabolic disorders; code first underlying disease as hemochromatosis (275.0)

790.6 Other abnormal blood chemistry, iron

964.0 Acute iron poisoning

999.8 Other transfusion reaction

V56.0 Extracorporeal dialysis

V56.8 Peritoneal dialysis
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://www.diabetesexplained.com/diabetic-conversions.html


Diabetic Conversion Factors.
If you are in a hurry and want instant answers skip to the conversions at the bottom of this page. Otherwise stay with me for, hopefully, education and entertainment.

When I was given my first glucose meter I did notice the small mmol/L at the bottom of the screen but it was under a large number and I paid it scant notice. It was the number that had my full attention - I am sure all you diabetics out there will understand.

In time I nicknamed this 'Naughty Number' because it behaved like a over active child - it was never still! It could have been measured in carrot tops for all I cared, as long as it behaved itself.

Then I started to educate myself and things changed - confusion crept in - now isn't that a surprise folks? It seemed every book or website I read was giving blood glucose readings in mg/dl and the figures were so high.

Made mine look good but I knew that there was no way these people would still be alive with those figures so the conversion rate was obviously not 1-1, but what was it and why were there two different ways of measuring the same thing?

Now mmol/L were familiar to me. Way back when I had met up with them in biochemistry classes (and was not that enamoured with them then - must have had a premonition or something) but I knew mmol/L stood for millimoles per litre and I was aware that the mole bit had nothing to do with cute furry critters that made unsightly humps in lawns but was short for the molecular weight of a substance.

For the really technically inclined a mole is the number of atoms in exactly 12 g of carbon-12.

A millimole is a thousandth of a mole, and is 602,253,000,000,000,000,000 molecules of glucose - see, I did learn something in those classes after all.

mmol/L is the SI or Systeme International unit, the world standard for measuring blood glucose. It is normally expressed to one decimal place eg 6.7

So where did the mg/dL come in, what did it mean and why is it used? Turned out it stood for milligrams per decilitre (which is thousandth's of a gram per tenth of a litre).

So basically they are finding the weight of the glucose present in each decilitre of blood. mg/dL is normally expressed to the nearest whole number eg 120. This is the traditional way of measuring blood glucose in the US and in a number of other countries.

It is becoming less common in it's usage in scientific circle but will undoubtedly be with us for a long time as so many people are accustomed to it. The way to convert from one to another is really easy.

You just multiply the mmol/L by 18 to get mg/dL.
You just divide the mg/dL by 18 to get mmol/L.
(Remember, this conversion figure is for blood glucose only)

Confused about whether your country measures blood glucose in mg/dL or mmol/L? Check on the list here. If I missed the place where you live I am sorry, the world seems to be changing faster than I can keep up!

So that appeared to be that. I had worked out how to convert mmol/L to mg/dL and all was tiddly - or was it? It was until my first blood test results came back and I had a look at my cholesterol levels. These were also expressed as mmol/L.

Wanting to know how they looked in relation to normal figures I checked my handy books and websites for information on the normal ranges - needless to say they were in mg/dL.

At this time I am seriously beginning to wonder about the term 'world standard'! However this does not phase me, after all I know how to convert, just times or divide by 18. Yea right! You want to try this sometimes, it makes for interesting numbers, to say the least.

More head scratching and plenty of unladylike language before the penny drops - we are looking at molecular weights here and naturally the weight of a molecule of glucose is not going to be the same as the weight of a molecule of fat. The number we multiply by is going to have to be different. Okay, okay, I know I should have known this but those biochem. classes were a long way back!

A lot of searching later I found the multiplication factors and the results of my tests became blindingly clear. At that moment I did sort of wish that I could roll the clock back and live in blissful ignorance but it was to late - damn. I try to comfort myself with the thought that some of you out there will be grateful for the knowledge.



The conversion from SI to US units.
Please note that I have sometimes given two ways of doing these. Both come up with identical results. My mathematically orientated husband felt it was not necessary to give both versions as it was obvious that they were the same anyway.

I, to whom mathematics is a closed book, did not see the connection and felt that there might be others of you out there who felt the same and who may have seen one or other method used in another publication and wondered why mine was different - result, you get both and can take your pick which one you use.

I have given the conversions for the tests done on my blood here in the UK. If you have others you are wondering about please let us know and we will do our best to find the answers for you.

Understanding prefixes:-
In most calculations you will see abbreviations like g or gm for grams and mol for moles. Usually there will be another letter, or sometimes two, in front of these. Many of you may be familiar with the letters and know what they mean but for those of you who, like me, have trouble remembering what is what here is a brief reminder.

G giga 10*9 = one billion
M mega 10*6 = one million
k kilo 10*3 = one thousand
h hecto 10*2 = one hundred
da deka 10 = one ten
d deci 10*-1 = one tenth
c centi 10*-2 = one hundredth
m milli 10*-3 = one thousandth
µ micro 10*-6 = one millionth
n nano 10*-9 = one billionth
p pico 10*-12 = one trillionth
f femto 10*-15 = one quadrillionth

In other word µmol (micromole) stands for one millionth of a mole and pg (picogram) stands for one trillionth of a gram. Get the idea?

Note:-
U/L stands for units per litre
mIU/1 stand for milli International Units/litre

Sometimes the conventional units are given in g/L instead of g/dl. If that is the case do the following conversion first.
Divide g/L by 10.0 to get g/dL
Multiply g/dL by 10.0 to get g/L.

The list below is in alphabetical order.
To convert Acetoacetic acid readings:-
Divide mmol/L by 0.098 to get mg/dL
Multiply mg/dL by 0.098 to get mmol/L

To convert Acetone readings:-
Divide mmol/L by 0.172 to get mg/dL
Multiply mg/dL by 0.172 to get mmol/L

To convert Albumin readings:-
Divide the g/L by 10 to get g/dL.
Multiply the g/dL by 10 to get g/L.

To convert Bilirubin readings:-
Divide the mol/L by 17.1 to get mg/dl.
Multiply the mg/dl by 17.1 to get mol/L.

Note -
in the red blood cell, white blood cell and platelet count, because of the different units being used, the two readings are identical and don’t actually need conversion. If you want to do the maths yourself (Multiply or devide by 1) the factors are as set out below.

To convert Red blood cell count readings:- (see note above)
Conventional units use ‘cells x 10*6/µL’
SI units use ‘cells x 10*12/L’
Divide ‘cells x 10*12/L’ by 1.0 to get ‘cells x 10*6/µL’
Multiply ‘cells x 10*6/µL’ by 1.0 to get ‘cells x 10*12/L’

To convert White blood cell count readings:- (see note above)
Conventional units use ‘cells x 10*3/µL’
SI units use ‘cells x 10*9/L’
Divide ‘cells x 10*9/L’ by 1.0 to get ‘cells x 10*3/µL’
Multiply ‘cells x 10*3/µL’ by 1.0 to get ‘cells x 10*9/L’

To convert Platelets (thrombocytes) readings:- (see note above)
Conventional units use ‘number of platelets x 10*3/µL’
SI units use ‘number of platelets x 10*9/L’
Divide ‘number of platelets x 10*9/L’ by 1.0 to get ‘number of platelets x 10*3/µL’
Multiply ‘number of platelets x 10*3/µL’ by 1.0 to get ‘number of platelets x 10*9/L’

To convert Blood Glucose readings:-
Divide the mg/dL by 18 to get mmol/L.
Multiply the mmol/L by 18 to get mg/dL.
OR
Divide the mmol/L by 0.0555 to get mg/dL
Multiply the mg/dL by 0.0555 to get mmol/L

To convert BUN readings:-
Divide the mmol/L by 0.357 to get mg/dL.
Multiply the mg/dL by 0.357 to mmol/L.

To convert Bromide readings:-
Divide mmol/L by 0.125 to get mg/dL
Multiply mg/dL by 0.125 to get mmol/L

To convert Calcium readings:-
Divide mmol/L by 0.25 to get mg/dL
Multiply mg/dL by 0.25 to get mmol/L
Divide mmol/L by 0.05 to get mEq/L
Multiply mEq/L by 0.05 to get mmol/L

To convert Total Cholesterol readings:-
Divide the mmol/L by 0.0259 to get mg/dL
Multiply the mg/dL by 0.0259 to get mmol/L

To convert HDL and LDL readings:-
Divide the mg/dL by 38.67 to get mmol/L.
Multiply the mmol/L by 38.67 to get mg/dL.
OR
Divide the mmol/L by 0.0259 to get mg/dL
Multiply the mg/dL by 0.0259 to get mmol/

To convert Copper readings:-
Divide µmol/L by 0.157 to get µg/dL
Multiply µg/dL by 0.157 to get µmol/L

To convert Cortisol readings:-
Divide nmol/L by 27.95 to get µg/dL
Multiply µg/dL by 27.95 to get nmol/L (nanomoles per litre)

To convert C-peptide readings:-
Divide the nmol/L by 0.333 to get ng/mL.
Multiply the ng/mL by 0.333 to get nmol/L

To convert Creatine readings:-
Divide mol/L by 76.26 to get mg/dL
Multiply mg/dL by 76.26 to get mol/L.

To convert Creatinine readings:-
Divide the mol/L by 88.4 to get mg/dL.
Multiply the mg/dL by 88.4 to get mol/L.

To convert Creatinine clearance readings:-
Divide the ml/s by 0.0167 to get ml/min.
Multiply the ml/min L by 0.0167 to get ml/s.

To convert degrees C to degrees F
Take the degrees C, multiply by 9. Divide the answer by 5. Add 32. That will give you your degrees F.
Eg. 37 x 9 = 333.
333 / 5 = 66.6.
66.6 + 32 = 98.6 degrees F

(Therefore 37 deg C equals 98.6 deg F)

To convert degrees F to degrees C
Take the degrees F, minus 32, divide the answer by 9, multiply that answer by 5.That will give you your degrees C.
Eg. 98.6 - 32 = 66.6
66.6 / 9 = 7.4
7.4 x 5 = 37 degrees C

To convert Fluoride readings:-
Divide µmol/L by 52.6 to get µg/mL
Multiply µg/mL by 52.6 to get µmol/L

To convert Glycated haemoglobin (glycosylated hemoglobin A1, A1C)
Conventional units use term - % of total hemoglobin
SI units use term - proportion of total haemoglobin
Divide ‘proportion of total haemoglobin’ by 0.01 to get ‘% of total hemoglobin’.
Multiply ‘% of total hemoglobin’ by 0.01 to get ‘proportion of total haemoglobin’

To convert Haemoglobin readings:- (See Note below)
Divide mmol/L by 0.6206 to get g/dl
Multiply g/dl by 0.6206 to get mmol/L

Note -
Sometimes the conventional units are given in g/L instead of g/dl. If that is the case do the following conversion first.
Divide g/L by 10.0 to get g/dL
Multiply g/dL by 10.0 to get g/L.

To convert Hematocrit readings:-
Conventional units use %
SI units use ‘proportion of 1.0’
Divide ‘proportion of 1.0’ by 0.01 to get %
Multiply % by 0.01 to get ‘proportion of 1.0’

To convert Insulin readings:-
Divide pmol/L by 6.945 to get µIU/mL
Multiply µIU/mL by 6.945 to get pmol/L

To convert Iron (total ) readings:-
Divide µmol/L by 0.179 to get µg/dL
Multiply µg/dL by 0.179 to get µmol/L

To convert LDL and HDL readings:-
Divide the mg/dL by 38.67 to get mmol/L.
Multiply the mmol/L by 38.67 to get mg/dL.
OR
Divide the mmol/L by 0.0259 to get mg/dL
Multiply the mg/dL by 0.0259 to get mmol/

To convert Magnesium readings:-
Divide the µg/dl by 5.494 to get µmol/L
Multiply the µmol/L by 5.494 to get µg/dl

To convert Platelets (thrombocytes) readings:-
See Blood cells above.

To convert Potassium readings:-
Divide the mEq/l by 1 to get mmol/L.
Multiply the mmol/L by 1 to get mEq/l. This is therefore a 1-1 conversion. (The two are the same!)

To convert Protein (serum total) readings:-
Divide g/dl by 0.1000 to get g/L
Multiply g/L by 0.1000 to get g/dl
OR
Divide g/L by 10.0 to get g/dL
Multiply g/dL by 10.0 to get g/L

To convert Protein (urine/fluid total) readings:-
Divide mg/dl by 0.1000 to get mg/L
Multiply mg/L by 0.1000 to get mg/dl

To convert Red Blood cell count:-
(see note above under blood cells)
Because of the relationship of the different units being used the two readings are actually identical and don't need conversion.

To convert the Reticulocyte count readings:-
Conventional units use ‘% of RBCs’
SI units use ‘Proportion of 1.0’
Divide ‘Proportion of 1.0’ by 0.01 to get ‘% of RBCs’
Multiply ‘% of RBCs’ by 0.01 to get ‘Proportion of 1.0’

To convert Selenium readings:-
Divide the µg/dl by 7.896 to get µmol/L
Multiply the µmol/L by 7.896 to get µg/dl

To convert Serum Magnesium readings:-
Divide mmol/L by 0.411 to get mg/dl
Multiply mg/dL by 0.411 to get mmol/L
OR
Divide the mg/dl by 2,430 to get mmol/L
Multiply the mmol/L by 2.430 to get mg/dl
Or if your results are given in mEq/L then:-
Divide mmol/L by o.5o to get mEq/L
Multiply mEq/L by 0.50 to get mmol/L
OR
Divide mEq/L by 2.0 to get mmol/L
Multiply mmol/L by 2.0 to get mEq/L

To convert Serum Zinc readings:-
Divide the µg/dl by 6.541 to get µmol/L
Multiply the µmol/L by 6.541 to get µg/dl.
OR
Divide µmol/L by 0.153 to get µg/dL
Multiply the µg/dL by 0.153 to get µmol/L

To convert Sodium readings:-
Divide the mEq/l by 1 to get mmol/L.
Multiply the mmol/L by 1 to get mEq/l. This is therefore a 1-1 conversion. (The two are the same!)

To convert Thyroxine, free (T4) readings:-
Divide pmol/L by 12.87 to get ng/dL (nanograms per decilitre)
Multiply ng/dL by 12.87 to get pmol/L (picomoles per litre)

To convert Thyroxine, total (T4) readings:-
Divide nmol/L by 12.87 to get µg/dL
Multiply µg/dL by 12.87 to get nmol/L

To convert Total Cholesterol readings:-
Divide the mmol/L by 0.0259 to get mg/dL
Multiply the mg/dL by 0.0259 to get mmol/L

To convert Triiodothyronine free (T3) readings:-
Divide pmol/L by 0.0154 to get pg/dL
Multiply pg/dL by 0.0154 to get pmol/L

To convert Triiodothyronine total (T3) readings:-
Divide nmol/L by 0.0154 to get ng/dL
Multiply ng/dL by 0.0154 to get nmol/L

To convert Triglyceride readings:-
Divide the mg/dL by 88.57 to get mmol/L.
Multiply the mmol/L by 88.57 to get mg/dL.
OR
Divide mmol/L by 0.0113 to get mg/dL
Multiply mg/dL by 0.0113 to get mmol/L

To convert Urea Nitrogen (BUN) readings:-
Divide mmol/L by 0.357 to get mg/dL
Multiply mg/dL by 0.357 to get mmol/L

To convert Uric Acid readings:-
Divide µmol/L by 59.48 to get mg/dL
Multiply mg/dL by 59.48 to get µmol/L

To convert Vitamin A (retinol) readings:-
Divide µmol/L by 0.0349 to get µg/dL
Multiply µg/dL by 0.0349 to get µmol/L

To convert Vitamin B6 (pyridoxine) readings:-
Divide nmol/L by 4.046 to get ng/mL
Multiply ng/mL by 4.046 to get nmol/L

To convert Vitamin B12 (cyanocobalamin) readings:-
Divide pmol/L by 0.738 to get pg/mL
Multiply pg/mL by 0.738 to get pmol/L

To convert Vitamin C (ascorbic acid) readings:-
Divide µmol/L by 56.78 to get mg/dL
Multiply mg/dL by 56.78 to get µmol/L

To convert Vitamin D readings:-
a)1,25-Dihydroxyvitamin D
Divide pmol/L by 2.6 to get pg/mL
Multiply pg/mL by 2.6 to get pmol/L
Or
b)25-Hydroxyvitamin D readings:-
Divide nmol/L by 2.496 to get ng/mL
Multiply ng/mL by 2.496 to get nmol/L

To convert Vitamin E readings:-
Divide µmol/L by 23.22 to get mg/dL
Multiply mg/dL by 23.22 to get µmol/L

To convert Vitamin K readings:-
Divide nmol/L by 2.22 to get ng/mL
Multiply ng/mL by 2.22 to get nmol/L

To convert White Blood cell count:-
(see note above under blood cells)
Because of the relationship of the different units being used the two readings are actually identical and don't need conversion.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://mus clechatroom.com/forum/showpost.php?p=49405&postcount=17

Originally Posted by agoraphobe
for some reason when you say that it makes me cringe, why not just say doctor? lol
Because the word doctor has lost it's original meaning - which was teacher - and has become:

doctor = professional with qualifications in medicine to whom you subcontract the management of your health

###

Since I encourage others to take ownership of their own health management, therefore by doing so they cannot subcontract the management of their health to another individual.

Since I encourage others to take ownership of their own health management, therefore by doing so they must treat other individuals as advisers only.

Therefore I encourage others to treat doctors only as an adviser, not a controller, and not a prescriber.

(prescriber = someone who prescribes / mandates what you must do)

###

So I have to replace the word doctor (whose meaning has been corrupted) with another word which means:

doctor = professional with qualifications in either biology and/or medicine, who advises you on how you can manage your own health, but doesn't believe it's "my way or the highway".

If there were a a single word, or two words, which I considered universally conveyed that concept, then I would use the single word, or the two words.

As it is, I could only find a minimum of three words, medical professional adviser, and I have to accept that some people won't be able to read into those three words what I'm trying to convey.

Life's a compromise.
 
The Matrix

The Matrix

Well-known member
Awards
1
  • Established
This post is a place maker,

I will not answer any questions here.


thank you.


----------------------------- =========
What are your IGF-1 levels looking like?
As my igf-1 levels got into 300's my reverse t-3 dropped from 28 to 18 and felt better and was building muscle mass like crazy.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://muscle chatroom.com/forum/showpost.php?p=55916&postcount=66

Omnitrope Pen5:
http://www.omnitrope.com/omnitrope/resource/LR11631OmniPenPlcmat_2lang.pdf



The Omnitrope Pen5 uses the same needle-inside-a molded-housing as all insulin pens, 31G.

I have two such pens (both identical):
1) a Pen5 device with a disposable Omnitrope GH cartridge with GH in it - let's call this my "GH pen"
2) a Pen5 device with a disposable Omnitrope GH cartridge, refilled with HCG - let's call this my "HCG pen".

I attach a fresh (unused) needle-inside-a molded-housing to my HCG pen, and I inject myself with HCG.

I remove the used needle-inside-a molded-housing from my HCG pen, and I attach it to my GH pen, and I inject myself with GH.

I then discard the needle-inside-a molded-housing (in my sharps bin).

Repeat daily.

.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Omnitrope Pen5

http://hallandalerx.com/growth_hormone.html

Common Formulations:
Omnitrope Pen5 5mg pen
Omnitrope 5.8 mg vial
Genotropin 0.2 mg MiniQuick
Genotropin 0.4 mg MiniQuick
Genotropin 0.6 mg MiniQuick
Genotropin 0.8 mg MiniQuick
Genotropin 1 mg MiniQuick
Genotropin 1.2 mg MiniQuick
Genotropin 1.4 mg MiniQuick
Saizen 5 mg Vial
Saizen 8.8 mg Vial
Norditropin 5 mg Pen
Norditropin 10 mg Pen
===============================================

Phil uses/will use

http://www.humatrope.com/index.jsp
http://mus clechatroom.com/forum/showpost.php?p=64975&postcount=11
Humatrope 6mg. pen got approved so my cost is $10 per month.
My Dr. has me starting on .3 mgs for the first week 6 days on and one off.
Then If I don't have any problems I go up to .4 mgs.
I get meds for 90 days but on this one it's only for 30 days so the script said
6 pens and all this crap to get 2 pens for 30 days.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Generic Arimidex

When Will Generic Arimidex Be Available?
The first patent for Arimidex currently expires in June 2010. Although this patent originally was set to expire in December 2009, the manufacturer was given an extension for performing much needed pediatric studies.

June 2010 is the earliest predictable date that a generic version of Arimidex could become available.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
Phil
says:
http://mus clechatroom.com/forum/showpost.php?p=74354&postcount=34
I am on HGH Humatrope the 6mg. Pen and I do .3mgs or .9 IU's a day. I had a IGFBP-1 test below the normal range and my IGF-1 was 130 before I started on HGH. I was due to test my Testosterone and Thyroid levels after only 3 shots. My IGF-1 went up from 130 to 262 over the top of the range for my age. And I went hyper on my throid meds and had to lower them. I am inpressed with this only 3 shots.
===============================================
http://mus clechatroom.com/forum/showpost.php?p=73356&postcount=71
Quote:
Originally Posted by BigJimcalhoun
How about a link to Dr. Mark Gordons Secetatropon or whatever it was? I searched, but could not find a place that actually sold it.
http://secretropinrx.com/

the site lists a pharmacy.
=================================================
http://mus clechatroom.com/forum/showpost.php?p=73365&postcount=74

Just in case anyone has not seen this yet, I thought it might be of interest. I have not watched it yet.

Secretropin: A Growth Hormone Secretagogue
Secretropin: A Growth Hormone Secretagogue

USE MS IE to view this presentation.

Thank you.

I use Google Chrome, presentation does not work on Chrome.
MS IE works ok.

Sound and slides.
..
Subject: Secretropin: A Growth Hormone Secretagogue
Speaker(s): Mark Gordon, MD
Date/Time: 2/27/2009 12:44:24 PM

===============================================

Arginine

E2----helps in increse of GH (guys, easy on Arimidex).

....
dr Gordon's practice used to do 15000-20000iu GH per year
now 90% people are on Secretropin, rest on combnation.

5% failure rate,

----------------------------------------
GH supplementatin works only if there is good support in other hormone levels.

looking at time 17.39, slide for (my NJ neighbor) 34 y/o
I note in dr Gordon's "Ideal Range" column

TT=1000(patient have 1853 note this and do not be scared of high TT when required)CrazyCrew are you listening??
SHBG=40(hopefully mistake, anybody able to contact dr Gordon, plus dr Gordon have not checked this guy's SHBG)(looks like he follows FreeT only)
DHT=35ng/dL(isn't it too low??)
E2=40pg/mL(lets take a note on this, high E2 helps in GH)(patient actually have 33.9)
cortisol<15(what is going on??? chllin help)

If DHEA is not corrected the response is lower, (bummer, I am not able to move the darn DHEAs wit 200-300mg/day)

....
=======================================================================
http://mus clechatroom.com/forum/showpost.php?p=74442&postcount=3

secretropin
Dadnatron writes:

I am using the recommended dosing for > 200 lbs.

I take 2 sprays sublingual in the AM... first thing when I get up.

4 sprays at night... just before I sleep.

I typically have about a 90% consistency, although I typically drink MUCH SOONER than 30 minutes have elapsed in the AM. I am good at night... given that I don't drink much while asleep.
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://mus clechatroom.com/forum/showpost.php?p=77041&postcount=19

CPAP

You have the wrong mask. This is REALLY common. A lot of people have to try many different masks before they find one that works for them.

There are probably around 30 different kinds and there is a top 10 list of the best sellers where 1 of which will end up working for almost anybody.

I tried at least 8 masks before i found one that is GREAT and works _perfect_ for me once I put electrical tape around the pivot on the tube to keep it from moving.

Go here:

http://www.cpap.com/simple-find-cpap-products.php?sortBy=popularity&shopbybrand=&selected=103

Click on "top seller" or "user reviews" to find the best masks that are most likely to work for you.

One way you can sample masks is that they have an option where you can pay something like $15 or $30 (dont remember off the top of my head) for the right to return it within 14 days.

You also may be able to talk to the company that gave you your cpap machine to see if they have the masks you want to try in stock and whether they will let you try them.

I bit the bullet when I got serious about treating my fatigue and just paid cash for all the masks until i found the best one.

Here is what i use:

http://www.cpap.com/productpage/resmed-mirage-swift-lt-nasal-plllow-system.html

Once i taped the connector at the top to keep it from swiveling, it's been a dream. But, different masks work for different people.

This is where you gotta start, you have to get the right mask, one that doesn't lose its seal, or move on your head, etc.

It's a common and minor problem and i promise you there is a 90% chance you'll find one that will work for you.

This is plan A. You gotta figure this out before you take the next step.
I use this:

http://www.cpap.com/cpap-machine/respironics-remstar-auto-cflex.html

the pressure drops up to 3 points on exhale, and it senses when you need higher pressure while inhaling and increases it for you automatically. It's got a smartcard that records various data about your sleep that you can review on your computer such as apnea events, waking up, pressure increases and drops, average pressure throughout the night, leaks, etc.

I would love to have the holy grail of cpaps, the bipap. Those are the crazy expensive ones. You can program completely different custom exhales and inhales. So you could have an exhale of 5 and an inhale of 20 if you wanted. I also wish the humidifier was twice its size, although i have a tip for this. Just buy two (they're $20), fill them both up before you go to bed, and keep one on standby. If you wake up and its been something like 7 hours or long enough to where it may run out of water soon, just swap the other one in and roll over.
This product no longer available and this information is for reference only. Newer Model Available: M Series Auto CPAP with A-Flex.

http://www.cpap.com/cpap-machine/remstar-auto-m-series-a-flex.html

M Series Auto CPAP with A-Flex
Aflex, Travel Friendly, Supports Software, Auto Adjusts
$649.00
This product ships for FREE! Shipping Policy
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
April 14/2010
Switched from Medrol 6mg + 75mcg
to
HC-30mg
1.5grain Armour + 50mcg-T3
T4-57
T3-13.5+50

30 days before any blood test change to 2grains Thyroid-s
using 1.5grains now is compromise so I can use up stores of Armour

Even all this is compromise
at first opportunity I will as doc for
100mcg-T4
50mcg-T3


Watch temperature and pulse, if both too low, add more Armour right away.

----------------------------- =========
 
JanSz

JanSz

Well-known member
Awards
1
  • Established
http://mus clechatroom.com/forum/showpost.php?p=99781&postcount=31

I've long since known that insulin sensitivity is important to health, but I don't think I've really grasped how important. Before I started desicated thyroid and adrenal support, over 5 years ago, I had an A1C of 4.9. Even with low testosterone/thyroid/adrenal function, I still had lots of energy and didn't get sick very often.

Since then, I've optimized my thyroid/testosterone/e2/adrenals, and even take GHRP-6. Unfortunately I don't have the same energy as I did before starting thryoid/adrenal support, AND I get sick every couple of months. Since then, I've had my A1C tested 3 times. It has progressed from 4.9, to 5.2, to 5.6, to 5.8. As you can see, it's not a great trend. I maintain a good diet, which is pretty much the same as it was 5 years ago (low-ish carbs, fruits, vegetables, 1 gram of protein per pound of bodyweight, and good fats like coconut oil, eggs, fish oil, butter, etc...). The only thing that has really changed is my thyroid/adrenal supplementation, and I don't exercise as much (I overtrain fairly easily nowadays. 5 years ago I was working out 6-7 days per week, and now it's 4-5. Any more than that and I get sick).

To summarize, since starting ANY hormonal therapy 5 years ago:

*Testosterone has gone from the 200's to the 700's. Currently taking 5 grams of Androgel plus 70 iu of hcg per day.
*E2 has gone from 35 to 20 without any AI
*Thyroid has been optimized with 4 grains of desicated thyroid daily
*Adrenals are supported on 6mg of medrol daily, split as 4mg upon waking, 2mg before bed
*DHEA and Preg supported with transdermal cream, 50mg of each daily.
*IGF-1 without GHRP-6 was about 220. I'm waiting to do Rhein's urine for GH.

With all of this improvement, the only thing that definitely hasn't improved is my insulin sensitivity. According to Chillin, this is THE most important thing to fix. If that's the case, I'm wondering if my issues of less energy, weaker in the gym, losing muscle, and weakened immune system are in fact due to my blood sugars.

For those who have successfully improved your insulin sensitivity, what benefits have you noticed? My guess is that I'll end up increasing my run workouts (studies have proven aerobic exercise reduces A1C moreso than lifting weights. It also seems to stimulate my immune system), and taking my daily carbs even lower.

This is alot of me just talking out loud, so if you have a comment, feel free.


Your poor glucose metabolism is most likely due to a reduction in cortisol metabolism, rather than poor carbohydrate management.

ie: glucose metabolism requires plenty of cortisol to process the energy. If you can't process the energy, then your cells can't use the excess glucose, and they become insulin resistant.

While I see you're supplementing with medrol, you and your medical professional adviser should have first performed a dosage response trial with pregnenolone, and if that went nowhere then you and your medical professional adviser should have considered a therapeutic trial of progesterone long before trialling medrol.

I recognise the need for a short term course of HC, but the adrenals rarely need a "permanent vacation" which is what medrol delivers. Medrol is great for those with genetically downregulated cortisol, but I doubt that we need it if our adrenals are only fatigued.

At this point you and your medical professional adviser should consider tapering off the medrol while replacing the cortisol supplement with regular HC. Then introduce pregnenolone, and gradually back off the HC while introducing pregnenolone.

###

If you'r totally glued to medrol, then discuss with your medical professional adviser to boost your dose until your energy levels are restored. BUT please also read the following detailed discussion re cortisol supplementation, with a heavy emphasis on medrol:

part 1: http://mus clechatroom.com/forum/showpost.php?p=86236&postcount=2

part 2: http://mus clechatroom.com/forum/showpost.php?p=86237&postcount=3

.


Thank you for that resonse, Chillin. You may be on to something, however I currently believe that 6mg of medrol is too much, combined with my dietary habits (I also believe candida to be involved here). I'm having strong immune reactions that I believe are tied to my consistantly elevated blood glucose levels. In the past, I've tried to lower medrol and/or switch a little of it to HC. I've usually gotten sick as a result. I think I need to work on my blood glucose first, and then try either lowering the medrol or switching a little to HC.

My goal is to have my A1C below 5.0% before I do so. This will first help strenghthen my immune system. To aid in this, I'll focus on the following supplements:

*Ribose - 10-15 grams per day

*Chromium - 600mcg to start for 1 month, then 400mcg/day after that. Research shows that steroids (of the cortisol variety) decrease chromium stores in the body, which negetively impacts blood glucose.

*Bitter Melon Tea - I already bought some of this at a local Asian grocery store. I get a noticeable drop in blood glucose from it.

*Metformin - Although not a supplement, I plan on using low doses (200-400mg/day).

Of course increasing exercise should always be first, I simply can't increase due to my poor immune system. I think this is a pretty potent stack, and don't plan on continuing all indefinitely if I get my A1C below 5.0.


Overview:

I doubt that the over-the-counter supps in your proposed list (metformin is not over-the-counter) will increase your insulin sensitivity sufficiently to make a noticeable health difference.

The reasons are not obvious among the supps fans because of a poor understanding of how insulin resistance comes about, and therefore how best to address it. I've explained some of that further below.

My experience and understanding is that the most reliable way to increase insulin sensitivity, via over-the-counter supps, is to regularly take low dosages of phyto-sourced free-radical suppressants, and to regularly take low dose SOD (superoxide dismutase) and glutathione boosters.

"Continually at low doses" would best describe the dosing strategy, not "take a big hit at the start of the day".

This is also described in more detail furhter below.

The meat-and-potatoes of insulin sensitivity management:

The management of insulin sensitivity is not via a hormone or immune signalling system which signals that glucose levels are adeqaute. We wish we had evolved (or been created with?) such a mechanism !

The management of insulin sensitivity is left to individual cells to "lock out" insulin, when glucose levels within cells gets too high. But even this mechanism is badly flawed, because even this mechanism is nowhere near sensitive enough to ensure insulin is locked out before glucose levels get too high. Ouch!

As a result the net effect of insulin resistance is that the setpoint for when it kicks in is set way too high for optimum health, so by the time insulin resistance has obviously kicked in, our cells are badly damaged by the free radicals generated because glucose metabolism is badly impaired.

The free radical damage creates a double whammy to kill the efficiency of glucose metabolism in our cells, because the free radicals not only damage the enzymes which trigger each step of the chemical reactions, but the damage causes a flood of repair materials into the damaged areas (inflammation) which physically obstructs normal processing.

So the fundamental and therefore most critical issue relating to insulin resistance, is to address the problems caused by the free radical damage within the insulin resistant cells.

We must first clean up the free radical damage (before trying the address the insulin sensitivity issue) so that the metabolism of glucose within those cells is as efficient as it can be, even with the restrictions imposed by insulin resistance.

As a result of the reduction in the free radical damage, the glucose metabolism efficiency does indeed increase, primarily because the inflammation decreases, and because all newly generated free radicals are "mopped up".

At the same time, because the cells glucose metabolism efficiency has improved, you will feel more energetic after eating less food, and you should translate that into eating fewer high GI carbs, and fewer fats and oils (minimize, don't go to zero).

Over time, the cells reduce their insulin resistance (an increase in insulin sensitivity).

##

Finally, you must independently manage the other aspects which feedbck strongly on glucose metabolism, such as optimum thyroid hormone T3 levels, and optimum cortisol levels.

What are phyto-sourced free radical suppressants ?

These are concentrated plant-sourced extracts of anthocyanins and other polyphenols.
The most reliable ones are:

LEF Enhanced Berry Complete
Masquelier's OPCs
Resveratrol (low dose)
green tea extract (low dose) (high polyphenols)


What are SOD and Glutathione boosters ?

LEF Endothelial Defense with GliSODin and CocoaGold
NAC, ie: N-acetyl-cysteine (to provide cystine, for glutathione synthesis)
Whey (to provide glutamate and glycine, for glutathione synthesis)
R-lipoic acid (to maintain glutathione in its non-oxidized state)

..
 

Similar threads


Top