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Goin' Ape fukcing $hit cycle

What's the name of your basketball team?

HAhaha! Drunken mic bastard! So whats up with your cycle? Still playing the waiting game or what? I had to nut up and order some prami and adex yesterday, Ive been on letro but Im almost out so Im making the switch to adex soon. I know you used prami before, how did you dose that? Did you use letro+aromasin? On MD you mentioned you used both while on tren.
 
HAhaha! Drunken mic bastard! So whats up with your cycle? Still playing the waiting game or what? I had to nut up and order some prami and adex yesterday, Ive been on letro but Im almost out so Im making the switch to adex soon. I know you used prami before, how did you dose that? Did you use letro+aromasin? On MD you mentioned you used both while on tren.
Ha ha...still 'ballin huh?

Nah, I never used Prami dude. I haven't had enough time to research it thoroughly. I used Aromasin throughout the cycle and Letro stayed on the shelf as no gyno appeared during the cycle. I kept the Letro for PCT, with Nolva.
 
Ha ha...still 'ballin huh?

Nah, I never used Prami dude. I haven't had enough time to research it thoroughly. I used Aromasin throughout the cycle and Letro stayed on the shelf as no gyno appeared during the cycle. I kept the Letro for PCT, with Nolva.

sorry hijack Tim

Never had any sides estro or prolactin but this is my first run with Tren E ive used oral progestins and ive run Vitex at 1200mg ED as i am right now

What to you guys think? I have Aromasin and Nolva on hand

PCT not an issue i will be cruising at 300mg Test Cyp
 
sorry hijack Tim

Never had any sides estro or prolactin but this is my first run with Tren E ive used oral progestins and ive run Vitex at 1200mg ED as i am right now

What to you guys think? I have Aromasin and Nolva on hand

PCT not an issue i will be cruising at 300mg Test Cyp
What exactly is your question?
 
What exactly is your question?

I think hes asking if Vitex aromasin nolva will be enough for prolactin control. From what I have read, if you control estrogen you will indirectly control prolactin. Prolactin needs and estrogen rich environment in order to become elevated. Im going to use the prami, some of the sides actually sound fun lol. Im gonna start compulsive gambling and asking women to engage in weird/freaky sex acts haha
 
I think hes asking if Vitex aromasin nolva will be enough for prolactin control. From what I have read, if you control estrogen you will indirectly control prolactin. Prolactin needs and estrogen rich environment in order to become elevated. Im going to use the prami, some of the sides actually sound fun lol. Im gonna start compulsive gambling and asking women to engage in weird/freaky sex acts haha
I'm not familiar with Vitex. Is this some sort of P5P product? When I ran my cycle I asked a million questions to HI. His suggestion was to up the dose of Aromasin if problems arose.

Prami does sound like fun, but are there any long-term effects? How many years of research are behind it?
 
I think hes asking if Vitex aromasin nolva will be enough for prolactin control. From what I have read, if you control estrogen you will indirectly control prolactin. Prolactin needs and estrogen rich environment in order to become elevated. Im going to use the prami, some of the sides actually sound fun lol. Im gonna start compulsive gambling and asking women to engage in weird/freaky sex acts haha

Y Taking Vitex currently at 1200mg ED for prolactin control do you think that will be enough, this has worked for me with oral progestins just fine

Have had no estrogen issues so far in cycle day 106

or do you think i should start dosing my AI

Have Aromasin and Nolva on hand
 
Y Taking Vitex currently at 1200mg ED for prolactin control do you think that will be enough, this has worked for me with oral progestins just fine

Have had no estrogen issues so far in cycle day 106

or do you think i should start dosing my AI

Have Aromasin and Nolva on hand

While I don't have the experience I have heard that pramipexole is good to have along side for tren. I have also read that caber is good to have on standby. Then again, if you keep the estrogen low (AI) you should not have any prolactin based sides.
 
I forgot to mention I started a bottle of Alpha-T2 by PES on Monday, I was sweating more than I have been lately while training and cardio. Im taking 2 caps ED, I have high hopes for this product.
 
I'm not familiar with Vitex. Is this some sort of P5P product? When I ran my cycle I asked a million questions to HI. His suggestion was to up the dose of Aromasin if problems arose.

Prami does sound like fun, but are there any long-term effects? How many years of research are behind it?

Its OTC like p5p but I believe its different. HI posted a real good read about it(prami and its effects explored is what its called) on MD

EDIT: Invalid Link Removed


Y Taking Vitex currently at 1200mg ED for prolactin control do you think that will be enough, this has worked for me with oral progestins just fine

Have had no estrogen issues so far in cycle day 106

or do you think i should start dosing my AI

Have Aromasin and Nolva on hand

Its really up to you, I would just to be safe, but if you can get blood work, that would be your best bet.
 
Its OTC like p5p but I believe its different. HI posted a real good read about it(prami and its effects explored is what its called) on MD

EDIT: Invalid Link Removed




Its really up to you, I would just to be safe, but if you can get blood work, that would be your best bet.

Bingo. Yes why didn't I think of that, blood work of course

Cheers
 
Have had no estrogen issues so far in cycle day 106

or do you think i should start dosing my AI

Have Aromasin and Nolva on hand

Its really up to you, I would just to be safe, but if you can get blood work, that would be your best bet.

Bingo. Yes why didn't I think of that, blood work of course

Cheers

Yes, bloodwork will tell the tale. You might not "feel" like your estrogen is high and may not be experiencing water retention/bloat and all that, but your estrogen levels could still be higher than what you desire them to be. It's the best scenario to get the bloodwork reading on your E levels and then make the decision on whether or not to open up that bottle of Aromasin.

I've been relying on low-dose ATD (25mg. per night) to keep my estro levels low while on the injectable cruises. It has been doing a good job so far. I can score some more, but ATD is no longer being produced.
So my next AAS order will have to include some A-Dex......and some more Proviron. :veryhappy:

On my next blasts I already have some Proviron to take to keep estro sides at bay and to free up more testosterone!! For the following cruises, I'll go back to using the ATD until it's all gone and then switch over to the A-Dex.

So: Proviron for blasts and A-Dex for cruises. This will be my protocol unless I can find a better one.


I like to take the lowest possible dose of an AI in order to lower estrogen to acceptable levels instead of totally crushing it. That is always a bad idea!!! DON'T CRUSH YOUR ESTROGEN LEVELS!!! :nono:

Estrogen also has many helpful benefits even in men. It aids with muscle growth, GH release, dry joint relief, and aids in maintaining a healthy libido. :thumbsup:


THE FOLLOWING IS A VERY LONG READ, BUT WELL WORTH THE TIME EXPENDED!!

Aromatizable Androgens and Anabolism:
The Role of Estrogen in Muscle Growth
by William Llewellyn

Can estrogen work to augment muscle growth? Is this hormone always unwanted when we are taking anabolic steroids? Anecdotal reports from athletes suggest that the use of estrogen maintenance drugs such as tamoxifen (anti-estrogen) or aminoglutethimide (anti-aromatase) may slightly hinder muscle mass gains during steroid therapy. An explanation or even clarification for this observation has not been easy to come by. Here I would like to take a look at the comparative effectiveness of certain aromatizable and non-aromatizable drugs, as well as the possible mechanism in which estrogen can play a beneficial role to the athlete.

The Androgen Receptor
All anabolic/androgenic steroids promote muscle growth primarily via the cellular androgen receptor (abbreviated as AR in this article). The steroid attaches to and activates the androgen receptor, which ultimately gives the cell an order to increase protein synthesis. This process is well understood. But it has been suggested that other mechanisms may foster muscle growth during steroid therapy as well, which lie outside of the androgen receptor. One way this is evidenced is by the fact that steroids displaying a high affinity for the AR in muscle tissue do not always promote an equally high level of muscle growth. In other words, anabolic potency does not always correspond perfectly to receptor affinity. Clearly there are some disparities that lead into question whether or not the androgen receptor is the only thing at work concerning growth.

Testosterone, Nandrolone and Methenolone
Testosterone is without question one of the most effective steroids for building muscle mass available to athletes. However it does not have the highest affinity for the androgen receptor compared to some other steroids. For example, it has been shown that by eliminating the 19-methyl group (nandrolone) the affinity of the steroid for the androgen receptor is greatly enhanced. Nandrolone thus displays approximately 2-3 times greater affinity for the androgen receptor compared to testosterone, yet its ability to promote muscle growth seems to be considerably lower than testosterone at an equal dosage. One discussed possibility for this occurrence is the reduced androgenic potency of nandrolone. While testosterone converts to the more active steroid dihydrotestosterone (3-4 times greater AR affinity) upon interaction with the 5-alpha reductase enzyme in various androgenic target tissues such as the skin, scalp, prostate, CNS and liver, nandrolone drops to a third of its original potency by converting to the weak steroid dihydronandrolone[ii]. However this action is very site specific, and in muscle tissue nandrolone dominates as the active form of the steroid. Therefore this explanation may not suffice.

Nandrolone also differs from testosterone in its ability to be converted by the aromatase enzyme to estradiol (an active estrogen). In comparison, nandrolone aromatizes at approximately 20% of the rate testosterone does, and as such is not known as a very estrogenic steroid. It is likewise favored when reduced estrogenic side effects such as water retention, fat deposition and gynecomastia are desired. However athletes know that there is a trade off with the reduced tendency for nandrolone to promote side effects, in that it is a less anabolic steroid. With its known high affinity for the AR in muscle tissue, could this suggest that estrogen may also be a key mediator of muscle growth?

When we look at Primobolan® (methenolone) we see a similar trend. Methenolone is at least as good a binder of the androgen receptor as testosterone. By some accounts it is on par with nandrolone[iii]. However it is known to be much weaker than both steroids at promoting muscle growth. We know that methenolone does not interact with 5-alpha reductase, and as such its affinity for the AR does not increase or decrease in androgen target tissues. This would logically seem like a more favorable trait for anabolism over the weakening we see with nandrolone. However methenolone is a markedly weaker anabolic, and requires relatively high doses to promote growth. This also brings into question the role of 5-alpha reductase in promoting an anabolic state. Perhaps the fact that Primobolan® is a non-aromatizable steroid is more relevant.

Estrogen and GH/IGF-1
To date the most common explanation for why anti-estrogens may be slightly counterproductive to growth in the sports literature has been the suggestion that estrogen plays a role in the production of growth hormone and IGF-1. IGF-1 (insulin like growth factor 1, formerly known as somatomedin C) is of course an anabolic product released primarily in the liver via GH stimulus. IGF-1 is responsible for the growth promoting effects (increased nitrogen retention, cell proliferation) we associate with growth hormone therapy. We do know that women have higher levels of growth hormone than men, and also that GH secretion varies over the course of the menstrual cycle in direct correlation with estrogen levels[iv]. Estrogen is likewise often looked at as a key trigger in the release of GH in women under normal physiological situations.

It is also suggested that the aromatization of androgens to estrogens in men plays an important role in the release and production of GH and IGF-1. This was evidenced by a 1993 study of hypogonadal men, comparing the effects of testosterone replacement therapy on GH and IGF-1 levels with and without the addition of tamoxifen[v]. When the anti-estrogen tamoxifen was given, GH and IGF-1 levels were notably suppressed, while both values were elevated with the administration of testosterone enanthate alone. Another study has shown 300mg of testosterone enanthate weekly (which elevated estradiol levels) to cause a slight IGF-1 increase in normal men, whereas 300mg weekly of nandrolone decanoate (a poor substrate for aromatase that caused a lowering of estradiol levels in this study) would not elevate IGF-1 levels[vi]. Yet another study shows that GH and IGF-1 secretion is increased with testosterone administration on males with delayed puberty, while dihydrotestosterone (non-aromatizable) seems to suppress GH and IGF-1 secretion, presumably due to its strong anti-estrogenic/gonadotropin suppressing action[vii]. All of these studies seem to support a direct, estrogen-dependant mechanism for GH and/or IGF-1 release in men. It is difficult to say at this point just how important estrogen is to IGF-1 production as it relates to the promotion of anabolism in the steroid using athlete, however it remains an interesting subject to investigate.

Glucose Utilization and Estrogen
Estrogen may play an even more vital role in promoting an anabolic state by affecting glucose utilization in muscle tissue. This occurs via an altering the level of available glucose 6-phosphate dehydrogenase. G6PD is an important enzyme in the support anabolism, as it is directly tied to the use of glucose for muscle growth and recuperation[viii] [ix]. During the period of regeneration after skeletal muscle damage, levels of G6PD are shown to rise dramatically. G6PD enzyme plays a vital role in what is known as the pentose phosphate pathway, and as such this rise is believed to enhance the PPP related process in which nucleic acids and lipids are synthesized in cells; fostering the repair of muscle tissue.

A 1980 study at the University of Maryland has shown that levels of glucose 6-phosphate dehydrogenase rise after administration of testosterone propionate, and further that the aromatization of testosterone to estradiol is directly responsible for this increase.[x] In this study neither dihydrotestosterone nor fluoxymesterone could mimic the affect of testosterone propionate on levels of G6PD, an affect that was also blocked by the addition of the potent anti-aromatase 4-hydroxyandrostenedione to testosterone. 17-beta estradiol administration caused a similar increase in G6PD, which was not noticed when its inactive estrogen isomer 17-alpha estradiol (unable to bind the estrogen receptor) was given. An anti-androgen could also not block the positive action of testosterone. This study provides one of the first palatable explanations for a direct and positive effect of estrogen on muscle tissue.

What does this all mean?
It is a long held belief among athletes that estrogen maintenance drugs can slightly hinder muscle gains during steroid therapy with a strong aromatizable steroid such as testosterone. Whether or not we have plausibly explained this remains to be seen, however the above evidence certainly does provide strong support for a direct and positive affect of estrogen on growth. Does this mean we should abandon estrogen maintenance drugs? I don’t think that should be the case. It is important to remember that estrogen can deliver many unwanted effects such as increased water retention, fat deposition and the development of female breast tissue when it becomes too active in the male body. Clearly if we plan a high-dose cycle with an aromatizable steroid, anti-estrogens will be an important inclusion. However we cannot ignore the suggestion of using estrogen maintenance drugs only when they are necessary to combat visible side effects during mild to moderately dosed cycles,especially if bulk is the ultimate goal of the athlete.
 
Thursday's w/o Bicep/Tricep/Delt


superset #1
Standing db curls 8 reps 8 sets no rest
Lying tricep ext. 8 reps 8 sets no rest
BB shoulder press 8 reps 8 sets 1 minute rest
superset #2
Neutral Grip Chin-ups 15-20 reps 2 sets No rest
Close grip bench press 15-20 res 2 sets 1 minute rest
Bent-over lateral raises 15-20 resp 2 sets No rest

I was sweating like fricking crazy today! Im sweating just eating right now lol. Im liking this alpha-T2! 4th day in!
 
How's that other pre-work out supplement working for you? ;)

Actually I didn't notice much, I did have ADD as a kid. Im gonna try 2 tomorrow and see if I just wasn't taking enough cause generic adderall (amphetamine) gets me frickin' wired. But the generic is also 30mg per pill.
Oh and thanks for the pic moti-frickin-vation :box:
 
Thursday's w/o Bicep/Tricep/Delt


superset #1
Standing db curls 8 reps 8 sets no rest
Lying tricep ext. 8 reps 8 sets no rest
BB shoulder press 8 reps 8 sets 1 minute rest
superset #2
Neutral Grip Chin-ups 15-20 reps 2 sets No rest
Close grip bench press 15-20 res 2 sets 1 minute rest
Bent-over lateral raises 15-20 resp 2 sets No rest

I was sweating like fricking crazy today! Im sweating just eating right now lol. Im liking this alpha-T2! 4th day in!

Alpha-T2? what the Trens not enough for you? LOL.. im actually pulsing some Clen and Helios while i wait for the fireworks to go off

:32:
 
Alpha-T2? what the Trens not enough for you? LOL.. im actually pulsing some Clen and Helios while i wait for the fireworks to go off

:32:

Im eating alot more right now, and I snapped the cranks on my bike so I have to wait for Schwinn to send me the new set:rasp: So cardio is still good, but its not what it was, so Im thinking this Alpha-T2 will help offset fat gain. Im sweating right now lol. This may get old, being sweaty all the time can suck



ALPHA-T2: Targeted Fat Destroyer!

* TARGETED FAT DESTRUCTION
* INCREASE METABOLISM
* DECREASE FAT STORAGE
* INCREASE FAT BURNING
* NO JITTERS OR UNWANTED SIDE EFFECTS

Performance Enhancing Supplements is proud to announce the release of their first product, ALPHA-T2 (aT2).

With new fat burners coming to the supplement market just about every week, PES set out to develop a product that was better than the rest. We weren’t interested in making a product with 8 ingredients and unknown dosages with names you can’t even pronounce. We weren’t trying to bring something entirely novel to the market that has to prove itself. Instead we took three POWERFUL and well researched ingredients and combined them at the perfect doses. Synergy at its finest…

ALPHA-T2 is the only supplement you need to burn fat. Whether you are trying to lose 100lbs, 20lbs, or in the middle of a lean bulk, ALPHA-T2 belongs in your supplement regimen.

Let me explain…

Ever wonder how some people can eat and eat and eat and never gain a pound? You always hear the same ol’ “they just have a fast metabolism”, and you ask yourself why you were stuck with the slowest metabolism on earth!

Well quit worrying, there is a solution!

Metabolism is basically the rate at which food is digested, absorbed, and used in your body. It is the main determinant of your natural physique.

By increasing metabolism you can shift your body to break down fat for energy as its primary source as well as utilizing your protein and carbohydrates for building lean muscle.

Normally, carbohydrates and protein are broken down for energy before fat. By shifting your metabolism, more fat is broken down and less protein, which means MORE protein for building muscle!

How do we do all this? With an ingredient known as 3,3-T2 (3,3-diiodo-l-thryonine).

Strictly changing your diet and increasing your cardio can help you lose weight, but it also will cause you to lose muscle mass. You will still have your same slow metabolism; you will just be throwing your body into overdrive to try to drop the weight. But that weight consists of A LOT of muscle. BAD!

Thankfully with 3,3-T2 you can now increase your metabolism just by taking it as a supplement! This will allow you to lose more fat and lose less muscle when you combine it with diet and exercise.

3,3-T2 is a naturally occurring iodo-thryonine that will kick your thyroid gland into action! You may have heard of people with an underactive thyroid suffering from weight gain. Well the opposite is true as well.

People with an active thyroid often do not have trouble with weight gain. 3,3-T2 will optimize your thyroid activity to help you LOSE WEIGHT, and best off all, it doesn’t negatively affect your TSH the way other thyroid hormones will.

Beware of supplements containing 3,5 diiodo-l-thryonine; this hormone has been shown to significantly suppress your natural TSH function, which is BAD! (5) All the benefits you need are in 3,3-T2.

3,3-T2 stimulates the enzyme cytochrome c oxidase, an important enzyme in the Electron Transport Chain of cellular respiration. By doing this it will uncouple proteins.

What benefit is that?

Uncoupling these proteins does exactly what you’re looking to do when you’re trying to lose weight. It breaks down and burns fatty acids to be used as energy!
Also, 3,3-T2 acts to counteract lipid accumulation, reducing cholesterol level, and increase lipid metabolism without cardiotoxic effects! (6)

The positive effects of 3,3-T2 are endless! Kick your metabolism into gear and increase your fat burning capabilities!

Well that’s one ingredient down…let’s keep the ball rolling…

Alpha-T2 has the ability to actually TARGET fat burning. That’s right! How do we do this? By using an ingredient called Rauwolscine HCl, which is an Alpha-2 receptor antagonist (Alpha-2-receptor blocker).

Why do we want to block this receptor in the body? Normally, when this evil receptor is active (which it’s going to be, unless you’re taking alpha-T2), it actually blocks your body’s natural ability to break down and burn fat! By not allowing your body to burn fat, you’re going to STORE more fat.

By blocking the alpha-2-receptor, your body is now INCREASING fat burning, and DECREASING fat storage.

Don’t worry; I didn’t forget to tell you how this TARGETS fat loss in the areas YOU are holding weight. Alpha-2 receptors are abundant in certain areas of the body; it is in those areas that the receptors are active, causing you to store fat and inhibiting fat burning.

These areas include the abdomen, the “love handles”, the chest (don’t worry ladies, not the breasts), lower back, buttocks, and neck. So by taking alpha-T2, you are TARGETING FAT LOSS in these areas!

This powerful Alpha-2 receptor inhibitor, Rauwolscine HCl, is chemically related to yohimbine, a very common fat loss ingredient, but it is FAR SUPERIOR to regular yohimbine.

Both Rauwolscine and yohimbine are very powerful Alpha-2 receptor antagonists, as well as beta adrenergic receptor agonists. Now here is the catch; yohimbine also activates Alpha-1 receptors, which can block the breakdown of fats! (1,2,3) POINTLESS! Yohimbine is a thing of the past!

Yohimbine also can vastly increase heart rate and anxiety, but Rauwolscine will not! In fact, Rauwolscine is a 5-HT1A receptor agonist, which can LOWER blood pressure and DECREASE heart rate. (4)

These two reasons alone are why some people are weary of taking fat burners. Increasing your blood pressure and heart rate can be dangerous. So the fact that Rauwolscine will not have these effects is VERY attractive. Users will also notice an increase in libido and a decrease an appetite!

Last, and certainly not least, let me introduce Methyl-Synephrine.
You make have heard of synephrine being sold in other supplements with the idea that it is the best ephedrine replacement. This is a LIE.

Regular synephrine is CRAP. It is useless. You burn as much fat taking regular synephrine as you do reading this right now!

Methyl-synephrine on the other hand is AMAZING. I wouldn’t say it is a second hand replacement to ephedrine, I would say that it’s BETTER.

Ephedrine gives you jitters, anxiety, increased blood pressure, and increased heart rate. None of this is actually what you are looking for when burning fat; those are just the SIDE EFFECTS. With methyl-synephrine, you get NONE of these side effects.

So what’s the difference? Although they are structurally similar, ephedrine acts on the beta-2-adrenergic receptor, where-as methyl-synephrine works on the beta-3-adrenergic receptor. The beta-2 receptor is primarily used in cardiac control. Both the beta-2 and beta-3 receptors are used by the sympathetic nervous system to activate the breakdown of fat stores for energy and increasing your resting metabolism.

So get all the benefits of beta-adrenergic fat loss, without the side effects!

Now you have it. Three perfectly dosed, perfectly combined, extremely synergetic ingredients designed to make you

BURN FAT by INCREASING your metabolism, and TARGETING the areas you hold fat!

P.S. – We know that some people do have their favorite stimulating fat burner and are hard pressed to let go, so we are not telling you to do so. One of the great things about aT2 is that even though it is an iconic fat destroyer on its own, it is designed to be stackable with your favorite stimulant or cortisol blocker.

References:

1. Berlan M, Galitzky J, Riviere D, et al. Plasma catecholamine levels and lipid mobilization induced by yohimbine in obese and non-obese women. Int J Obes. 1991 May;15(5):305-315

2. Galitzky J, Taouis M, Berlan M, et al. Alpha 2-antagonist compounds and lipid mobilization: evidence for a lipid mobilizing effect of oral yohimbine in healthy male volunteers. Eur J Clin Invest. 1988 Dec;18(6):587-594

3. Flechtner-Mors M, Jenkinson CP, Alt A, et al. In vivo alpha(1)-adrenergic lipolytic activity in subcutaneous adipose tissue of obese subjects. J Pharmacol Exp Ther. 2002 Apr;301(1):229-233

4. Partial agonist properties of rauwolscine and yohimbine for the inhibition of adenylyl cyclase by recombinant human 5-HT1A receptors. Arthur et al (1993) Biochem Pharmacol 45;2337.

5. Effect of 3,5-diiodo-L-thyronine on thyroid stimulating hormone and growth hormone serum levels in hypothyroid rats. Moreno M, Lombardi A, Lombardi P, Goglia F, Lanni A. Dipartimento diChimica, Facoltà di Scienze, Università degli Studi di Salerno, Italy. Life Sci. 1998;62(26):2369-77.

6. Metabolic effects of thyroid hormone derivatives. Moreno M, de Lange P, Lombardi A, Silvestri E, Lanni A, Goglia F. Dipartimento di Scienze Biologiche ed Ambientali, Università degli Studi del Sannio, Via Port'Arsa, Benevento, Italy. Thyroid. 2008 Feb;18(2):239-53.

Alpha T2
 
^^^:goodpost:

Nice read through on the Alpha-T2 brosenhauf!!!:deal:
 
Friday, oh glorious Friday!

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Some Asian action...
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I wonder what she is going to do with that bottle?
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Well tomorrow is the 1st day of my new program, kinda stoked here now. I took friday,saturday and today off, so I should be pretty fresh tomorrow.
 
Monday: Chest/Back/Traps/Rear Delts
Superset#1 Incline BB or DB press 6-8 reps 3 sets No Rest
Pull-up to front(wide grip) 6-8 reps 3 sets 1 Minute Rest
Superset#2
Chest Dips 6-8 reps 3 sets No Rest
Close Grip Chins 6-8 reps 3 sets 1 minute
Superset#3
Cable Incline Flies 10-12 reps 3 sets
Low Pulley Rows 10-12 reps 3 sets No Rest
Dumbbell Pullovers 10-12 reps 2 sets 1 Minute Rest
Superset#4
Stiff-arm Pull downs 15-20 reps 2 sets No Rest
Bent Arm Bent-over Rows 15-20 reps 2 sets No Rest
Upright Rows 15-20 reps 2 sets No Rest


BRUTAL!!!
 

You call him a slacker and this is what he goes and does? :

Monday: Chest/Back/Traps/Rear Delts
Superset#1 Incline BB or DB press 6-8 reps 3 sets No Rest
Pull-up to front(wide grip) 6-8 reps 3 sets 1 Minute Rest
Superset#2
Chest Dips 6-8 reps 3 sets No Rest
Close Grip Chins 6-8 reps 3 sets 1 minute
Superset#3
Cable Incline Flies 10-12 reps 3 sets
Low Pulley Rows 10-12 reps 3 sets No Rest
Dumbbell Pullovers 10-12 reps 2 sets 1 Minute Rest
Superset#4
Stiff-arm Pull downs 15-20 reps 2 sets No Rest
Bent Arm Bent-over Rows 15-20 reps 2 sets No Rest
Upright Rows 15-20 reps 2 sets No Rest


BRUTAL!!!
wow that was brutal

what kind of workout would you have if someone called you a punk? lol jk jk jk

Good workout. Would have killed me
 

WTF? Ed is banned now?!!!

You call him a slacker and this is what he goes and does? :


wow that was brutal

what kind of workout would you have if someone called you a punk? lol jk jk jk

Good workout. Would have killed me

Thanks man, that $hit was sick! I had to lay down for about 30minutes after that workout, I knew better than to try and down a 16oz shake at that point. The last set(super set # 4) took the wind out of me, I was just gassed at that point, no rest between 3 sets and you repeat that 2x with no rest.
 
Thought I would mention that I upped my dose of Aplha T-2 on Saturday to 4 caps a day. 2 upon waking and 2 later on around 2:30-3:00pm about 30-60 minutes before a meal. I not noticing much of a stimulant effect from this stuff, I get a little something which would have to be from the methyl-synephrine when I dose 2 caps, 1 cap no stim effect.
I change my shirt 2-3 times a day because of the sweating right now, which kinda sucks. My temp feels higher on this stuff all though out the day, Ive been eating more and more lately and should be putting on some fat at this point. For the past 2 days a few of the veins on my lower abs have disappeared, so I have put on some fat. My bike is broke and out of commission for the foreseeable future, so cardio is suffering, but I am still walking on my treadmill almost every evening and doing fasted walks on off days. I can't/don't push myself on a tredmill like I can a bike, partly because I don't enjoy being on a tredmill in the first place. Sorry for the rant, I want my dam bike fixed! I meant to stay lean on this cycle dammit, Im still lean but now as lean as I was.
 
Lately Ive been playing with some cycle ideas lol. Im thinking of ending the tren after this week and saving the other 10ml vial I have for next summer. Im thinking of starting up decca and going into an all out bulker. I have 30ml of decca 250 so I have more than enough to get a good run of it. I have 10ml of sust325 and 20ml of sust450 and 30+ml of Test E, dbol, superdrone, so I have enough Test and orals as well. I was thinking of using 375-500mg decca ew for 12+ weeks and 600-900mg Test and at some point hit up some of those orals.

I want to do an all out cutter next spring/summer with Test/Tren E/T3/Clen/ and some type of oral(maybe var), so that is partly my motivation for ending the tren when this vial runs out(because I'll have 10ml for next year lol). I really want to cut on tren/test, I always lose way too much muscle when I cut so these things should help. I was 225-230lbs last winter, when I cut I lost alot muscle and fat and ended up under 200lbs(and didn't even meet my cutting goals).

Part of my problem has been that I start bulking in the fall and bulk until Im uncomfortably fat and instantly go into cut mode, my frame hasn't acclimated to all this new weight and I go ahead and start cutting calories anyways. So the first to go is the new fat/muscle I gained that season, I only end up 5-10lbs heavier than the year before(but leaner too).
This is why I transitioned into a lean bulker over 6 weeks ago, I wanted to start bulking sooner so that I could walk around with the new weight longer before going into cut mode, I feel I will lose less muscle this way(at least I hope). This bulk will be more than 2 months longer than my normal bulks last, I want to be chemically enhanced during this time haha. So I guess I may blast and cruise for the next 8 months
 
Lately Ive been playing with some cycle ideas lol. Im thinking of ending the tren after this week and saving the other 10ml vial I have for next summer. Im thinking of starting up decca and going into an all out bulker. I have 30ml of decca 250 so I have more than enough to get a good run of it. I have 10ml of sust325 and 20ml of sust450 and 30+ml of Test E, dbol, superdrone, so I have enough Test and orals as well. I was thinking of using 375-500mg decca ew for 12+ weeks and 600-900mg Test and at some point hit up some of those orals.
I want to do an all out cutter next spring/summer with Test/Tren E/T3/Clen/ and some type of oral(maybe var), so that is partly my motivation for ending the tren when this vial runs out(because I'll have 10ml for next year lol). I really want to cut on tren/test, I always lose way too much muscle when I cut so these things should help. I was 225-230lbs last winter, when I cut I lost alot muscle and fat and ended up under 200lbs(and didn't even meet my cutting goals).

Part of my problem has been that I start bulking in the fall and bulk until Im uncomfortably fat and instantly go into cut mode, my frame hasn't acclimated to all this new weight and I go ahead and start cutting calories anyways. So the first to go is the new fat/muscle I gained that season, I only end up 5-10lbs heavier than the year before(but leaner too).

This is why I transitioned into a lean bulker over 6 weeks ago, I wanted to start bulking sooner so that I could walk around with the new weight longer before going into cut mode, I feel I will lose less muscle this way(at least I hope). This bulk will be more than 2 months longer than my normal bulks last, I want to be chemically enhanced during this time haha. So I guess I may blast and cruise for the next 8 months

It's good when we learn from our past mistakes. You know what you've been doing wrong with the cutting phases, so now is the time to do something different.

Ronnie Coleman has a plaque on his home-gym wall that reads:

If you do what you've always done, you will get what you always got!!

SO TRUE!!!

I also like this: From tim1985-------"I was thinking of using 375-500mg decca ew for 12+ weeks and 600-900mg Test and at some point hit up some of those orals!!" :32:


Thunder-Likey!!!!:hammer:
 
It's good when we learn from our past mistakes. You know what you've been doing wrong with the cutting phases, so now is the time to do something different.

Ronnie Coleman has a plaque on his home-gym wall that reads:

If you do what you've always done, you will get what you always got!!

SO TRUE!!!

I also like this: From tim1985-------"I was thinking of using 375-500mg decca ew for 12+ weeks and 600-900mg Test and at some point hit up some of those orals!!" :32:


Thunder-Likey!!!!:hammer:

Haha, yeah man I try and learn form my mistakes. I do a little better each year, but this year is gonna be different. Blood work is a must on this one though.
 
I don't know, I checked his last few posts and nothing out of the ordinary from him. His posts may have been deleted though

It doesn't look like he's banned. It still says, "Registered User" under his name. I know he was going through something when a close friend of his was critically injured in an accident.
 
Tuesday: Hamstrings/Calves /Abs
Superset#1
Hamstring Leg Press substituted with wide stance squats 6-8 reps 4 sets No Rest
Hanging Leg Raises 10-15 reps 3 sets 1 Minute Rest
Superset#2
DB Stiff-Legged Deadlifts 10-12 reps 4 sets
Bicycle Crunches 15 reps 3 sets 1 Minute Rest
Superset#4
Donkey Calf Raises 10-15 reps 4 sets No Rest
Standing Calf Raises 8 reps 3 sets 30 seconds Rest
 
Tuesday: Hamstrings/Calves /Abs
Superset#1
Hamstring Leg Press substituted with wide stance squats 6-8 reps 4 sets No Rest
Hanging Leg Raises 10-15 reps 3 sets 1 Minute Rest
Superset#2
DB Stiff-Legged Deadlifts 10-12 reps 4 sets
Bicycle Crunches 15 reps 3 sets 1 Minute Rest
Superset#4
Donkey Calf Raises 10-15 reps 4 sets No Rest
Standing Calf Raises 8 reps 3 sets 30 seconds Rest

You're a freakin' machine! Not just this workout, but most of them.
 
You're a freakin' machine! Not just this workout, but most of them.

Thanks man, I really do appreciate that!

So heres whats going on right now, my chest and back are fuct(in a really good way)and my hammy's are already sore(i just worked them out this morning haha. I don't remember the last time my chest was this tore up, Im really liking this.

I have my bike in the shop, so cardio will be back in gear fully by tomorrow.

Yesterday I shot 500mg Decca! Front load FTW!
 
Thanks man, I really do appreciate that!

So heres whats going on right now, my chest and back are fuct(in a really good way)and my hammy's are already sore(i just worked them out this morning haha. I don't remember the last time my chest was this tore up, Im really liking this.

I have my bike in the shop, so cardio will be back in gear fully by tomorrow.

Yesterday I shot 500mg Decca! Front load FTW!

With you working out at that pace, do you really need additional cardio? lol
 
With you working out at that pace, do you really need additional cardio? lol

I think I do, its healthy, and I have been eating alot more lately. I have noticed some loss in definition(ie: some of the veins in my abs are no longer visible and the veins in my biceps only pop sometimes, not all the time like they were).
 
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