Dr. D PCT question
- Thread starter dertynasty
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JonesersRX7
Registered User
It will do the trick to get you on the liver transplant list.Methyl1 said:
Do not add a Methyl with Dbol.
:trout:
DR.D
Well-known member
Methyl1 said:
JonesersRX7 is right, don't stack those two. Good results but you have to really know what your doing because it's a pretty toxic combo. You can stack M1T with test or Dbol with test though. I thought you were going for a non-suppressive, low dose dbol only cycle though? I would personally stack it with test, but now your talking about a real cycle with high suppression, PCT requirements, and gyno concerns.
IronPimper
New member
I am halfway through a 24 day sd cycle. week 1, 10mg/day,
week 2 and 3 20mg/day. I was going to do the following for pct, any thoughts:
week 3: 20mg/sd, 2 caps of ultra hot
week 4: 40mg nolva, 1.5g fenu, 100mg dhea + blue rhino
week 5: 20mg nolva, 2.2g fenu, 100mg dhea + blue rhino
week 6: 10mg nolva, 2.8g fenu, 100mg dhea, 3 caps Ultra Hot + blue rhino
week 7: 0mg nolva, 3.5g fenu, 100mg dhea, 4 caps Ultra Hot + blue rhino
Blue rhino is avenia sativa, longjack, trib, caffeine. I would also be taking
5g per day of cee, zma w/ added trib, proliver, and policosinol.
week 2 and 3 20mg/day. I was going to do the following for pct, any thoughts:
week 3: 20mg/sd, 2 caps of ultra hot
week 4: 40mg nolva, 1.5g fenu, 100mg dhea + blue rhino
week 5: 20mg nolva, 2.2g fenu, 100mg dhea + blue rhino
week 6: 10mg nolva, 2.8g fenu, 100mg dhea, 3 caps Ultra Hot + blue rhino
week 7: 0mg nolva, 3.5g fenu, 100mg dhea, 4 caps Ultra Hot + blue rhino
Blue rhino is avenia sativa, longjack, trib, caffeine. I would also be taking
5g per day of cee, zma w/ added trib, proliver, and policosinol.
DR.D
Well-known member
pu12en12g said:
Yeah, I've experimented with it like that before. It helps with prostate issues and attenuates suppression. But it has to be cycled, so I really just save it for PCT or use it right at the start of a cycle. Just don't take it at the same time of day as DHEA or Preg, it hurts the absorption. I take fen all in one dose at night.
DR.D
Well-known member
raylewis said:
Yes sir, I'm in wk2 doing the invert method like I planned, but I'm using the bad lot of RXT so my results are invalid! :frustrate Looks like I'll have to wait till my next PCT to try it. I am having really killer results with my PCT though. Using Ral(from custom), fen, DHEA, creatine, LX, MP, and ACT. Wow! I feel like I'm still on. I'm strong, hard and pumped and recovering still recovering well. Plus I'm shredding up like I was on a diet and diuretics or something. MP and ACT are awesome.
IRserge
Board Supporter
So after a 6 week 1-ad/m4ohn cycle at 400mg 1-ad/16mg m4ohn would a PCT like this be overkill if you wanted to get your nuts back and keep most of your gains:
W1: 40mg Nolva, 25mg RXT, 3caps Fen., 10g Creatine
W2: 40mg Nolva, 25mg RXT, 4caps Fen., 10g Creatine
W3: 20mg Nolva, 50mg RXT, 5caps Fen., 10g Creatine
W4: 20mg Nolva, 50mg RXT, 6caps Fen., 10g Creatine
*Note*: I'm just planning this for the future, I have no intentions of using any PH's at this time.
W1: 40mg Nolva, 25mg RXT, 3caps Fen., 10g Creatine
W2: 40mg Nolva, 25mg RXT, 4caps Fen., 10g Creatine
W3: 20mg Nolva, 50mg RXT, 5caps Fen., 10g Creatine
W4: 20mg Nolva, 50mg RXT, 6caps Fen., 10g Creatine
*Note*: I'm just planning this for the future, I have no intentions of using any PH's at this time.
DR.D
Well-known member
wideguy said:
Creatine, Milk Thistle and ACT all the way through too.
wk1: Clomid 150mg/d, LX 100md/d, DHEA 200mg/d, Fen 4 caps/d, RXT 25mg/d
wk2: Clomid 100mg/d, LX 100mg/d, DHEA 200mg/d, Fen 5 caps/d, RXT 25mg/d
wk3: Clomid 50mg/d, Nolva 40mg/d, LX 50mg/d, DHEA 200mg/d, Fen 6 caps/d, RXT 25mg/d
wk4: Nolva 60mg/d, DHEA 200mg/d, RXT 50mg/d
wk5: Nolva 40mg/d, DHEA 100mg/d, RXT 50mg/d
wk6: Nolva 20mg/d, DHEA 100mg/d, Fen 4 caps/d, RXT 50mg/d
wk7: Nolva 20mg/d, DHEA 100mg/d, Fen 5 caps/d, RXT 75mg/d
wk8: DHEA 100mg/d, Fen 6 caps/d, RXT 75mg/d
Sky9
Tan from the Island
I just read this, now I wish I had gotten that instead of nolva for my SD PCT
Raloxifene: A Selective Estrogen Receptor Modulator
JANINE A. SCOTT, M.D., Duke/Southern Regional Area Health Education Center, Fayetteville, North Carolina CARLOS C. DA CAMARA, PHARM.D., Campbell University School of Pharmacy, Buies Creek, North Carolina J. ELIZABETH EARLY, PHARM.D., University of North Carolina School of Pharmacy, Chapel Hill, North Carolina Invalid Link Removed[size=-1]Invalid Link Removed[/size]
Raloxifene: A Selective Estrogen Receptor Modulator
JANINE A. SCOTT, M.D., Duke/Southern Regional Area Health Education Center, Fayetteville, North Carolina CARLOS C. DA CAMARA, PHARM.D., Campbell University School of Pharmacy, Buies Creek, North Carolina J. ELIZABETH EARLY, PHARM.D., University of North Carolina School of Pharmacy, Chapel Hill, North Carolina Invalid Link Removed[size=-1]Invalid Link Removed[/size]
[size=-1]Raloxifene is a selective estrogen receptor modulator that produces both estrogen-agonistic effects on bone and lipid metabolism and estrogen-antagonistic effects on uterine endometrium and breast tissue. Because of its tissue selectivity, raloxifene may have fewer side effects than are typically observed with estrogen therapy. The most common adverse effects of raloxifene are hot flushes and leg cramps. The drug is also associated with an increased risk of thromboembolic events. The beneficial estrogenic activities of raloxifene include a lowering of total and low-density lipoprotein cholesterol levels and an augmentation of bone mineral density. Raloxifene has been labeled by the U.S. Food and Drug Administration for the prevention of osteoporosis. However, its effects on fracture risk and its ability to protect against cardiovascular disease have yet to be determined. Studies are also being conducted to determine its impact on breast and endometrial cancer reduction. (Am Fam Physician 1999;60:1131-9.)[/size]
Sky9
Tan from the Island
Vopr Onkol. 1997;43(6):587-95.Invalid Link Removed LinksDR.D said:
[size=+1][Results of phase II clinical trial of Tamoxifen and Toremifen in two different doses in advanced breast cancer in postmenopausal women][/size]
[Article in Russian]
Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed, Invalid Link Removed.
Prof. N.N.Petrov Institute of Oncology, Ministry of Health of the RF, St.Petersburg.
Efficacy and safety of toremifene 60 and 240 mg daily (TOR 60 and TOR 240) are compared to 40 mg tamoxifen daily (NFV 40) in postmenopausal women with advanced estrogen receptor (ER) positive of ER unknown breast cancer. The study is randomized in three parallel groups. Primary efficacy variables are response rate and time to progression. WHO and ECOG criteria were used for measurable and nonmeasurable disease assessment, respectively. Safely was reported according to WHO criretia. Altogether 463 patients were randomized (157 to TOR 60, 157 to TOR 240 and 149 to TAM 40). By data cut-off, after 20.5 months medianfollow-up time, over 70% of the patients had experienced disease progression. Response rates are 20.4%, 28.7% in TOR 60, TOR 240 and TAM 40, respectively. TOR 60 and TAM 40 show statistically equivalent efficacy and the difference between TOR 240 and TAM 40 is not significant (P = 0.112). Median times to progression are 4.9 (TOR 60), 6.1 (TOR 240) and 5.0 (TAM 40) months and corresponding hazard ratios (TAM:TOR) 1.015 and 1.124. Again, TOR 60 and TAM 40 are statistically equivalent and the difference between TOR 240 and TAM 40 is not significant (P = 0.374). All treatments were well tolerated. As a conclusion TOR 60 and TAM 40 show equivalent clinical efficacy and tolerability. The higher dose of toremifene slightly but not significantly improved response rate and time to progression. In postmenopausal women, toremifene (60 mg) daily is an effective and safe treatment of advanced ER positive or ER unknown breast cancer.
Publication Types:
- Clinical Trial
- Clinical Trial, Phase III
- Randomized Controlled Trial
Sounds by this study that tamoxifen may be better because it takes a lower dosage to show effectiveness, but that at 60mgED it may be a little better. Can you better explain your experience with nolva vs. raloxifene. Personally I wish I could just use clomid for PCT, I really like that stuff.
The Paper Route
Member
What should my dosing of U.H. be on the last week with of a 3 week cycle of sd at 10/20/20 ?
TrainTilUDrop
New member
Yeah but clen burns calories like its going out of style so if he is an ecto he is going to burn all his gains away. That is how it is for me anyways. I don't believe using clen would be a very good idea. I think just using DHEA would be a better idea then adding in a metabolic enhancer like clen.DR.D said:
DR.D
Well-known member
TrainTilUDrop said:
You have a point. I'm an ecto too, but it isn't too bad for me. I always encourage my loss of gains though. People try to hold on to them, but the way I see it, if they are for real then you shouldn't have to walk on egg shells to keep them. If you do, then screw it. It was just temp gains anyway. I may be over generalizing a little.
Gethuge
Member
DR.D said:
That's a good and valid point I believe. It's one of the arguements I make for the differences in net gains for different drugs. Tren leads to more solid and "slower" gains in some peoples minds than test. However if you take the net gained from each cycle into account there's really not a lot of difference. YOu gain more while on a test cycle but you lose a lot post cycle because of water loss. In the end there really isn't a whole lot of difference.
Gethuge
Member
Differences in perspective for different people I guess. Younger guys usually love the huge, bloated, over the top look while they are on a test/dbol or similar cycle. For us "more experienced" (i.e. older) fellows slower yet more permanent gains are more desired.
P.S. my appologies for using true steroids in my examples in the PH section.....oh well...it's all the same now anyway really.
P.S. my appologies for using true steroids in my examples in the PH section.....oh well...it's all the same now anyway really.
SilentScream27
Member
Dr. D, what do you think of using ralixofene (instead of nolva) for PCT for a 4-6 weeker? I'm impressed by its gyno-reducing properties, I just want to make sure that it will be equally effective at restoring natural test at appropriate doses.
DR.D
Well-known member
SilentScream27 said:
I started using it @ 180mg/d 3 weeks ago for my recent PCT. It has not produced dramatic results, so I think I'll switch to a 150mg Clomid for a week and finish with Nolva the last two weeks @ 60,40. Ral is a great gyno treatment and a good calcium loader in bone, also mildly improves lipid ratios and is one of the most non-toxic SERM's out there. But it seems to lack the potency needed for rapid PCT effects. It is actually a very potent anti-e so this can probably be explained by it's very low oral bioavailability, only about 2%. It is very water soluble so theoretically, an isotonic aqueous solution could be delivered IM @ about 15-30mg/d to give awesome PCT effects, but it's only speculation.
SilentScream27
Member
ok, thank you very much for that information, I will continue using nolva for PCTs. I just have one more question... is nolva still better for on-cycle gyno control? As in, "****, my gyno is flaring up, better take some ______"; would ralixofene be better/worse than nolva here?DR.D said:
intv
Board Supporter
Any chance of using ral trandsdermally? Could custom's solution be mixed into lipderm-y for instance? I know lipo is a local carrier, but could it be applied (w/ral) to the chest? From a quick search it looks like the MW of ral is around 550. Does custom use PEG for his ral? Tastes like it to me. Also looks like it has the binders from the pills in the solution. Sorry, just thinking out loud.
DR.D said:
DR.D
Well-known member
raylewis said:
Yeah, I still am advocating the inverse ramp. It just makes sense to me that as PCT moves into the last half, test levels start getting higher again. That means estrogen levels are getting higher too but your SERM dose keeps getting lower! See what I mean? It's just to avoid upregulating estro at the end and getting any SERM rebound effect. I'm back on a good batch of RXT again so hopefully I can salvage this PCT and still get some good data on Ral+Clomid+Nolva, MP, RXT, and ACT (all the new stuff and new combo's I'm using!!)
DR.D
Well-known member
SilentScream27 said:
Nolva is tried and true. If your 'preventing' I'd say Ral or letro, but if your 'aborting' I'd say Nolva. Once you get gyno during a cycle and decide to abort, Ral may work just as well, but I have not used it in that capacity so I can not say. I suspect it would, at the right dose.
DR.D
Well-known member
intv said:
It has no binders. I tested his first batch and it was of pure quality. He probably put it in the same carrier that he does the nolva and clomid with (could be PEG or PP), but it is very water soluble and his solution turns clear yellow when diluted. Ral is a hydrochloride amine salt. I don't know how trans would work for that, maybe w/ DMSO or if you alkalinize the solution first? Also, the molecular weigh is a bit high, but it probably could be done with some research, trial, and error. It seems best suited for Sub-q or IM injection IMO. Good, long 24hr half-life, so ED or EOD would afford steady state.
Last edited:
DARREN CROLLA
New member
DAZ
ANYONE
I HEAR A LOT ABOUT NOLVA PCT, BUT WHAT'S THE DIFFERANCE
BETWEEN NOLVA AND TAMOXIFEN. I HAVE 20 MG TABS OF TAMOXIFEN
WHICH I WAS GOING TO USE FOR MY PCT AFTER MY 3 WEEK SUPERDROL CYCLE
ANYONE
I HEAR A LOT ABOUT NOLVA PCT, BUT WHAT'S THE DIFFERANCE
BETWEEN NOLVA AND TAMOXIFEN. I HAVE 20 MG TABS OF TAMOXIFEN
WHICH I WAS GOING TO USE FOR MY PCT AFTER MY 3 WEEK SUPERDROL CYCLE
DARREN CROLLA
New member
DAZ
Thanks for the advice Scottyo, it's just that i thought tamoxifen was a stronger version of nolva and if so, what dosage to take compared to nolvaScottyo said: