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Solo Run - DAA ???

musclebound said:
anyone ever tried DAA MAX by Vital Labs or DAA by another company by itself?... is it worth a shot if your over 30?
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No. It has shown to boost testosterone in only 1 study the rest show it having no effect or even suppressing testosterone. Not to mention the raise in prolactin levels which isn't good.
 
ok , thanks for the heads up, fairly new to these supplements, i'm reading reviews on websites and then asking questions to get real time responses

rtmilburn said:
No. It has shown to boost testosterone in only 1 study the rest show it having no effect or even suppressing testosterone. Not to mention the raise in prolactin levels which isn't good.
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I will tag on here. I have noticed little to nothing good or bad when using DAA products. That's only my experience.
 
Yea dont waster ur money. Pick up a natural test booster or a natural anabolic.
 
Over 30 is vague if your closer to 40 maybe dhea will work but most test boosters are not enough for you too see a difference.
 
I suggest trying Follidrone or some other natural anabolic. Like mentioned above, I don't use natty test boosters unless during PCT.
 
rtmilburn said:
No. It has shown to boost testosterone in only 1 study the rest show it having no effect or even suppressing testosterone. Not to mention the raise in prolactin levels which isn't good.
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Incorrect there are two studies showing an increase
 
mw1 said:
We'll it's realistic...no test booster will increase levels 200 -500% as some claim
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I know. Which is funny that they say 200-500+% because those are mostly based on rats. Although none of them mention the 1000% increase that TRIB ATALUS had on rats.
 
snyderd1972 said:
What is trib atalus?
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It is a form of trib that's is supposed to contain more testosterone boosting saponins and in few studies done on rats it showed an increase of testosterone of 1000%. Unfortunately there is no human data on it. I think its just as worthless as trib terrestris tho.
 
Don't forget the DAA Boar and Lizard studies. IIRC (have to check to be 100%) it also increased aromatase to estrogen.

Added:

Recently an in vitro study carried out on boar testes revealed that endogenous testicular D-Asp enhances gonad aromatase activity, the key enzyme that converts testosterone into 17β-estradiol. In addition, studies done on the testes and ovary of the lizard Podarcis s. sicula have shown similar findings, confirming that D-Asp is involved in the local production of estrogen.

Lamanna C, Assisi L, Botte V, Di Fiore MM: Involvement of D-Asp in P450 aromatase activity and estrogen receptors in boar testis.

Amino acids 2007, 32:45-51.

Raucci F, D'Aniello S, Di Fiore MM: Endocrine role of D-aspartic acid in the testes of lizard Podarcis s. sicula.

J Endocrinol 2005, 187:347-59.

Assisi L, Botte V, D'Aniello A, Di Fiore MM: Enhancement of aromatase activity by D-aspartic acid in the ovary of the lizard Podarcis s. sicula.

Reproduction 2001, 121:803-808.
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Okay here is the thing about DAA that seems to be ignored.

First and foremost there is 1 study...yes 1 study that used a different form of DAA at a much higher dose that showed the decrease. The study gave 6g of regular DAA. The original study which had 28 participants used 3.12g of SODIUM DAA experienced a 40% increase. Furthermore it is to note that these where sedantary individuals(aka not producing optimal levels). A second study that uses SODIUM DAA shows that individuals who where at a deficit recieved a 60% increase in the levels, they used it for 90 days. So we got 2 studies using Sodium DAA that show an increase and 1 study that doubles the dose of regular DAA that shows a decrease or no change at 3g.

Can we see a theme here?

First off you need to use the right kind of DAA. Regular old DAA or NMDA doesnt cut it, you need to use NA-DAA. Second off people who are at a deficit and are not producing test at optimum levels are the ones who recieve the most benefit from this product(with a 12 day and a 90 day study to verify this using the correct form of DAA). So the appropriate response is not to just blindly bash this ingredient, but to realize it's merit as a post cycle aid to recovery when using the proper amount and the proper type.

Lastly, keep your estrogen in check and your prolactin in check to maximize the use of Sodium DAA. This is an ingredient that works in the pituatary, so the fact you get an increase in prolactin is a by product of it working to increase signaling. A little but of mucuna or b6 and you avoid this all together.

And if you still want to use the one flawed study that used health, athletic individuals not at a deficit...you have to realize that 3g showed no significant change and doubling the dose is where there is a problem. So if you want to be EXTRA nit-picky....dont double the dose. Stay between 2.66 and 3.12g and you will optimize your bodies ability to product test if you are indeed at a deficit. Ideal for when you are coming off a suppressive cycle.

Personally, I see DAA as an awesome PCT aide with good research(right type and right dose) to support it being used during this time.
 
GoHardOrGoHme said:
Okay here is the thing about DAA that seems to be ignored.

First and foremost there is 1 study...yes 1 study that used a different form of DAA at a much higher dose that showed the decrease. The study gave 6g of regular DAA. The original study which had 28 participants used 3.12g of SODIUM DAA experienced a 40% increase. Furthermore it is to note that these where sedantary individuals(aka not producing optimal levels). A second study that uses SODIUM DAA shows that individuals who where at a deficit recieved a 60% increase in the levels, they used it for 90 days. So we got 2 studies using Sodium DAA that show an increase and 1 study that doubles the dose of regular DAA that shows a decrease or no change at 3g.

Can we see a theme here?

First off you need to use the right kind of DAA. Regular old DAA or NMDA doesnt cut it, you need to use NA-DAA. Second off people who are at a deficit and are not producing test at optimum levels are the ones who recieve the most benefit from this product(with a 12 day and a 90 day study to verify this using the correct form of DAA). So the appropriate response is not to just blindly bash this ingredient, but to realize it's merit as a post cycle aid to recovery when using the proper amount and the proper type.

Lastly, keep your estrogen in check and your prolactin in check to maximize the use of Sodium DAA. This is an ingredient that works in the pituatary, so the fact you get an increase in prolactin is a by product of it working to increase signaling. A little but of mucuna or b6 and you avoid this all together.

And if you still want to use the one flawed study that used health, athletic individuals not at a deficit...you have to realize that 3g showed no significant change and doubling the dose is where there is a problem. So if you want to be EXTRA nit-picky....dont double the dose. Stay between 2.66 and 3.12g and you will optimize your bodies ability to product test if you are indeed at a deficit. Ideal for when you are coming off a suppressive cycle.

Personally, I see DAA as an awesome PCT aide with good research(right type and right dose) to support it being used during this time.
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I could be wrong but i dont think there is any human studies on NMDA.
 
Yes there is.

One study used NMDA in 20 heavy resistance training men and showed no change in test levels or overall muscle mass or strength. So like I said NMDA doesnt cut it(along with plain DAA which is found in many DAA supplements)
 
GoHardOrGoHme said:
Yes there is.

One study used NMDA in 20 heavy resistance training men and showed no change in test levels or overall muscle mass or strength. So like I said NMDA doesnt cut it(along with plain DAA which is found in many DAA supplements)
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Still perplexes me why anyone would want to put NMDA in a supplement. It can be neurotoxic.
 
AlphaMax is the top t-booster on the maps rn. The inclusion of arimistane and forskolin helps greatly to tighten you up. Add in some -(-)Epi and you're gtg m8 :D
 
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