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Non methyls

mugen112

mugen112

Member
What are some non methyl PHs? I'm using katanadrol but am hoping to get some different ones for the next cycle. Are there any good ones available right now?
 
Look into Primordial Perfomance and Frontrunner labs products. All are non-methyls. Also, pstanz, stanodrol, 11oxo, 5aOHP, trenezone just to name a few. Look and you shall find.
 
stanodrol, non methyl ph to dht
11-sterone, non methyl ph to 11-keto testosterone
delta 2, ph to non methylated desoxytestosterone (non methylated phera)
4-dhea, ph to testosterone
1-dhea, ph to 1-test
19-nor dhea, ph to nor test
pstanz, non methylated winstrol
trenazone, transdermal dienolone
 
Also coming, but not yet available:

Mentdione - Precursor to Trestolone (MENT)
Trendione - Precursor to Trenbolone
 
kevinhy said:
Also coming, but not yet available:

Mentdione - Precursor to Trestolone (MENT)
Trendione - Precursor to Trenbolone
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this!

also 1 test seems like a nice non methyl
 
vpower said:
1 Test can also cause some lethargy in some cases, it would be a nice idea to stack it with DHEA or some pregnenelone.
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hit it right on the head, i have never used it but im looking forward to getting around to it if i can get some via trade or cheap sale
 
rochabp said:
hit it right on the head, i have never used it but im looking forward to getting around to it if i can get some via trade or cheap sale
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PP's old 1-t had it, and I believe they still some bottles of v2 Andromass left, which has it in there as well. I have seen that they are taking it out of their new formula next month tho.
 
i was looking more into ironmags 1 andro
which looks to be high quality stuff its just too rich for my liking if i wanted to try it at a high dose...which i probably would wanna high dose it.
 
maybe its me, and i dont have lab results to prove it but epi seems to be very mild on my liver, i dont get that nasty yellow pee or any other symptoms on stronger phs but then again i cant really prove it, but i am not the only to report this
 
max lmg wasn't put in the list because it is still an alkylated compound.

13b ethylated to be exact.

but it, like metnadione is pretty mild, nothing like a 17a methyl
 
Epistane is harsh for me tho I do like it. It is more mild than superdrol tho ;)

Are there any max lmg products that don't have dimethazine in it?
 
i would rec natadrol and M1D for non liver toxic orals.
 
T-Bone said:
The tren type products are hard on the liver even though they are not methyl.
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I dont agree. even straight tren is not liver NORE kidney toxic, it CAN lead to dehydration if not taking in extra water which would in turn cause kidney issues but its not direcly from tren.
 
mugen112 said:
Epistane is harsh for me tho I do like it. It is more mild than superdrol tho ;)

Are there any max lmg products that don't have dimethazine in it?
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also M1D with katanadrol would be nice
 
I would be interested to see if m-lmg puts stress on the liver. the usual points of liver stress come from areas of the compound which are more difficult to metabolize or unable to be metabolized due to stereochemical alignment / steric hindrance.

but, I don't think the 13b-ethyl is any area of metabolism in max-lmg, although it seems to be fairly orally active, so who knows.

mugen112 said:
Are there any max lmg products that don't have dimethazine in it?
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CEL M-LMG bruh
 
jbryand101b said:
hmmm, seems like something to be discussed with pa and henryv
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FWIW you may very well be correct about the 13b-ethyl-liver stress association. I remember seeing some metabolites in a paper I read where a hydroxyl group would attach to the C-18 in certain steroids. So it's possible m-lmg's 13b-ethyl may inhibit such a process from occuring.

Here's an example just by googling:
Invalid Link Removed

^18-hydroxy-1 alpha-methyl-5 alpha-androstan-3,17-dione
 
I'm confused now. So is mlmg harsh on your liver/kidneys or not? How does it does it compare to a traditional methyl like hdrol?
 
im pretty sure when i've commented on it before, pa stated it has hepatotoxicity, though on a much lesser scale than a 17a methyl or 17a ethyl.

but where bry is getting at, i'd have to research, im way off my game on the chemical stuff.
 
romapwr88 said:
I'm confused now. So is mlmg harsh on your liver/kidneys or not? How does it does it compare to a traditional methyl like hdrol?
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Put it this way. An easy way to determine how relatively liver toxic an AAS is, is to determine how many mg's is needed to get the desired anabolic effect orally. Those that take 20mg's a day and under like m1t, superdrol, etc. are usually the toxic monsters. those that require 750+mg's a day are usually much less liver stressful. m-lmg is probably somewhere behind x-tren with respect to liver toxicity. <<pretty general, but hey it seems pretty consistent with a large number of anabolics

jbryand101b said:
im pretty sure when i've commented on it before, pa stated it has hepatotoxicity, though on a much lesser scale than a 17a methyl or 17a ethyl.

but where bry is getting at, i'd have to research, im way off my game on the chemical stuff.
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sorry sometimes i go off on a tangent, didnt mean to confuse anyone.
 
So is stacking M-lmg with D-zine okay or is that frowned upon? Granted I'll be taking all of my supporting supplements and liver care but still, just curious.
 
JerseyDrew77 said:
So is stacking M-lmg with D-zine okay or is that frowned upon? Granted I'll be taking all of my supporting supplements and liver care but still, just curious.
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actually ive seen that ran on more than one occasion seems fine to me, like i said u wouldnt be the first to run it i thinks a pretty popular stack a lot like epi and deinolone
 
i've ran a stacked product with those two, diesel, and liked it.

def recomend getting them seperately though so you can increase/decrease seperately.
 
What have results been like on furuza-a standalones?

Im definitely intrigued by the compounds write up but haven't come across to many solo logs
 
fura products should be ran at 300 plus mg's for 6-8 weeks in reality.

if you can afford to do this, then it is a awsome compound.
 
Bry17 said:
the usual points of liver stress come from areas of the compound which are more difficult to metabolize or unable to be metabolized due to stereochemical alignment / steric hindrance.
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well said

jbryand101b said:
fura products should be ran at 300 plus mg's for 6-8 weeks in reality.

if you can afford to do this, then it is a awsome compound.
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i remember being lucky enough to have a bunch and running it at somwhere between 350-450mg/day for close to 8 weeks. i loved it.
 
jbryand101b said:
fura products should be ran at 300 plus mg's for 6-8 weeks in reality.

if you can afford to do this, then it is a awsome compound.
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agreed, sadly this is not a very cost effective supp.....even 450mg is good but pricey, might as well use winstrol
 
maybe an amazing cutter at that dose but i doubt people gained a lot, imo there are better options if your gonna run it solo unless you are scared for your liver and even then you still have alternatives such as stano but i like a lot too, but to me its biggest asset is its stack-ability
 
gymrat827 said:
450 is a bit high, 350 gives awesome effects. F winny, terrible on the joints.
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i just mentioned winny as its similarly priced....im with you tho, ill pay up for var anyday.....used letro on my tren cycle as nothing else worked, killed my joints, and my fkn workouts for 2wks....illl never touch winny
 
Bry17 said:
FWIW you may very well be correct about the 13b-ethyl-liver stress association. I remember seeing some metabolites in a paper I read where a hydroxyl group would attach to the C-18 in certain steroids. So it's possible m-lmg's 13b-ethyl may inhibit such a process from occuring.

Here's an example just by googling:
Invalid Link Removed

^18-hydroxy-1 alpha-methyl-5 alpha-androstan-3,17-dione
Click to expand...

turns out there may be some truth to metabolism interference. m-lmg's 13b-ethyl may inhibit 11-hydroxylation once converted(or in present state) to the active steroid it's believed to convert to.


Taken from an in vitro (or ex vivo) microbiological analysis:
Homologation of the 13b-alkyl group affects sites of hydroxylation and proportions of hydroxylated products. Hydroxylation products from racemic 13b-ethyl-17b-hydroxygon-4-en-3-one are essentially of analagous structure to those from the parent 17b-hydroxyoestr-4-en-3-one. However 11b-hydroxylation of the racemic 13b-ethyl analogue did not occur with Curvularia lunata. The 13b-ethyl group clearly sterically hindered 11b-hydroxylation in these cases.
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source: terpenoids and steroids; Volume 4; pg.451 Invalid Link Removed

^Whether or not this takes place in man i don't know, but I suspect the 13b-ethyl may truly inhibit metabolism to some degree, therefore liver toxicity is a possibility with M-LMG.
 
Bry17 said:
turns out there may be some truth to metabolism interference. m-lmg's 13b-ethyl may inhibit 11-hydroxylation once converted(or in present state) to the active steroid it's believed to convert to.


Taken from an in vitro (or ex vivo) microbiological analysis:


source: terpenoids and steroids; Volume 4; pg.451 Invalid Link Removed

^Whether or not this takes place in man i don't know, but I suspect the 13b-ethyl may truly inhibit metabolism to some degree, therefore liver toxicity is a possibility with M-LMG.
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i read somewhere that lmg has a fairly long half-life which should indicate that it is not easily metabolized. also it is effective at fairly low doses.the 13 ethyl is probably the reason.
xmass.gif

xmass2.gif


the 17 ketone should convert to the 17 hydroxl. i dont know why the didn't put that in the drawings. i guess u could call it 13 ethyl nandrolone as the target molecule.
 
rebelhead said:
i read somewhere that lmg has a fairly long half-life which should indicate that it is not easily metabolized. also it is effective at fairly low doses.the 13 ethyl is probably the reason.
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my thoughts as well. not to mention the C-3 methoxy sort of acts as an ester and possibly extends the half-life.

I kind of always thought it was weird the compound works at such low doses for a non-methyl, especially when the vida data of its active appears weak and it takes quite a few biological processes before becoming 'active.' I wonder how orally active 13b-ethyl nortestosterone is then.
 
Bry17 said:
my thoughts as well. not to mention the C-3 methoxy sort of acts as an ester and possibly extends the half-life.

I kind of always thought it was weird the compound works at such low doses for a non-methyl, especially when the vida data of its active appears weak and it takes quite a few biological processes before becoming 'active.' I wonder how orally active 13b-ethyl nortestosterone is then.
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i dont know. weird. the site i got got the drawing for the conversion states that the methyl at the 3 position is cleaved by stomach acid.
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maybe they are wrong about the double bond shifting to the 4 position.
the 7 alpha methyl version is fairly anabolic.
Friday, October 14, 2011 (2).jpg

maybe somone should make a ph to it. or a transdermal of it
 
Bry17 said:
turns out there may be some truth to metabolism interference. m-lmg's 13b-ethyl may inhibit 11-hydroxylation once converted(or in present state) to the active steroid it's believed to convert to.


Taken from an in vitro (or ex vivo) microbiological analysis:


source: terpenoids and steroids; Volume 4; pg.451 Invalid Link Removed

^Whether or not this takes place in man i don't know, but I suspect the 13b-ethyl may truly inhibit metabolism to some degree, therefore liver toxicity is a possibility with M-LMG.
Click to expand...

quick revision: 11b-hydroxylation does not readily occur in the liver and the only steroidal thing i could find on 11b-hydroxylation in liver was on testosterone, but it doesn't happen to a large extent.

Invalid Link Removedo any liver impairment from m-lmg may not result specifically from attempted 11b-hydroxylation of m-lmg, but that doesn't mean the 13b-ethyl won't sterically hinder other forms of metabolism.
 
13b ethyl methoxygonadiene-> 13b-ethyl-norandrostenedione->13b-ethyl nortestosterone.

norbolethone-> 13b,17a-diethyl-17b-hydroxygon-4-en-3-one

similar, but different.

 
jbryand101b said:
or maybe a 13a,17b diethyl norandrostenedione ph! eh eh?
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that couldn't exist ;) the diol could, but it'd be 17a-hydroxy

there's a sh1t-ton of norandrogens both ph and not that would likely rape in the anabolic department, but most would be no more unique than what's already available

in any case, how bout' a 2-oxa-13b-ethyl-7a, 17a-dimethyl trenbolone?
 
kevinhy said:
Also coming, but not yet available:

Mentdione - Precursor to Trestolone (MENT)
Trendione - Precursor to Trenbolone
Click to expand...

Oh man, can't wait till trendione hits!
 
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