WIN A X FACTOR T SHIRT or EVEN A CYCLE!!!
- Thread starter jjohn
- Start date
Cellardude
Well-known member
Here we go for another contest: What is the best recipe you have made that would follow with X Factor's effects recommendations? Best idea will win a T-Shirt
Hint: Coconut and MCT's are highly recommended as fat sources
whole wheat pasta on slightly grilled chicken on marinara sauce yum.
jjohn
Registered User
What's marinara sauce?? Sorry I look stupid lol
Cellardude
Well-known member
I make my own marinara sauce, not authentic really without olive oil for it to be marinara sauce but I buy tomato's and the heavy tomato paste and throw in some salt peper and other goodies. Doesn't taste like authentic italian cooking or sauce, but it sure is healthy 
The chicken is also flavored with a bunch of stuff like soy sauce, chopped up ginger corn oil (sorry no coconut oil) and flavored to perfection.
If I get sick of the marinara sauce, Ill just use my own secret bottle of franks red hots and drench the chicken in it and eat it down.
Invalid Link Removed
thats not my own picture but thats how my meal usually looks with an extra dish of chicken on the side or ill mix it into the pasta. really good stuff.
The chicken is also flavored with a bunch of stuff like soy sauce, chopped up ginger corn oil (sorry no coconut oil) and flavored to perfection.
If I get sick of the marinara sauce, Ill just use my own secret bottle of franks red hots and drench the chicken in it and eat it down.
Invalid Link Removed
thats not my own picture but thats how my meal usually looks with an extra dish of chicken on the side or ill mix it into the pasta. really good stuff.
Cellardude
Well-known member
Man this looks good. Don't forget this small contest will last until tonight
it taste pretty good too. If I get the chance, Ill ask my parents on how they flavor the chicken. My parents are all about flavoring. I found that slow heat cooked with fresh chicken has a BIG impact on the way it taste. for some reason unfresh chicken with big heat causes the chicken to get a bit hard while fresh slow pan fried or whatever its called yeilds chicken that literally melts in your mouth with the pasta.
EasyEJL
Never enough
8oz of shrimp
1/4 cup coconut oil
1/2 red onion
1/2 cup mixed chopped red, yellow and green peppers
1 cups spinach
jamaican curry powder
Invalid Link Removed
1 cup brown rice cooked
optional adds (any 1 is ok)
1/2 cup chopped mango
1/2 cup chopped pineapple
1/2 cup green plantains
heat coconut oil on med high. Add onions and peppers stir fry rapidly for 1-2 minutes. If using fruit, add at this point. Add shrimp and sprinkle curry powder, shake pan every 30 sec, flip shrimp after 3 minutes and sprinkle all with curry powder again. contiue shaking pan every 30 sec for another 3 minutes. Add spinach, and stir, continuing to cook until spinach is softened. Serve over brown rice
1/4 cup coconut oil
1/2 red onion
1/2 cup mixed chopped red, yellow and green peppers
1 cups spinach
jamaican curry powder
Invalid Link Removed
1 cup brown rice cooked
optional adds (any 1 is ok)
1/2 cup chopped mango
1/2 cup chopped pineapple
1/2 cup green plantains
heat coconut oil on med high. Add onions and peppers stir fry rapidly for 1-2 minutes. If using fruit, add at this point. Add shrimp and sprinkle curry powder, shake pan every 30 sec, flip shrimp after 3 minutes and sprinkle all with curry powder again. contiue shaking pan every 30 sec for another 3 minutes. Add spinach, and stir, continuing to cook until spinach is softened. Serve over brown rice
TripDog
Bananas
Easy is like the new pimp :clap2: congrats E@$Y..
jjohn
Registered User
Lucky bastard. I live in Canada, but I still am always a T junkie lol
grila jujitsu
Well-known member
bumpity hope i win the bottle bump!
jjohn
Registered User
Me too. I'm stealing all the winner's t-shirts Muhahahahah
jjohn
Registered User
Easy IS justice
jjohn
Registered User
Ah this is why I woke up under my neighbor's deck in the snow! Thanks Easy! :toofunny:
TripDog
Bananas
You must spread some Reputation around before giving it to EasyEJL again.
TripDog
Bananas
Thank-you,Thank-you.....I'd like to thank all the little people behind the scenes that made this possible.....you know who you are 
Dancebot 2000
Active member
Wait, what's going on with this thread? How do I win a Cycle of X-factor? I'd really like to win a cycle of x-factor.
jjohn
Registered User
Answer the first question
Winners will be in a poll to see if you win the cycle!
jjohn
Registered User
I still like my inflammation definition
I too like it, they're all good definitions!
Dancebot 2000
Active member
I have a good idea, and I might do it for fun, but going up against TripDog in a poll based contest is kind of like trying to staple diarrhea to wall; completely futile. :lol:
DerekAdams1
Banned
I want to try some x-factor.
-Derek
-Derek
EasyEJL
Never enough
Well, this is the contest, so jjohn will be picking it dancebot, so you have a chance vs trip
Dancebot 2000
Active member
Well, this is the contest, so jjohn will be picking it dancebot, so you have a chance vs trip
He said earlier in a post just above here that it was going to be a poll. Either way I might do it because I have a fun idea, it's just whether or not I find the time to write it.
jjohn
Registered User
TripDog
Bananas
It's ON baby!!! Bwahahahahahahah :twisted:
Cellardude
Well-known member
king1033
Active member
INflimation - It occurs when you have a REAL WORKOUT :thumbsup:
jjohn
Registered User
True dat!
EasyEJL
Never enough
and its ouflamation when you have a burrito with too many jalapenos
nolinenowait
Banned
Inflammation (Latin, inflammatio, to set on fire) is the complex biological response of vascular tissues to harmful stimuli, such as pathogens, damaged cells, or irritants. It is a protective attempt by the organism to remove the injurious stimuli as well as initiate the healing process for the tissue. Inflammation is not a synonym for infection. Even in cases where inflammation is caused by infection it is incorrect to use the terms as synonyms: infection is caused by an exogenous pathogen, while inflammation is the response of the organism to the pathogen. -taken from wikipedia
In the absence of inflammation, wounds and infections would never heal and progressive destruction of the tissue would compromise the survival of the organism. However, inflammation which runs unchecked can also lead to a host of diseases, such as hay fever, atherosclerosis, and rheumatoid arthritis. It is for this reason that inflammation is normally tightly regulated by the body.
Muscle Inflammation is Necessary for Muscle Hypertrophy
Wolverine from the movie ‘X-men’ is quite possibly the ultimate soldier. He has adamantium claws that can turn anyone that pisses him off into chop suey, an unbreakable skeleton, and a bad attitude to match. His primary mutant power is an accelerated ‘healing factor’ that regenerates damaged or destroyed areas of his body far beyond the capabilities of an ordinary human and renders him immune to physical damage. Wouldn’t it be nice if we had an accelerated ‘healing factor’ which would allow us to go to the gym and have an intense workout with some heavy negatives and not be sore? That sore feeling you get after an intense workout is because you damaged skeletal muscle and your body’s own ‘healing factors’ are regenerating muscle fibers. Believe it or not your immune system plays an important part in the role of building muscle; anything that compromises its function can impede muscle growth.
The Role of Macrophages in Muscle Repair and Growth
When resistance exercise involves heavy eccentric muscular contractions (lowering the weight), it is associated with overloading skeletal muscle —that is, the force requirement of the muscle exceeds what it is accustomed too—and results in injury to skeletal muscle. It has traditionally been felt that the events following the initial injury, including inflammation, are necessary for optimal repair and growth of new muscle. Exercise-induced muscle damage stimulates an acute-phase inflammatory response, which includes infiltration into skeletal muscle by macrophages1. Macrophages (Greek: ‘big eaters’) are cells within the tissues that originate from specific white blood cells. One important role of macrophage is the removal of damaged tissue by heavy resistance exercise. Repairing damaged muscle cells is an important function of macrophages in the early stages of muscle damage and inflammation. This inflammatory response coincides with muscle repair, regeneration, and growth, which involves the activation of satellite cells (satellite cell activation is essential for muscle hypertrophy). The number of macrophages per square millimeter of muscle tissue is increased after heavy eccentric exercise compared to concentric exercise (lifting the weight) which may play an important role as to why eccentric contractions produce greater muscle hypertrophy2. Macrophages are also able to promote muscle growth and repair. In vitro studies (test tubes studies) show macrophages can increase muscle cell growth factors3,4, which indicates a role for macrophage-derived factors in muscle growth.
What happens if you suppress your ‘Healing Factor’?
The healing process that occurs during rest is an important adaptation to building muscle. You may be sore as hell after a heavy leg workout but that means your shocking your muscles into new growth. The inflammatory process although painful for a bodybuilder is an essential part of the muscle growth process. For instance, mediators such as IGF-1 are stimulated by the release of inflammatory cells5. So what happens if you suppress macrophages after injury? Just like Wolverine in the movies, if you lose your ‘healing factors’ you’re screwed in terms of building muscle. Researchers injected mice with an antibody that reduced macrophage concentrations after a couple of days of muscle overloading (86% reduction in macrophage concentration) and found that muscle fiber repair and growth was significantly impaired after muscle overload6. Macrophage-depletion also reduced muscle regeneration and prevented growth of muscle fibers that normally occurs with muscle overload. These findings suggest that macrophage first invades injured muscle serves to remove cellular debris, after which the subsequent invasive population participates in repair, regeneration and growth.
A strain of mice was recently genetically engineered to test the hypothesis of macrophage invasion to the site of injury is important for muscle regeneration and growth. A strain of mice was bred to be deficient in a protein called MCP-1 (Monocyte chemotactic protein-1), a potent activator for macrophage invasion and inflammation. Researchers damaged muscle fibers of these mice and compared them to normal mice; the mice that had impaired muscle macrophage activity due to a MCP-1 deficiency had impaired muscle regeneration and growth. The researchers speculated that the impaired muscle regeneration was due to macrophages not being able to repair the damaged muscle. Remember, muscle growth takes place during the recovery phase, lifting weights only serves as the stimulus. The research suggests that unless the damaged muscle fiber becomes invaded by macrophages and other repair mediators, it becomes stagnant and muscle repair is halted and the muscle tissue stays damaged. Interestingly, macrophages can increase nitric oxide which expands blood vessels and open up the muscle tissue to blood flow which allows for more macrophages to repair damaged muscle tissue10. NSAIDS Suppress Muscle Growth
You may feel the urge to take an Aleve or Ibuprofen tablet after an intense workout to reduce muscle soreness or you may suffer from a chronic knee or elbow injury from years of heavy lifting but only take NSAIDS when absolutely necessary. According to a new study in Medicine & Science in Sports & Exercise, taking ibuprofen can inhibit muscle hypertrophy. In the study, rats had surgeries performed in which their leg muscles are chronically overloaded to cause muscle hypertrophy. One group of rats received ibuprofen while the other group received nothing. At the end of the study, rats that were administered ibuprofen had a whopping 50% reduction in muscle hypertrophy8. Another study reported that when Non Steroidal Anti-Inflammatory Drugs (NSAIDS) were examined after muscle injury, not only was satellite cell (muscle stem cells) activity in muscle inhibited by their was a increase in myostatin (inhibits muscle growth) production. Examples of NSAIDs include Aspirin, indomethacin (Indocin), ibuprofen (Motrin), naproxen (Naprosyn), piroxicam (Feldene), and nabumetone (Relafen). Recent research suggests that regular use of NSAIDS or COX-2 inhibitors such as Celebrex or Vioxx can significantly reduce muscle hypertrophy by reducing expression of the enzyme COX-2. In conjunction with other research, the COX-2 pathway serves as an important mediator of the inflammation response after exercise serving to repair damaged tissue and is an essential for muscle hypertrophy to occur.
More recent findings have shown cyclooxygenase-2 (COX-2) oral administration of COX-2 inhibitors can slow muscle regeneration and reduce muscle growth after acute injury of muscle7. Interestingly, mice that are genetically engineered to be COX-2 deficient showed less macrophage invasion of injured muscle during regeneration7, which may indicate that macrophages normally promote muscle cell proliferation and muscle regeneration following injury. Alternatively, COX-2 may have a direct effect on muscle cells to affect muscle repair. Additionally, it is well known that in order to build muscle there must be an increase in muscle protein synthesis rates. Studies have reported that if the maximal dosage of ibuprofen or acetaminophen is taken before exercise that protein synthesis rates are blunted9. In that study, subjects performed 10 sets of eccentric exercise performed at 120% of a 1-RM, subjects that received a placebo had average of 76% increase in protein synthesis rates, whereas the subjects whom received either ibuprofen or acetaminophen had no increase in protein synthesis.
In conclusion, don’t take any NSAIDS unless it’s absolutely necessary for alleviating pain. NSAIDS reduce muscle protein synthesis and inhibit muscle hypertrophy. Muscle inflammation and repair is an essential part of hypertrophy process, and although you may be sore as hell, the benefits you will reap from the pain are bigger and stronger muscles.-Trainwiser.com
In the absence of inflammation, wounds and infections would never heal and progressive destruction of the tissue would compromise the survival of the organism. However, inflammation which runs unchecked can also lead to a host of diseases, such as hay fever, atherosclerosis, and rheumatoid arthritis. It is for this reason that inflammation is normally tightly regulated by the body.
Muscle Inflammation is Necessary for Muscle Hypertrophy
Wolverine from the movie ‘X-men’ is quite possibly the ultimate soldier. He has adamantium claws that can turn anyone that pisses him off into chop suey, an unbreakable skeleton, and a bad attitude to match. His primary mutant power is an accelerated ‘healing factor’ that regenerates damaged or destroyed areas of his body far beyond the capabilities of an ordinary human and renders him immune to physical damage. Wouldn’t it be nice if we had an accelerated ‘healing factor’ which would allow us to go to the gym and have an intense workout with some heavy negatives and not be sore? That sore feeling you get after an intense workout is because you damaged skeletal muscle and your body’s own ‘healing factors’ are regenerating muscle fibers. Believe it or not your immune system plays an important part in the role of building muscle; anything that compromises its function can impede muscle growth.
The Role of Macrophages in Muscle Repair and Growth
When resistance exercise involves heavy eccentric muscular contractions (lowering the weight), it is associated with overloading skeletal muscle —that is, the force requirement of the muscle exceeds what it is accustomed too—and results in injury to skeletal muscle. It has traditionally been felt that the events following the initial injury, including inflammation, are necessary for optimal repair and growth of new muscle. Exercise-induced muscle damage stimulates an acute-phase inflammatory response, which includes infiltration into skeletal muscle by macrophages1. Macrophages (Greek: ‘big eaters’) are cells within the tissues that originate from specific white blood cells. One important role of macrophage is the removal of damaged tissue by heavy resistance exercise. Repairing damaged muscle cells is an important function of macrophages in the early stages of muscle damage and inflammation. This inflammatory response coincides with muscle repair, regeneration, and growth, which involves the activation of satellite cells (satellite cell activation is essential for muscle hypertrophy). The number of macrophages per square millimeter of muscle tissue is increased after heavy eccentric exercise compared to concentric exercise (lifting the weight) which may play an important role as to why eccentric contractions produce greater muscle hypertrophy2. Macrophages are also able to promote muscle growth and repair. In vitro studies (test tubes studies) show macrophages can increase muscle cell growth factors3,4, which indicates a role for macrophage-derived factors in muscle growth.
What happens if you suppress your ‘Healing Factor’?
The healing process that occurs during rest is an important adaptation to building muscle. You may be sore as hell after a heavy leg workout but that means your shocking your muscles into new growth. The inflammatory process although painful for a bodybuilder is an essential part of the muscle growth process. For instance, mediators such as IGF-1 are stimulated by the release of inflammatory cells5. So what happens if you suppress macrophages after injury? Just like Wolverine in the movies, if you lose your ‘healing factors’ you’re screwed in terms of building muscle. Researchers injected mice with an antibody that reduced macrophage concentrations after a couple of days of muscle overloading (86% reduction in macrophage concentration) and found that muscle fiber repair and growth was significantly impaired after muscle overload6. Macrophage-depletion also reduced muscle regeneration and prevented growth of muscle fibers that normally occurs with muscle overload. These findings suggest that macrophage first invades injured muscle serves to remove cellular debris, after which the subsequent invasive population participates in repair, regeneration and growth.
A strain of mice was recently genetically engineered to test the hypothesis of macrophage invasion to the site of injury is important for muscle regeneration and growth. A strain of mice was bred to be deficient in a protein called MCP-1 (Monocyte chemotactic protein-1), a potent activator for macrophage invasion and inflammation. Researchers damaged muscle fibers of these mice and compared them to normal mice; the mice that had impaired muscle macrophage activity due to a MCP-1 deficiency had impaired muscle regeneration and growth. The researchers speculated that the impaired muscle regeneration was due to macrophages not being able to repair the damaged muscle. Remember, muscle growth takes place during the recovery phase, lifting weights only serves as the stimulus. The research suggests that unless the damaged muscle fiber becomes invaded by macrophages and other repair mediators, it becomes stagnant and muscle repair is halted and the muscle tissue stays damaged. Interestingly, macrophages can increase nitric oxide which expands blood vessels and open up the muscle tissue to blood flow which allows for more macrophages to repair damaged muscle tissue10. NSAIDS Suppress Muscle Growth
You may feel the urge to take an Aleve or Ibuprofen tablet after an intense workout to reduce muscle soreness or you may suffer from a chronic knee or elbow injury from years of heavy lifting but only take NSAIDS when absolutely necessary. According to a new study in Medicine & Science in Sports & Exercise, taking ibuprofen can inhibit muscle hypertrophy. In the study, rats had surgeries performed in which their leg muscles are chronically overloaded to cause muscle hypertrophy. One group of rats received ibuprofen while the other group received nothing. At the end of the study, rats that were administered ibuprofen had a whopping 50% reduction in muscle hypertrophy8. Another study reported that when Non Steroidal Anti-Inflammatory Drugs (NSAIDS) were examined after muscle injury, not only was satellite cell (muscle stem cells) activity in muscle inhibited by their was a increase in myostatin (inhibits muscle growth) production. Examples of NSAIDs include Aspirin, indomethacin (Indocin), ibuprofen (Motrin), naproxen (Naprosyn), piroxicam (Feldene), and nabumetone (Relafen). Recent research suggests that regular use of NSAIDS or COX-2 inhibitors such as Celebrex or Vioxx can significantly reduce muscle hypertrophy by reducing expression of the enzyme COX-2. In conjunction with other research, the COX-2 pathway serves as an important mediator of the inflammation response after exercise serving to repair damaged tissue and is an essential for muscle hypertrophy to occur.
More recent findings have shown cyclooxygenase-2 (COX-2) oral administration of COX-2 inhibitors can slow muscle regeneration and reduce muscle growth after acute injury of muscle7. Interestingly, mice that are genetically engineered to be COX-2 deficient showed less macrophage invasion of injured muscle during regeneration7, which may indicate that macrophages normally promote muscle cell proliferation and muscle regeneration following injury. Alternatively, COX-2 may have a direct effect on muscle cells to affect muscle repair. Additionally, it is well known that in order to build muscle there must be an increase in muscle protein synthesis rates. Studies have reported that if the maximal dosage of ibuprofen or acetaminophen is taken before exercise that protein synthesis rates are blunted9. In that study, subjects performed 10 sets of eccentric exercise performed at 120% of a 1-RM, subjects that received a placebo had average of 76% increase in protein synthesis rates, whereas the subjects whom received either ibuprofen or acetaminophen had no increase in protein synthesis.
In conclusion, don’t take any NSAIDS unless it’s absolutely necessary for alleviating pain. NSAIDS reduce muscle protein synthesis and inhibit muscle hypertrophy. Muscle inflammation and repair is an essential part of hypertrophy process, and although you may be sore as hell, the benefits you will reap from the pain are bigger and stronger muscles.-Trainwiser.com
Dancebot 2000
Active member
DB2000: Inflimation is bad. It's hurts. There is nothing good about it. I hate inflimation.
Jjohn: But Dance, inflimation is needed for effective muscle growth.
DB2000: Yeah right.
Jjohn: Seriously. It's part of the process called hypertrophy.
DB2000: I once won the hyper trophy for being the most hyperactive kid in the second grade.
Jjohn: No, not the hyper trophy. Hypertrophy. It means muscle growth, and inflimation is one of the catalysts to set this in motion.
DB2000: Pffff.... That's the craziest thing I've ever heard. Why would you say something so crazy? You big crazy. You and I and everyone on this forum knows that inflimation is a response to harmful stimuli, like damaged cells and pathogens. It can be a result of increased free radicals, leakage of blood and other stuff too. It is uncomfortable and stupid. In conclusion, inflimation sucks.
Jjohn: That's not entirely true. Exercise causes musclar damage, which induces an "acute-phase inflammatory response", which in turn causes skeletal muscle to be penetrated by macrophages.
DB2000: Macro-whatits?
Jjohn: Macrophages. These are cells that originate within white blood cells. One of their primary functions is to remove damaged tissues, like the ones caused by training. You see, hypertrophy is caused by the damaging and then repairing of muscles. Inflimation is the initiating process by which this regeneration occurs.
DB2000: Regeneration! Like Wolverine?
Jjohn: Uh...sure. This inflimation also stimulates the activation of satallite cells, and the release of IGF-1, an important anabolic hormone and prostaglandins, which regulate protien synthisis. All of these factors contribute to muscle growth.
DB2000:....Go on.
Jjohn: Um, that's pretty much it, short of me going into far too much detail.
DB2000: Wow Jjohn, You're a good rep. Thanks for your help.
Jjohn: No problem. And remember, you should embrace inflimation, and know that even though it's uncomfortable, it's a sign that your getting bigger and looking great!
DB2000:...Are you coming on to me?
Jjohn: What?
DB2000: What's all this about me "looking great"? Are you hitting on me?
Jjohn: What? No! That's not what I'm saying. This is a misunderstanding. I'm French.
DB2000: Tsk-tsk, Jjohn. You represent Molecular Nutrition, a professional company. What would Bill Llwenlyn think if he knew you were out here hitting on potential customers, telling them they look good?
Bill Llwenlyn: I also think you look good, Dance.
DB2000: (pause) Okaaay....This is awkward....how 'bout you give me a t-shirt, and I'll get out of your hair. Thanks.
DB2000
Jjohn: But Dance, inflimation is needed for effective muscle growth.
DB2000: Yeah right.
Jjohn: Seriously. It's part of the process called hypertrophy.
DB2000: I once won the hyper trophy for being the most hyperactive kid in the second grade.
Jjohn: No, not the hyper trophy. Hypertrophy. It means muscle growth, and inflimation is one of the catalysts to set this in motion.
DB2000: Pffff.... That's the craziest thing I've ever heard. Why would you say something so crazy? You big crazy. You and I and everyone on this forum knows that inflimation is a response to harmful stimuli, like damaged cells and pathogens. It can be a result of increased free radicals, leakage of blood and other stuff too. It is uncomfortable and stupid. In conclusion, inflimation sucks.
Jjohn: That's not entirely true. Exercise causes musclar damage, which induces an "acute-phase inflammatory response", which in turn causes skeletal muscle to be penetrated by macrophages.
DB2000: Macro-whatits?
Jjohn: Macrophages. These are cells that originate within white blood cells. One of their primary functions is to remove damaged tissues, like the ones caused by training. You see, hypertrophy is caused by the damaging and then repairing of muscles. Inflimation is the initiating process by which this regeneration occurs.
DB2000: Regeneration! Like Wolverine?
Jjohn: Uh...sure. This inflimation also stimulates the activation of satallite cells, and the release of IGF-1, an important anabolic hormone and prostaglandins, which regulate protien synthisis. All of these factors contribute to muscle growth.
DB2000:....Go on.
Jjohn: Um, that's pretty much it, short of me going into far too much detail.
DB2000: Wow Jjohn, You're a good rep. Thanks for your help.
Jjohn: No problem. And remember, you should embrace inflimation, and know that even though it's uncomfortable, it's a sign that your getting bigger and looking great!
DB2000:...Are you coming on to me?
Jjohn: What?
DB2000: What's all this about me "looking great"? Are you hitting on me?
Jjohn: What? No! That's not what I'm saying. This is a misunderstanding. I'm French.
DB2000: Tsk-tsk, Jjohn. You represent Molecular Nutrition, a professional company. What would Bill Llwenlyn think if he knew you were out here hitting on potential customers, telling them they look good?
Bill Llwenlyn: I also think you look good, Dance.
DB2000: (pause) Okaaay....This is awkward....how 'bout you give me a t-shirt, and I'll get out of your hair. Thanks.
DB2000
Dancebot 2000
Active member
Thanks Jjohn. I'm glad I found the time to write it. It was fun.
jjohn
Registered User
Cool anyone else?
