By the post, I'm not sure what you had checked. Was it only SHBG, and Estro?
Is this mid cycle? Post Cycle? You don't specify.
Mid cycle. First test cycle. 600mg a weekIs this mid cycle? Post Cycle? You don't specify.
E2 is at the high end of normal for this tests range.
Low SHBG is good. (Free Test) as noted already.
Your Total T is probably higher than the range permitted to test. (Beyond 2100)?
So, this is mid cycle? I'm guessing yes.
Letrone by BLR would be good to keep that E2 in check.
(Last day of our March 30% off sale! MADMARCH)
Not knowing what SHBG is; is kind of telling here. You should
do some research on all the major components of blood tests
for your own safety. Do you have a PCT lined up?
Oh, my bad. I saw your other post and didn't connect them.Mid cycle. First test cycle. 600mg a week
SourceSupplemental L-Carnitine L-Tartrate at 2g daily has been shown in vivo to increase the density of Androgen Receptors in muscle cells over 21 days. Although this mechanism would not increase testosterone levels per se, it may increase the effects of testosterone as they are vicarious through its receptors.
Med Sci Sports Exerc. 2006 Jul;38(7):1288-96.
Androgenic responses to resistance exercise: effects of feeding and L-carnitine.
Kraemer WJ1, Spiering BA, Volek JS, Ratamess NA, Sharman MJ, Rubin MR, French DN, Silvestre R, Hatfield DL, Van Heest JL, Vingren JL, Judelson DA, Deschenes MR, Maresh CM.
1Human Performance Laboratory, Department of Kinesiology, University of Connecticut, Storrs, CT 06269-1110, USA. [email protected]
- Med Sci Sports Exerc. 2006 Oct;38(10):1861.
The purpose of this investigation was to determine the effects of 3 wk of L-carnitine L-tartrate (LCLT) supplementation and post-resistance-exercise (RE) feeding on hormonal and androgen receptor (AR) responses.
Ten resistance-trained men (mean+/-SD: age, 22+/-1 yr; mass, 86.3+/-15.3 kg; height, 181+/-11 cm) supplemented with LCLT (equivalent to 2 g of L-carnitine per day) or placebo (PL) for 21 d, provided muscle biopsies for AR determinations, then performed two RE protocols: one followed by water intake, and one followed by feeding (8 kcal.kg body mass, consisting of 56% carbohydrate, 16% protein, and 28% fat). RE protocols were randomized and included serial blood draws and a 1-h post-RE biopsy. After a 7-d washout period, subjects crossed over, and all experimental procedures were repeated.
LCLT supplementation upregulated (P<0.05) preexercise AR content compared with PL (12.9+/-5.9 vs 11.2+/-4.0 au, respectively). RE increased (P<0.05) AR content compared with pre-RE values in the PL trial only. Post-RE feeding significantly increased AR content compared with baseline and water trials for both LCLT and PL. Serum total testosterone concentrations were suppressed (P<0.05) during feeding trials with respect to corresponding water and pre-RE values. Luteinizing hormone demonstrated subtle, yet significant changes in response to feeding and LCLT.
In summary, these data demonstrated that: 1) feeding after RE increased AR content, which may result in increased testosterone uptake, and thus enhanced luteinizing hormone secretion via feedback mechanisms; and 2) LCLT supplementation upregulated AR content, which may promote recovery from RE.
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