I've reviewed dozens and dozens of studies on this subject and it always seems to come back to a ratio of approximately 2 to 1 carbs to protein (up to possibly 4:1 ratio of carbs to protein). This seems to be the optimal combination to maximize muscle repair to boost that metabolism for long term fat loss.
Are you sure that it's ok? We pay the isolate much, because it has less fat and carbs. So if a add 60gr of maltodextrin and 30gr of isolate, i'll have a 33% protein powder!
Yes i take bcaa's pre and intra!Do you take BCAAs prior to working out fasted?
Protein alone has been proven to spike post workout.
First of all, it has been proven time and time again that if carbohydrates are insufficient, performance suffers. Thus it would be sub optimal to recommend such a high protein intake that inadequate carbohydrates were consumed leading to poor workouts and a lessened training stimulus for growth. This would defeat the whole point of eating more protein in the first place! In fact, if carbohydrates are too low, it does not matter how much protein you eat, you can lose muscle tissue!
Carbohydrates are equally important (if not more so) for muscle growth, muscle maintenance and performance when compared to protein. So although yes, many athletes should consume more protein than they do, they should not consume it at the expense of dietary carbohydrate.
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This article just came up today on simplyshredded.com
Yo, ive seen this posted b4 and do NOT agree. anyone got an article on this theory? I thought that b/c protein itself never enters the Krebs cycle it does not have an Insulin SPIKING responce. After protein is broken down to Amino acids it will respond with an Increase in insulin. Spikes happen thru carbs. Anything you intake your pancreas will secrete insulin to account for the rasie in blood sugar level, but spikes only happen thru carbs.
That is overall CHO intake, not CHO timing. We all know that carbs are necessary for muscle growth, this isn't new.
Carbohydrates
High intensity exercise places great demand on glycogen stores. Glycogen is the sugar stored in the liver and muscles. Because high intensity exercise burns energy at such a high rate, the body is unable to supply sufficient oxygen to be able to use fat for fuel. Instead, it must use sugar both stored in the muscle and brought in from the blood.
Consuming simple sugars right before training can reduce the amount of glycogen used during exercise. This can prolong performance. More importantly, higher blood sugar and insulin levels appear to create a hormonal milieu favorable to anabolism (growth).
During exercise, cortisol accelerates lipolysis, ketogenesis, and proteolysis (protein breakdown). This happens in order to provide additional fuel substrates for continued exercise. The effects of cortisol may also be necessary to provide an amino acid pool from which the muscle can rebuild new contractile proteins if there are insufficient amino acids delivered from the blood. This ensures that some degree of adaptation can occur regardless of the availability of dietary protein. Over time however, if this process is not balanced with additional dietary protein, the net effect will be only maintenance or even a decrease in functional muscle tissue, as is evident during periods of starvation or prolonged dieting. Fortunately, there is only a non-significant rise in cortisol levels when carbohydrates were consumed during exercise. (Tarpenning, 1998) The net effect is a more rapid increase in the cross sectional area of the muscle fibers with the greatest effect seen in type-II fibers.
This may be a less expensive option for those who were thinking of using phosphatidylserine. In this case, carbohydrate administration appears to down regulate the hypothalamic-pituitary-adrenal axis, probably through insulin or perhaps through the presence of carbohydrate itself. This would, in effect, greatly reduce the body’s catabolic response to exercise stress. All good news for bodybuilders.
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most nutrients create an insulin response.
2 Eat Plenty Of Carbs For Energy
Your body can use protein, carbs or fats for energy, but when you’re training hard, it preferentially uses carbs. Taking in ample carbs makes it easy for your body to fuel itself, sparing protein and fats for their more specific nutritional uses.
3 Emphasize Slow-Digesting Carbs
Complex carbs are constructed of long chains of sugars. Most, but not all complex carbs are slow digesting. Stress whole-grain products (whole-wheat bread and pastas, oatmeal, brown rice, etc.) and sweet potatoes, which are all slow digesting, as opposed to white breads and white potatoes, as well as sugars, which are all fast digesting. Slow-digesting carbs provide longer-lasting energy and there’s less chance they’ll be stored as bodyfat. During a mass-building phase, strive to take in a minimum of 2 g and up to about 3 g of carbohydrates per pound of bodyweight per day. During cutting phases, reduce total carbohydrate consumption to 1 g per pound of bodyweight.
4 De-Emphasize Simple Carbs
Except after workouts, a bodybuilder should minimize calorie consumption from simple carbohydrates (sugar). Simple carbs can be rapidly absorbed, especially in liquids with little or no fats or solid foods to slow down their passage through the gastrointestinal tract. Significant quantities of simple carbs provide the release of insulin, desirable after a workout, but not at other times, because it can encourage the body to store these sugars as bodyfat. Foods that are high in sugar are among the worst for bodybuilders seeking to keep bodyfat low.
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The hypoglycemic effect of fat and protein is not attenuated by insulin resistance.
Abstract
BACKGROUND: The glucose-lowering effect of fat and protein is attenuated or absent in diabetic patients, which suggests that the same may occur in insulin-resistant subjects without diabetes.
OBJECTIVE: The objective was to determine whether the postprandial metabolic responses elicited by fat and protein were influenced by the insulin sensitivity of the subjects and whether fat and protein modulate glucose responses through different mechanisms.
DESIGN: Healthy nondiabetic subjects aged 18-45 y took 50 g oral glucose with 0-30-g doses of canola oil and whey protein on 11 separate mornings after fasting overnight. The subjects were classified into 3 fasting serum insulin (FSI) groups: FSI < 40 pmol/L (n = 9), 40 < or = FSI < 70 pmol/L (n = 8), and FSI > or = 70 pmol/L (n = 8). The relative glycemic response was expressed as the incremental area under the curve (AUC) after each test meal divided by the mean AUC of the glucose control in each subject.
RESULTS: Protein significantly decreased glucose (P < 0.0001) and hepatic insulin extraction (P <0.0001) and increased insulin (P < 0.0001) and glucagon-like peptide 1 (P = 0.004); however, protein had no significant effect on C-peptide (P = 0.69) or on the insulin secretion rate (P = 0.13). No significant FSI x fat (P = 0.19) or FSI x protein (P = 0.08) interaction effects on glucose AUC were observed. In addition, the changes in relative glycemic response per gram of fat (r = -0.05, P = 0.82) or protein (r = -0.08, P = 0.70) were not related to FSI.
CONCLUSIONS: The hypoglycemic effect of fat and protein was not blunted by insulin resistance. Protein increased insulin but had no effect on C-peptide or the insulin secretion rate, which suggests decreased hepatic insulin extraction or increased C-peptide clearance.
PMID:19923374
Carb intake is truly overrated in the BB community IMO.
If you dnt replenish the glycogen stores after a w.o you risk going catabolic, no matter how much protein you intake.
Remember that the body breaks down fat and protein to restore glycogen levels IF needed as well. It's called gluconeogenesis.
gluconeogenesis:
the synthesis of glucose from noncarbohydrate sources, such as amino acids, propionate and glycerol. It occurs primarily in the liver and kidneys whenever the supply of carbohydrates is insufficient to meet the body's metabolic demands or in the rumen by the action of bacteria in well-fed ruminants. Gluconeogenesis is stimulated by cortisol and other glucocorticoids and by glucagon. Formerly called glyconeogenesis.