ZiR RED
Well-known member
ok, i had mistakenly thought you were completely against the concept of higher concentrations of oxygen use as a supplement
i tried accessing the first study i posted through my uni but they dont have it... it would be interesting to see how the subjects were using the 33-100% oxygen and in what duration during their heavy work
I downloaded the second review. The major crux of their hypothesis is that subjects must have exercise induced arterial hypoexmia (EIAH). I'm not sure the exact figures, but most people don't experience EIAH during intense exercise, especially not resistance exercise. The fainting or light headedness during resistance training is due to changes in blood pressure, specifically with altered posture (going from leg press to standing - vaso vagal syndrome) or the valslva manuevar (squats and dead lifts).
I find it interesting they implicate CO2 in the central fatigue hypothesis. CO2 and its conversion to H+ (Acid) does cause vasorelaxation and increased blood flow. Since CO2 production is not going to increase significantly in the brain, I can see how this may cause central fatigue in people who hyperventilate with the exercise.
If we are going to use this hypothesis in our discussion, we need to delimitate between simple exercise induced hyperpnia (increased rate and depth of breathing to supply O2 and remove CO2) and actual hyperventilation.
Br