QUESTION? if EPHEDRA was unbanned tommorrow... how fast would AX have a ECA based Fatburner on the marker?
Agreed, SX wipes the floor with ECA/Clen.
Especially in terms of risk Vs reward.
bump...SX-Slim Xteme?
i read somewhere that it should be like 1 mg per 10 lbs. so 22/23 mg for u daily?start off with 2 or 3 caps and work ur way up id say. im up to 3-4 4x a day at 2.5 mg each but i think im kind of desensitized to it.I have a dumb question. Ive been trying to figure out my dosage for Y. I weigh 220-225lbs and one cap is 2.5mg of Y....so what should my daily total be to take?
I read that also, I did start out with just E&C and just started adding in Y...I can tell somewhat of a differencei read somewhere that it should be like 1 mg per 10 lbs. so 22/23 mg for u daily?start off with 2 or 3 caps and work ur way up id say. im up to 3-4 4x a day at 2.5 mg each but i think im kind of desensitized to it.
ive also read that u should start with just ec for a week or so and then add in the y after to get better results. i started off with ec and y and the effects diminished a bit.
QUESTION? if EPHEDRA was unbanned tommorrow... how fast would AX have a ECA based Fatburner on the marker?
i read somewhere that it should be like 1 mg per 10 lbs. so 22/23 mg for u daily?start off with 2 or 3 caps and work ur way up id say. im up to 3-4 4x a day at 2.5 mg each but i think im kind of desensitized to it.
ive also read that u should start with just ec for a week or so and then add in the y after to get better results. i started off with ec and y and the effects diminished a bit.
I need your advice on a thought I have...can I send you a PM?It's really tough to dose a stimulant because we have to incorporate available adrenergic receptors as well as body size. There's a wide range of body sizes, literally ranging from midgets (or little people, to be more PC) to giants, and everyone in between. Unfortunately, receptor availability/density has a similarly wide range.
One guy can take 200mcg of clen and be fine, another guy will take it and wind up in the hospital.
I got mine at a pharmacy, Bronkaid....but it also has 400mg of GuaifenesinSorry if this is a stupid question but where the heck do you get E from?
Also, haven't we learned that any kind of anti-inflammatory like aspirin impairs muscle growth? Or do people doing ECA not care about that so long as they are cutting?
So the only way to get ephedrine is in cold medicine?I got mine at a pharmacy, Bronkaid....but it also has 400mg of Guaifenesin
thats why Im not using Aspirin in mine, instead doing Yohimbine
research it man, every component has a purpose...as stated above, you figure out your own dosesI got a question regarding the EC stack. Do you necessarily need the caffeine or is it just to help aid in fatloss? Also, has anyone ran EC at half doses instead of the full doses.
I guess so, the G helps with respiratory issues, lately when I get short of breath, I can tell its from the G, but it feels really good, its scary but in a good waySo the only way to get ephedrine is in cold medicine?
Is there a safety issue or possible side effects from regularly taking cold medicine? If cold medicine is the only option it's unappealing to me.
Very broad and sweeping statement.No fat burner is more effective than the ECA stack...or if u can stand it the ECY stack...so why waste money on fat burners that dont stack up...
i dont get it? none of them can touch the ECA..
if they could...they would be banned for fat loss just like Ephedra is
Green Tea Extract (ECGC) will provide similar benefits as it is a COMT inhibitor with the added benefit of being a potent anti-oxidant.
Take Green Tea Extract (ECGC) and you don't need the caffeine or the aspirin.
Invalid Link Removed
guys add aspirin every other day,it was used in the original eca for a reason and is has a patent on it.
There is scientific testing done on it,It keeps the body from stopping the norephrine down regulation.
add yohimbe to block the bad receptors if you can handle the stim.Here is a paste of an old write up.
Ephedrine enhances the release of the hormone norepinephrine in the body, and also binds to the same receptors as that hormone, causing excess calories to be converted to heat instead of being stored as fat. It also raises blood pressure. Epinephrine and norepinephrine are also known as adrenaline and noradrenaline. Ephedrine thus acts to stimulate an "adrenaline rush".
Interactions with caffeine and aspirin can increase the effects of ephedrine. Norepinephrine works in part by increasing the levels of cyclic AMP in cells. Caffeine inhibits the enzyme that breaks down cyclic AMP. Together, ephedrine makes more cyclic AMP, and caffeine prevents it from breaking down. Aspirin inhibits the receptors that turn off release of norepinephrine. So ephedrine releases norepinephrine, and aspirin prevents the release from being turned off.
Ephedrine by itself has been shown to be ineffective as a weight loss treatment. Ephedrine combined with either caffeine or aspirin was effective. The effect appears to be caused by a reduction in appetite, and preventing the metabolic rate decrease usually associated with a reduced calorie diet.
so your saying Aspirin enhances the effects? therefore it is required? what if you did E/C/A/YGreen Tea Extract (ECGC) will provide similar benefits as it is a COMT inhibitor with the added benefit of being a potent anti-oxidant.
Take Green Tea Extract (ECGC) and you don't need the caffeine or the aspirin.
Invalid Link Removed
"I" am saying don't waste your time with aspirin.
If you must use E and Y together I would use E/Y/G. You don't need C as Green Tea Extract has C.
GTE is a COMT inhibitor. This means it inhibits the downregulation of the stimulants. So it inversely increases the duration of their effects.
damn, I just got some Bayor baby Aspirin"I" am saying don't waste your time with aspirin.
Ill start using my GTE today, how should it be dosed?If you must use E and Y together I would use E/Y/G. You don't need C as Green Tea Extract has C.
GTE is a COMT inhibitor. This means it inhibits the downregulation of the stimulants. So it inversely increases the duration of their effects.
this what I hope on doingInteresting. So one could actually goes as far as just an E/G and throw out the Y and still be effective?
ok, Ive been dosing everything accordingly...Im not dumb lol. Im going to do one week of C, then do the GTE on my taper down 3/2/1...I think that'll be a good combination. So then it will be E/Y/A/GTEI'm not saying throw out the Y. E targets beta receptors and Y targets alpha receptors. If you have adipose that is either, or, then use either, or, accordingly.
I use one gram in the morning and one gram post workout.Ill start using my GTE today, how should it be dosed?
Is that legal?I wish NP would carry 12.5mg ephedrine caps.
Adams
Is that legal?
Is there anywhere you can buy that now? I thought the only way to legally get ephedrine is in cold medicine?
post WO...never thought of that, why post?I wish NP would carry 12.5mg ephedrine caps.
Adams
post WO...never thought of that, why post?
no, lol, this is weird...i meant to reply to LAGears post...??? Did I say Post WO somewhere?
Adams
post WO...never thought of that, why post?
"I" am saying don't waste your time with aspirin.
I use one gram in the morning and one gram post workout.
The more important thing is how much EGCG you are getting per dose of GTE. Aim for about 400mg EGCG per dose. Take twice daily.
NP GTE powder is 50% ECGC so I'm getting 500mg twice a day.
I just have the Vitmain Shoppe brand of GTE, never really considered GTE an important supp, until nowThe more important thing is how much EGCG you are getting per dose of GTE. Aim for about 400mg EGCG per dose. Take twice daily.
so ECA is better than ECY....Why would you sugest this,I have tried eca many times and with aspirin it worked better every time.
There are many studies done at countless universities that used aspirin in there testing.
it blocks Prostaglandin in the cycle for thermogenesis.
here is a picture from 1993 that was used in a study.ECA has been tested for up to 24 weeks and it showed that it kept on increasing ne while increasing the camp cycle.Where not taking about receptor downregulation,with eca you can get used to the stim affect after a week but there thermogenisis lasts for over 6 months.The aspirin plays a huge role in that.
Invalid Link Removed
An additional claim made by proponents of ECA-induced weight loss is that the thermogenic effects are
limited to fat catabolism, and that there is no protein catabolism, increased heart rate, or tremors which are
associated with other sympathetic stimulation. One hypothesis is that the main stimulation by ephedrine is
through beta2- and beta3-adrenergic receptor subtypes (2, 7), both of which are predominantly responsible
for lipolysis and protein synthesis, but are not associated with cardiovascular and central nervous system
effects mediated by beta1-receptor (2). Tolerance rapidly develops to the effects of ephedrine on heart rate,
but does not develop to the thermogenic effects (2, 8-12), suggesting that different mechanisms are
responsible for these different effects, and that ephedrine has longer-acting effects on thermogenesis.
The third component of the ECA combination, aspirin, enhances the peripheral actions of ephedrine and
caffeine by inhibiting prostaglandin (PG) synthesis. PGs, like adenosine, have been implicated in inhibiting
NE release from the post-synaptic nerve terminal, and in inhibiting the lipolytic actions of sympathetic
stimulants (2). However, these effects have been limited to only one study, and more experiments must be
performed before this effect can be conclusively linked to aspirin.
In summary, the effects of ephedrine, caffeine, and aspirin in rat brown adipose tissue involve a significant
central component of increased NE release, and a contributing peripheral part that has direct action on the
target tissue. These peripheral actions act to directly increase stimulation of beta-adrenergic receptors
(especially the beta2- and beta3-subtypes), to inhibit the regulatory mechanisms of negative feedback by
adenosine and PGs extracellularly, and to inhibit the destruction of cAMP by PDE intracellularly. This
overall scheme is depicted in Figure 1.
Efficacy
I understand, I want to know if E/C/A/Y is safe? if it is, I will dose all, or EOD..You keep mising the point...E stimulates beta receptors, Y stimulates alpha receptors. Different receptors different adipose.
Invalid Link Removed