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ANABOLIC-PUMP™ Real Time Q&A to form a FAQ

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Because you would be theoretically using your insulin better then adding AP and not adjusting your Insulin dose would cause Hypo
 
Cool, thanks Bio and JayHawk. Whenever I can get my hands on some AP I'll take my slin dose down to like 4iu's the first couple times to assess tolerance. I generally dont need as many carbs to stay out of hypo as some people do, but I'll start out a bit higher than I normally would and adjust slin dose and carb intake according to hypo symptoms or lack thereof (once AP is released). Wich is cool with me. Cuz I like carbs.
 
psychospic said:
How would this work with Superdrol which also relies on high carb intake?
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Based on what I've read, it would make Suoerdrol more effective. I have a friend who is planning to combine the two. Maybe I can convince him to post a log.
 
colkurtz_spf said:
Based on what I've read, it would make Suoerdrol more effective. I have a friend who is planning to combine the two. Maybe I can convince him to post a log.
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That would be Flucking awesome. I am loving my results on SD now, I can't imagine it being even better. I might just have to give this combo a run in a few months.
 
"Im taking GH and insulin currently. (The former of course is known to decrease insulin sensetivity).

1) Can I expect to achieve a more efficient utilization of my insulin shots w/ use of AP?

2) Will I go hypo more easily if I combine a dose of AP w/ my pre-meal insulin (humalog) dose?
If so, should I eat more carbs w/ it, or just take a lower dose of insulin?

3) is there anything else I should be wary of when combining these 3?"

First off, good stack :twisted:

1. You will be more insulin sensitive thus requiring less insulin

2. YUP, carb intake will have to go up in the ball park of 100g's+

3. No, just make sure your not eating too little carbs, too much in this case would be alot safer then too little. So lets say you combining AP with your SLin shot and originally where taking in 100g's of carbs, i'd bump it right up to 200g's.
 
We've gone over the advanced stuff but I don't think the basics have been covered:

How do you dose Anabolic Pump:

Take a Sunday through Saturday 7 day week.

1) When do I take it on training days? And how much?
2) When do I take it on off (or cardio) days? And how much?

Thanks
 
Ohhh I got one

Regarding the carb/hypo thing

Pre and postworkout for my carbs I have oats or sweet potatoe

Now, I usually have alot of milk with that

Should I be cutting out the milk too ? Or are we looking at the overall makeup of the meal? Like if most of your meal is complex carbs is it okay to throw in stuff like honey, milk, fruit, etc, as a topper?
 
Well, from experience I drink 1 % lactaid milk with most of my meals and I've had any problems as far as feeling hypo is concerned.

I'm using the bulk powder version that was sold through nutraplanet.com
 
icey said:
Ohhh I got one

Regarding the carb/hypo thing

Pre and postworkout for my carbs I have oats or sweet potatoe

Now, I usually have alot of milk with that

Should I be cutting out the milk too ? Or are we looking at the overall makeup of the meal? Like if most of your meal is complex carbs is it okay to throw in stuff like honey, milk, fruit, etc, as a topper?
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All sugars aren't made equal, which is something to keep in mind. Lactose will be broken down far slower than glucose for example, so you are fine in doing that.
 
protomike said:
mine has been very healthy over the last two months on ap. nothin to complain about at all.
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I don't have the final product but on YG it doesn't affect my libido at all either.
 
I have a question.When will we know won some?There have been a couple of good mock write ups in the supp section.:study: :thumbsup:
 
Vipersg123 said:
Q: Is it safe to take this product if you suffer from diabetes?
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Yes, absolutely. Jacob discovered this herb through a study done on Diabetics, specifically obese diabetics, as a functional herbal replacement for insulin regimine. We are just using it for our purposes, it is, in fact, meant for them.
 
Mulletsoldier said:
Yes, absolutely. Jacob discovered this herb through a study done on Diabetics, specifically obese diabetics, as a functional herbal replacement for insulin regimine. We are just using it for our purposes, it is, in fact, meant for them.
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I'm very curious about the feedback on overall weight gain. What's everybody else noticing in terms of their weight? It increases glycogen stores, yet supposedly increase fatty acid oxidation. So has the net effect been an overall decrease in wgt or the opposite?
 
Is there a big advantage between AP and say, Designer Supplement's GXR? It seems to me like they are kind of similar. If I am missing something please set me straight.
 
While GXR is a fine product, and I have used it myself in the past, I cannot say that AP and GXR are in the same supplement category. Now, that is not meant in a derogatory manner either, just at USP Labs we feel that Anabolic Pump is the only legal product which is proven to shuttle nutrients into muscle cell mitochondria while preventing the same process in fat cells and causing lypolysis.
 
prld2gr8ns said:
I'm very curious about the feedback on overall weight gain. What's everybody else noticing in terms of their weight? It increases glycogen stores, yet supposedly increase fatty acid oxidation. So has the net effect been an overall decrease in wgt or the opposite?
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I think the differences in weight have been totally diet dependent. In a deficiet, your net weight may drop, but may also likely remain static as you recomp so to speak. In a surplus, net weight should increase, however, with a very small amount of fat gained.
 
Mulletsoldier said:
While GXR is a fine product, and I have used it myself in the past, I cannot say that AP and GXR are in the same supplement category. Now, that is not meant in a derogatory manner either, just at USP Labs we feel that Anabolic Pump is the only legal product which is proven to shuttle nutrients into muscle cell mitochondria while preventing the same process in fat cells and causing lypolysis.
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While lowering LDL cholesterol and anti -imflammatory and prevents adipogenesis....
 
Mulletsoldier said:
I think the differences in weight have been totally diet dependent. In a deficiet, your net weight may drop, but may also likely remain static as you recomp so to speak. In a surplus, net weight should increase, however, with a very small amount of fat gained.
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Not to bug to much but what would you say about someone running a maintainence diet?
 
prld2gr8ns said:
Not to bug to much but what would you say about someone running a maintainence diet?
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Your weight should stay fairly stationary, you may gain or lose a few lbs depending on how you spend your day, workout etc.
 
prld2gr8ns said:
Not to bug to much but what would you say about someone running a maintainence diet?
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You would lose weight. But why...think about it like this...

You can increase your caloric intake and still be at maintenance level but lose fat and gain muscle.
 
I'm pretty much just on maintenance calories though I have "cheated" with far more carbs than usual. Doing this on YG initially caused me to lose a little fat, but now I am holding stedy in body composition which is actually saying a lot considering I am not putting any real effort into it.

Been lifting 2x per week with no time for cardio. I have a strong feeling that AP would allow for a faster more efficient cut if doing cardio every morning or even at night as you could ensure that you'd be in a fasted state whenever you do your cardio.
 
prld2gr8ns said:
I'm very curious about the feedback on overall weight gain. What's everybody else noticing in terms of their weight? It increases glycogen stores, yet supposedly increase fatty acid oxidation. So has the net effect been an overall decrease in wgt or the opposite?
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I started 2months ago on YG at 185lbs in the morning I'm now Im 193lbs in the morning. And I have lost over 0.5'' from my waist.
 
1]in the average 40 year old male what would be normal ratio between the nutrients that go to fat or muscle--is it like an age thing , as you get older more goes to fat, or more like genetics?------------------------------------------------------- 2]if its not GLXR or NOH type products where the pump goes away[I think you claim perment gains?]then are you guys claiming HYPERPLASIA? --------------------------------------3] if my diet and training is in check , then whats stopping my muscle cells from getting propper nutrition on its own?
 
skull said:
1]in the average 40 year old male what would be normal ratio between the nutrients that go to fat or muscle--is it like an age thing , as you get older more goes to fat, or more like genetics?------------------------------------------------------- 2]if its not GLXR or NOH type products where the pump goes away[I think you claim perment gains?]then are you guys claiming HYPERPLASIA? --------------------------------------3] if my diet and training is in check , then whats stopping my muscle cells from getting propper nutrition on its own?
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No not claiming hyperplasia. You bring up an interesting point about weithlifting causing Hypertrophy.a bigger muscle cell! A bigger muscle Cells means increase in Glycogen storage which Could trigger many anabolic processes.

Weight training + AP = (I will let you fill that in after a run with AP).
 
Weight training + AP = more muscle, less fat, more strength, more endurance, better pumps.
 
If I'm not mistaken, Metformin has some increasing insulin sensativity advantages to it, but it's primary use is for the control of liver glucose storage release. That being said, I'd say AP(strictely based on some of the blood work seen so far with NON-DIABETIC people) is comparable.
 
I'm pretty sure that Metformin has been said to increase glucose uptake of adipose tissue as well, while AP is supposed to do the opposite.
 
Speedbacker said:
Do you think AP would be a better nutrient partitioning agent than glucophage (metformin)?
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Yes and Metformin as many drugs are Herbs first. This is the reason I love the Herbal market.
 
rpen22 said:
I'm pretty sure that Metformin has been said to increase glucose uptake of adipose tissue as well, while AP is supposed to do the opposite.
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I've never heard that, but you very well could be right. I'm a non-diabetic and have been taking metformin with my higher carb meals while eating in a caloric surplus and it is really helping me to stay lean despite eating quite a bit of carbs and calories. My pumps in the gym are great after taking this. If AP is anything like glucophage then that would be great.
 
Speedbacker said:
I've never heard that, but you very well could be right. I'm a non-diabetic and have been taking metformin with my higher carb meals while eating in a caloric surplus and it is really helping me to stay lean despite eating quite a bit of carbs and calories. My pumps in the gym are great after taking this. If AP is anything like glucophage then that would be great.
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I looked further into it, and Metformin increases insulin-stimulated glucose uptake of VAT, but not subcutaneous fat.
 
USPLabs said:
Yes and Metformin as many drugs are Herbs first. This is the reason I love the Herbal market.
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I love my herbals. For me it seems the more processed something is the harder it is for me to absorb it. This goes for foods as well (I have a hard time processing a lot of stuff and it just makes me feel ill).
 
SIDE EFFECTS: The most common side effects with metformin are nausea, vomiting, gas, bloating, diarrhea and loss of appetite. These symptoms occur in one out of every three patients. These side effects may be severe enough to cause therapy to be discontinued in one out of every 20 patients. These side effects are related to the dose of the medication and may decrease if the dose is reduced.

A serious--though rare--side effect of metformin is lactic acidosis. Lactic acidosis occurs in one out of every 30,000 patients and is fatal in 50% of cases. The symptoms of lactic acidosis are weakness, trouble breathing, abnormal heartbeats, unusual muscle pain, stomach discomfort, light-headedness and feeling cold. Patients at risk for lactic acidosis include those with reduced function of the kidneys or liver, congestive heart failure, severe acute illnesses, and dehydration.

Metformin acts by increasing the sensitivity of liver, muscle, fat, and other tissues to the uptake and effects of insulin. These actions lower the level of sugar in the blood. Unlike glucose-lowering drugs of the sulfonylurea class, e.g. glyburide (Micronase; Diabeta) or glipizide (Glucotrol), metformin does not increase the concentration of insulin in the blood and, therefore, does not cause excessively low blood glucose levels (hypoglycemia) when used alone. In scientific studies, metformin reduced the complications of diabetes such as heart disease, blindness and kidney disease. Metformin was approved by the FDA in December of 1994--------------------------------does AP have these same sides?--how about price compared to metformin?--I think you could get 100 caps[500mg] for 34$
 
Titre du document / Document title
Metformin restores responses to insulin but not to growth hormone in Sprague-Dawley rats
Auteur(s) / Author(s)
BORST Stephen E. ; SNELLEN Harold G. ; ROSS Henry ; SCARPACE Philip J. ; KIM Yong-Woon ;
Résumé / Abstract
Administration of growth hormone (GH) increases muscle mass in F344 x BN rats, but not in Sprague-Dawley (S-D) rats. S-D rats are insulin-resistant and insulin responsiveness is required for the anabolic actions of GH. Wehypothesized that correction of insulin resistance with metformin might also restore anabolic effects of GH. Treatment with GH (0.25 or 1.0 mg/kg twice daily for 9 days) had limited anabolic effects, reducing weight gain by 14%, increasing muscle glycogen content by 40% and increasing exercise capacity by 24%, but failing to increase muscle mass or to reduce fat mass. GH also impaired insulin responsiveness and increased visceral fat TNF content of visceral fat by 77%. Metformin enhanced insulin responsiveness in skeletal muscle, but failed to enhance anabolic effects of GH. Rats aged 14 weeks were treated for 21 days with metformin (320 mg/kg/day) and for the last 9 days, with GH (0.25 mg/kg, twice daily). Metformin caused a 2.3-fold increase in insulin-stimulated muscle glucose transport and a 20% reduction in muscle fatty acid oxidation, indicating increased glucose utilization. However, metformin did not augment GH-induced weight reduction. Metformin decreased visceral fat by 22% and subcutaneous fat by 20%, but no decreases were observed in the GH/metformin group. GH increased muscle glycogen by 40%, but the effect was reversed by metformin. VO[2][m][a][x] was increased 24% by GH and 17% by metformin, but was not elevated in the GH/metformin group. GH increased TNF in visceral fat and the effect was augmented by metformin (144% increase). We conclude that metformin enhances some aspects of insulin responsiveness, but does not enhance anabolic responses to GH. The latter may, in part, be explained by the failure of metformin to prevent GH-induced elevation of TNF in visceral fat.
Revue / Journal Title
Biochemical and biophysical research communications (Biochem. biophys. res. commun.) ISSN 0006-291X CODEN BBRCA9
Source / Source
2002, vol. 291, no3, pp. 722-726 [5 page(s) (article)]
Langue / Language-----------------------------------------------So I guess the question is can AP beat the 20% differental and can USP beat the price?
 
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