For everyone that is wondering about fd2 and ArA the are a couple questions that need to be answered.
1: Is ArA muscle building effects truely from inflammation?
"In fact, one of the initial theories related to the potential ergogenic value of AA(ArA)for athletes was that dietary supplementation of AA(ArA) during training would promote greater localized inflammation thereby stimulating subsequent intramuscular
protein synthesis during resting/repair phases between workouts.
Nonetheless, present findings do not support this hypothesis. Rather, results suggest that AA(ArA) supplementation during training may reduce chronic levels of IL-6 thereby reducing associated inflammation that may occur during high volume training".
(This study could be very wrong but the main thing is there is controversy on what the true mechanism of which of its anabolic effects are. This is kind of a older study there could be new information that I'm not aware of. Also im sorry i don't have the link to the study, at the moment. I just can't find it right now, but I have read it before)
2: If its truely is from increased inflammation is 1 imparticular pathway of which its anabolic effects? Is a few of them? Or just of all them?(I really REALLY doubt it's the last one, as both your body and ArA have a plethora of pathways to increase inflammation. Furthermore some of the pathways of inflammation have shown to inhibition myotrophy, some of which ArA is involved in. Last but not least on this question is: to my understanding is we don't truely know all the mechanism of which both human body and ArA increase/decrease inflammation. Yes I said decrease as there is some pathways of which ArA reduces specific types of inflammation, as shown in the previous study.)
3: Are the pathways that ArA work infacted decrease by EC?(This is a tough one to answer as it requires us to 100% know what pathways ArA is anabolic. Is it actually inflammation that makes ArA work? If it is what imparticular type(s) of inflamtion is it? What pathway(s) increases that inflammation? Also if it is inflammation, does EC's anti-inflamatory pathway(s) overlap with that/those certian anabolic pathway(s) of ArA? Make this more difficult I'm assuming but pretty sure that not all of ECs anti-inflamatory are known. Plus there are other ingredients in fd2. Do they effects these pathways and if so in what way?)
4: Even if it is inflammation and EC's, or fd2's, pathways inhibt ArA's pathways, is it enough to fully prevent ArA from being anabolic at all?(Yet again this causes us to answer all other previous question assuming that all answers are yes, and then know the degrees of which of how each do so.)
Do you see were I'm going with this?
There is just WAY WAY WWWAAAAAYYYY to many IFs here. I can't answer pretty much any of these questions. Sure their might be some that might be able to answer these questions, but I doubt it. Even if there is a few who can, I doubt any of them are on this or any forum.
Also there are quite a bit of people whom have used ArA while taking anti-inflamatory medication(s) (asprin advil, aleve etc etc) or supplements trib(Cox inhibitor), cissus, etc, etc and have claimed that they made great gains.
The only way to know for sure is just to try it out IMO.
Note: I might be blowing steam out of my @$$ but I feel like I'm spot on, on this. I willing invite anyone to correct me. Also I if there is anything confusing or can't understand just let me know. I often make big errors that change the sentence and dont notice it, because to me it makes sense.