Don't underrate how far this forum has come
Forskolin increases adenyl cyclase activity, which is best taken advantage of by using upstream activators of adenyl cyclase. B-agonism activates the b-adrenergic receptor (GPCR) -> Gs -> adenyl cyclase -> cAMP -> PKA -> effectors (lipolysis). Thus, forskolin, by further boosting adenyl cyclase activity, potentiates b-agonists. Caffeine and alpha-2-antagonists (yohimbine) do the same via adenosine receptors and disinhibition of postganglionic NT release, respectively.
So that's ABE + Alphamine so far. And that's one side of the coin: lipolysis (the freeing of fatty acids for adipose). Now you need to burn those fatty acids, which is where TTA comes into play be increasing b-oxidation. What else increases b-oxidation? Exercise.
In summary:
The stack you outlined works very well for covering all your fat loss bases. It works best if consumed prior to exercise or fasting, both of which are periods in which b-agonism and b-oxidation are peaking.
Bonus: There is a feedback mechanism in the body that prevents "runaway lipolysis." Fatty acids in the bloodstream increase insulin release, which feeds back on HSL and prevents further lipolysis. In other words, the maximum rate of lipolysis in your body has a ceiling after which it cannot be exceeded. How can you increase the height of this ceiling? Take all of these preworkout along with a weak, insulin-independent glucose disposal agent (if too strong, you risk hypoglycemia). By reducing blood glucose without insulin, you keep insulin levels low and HSL activity high. Alternatively, intense exercise will induce epinephrine release which strongly blocks insulin release form b-islets.
