the atd is what killed your libido. unless you took blood tests to see #'s, I doubt you were shut down.
I would recommend taking it in the morning and maybe afternoon. I would advice not taking it at night when melatonin levels are high, as it may interfere with your body's natural sleep paterns.
Like 10,20,30,40 basically. No ramp down used with the short cycle length.and how much SD did u run that you ran a PCT like that?
Yeah, divided doses on the DHEA. 100mg morning and 100mg noon. On days when I only use 100mg, I'll take it all in the morning. Never use DHEA too close to bed or after 6pm or it might delay recovery instead of helping with it. It's a great idea to use 3mg melatonin/night too when on DHEA. These two compounds balance one another. I start with 2-3 caps on the fen and add another cap every week I stay on it.Dr. D, Do you take those 200mg of DHEA all at once or do you divide the doses through the day? and how much Fen do you take?
I've used up to 500mg no problem. On paper it looks like the conversion rate of DHEA to 5AD would be high, but it must not be in real life because I have done 5AD and can tell the difference. I've read studies of some using up to 1600mg/d with no endocrine disruption, but that's just too much IMO! Stick to low doses (200mg or less) just to play it safe. It doesn't appear to cause shutdown in anyone I've ever talked to at that dose. Recovery is faster in fact and normal PCT sides are improved. DHEA is the most abundant steroid in the human body (male and female). If it caused suppression, then PCT would never be possible because it's always there normally! After a cycle, it's levels are too low and cortisol is too high. If you use DHEA in PCT, you'll keep more gains and your system bounces back more efficiently. This is my honest observation, regardless what else you may have heard about it.By the way, at what dose do you think dhea causes shutdown or problems?
hahaha, don't believe the hype. :type:Just an FYI on this whole SD "sticking with you" thing we're talking about.. I ended my SD/FiniGenx cycle over a week ago and I can't even tell you how much my bench-press has gone up since then without sounding ridiculous.. I'm just waiting for breasts now...
Dr. D,hahaha, don't believe the hype. :type:
It is possible that estrogen rebound caused it to happen. In the mean time I would advise the use of a SERM to avoid the same thing happening again while off cycle. Raloxifene is the most predominate gyno buster out there IMO and it is virtually non-toxic, even in large quantities. The same cannot be said for tamoxifen citrate. If you are having trouble finding it, PM me.Dr. D,
Have you found any evidence that this SD "delayed gyno" couldn't happen after PCT? When you show me that evidence, I'll stop claiming I got gyno/psuedogyno 3 months after the fact.
You're talking about my alpha run of SD? I know I did blood (I do on every new product) but have to dig it up. That was well over a year ago, but I remember off hand that my liver enzymes didn't move an inch. Cholesterol was hit but my test wasn't too low or I would have included a SERM, which I didn't. I hear people saying how suppressed they are after just a month of SD, it blows me away because I wasn't that suppressed at all.also, i might have missed this in an earlier post...but did u run a serm with all that or just the DHEA and fen, i might have missed that sorry if this is a repeat
I still do not understand the exact nature of these phenomena, but have advanced several hypotheses over many threads on several boards. I have yet to see any solid trends that would help me to narrow it down. Some used a SERM, some used an AI, some used both, some think it's the SD itself. If it is for real, I suspect ATD alone PCTs could share some of responsibility. DHEA is not the problem and I feel that it may actually have helped if used simultaneous with ATD alone PCTs, if in fact ATD is even the common link. If not, you guys being affected are doing something basically wrong while on or post-cycle or else are extra sensitive genetically to androgen manipulation.Dr. D,
Have you found any evidence that this SD "delayed gyno" couldn't happen after PCT? When you show me that evidence, I'll stop claiming I got gyno/psuedogyno 3 months after the fact.
Yeah, divided doses on the DHEA. 100mg morning and 100mg noon. On days when I only use 100mg, I'll take it all in the morning. Never use DHEA too close to bed or after 6pm or it might delay recovery instead of helping with it. It's a great idea to use 3mg melatonin/night too when on DHEA. These two compounds balance one another. I start with 2-3 caps on the fen and add another cap every week I stay on it.
Still new and learning like the other 50% that post here however when I first started reading up on PCT and Nolva/Clomid, I came across a few post on BB that mentioned Raloxifene. As it is suppose to be the top dog of estrogen killers and preventers. While I've learned Nolva is a receptor blocker but allows the estrogen to roam around freely until you cease dosing of Nolva. Seems to me it would just attach after the fact. Where as Raloxifene would prevent the estrogen from ever forming? Am I any where near right? If so why don't more people use Raloxifene or similar as their PCT?
If it is for real, I suspect ATD alone PCTs could share some of responsibility. DHEA is not the problem and I feel that it may actually have helped if used simultaneous with ATD alone PCTs, if in fact ATD is even the common link.
Based upon my experience of several cycles of various compounds and various PCT's, I definitely believe it is the ATD, although I don't believe it is only an ATD alone PCT.
For whatever reasons, I think ATD is having some bad effects on alot of people. I have never had any real problems with PCT until I included ATD as part of the PCT of my last cycle. It was the worst PCT I have ever had, I felt shutdown the longest, and libido was absolutely horrible. Even low does of ATD will trash my libido.
When I eliminated the ATD as part of the PCT protocol, things began to return to normal, but I had to do a second, shorter PCT after 6 weeks using only Clomid and Nolva to get fully recovered.
Just my opinion, but I will never use ATD again. There is something unusual going on with it.
Quick question..
I'm looking at an SD cycle down the road from now and this is what my PCT looks like, let me know if I need correction and if I should worry about gyno.
PCT as follows:
Day 1
- 60mg Nolva
- 1000mg Milk Thistle
- 1200mg RYR
- 60mg CoQ10
- 5g Taurine
Day 2-11
- 40mg Nolva
Day 2-7
- 1000mg Milk Thistle
- 1200mg RYR
- 60mg CoQ10
- 5g Taurine
Days 12-21
- 20mg Nolva
*Trib throught PCT if I feel it's needed*
so you havent run a SD cylce since then?You're talking about my alpha run of SD? I know I did blood (I do on every new product) but have to dig it up. That was well over a year ago, but I remember off hand that my liver enzymes didn't move an inch. Cholesterol was hit but my test wasn't too low or I would have included a SERM, which I didn't. I hear people saying how suppressed they are after just a month of SD, it blows me away because I wasn't that suppressed at all.
That's what I would have done too, separate I mean. I don't see a good reason to include both at the same time. They will compete with one another and there will almost always be an overall loss of effect. The weaker substrate often has the higher affinity, so it would work, but inefficiently.Both at he same time?? I dont think that is needed, I'm not sure if it could exacerbate things at all. perhaps Dr D could shed some light on this issue.
I personally would have tapered the Clomid over two weeks, than ran the Nolva consecutively as opposed to concurrently like you did.
At least you did in fact use Serms, and for more than one week like a few logs I've seen. Hopefully Dr D can offer more insight into what could have gone wrong here.
Yes, Sir, you're right on. It works the same as the the triphenylethylene class (Clomid, Nolva) but the half-live is only ~24hrs and it's much less toxic. It's better suited for women than Nolva because it has greater estrogen agonist activity in the bones but less in the endometrium while still being a good, strong antagonist in the breast. For us guys, the big advantage for use is the lack of emotional sides and the lack of liver stress when stacked with orals. It's the best on-cycle SERM you can use IMO.Raloxifene is still a SERM and works through the same mechanism of Nolvadex, a little different, but I dont know the specifics, Dr.D may know ...
Yeah, a few other times since then. I haven't associated it with any gyno on or off cycle though. Thanks for the link, I'll read through it this w/e probably.so you havent run a SD cylce since then?
http://anabolicminds.com/forum/anabolics/29911-sd-cycle-starts-monday-10.html
I can't PM you... Can you email it to me? The Raloxifene link.It is possible that estrogen rebound caused it to happen. In the mean time I would advise the use of a SERM to avoid the same thing happening again while off cycle. Raloxifene is the most predominate gyno buster out there IMO and it is virtually non-toxic, even in large quantities. The same cannot be said for tamoxifen citrate. If you are having trouble finding it, PM me.
Sky
Well, not to beat a dead horse, but I finally got my own copy of my bloodwork results back from the endo after chasing him for about a month. Thought I'd post it.The prolactin route is exactly my theory right now. I went to see an endocrinologist to discuss my gyno, lack of libido/worries of low Test, and generally feeling crappy all the time. His thought is that it is prolactin related.
Dr. D, what are your thoughts on SD maybe having an effect on prolactin and/or dopamine levels post cycle?
You can take 'em at the same time, no problemsJust a quick question...I've been searching endlessly and I havent been able to find an answer for this seemingly simple question. My PCT after 3 week SD cycle is Rbound XT Activate and lean extreme. My question is how should I time the dosing and is it bad to take them at the same time. For example taking RXT and Activate together or waiting an hour in between. Would they interfear with eachother if I took them together? Whats the best way to take them?
http://anabolicminds.com/forum/475819-post20.htmlWhat's with all that "Nolva PCT" talk? Nolva blocks estrogen. Nolva, AFAIK, doesn't boost the boys. Most designer orals do not aromatize, and are suppressive, which means your estrogen levels at the end of a cycle should be particularly low. Why nolva then? I cannot think of a good reason... :think:
Clomid is different, it stimulates either the HPTA or the boys themselves, not sure wich, but at least it does contribute positively to getting things back to normal... Taking nolvadex after a non-aromatizing cycle is useless IMO. Better to take nothing and save the money.
CorrectHmmmm.... OK so that says that nolva and clomid block estrogen from blunting HPTA. But since there is very little estrogen at the end of these cycles, then they are not needed in that capacity. I'm fairly sure that Dr. D explained that clomid has some actual testosterone-stimulating effect, other than simply blocking estrogen from blunting the HPTA.
Did you at least read the link in the post.Hmmmm.... OK so that says that nolva and clomid block estrogen from blunting HPTA. But since there is very little estrogen at the end of these cycles, then they are not needed in that capacity. I'm fairly sure that Dr. D explained that clomid has some actual testosterone-stimulating effect, other than simply blocking estrogen from blunting the HPTA.
"Studies conducted in the late 1970's at the University of Ghent in Belgium make clear the advantages of using Nolvadex instead of Clomid for increasing testosterone levels (1). Here, researchers looked the effects of Nolvadex and Clomid on the endocrine profiles of normal men, as well as those suffering from low sperm counts (oligospermia). For our purposes, the results of these drugs on hormonally normal men are obviously the most relevant. What was found, just in the early parts of the study, was quite enlightening. Nolvadex, used for 10 days at a dosage of 20mg daily, increased serum testosterone levels to 142% of baseline, which was on par with the effect of 150mg of Clomid daily for the same duration (the testosterone increase was slightly, but not significantly, better for Clomid). We must remember though that this is the effect of three 50mg tablets of Clomid. With the price of both a 50mg Clomid and 20mg Nolvadex typically very similar, we are already seeing a cost vs. results discrepancy forming that strongly favors the Nolvadex side
I dunno bro.BTW, who has bloodwork that shows that SD acts as an anti-estrogen?
Read what I quoted from his article when I posted it in that other thread:Now my question is geared towards a PCT that starts with very low estrogen and testosterone levels. Since the estrogen levels are low, an anti-estrogenic effect isn't needed.
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