Aha, all right, I understood your question wrong, sorry.Thanks.
I understand the general theory, im kind of eluding to what is it regarding lgd that impacts lipids more than say osta etc. my lipids never moved on osta fyi.
Unfortunately, the full mechanics are not discovered yet of those SARM. But your question is a very legitimate one, especially in a specific case like this.
Im case of AAS, such based on DHT are more aggressive on the lipids than non-DHT. I have no clue, how to classify the SARM, everyone is different and has nearly nothing common with the other ones (with exceptions, of course), while AAS are all based on cholesterole with litte modifications and clear - and known - impacts (e.g. DHT).
Maybe someone knows more about that. Normally, we should comparise the effect of LGD-4033 with one of more or less the same class like LGD-3303/-2226/etc. Then we could evaluate the effect on the lipids in comparison to another class like MK-2866 ect.
But honestly, I don't know why LGD has a stronger negative effect in comparison to Ostarine.