@GreenMachineX
I am not an expert on this of course, but from my limited experience with reading bloodwork and such, here are my thoughts.
It is hard to really say much when given a single value without seeing ALL of the bloodwork. A lot of these things don't happen in a vacuum and you can generalize what each value represents but it is all part of a bigger picture and a single reading can easily be taken out of context.
If everything else is inline and you had a complete CBC, etc. - then a slightly low WBC is not a concern off the bat to me.
Do you know your neutrophils, lymphocytes, and actual platelet counts?
I would be more concerned with elevated WBC during covid. It's my understanding that a cytokine storm is ultimately what causes death in Covid, and a cytokine storm will look a lot like sepsis with severely elevated WBC.
As for the platelets - this seems to be common in Covid patients and was really the problem a few months back when everyone was up in arms about strokes from the vaccines - increased cytokines will increase the destruction of platelets. If you have a stroke and low platelets, and doctors don't know this - they will give you a blood thinner for the stroke and cause an issue. This is pretty rare though and low on the concern list...especially if they are mildly suppressed. IMO at least.
The following paragraph from
this study is interesting:
Patients with COVID-19 pneumonia can present with blood coagulation abnormalities, commonly manifested by elevated levels of fibrinogen and D-dimer in tandem with mild thrombocytopenia.115,116 Rise in D-dimer levels has been linked to poorer prognosis and higher mortality rate.117,118 D-dimer levels, lung inflammation, and pulmonary hemorrhage are influenced by neutrophil elastase activity.119,120 As such, suppression of elastase and neutrophil activation may be helpful in hemorrhagic or thrombotic complications associated with COVID-19.121 N-acetylcysteine has been found to inhibit release of elastase and modulate neutrophil activity when used at high concentrations.122 In neutrophilic airway inflammation in cystic fibrosis, high-dose N-acetylcysteine decreases the neutrophil burden in airways and the number of airway neutrophils actively releasing elastase-rich granules.123 Treatment with N-acetylcysteine can also attenuate lung emphysema induced by elastase as depicted in amelioration of airspace enlargements, partial recovery of expiratory flows, and normalization of lung collagen content.124 This evidence sheds light on the possible role of N-acetylcysteine on mediating inflammation-mediated lung injury and abnormalities of blood coagulation in severe COVID-19.
I would maybe see if you can convince your doctors to test fibrinogen levels and this may be a way for NAC to be a benefit. I personally think NAC and Vitamin D are probably your best bet. I'm not really sold on the quercetin stuff.
Also, they are hard to find but there are studies that suggest Vitamin D may reduce "tolerance" to corticosteroids like prednisone or cortisol. This may mean that, since you've maintained sufficient levels, you are having a strong/appropriate response to a small amount of extra steroid hormones in your system, which means your body doesn't have to go nuts and release a bunch extra to get the effect - and extra tends to increase side effects - so you you've minimized some sides from the endogenous response. If you have an increase in immature white blood cells as well, this would indicate that you are losing them more quickly due to the infection; but also making more rapidly which is an appropriate response and this effect of Vitamin D would facilitate the process I believe.
All in all, I'm grasping at straws because I don't have a complete picture but I don't think there is any real reason for concern. Keep monitoring.