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Methylepithio the truth!

if it converts to phera at all, it's insignificant amounts. I respond to phera and make gains on it... I didn't (COULDN'T) make any gains on Epi.
 
I don't feel anything on EPI even at high doses for a 6 week run. I feel Phera at a much lower dose with great strength gains.
 
It has been proven that at least part of Epi activity is due to the Dmt conversion.








How much the unconverted epi contributes who knows ?










Quote Originally Posted by Patrick Arnold View Post
methylepistanol willl spontaneiously break down into dmt. that is why you they see a little dmt in the products. it has nothing to do with spiking. and in fact, its probably likely that dmt is the active compound once you ingest methylepistanol anyway

Quote Originally Posted by Patrick Arnold View Post
dmt is used to make methylepithiostanol.
and heat and low ph will catalyze the loss of a sulfur atom, which regenerates dmt
its pretty impossible to make me methylepi and not have at least a small part of it degenerate back into dmt upon storage i would think. cuz even at norml conditins you will still have the reaction proceeding. thats why you smell sulfur when you open the bottle


quote originally posted by patrick arnold
epi may conceivably slowly decompose to dmt, although its probably so slow its not of significance

Its also possible that epi is not even the active drug in the body but merely a pro-drug. that is, it converts to dmt in the body and that is what is active. but i said that is only possible, i dunno

if someone can find metabolite studies for epitiostanol then that might give some clues.
 

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I respect henryv and just99, they're probably the only people i ever ask for nomenclature advice. I also ask pa questions every so often. im not too fond of him from my days on bb.com

anyhow, I am suprised that it isn't known epi has some breakdown/conversion into dmt.

see what a little research can do?

so have you found out why you got gyno from epi. cause it doesn't have anything to do with it's insignificant conversion into dmt.

both steroids are non aromatizing androgens. :D

I'll leave this question for you to find....... :ntome:

on another note, i also didn't get anything from my bottle of cel estane. but im gonna give epi another shot someday. probably epi-strong.
 
my next time will be IBE epistane (white caps, white lid).

IBE epistane has been tested positive to tren in past. it was black lid, black caps.

I can pm the link to anybody who need it.

As i said to RPN the good companies are those that provide lab test for each batches.
 
coa's and lab test arent worth a shhit.

if you really believe in them, i'll sell you a 99% pure epistane, with a lab test to go with it. just shoot me a pm.

you may not get any results from it, but hey, i got that lab test showing it's 99% pure.
 
masterX said:
my next time will be IBE epistane (white caps, white lid).

IBE epistane has been tested positive to tren in past. it was black lid, black caps.

I can pm the link to anybody who need it.

As i said to RPN the good companies are those that provide lab test for each batches.
Click to expand...

wouldnt lot numbers be more accurate, instead of lids?

I would still go with IBE or Epi-Strong.
 
Mehtylepithiostanol gyno is quite simple to understand. Think this way: Epi isn't as androgenic as testosterone, plus the use of exogenous AAS will reduce the amount of testosterone you have, wich leads us to the second part of the problem: lower DHT, which is our natural estrogen antagonist, keeping it under control. Once you are on cycle and the hormone you're using isn't androgenic enough (like epi, wich scores in the anabolic:androgenic raio a prety 1100:91 and our famous methyl-testosterone has something between 115-150:94-130, making epistane extremely anabolic but low androgenic potential compared to testosterone - wich per si is 3 times less androgenic than the DHT that we need to keep our estrogen under control), it might cause relatively elevated estrogen levels, since you don't have enough DHT to counter its rising. This way you'll get estrogen related gyno. But you may say "Isn't methylepitiostanol, a methylated version of the japanese anti-aromatase epithiostanol and a 5a-reduced hormone?" to wich I answer, "Yes, you're right. But so does Superdrol and Masteron difer in the fact that the first is 17a methylated and the second isn't, and look at the diferences! Sdrol is used mostly to bulk up and Masteron is a "pre-contest drug" used to leaning out, and they're both 5a-reduced steroids, having the first reports of gyno precisely due to an induced lower production of DHT while someone is on it."
So Epistane might cause gyno, is just a matter of being predisposed to it.
 
Broly said:
Mehtylepithiostanol gyno is quite simple to understand. Think this way: Epi isn't as androgenic as testosterone, plus the use of exogenous AAS will reduce the amount of testosterone you have, wich leads us to the second part of the problem: lower DHT, which is our natural estrogen antagonist, keeping it under control. Once you are on cycle and the hormone you're using isn't androgenic enough (like epi, wich scores in the anabolic:androgenic raio a prety 1100:91 and our famous methyl-testosterone has something between 115-150:94-130, making epistane extremely anabolic but low androgenic potential compared to testosterone - wich per si is 3 times less androgenic than the DHT that we need to keep our estrogen under control), it might cause relatively elevated estrogen levels, since you don't have enough DHT to counter its rising. This way you'll get estrogen related gyno. But you may say "Isn't methylepitiostanol, a methylated version of the japanese anti-aromatase epithiostanol and a 5a-reduced hormone?" to wich I answer, "Yes, you're right. But so does Superdrol and Masteron difer in the fact that the first is 17a methylated and the second isn't, and look at the diferences! Sdrol is used mostly to bulk up and Masteron is a "pre-contest drug" used to leaning out, and they're both 5a-reduced steroids, having the first reports of gyno precisely due to an induced lower production of DHT while someone is on it."
So Epistane might cause gyno, is just a matter of being predisposed to it.
Click to expand...

Hmm that sounds like a good explanation. But it is very weird how some people's lumps shrivel up and others get gyno for the first time. But gyno seems a most complicated mechanism relying on a variety or hormones and each individual's body chemistry varies so much, a lot is possible.
 
UnrealMachine said:
Hmm that sounds like a good explanation. But it is very weird how some people's lumps shrivel up and others get gyno for the first time. But gyno seems a most complicated mechanism relying on a variety or hormones and each individual's body chemistry varies so much, a lot is possible.
Click to expand...

Thanks, but you're right. Gyno can come from the most diferent orgins and individual's body chemical reactions to certain hormonal changes. If you mess with progestins, you may get gyno from high progesterone; if you play with testosterone, you may also get gyno from excessive estrogen; if you play with anti-estrogens you can get gyno from estrogen rebound; play with pure DHT, you may also get gyno due to supressed endogenous DHT and consequently reduced hability to control natural estrogen levels. All in all, I honestly think that it depends from personal genetics, hormone used and how it is used.
 
masterone is methylated in the 2 alpha position.
2a-methyl-5a-androstan-17b-ol,3-one propionate


methyldrostanolone is methylated inthe 2 alpha position, and 17 alpha position. making it a di methyl.
2a,17a di-methyl-5a-androstan-17b-ol, 3-one

despite what it says on paper, epistane has shown strong androgenic side effects.

the most logical reasoning behind gyno caused from epistane, most likely from its ability to displace shbg, freeing up not only testosterone (which there will be little of) but also estrogen to cause it's unwanted side effects.

another cause for epistane reducing gyno in some is most likely d/t epi shifting the androgen/estrogen balance in favor of androgens.

as has been noted, there are many reasons gyno can be caused, but it all starts with totally screwing up your hormonal balance by taking steroids.
 
jbryand101b said:
as has been noted, there are many reasons gyno can be caused, but it all starts with totally screwing up your hormonal balance by taking steroids.
Click to expand...

Not only;) Think about all the guys that got gyno during their teens, and that's not due to taking steroid, that's from endogenous steroid production that have changed naturally:) So that's not the main reason, but I see your point;)

Plus, remind me of some pure DHT related sides (DHT not general androgen sides) that epi has shown to cause, because I don't see any specificly due to DHT's related androgenic effects. Sore joints, enlarged prostate or hair thining, might all be methylepithiostanol related issues and but that's not proof that it's as androgenic as DHT to cause them neither that has the needed DHT related androgenic effects in the body to suport an healthy function of it.

About the detailed info about the proper name to call the state of methylation of Superdrol, thanks for it I was being inaccurate in my explaination, but you get my point;)
 
UnrealMachine said:
Hmm that sounds like a good explanation. But it is very weird how some people's lumps shrivel up and others get gyno for the first time. But gyno seems a most complicated mechanism relying on a variety or hormones and each individual's body chemistry varies so much, a lot is possible.
Click to expand...

Genetics, predisposition and response from Compound X.

This subject still has me in awe, how some can respond so well or so poorly for that matter, to the same exact compound and dosages.

That goes from Test E to superdrol to epi.

I have a buddy that just responds awesome to test. Amazes me.
 
Im not following your reasoning behind the differentiation between androgenic side effects and dht.

dht is an anabolic/androgenic steroid, as well as epistane. the numbers of their androgenic ratings are taken from test done on rats.

an androgenic side effect is just that, an androgenic side effect. a compounds androgenic rating isn't going to make a difference.

there are a bunch of steroids, that on paper, look amazing, but fail to perform, or perform dfferently in humans.

dht side effects are androgenic side effect, and anabolic side effects.

the severity of the side effects is compound specific, all steroids known to man right now, have anabolic/androgenic side effects.

epistane has shown the ability to have an increased number of androgenic side effects, such as sore joints, hair thinning, enlarged prostate, enhanced body hair growth, acne, and the ability to displace estrogen, in low dosages.



all steroids are capable of these side effects, and will vary person to person.

in my opinion of epistane, it has shown to be a highly androgenic and moderately anabolic steroid.

epistane and dht, though two different steroids, aren't very different in how they act in people, to me atleast.
I think oral turinabol, or anavar are better representations of lowered androgenic steroids, that are highly anabolic.
 
Sorry, I haven't made myself clear (here I notice some lack of english vocabulary, my fault:D)

What I want to say is that there are some benifits that DHT, despite being an highly androgenic hormone, it gives us some benifits that other androgenic steroids (like epistane) don't give to us, but still they are androgenic enough to give us androgenic related sides. For instance, DHT gives us some androgenic benefits like increased strength, agression and can keep our estrogen levels controled enough to make us look leaner without compromising cholesterol levels due to extremely lowered estrogen levels and helps us keeping our woody friend up. But there are some relatively high androgenic steroids, that gives us much more issues than advantages, such as sore joints, thined hair and all the sides you've mentioned. So there's a problem specific to those androgenic hormones, that despite being more on the androgenic sides, they tend to give us more sides then benifits (while I'm writing this Mestanolone vs Epi come to my mind).

Trying to sum up what I meant to say is that among the androgenic steroids there are some that gives us more benefits without so many sides, while relying in their androgenic hability, as there are some highly anabolic substances that give the same final results as others but with much more sides:)
 
rpn havoc/primaforce havoc is rediculous for me. I get pumped as crap and gain alot of solid/dry mass each time.

I have taken PPlex, it bloated me and gave me INTENSE anxiety/panic attacks that lasted for weeks.

Totally totally different from havoc experiences.
 
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