Tren Acetate
150mg Everyday
Strength and Mass gain was through the roof, it was stacked with test.
Insane for hardening and leaning out.
who likes what when it comes to Tren... (as far as esters and dosages are concerned.)
Post the Ester used, the dosage used, and how often you inject, length of cycle, other anabolic steroids stacked
-Trenbolone Hexahydrobenzylcarbonate
-Trenbolone Enanthate
-Trenbolone acetate
Have you ever tried tren before to see how it affects you?
nope, but it will be in my next cycle.!
definitely ace then. Tren for me was a mixed bag. One hand it lead to AMAZING gains in strength, especially shoulders. It got to thep point that I maxed out the plate loading machine hammer strength and was knocking it out for reps. On the other hand, it made me have very extreme emotions. If I was mad, I was PISSED. If I was upset, I was in tears. Eventually, it lead to severe depression. Tren was amazing for my physique, even more so considering how bad my diet was toward the end of my use, but I feel it really did screw my life up. Well, I shouldn't say it screw my life up, but it made it far worse when things got bad. I tossed the rest of my stuff and I feel 100x's better now that I'm off. I never got acne or anything like that, no physical sides at all, they were all mental. The bottom line is, if your life is unstable or rocky at all, I would seriously rethink your use of tren.
Tren Ace hands down. Know too many bro's that have had prolactin issues and they always used enanth.
Any feedback from the H... ester? (used alone) I am attracted to the idea of tri tren. Seems like a good combo.
Thats interesting... I have taken a tren PH, which doesnt give the same results as the real deal, both physically and emotionally, but I didnt experience any emotional instability. I wonder if it was just a personal reaction of yours..Its just an individual process of trial and error really. Thanks for the input tho.
If I use it I was thinking Tren ace, or the tri-tren. I can get both. fewer injections with tri-tren would be nice.
Any feedback from the H... ester? (used alone) I am attracted to the idea of tri tren. Seems like a good combo.
I have no eperience with this whatsoever. It is interesting to read though. Of the things that I have read about this drug, it seems to be the one with the most horrible side effects and a no-no for beginners. Really everything I read about it, sounds like hell to me.
I'm not the only one that has suffered from emotional problems while on tren. Tren has been shown to affect serotonin levels.
What tren ph did you try? Finigenx and its clones really never could convert to tren. From my understanding, it was missing a double bone that the body could not possibly create. As per, methoxy-trn and its clones, well I still don't think anyone knows exactly what was in them. You may or may not have a bad reaction, but don't base that on experience with "similar" compounds is all I am saying.
If you want to know more about the hex ester you should look up parabolan. That was the pharm grade tren with the same ester.
I'd agree with Pudz (no personal experience), but depending on where your reading it seems that the Tren sides are a little overblown. I've seen a lot of guys that love the stuff.
Pudz you forgot the hair loss side...but that is pry a given.
by what means is hair loss truly exacerbated with tren. I mean it doesn't convert to DHT, so I assume its not a by product of 5a-reductase interaction.
Parabolan seems to be the best option (minus the price). I feel as though Tren ace would be pretty much equal but requires more frequent injections.
I also know that since its a progestin, then post cycle therapy would not necessarily be the same (meaning no SERMS?)....I have read this. What are Tren users doing for PCT, if this is the case?
I don't know what you wouldn't use a serm for post cycle therapy for a progestin, but for my cycles with tren I just made sure to end the tren a few weeks ahead of everything else. Then run a normal post cycle therapy. Tren A will pretty much be gone after 2 weeks, given its short half life so it won't interfere with recovery. This has worked great for me. I have personally given up cycles, but when I did do them I finished with test only until everything else cleared, tapering to lower serum levels and SHBG. Tren is severly suppressive, so letting it clear out completely is a good idea in my opinion.
I would definitely stack tren with test, but both being very androgenic, I would think they would compete for receptors. Tren has an extremely strong affinity, probably moreso than Test, For this reason I think it would be good to use less test. so under 1g for most people. (if injecting ace at 75mg per day to start out with)=525mg per week, then test should be aroudn the same depending on goal and the individual? some guys use a few gms of test per week with like 150mg tren per day....
Parabolan seems to be the best option (minus the price).
I feel as though Tren ace would be pretty much equal but requires more frequent injections.
I also know that since its a progestin, then post cycle therapy would not necessarily be the same (meaning no SERMS?)....I have read this. What are Tren users doing for PCT, if this is the case?
I would definitely stack tren with test, but both being very androgenic, I would think they would compete for receptors. Tren has an extremely strong affinity, probably moreso than Test, For this reason I think it would be good to use less test.
This doesn't happen. Believe me you can not reach a point of oversaturating androgen receptors. Not even at 3 grams of test per week. LOL
Also Tren is an active fat loss agent and this probably occurs from its affinity for binding directly to the androgen receptor in the fat cells (test will not do that).
So use whatever you feel you need from both compounds without fear of receptor competition.
This doesn't happen. Believe me you can not reach a point of oversaturating androgen receptors. Not even at 3 grams of test per week. LOL
Also Tren is an active fat loss agent and this probably occurs from its affinity for binding directly to the androgen receptor in the fat cells (test will not do that).
So use whatever you feel you need from both compounds without fear of receptor competition.
I would just like some clarification why you say that the androgen receptor can't be oversaturated. I'm not arguing with you, I really don't know one way or the other and I am very curious to become educated on this subject.
It would seem to me that their are alot of substrates and various enzymes needed for these processes to occur and that a point of saturation can certainly be reaced. But I'm a chemist not a biochemist so I certainly don't know this for sure.
I know that when dealing with hormones their is no receptor site on the cell wall. The hormone freely passes the cell membrane and is is carried into the nucleus by a carrier molecule(I can't remember the name). It would seem that this carrier molecule would eventually become saturated.
Is this not the case?
Strong androgens UPregulate AR's and some even increase the number of AR's, thus the AR's won't be oversaturated due to increased activity and number.
awmcdon said:I would just like some clarification why you say that the androgen receptor can't be oversaturated. I'm not arguing with you, I really don't know one way or the other and I am very curious to become educated on this subject.
Thanks alot for the insight aspire and datBtrue.....appreciate it.
Pubmed said:1: J Steroid Biochem Mol Biol. 1990 Nov 30;37(4):553-8.Links
Up-regulation of androgen receptor binding in male rat fat pad adipose precursor cells exposed to testosterone: study in a whole cell assay system.
De Pergola G, Xu XF, Yang SM, Giorgino R, Bjorntorp P.
Istituto di Clinica Medica, Università di Bari.
Binding of androgens to adipocytes has previously been evaluated using cytosol fractions without taking into account nuclear binding, although the latter is suggested to be close to the physiological site of action. In the present study, performed in differentiated fat pad adipose precursor cells, we describe a simple, reliable and reproducible androgen binding assay in a system with intact cells. Tritiated and unlabeled methyltrienolone (R1881) were used to define specific and unspecific androgen binding. Triamcinolone acetonide was added to prevent the binding of R1881 to other types of receptors. Differentiated adipose precursor cells contain a homogeneous class of high affinity androgen binding sites, and binding is saturable and reversible. Binding apparently occurs at one site, with a Kd in the range of physiological androgen concentration (about 4 nM). Competition studies indicate that the receptor is specific for R1881, testosterone and dihydrotestosterone, which have approximately the same affinity, while progesterone, estradiol and dexamethasone show much lower affinity. Androgen binding was markedly enhanced after cellular exposure to R1881 and testosterone but not dihydrotestosterone, and this increase was dependent on protein synthesis, suggesting the formation of new receptors by these androgens. In conclusion, fully differentiated adipocytes contain a specific, high affinity receptor, the density of which is dependent on androgens.
datbtrue, your experiment was with test only correct? I remember reading about compounds competing each other for the AR, what does that mean then. If its true that they are competing then wouldnt you be getting less out of your doses?
Possible only if you are using a huge dose of tren and another non-aromatising androgen. However you are not taking into account the occurance of upregulation of the androgen receptor through different pathways. Also keep in mind that the binding of a steroid molecule and androgen receptor is temporary, not permanent. The steroid molecule binds, delivers its message and unbinds in a predictable time limited fashion. Also note that tren has an affinity for androgen receptor-sites within adipose tissue as well which is not a characteristic of testosterone.
My "experiment" was with a compound aproximately 1/5 as androgenic as trenbolone (androgenic ratio is 500 vs testosterone's 100). There are other variables of course but we could generalize and say that 600mg of trenbolone is about as androgenic as 3000mg of testosterone. In my one man experiment I was still growing tremendously at that level. This indicated to me that I was not out of available androgen receptors (or better yet androgen receptor binding opportunities).
Extrapolating using the androgenic ratios of the two compounds...I could have used 400mg of tren and 1000mg of test per week (equivalent to 3 grams of test) and not had to worry about androgen receptors.
First, if you look at the study I posted above testosterone DOES have affinity for adipose androgen receptors.
Second, Trenbolone is actually roughly twice as anabolic and three times as androgenic as testosterone.
Third, comparing androgenic values with binding availibility doesn't make much since since that shows affinity. Tren binds stronger to AR's, not to more of them.
However, I do agree with your message. It might be possible to overload receptors, but it would take a dose so high it would be ridiculous and you would likely be miserable or dead first.
Practically speaking perhaps we should say "several times stronger."
However they are listed as:
Trenbolone Anabolic/Androgenic ratio: 500/500
Testosterone Anabolic/Androgenic ratio: 100/100
Pubmed said:Environ Qual Saf Suppl. 1976;(5):253-64.Links
Pharmacological and endocrinological studies on anabolic agents.
Neumann F.
When used in connection with animal production the term "anabolic agents" covers a wide range. Ther steroidal male and female sex hormones are included in this list, as are the nonsteroidal estrogens. For the clinician and for the endocrinologist, anabolics are only steroids chemically related to testosterone and 19-nortestosterone. Estrogens, though possessing anabolic properties, too, do not belong to this class. This paper will deal with anabolic agents in in the stricter sense of which mainly trenbolone acetate combined with hexestrol has been recommended for bull and heifer fattening. To consider possible consumer injury from ingestion of meat from anabolic agent treated animals, it is necessary to know the pharmacological properties of the agents, the doses producing certain effects or might produce, and the levels of residues in the meat. Trenbolone acetate will be compared with the following anabolic agents: methenolone acetate, methandrostenolone, nandrone, androstanazole, and 19-nortestosterone. The activity spectrum of trenbolone acetate is similar to that of 19-nortestosterone or those anabolics that are derived from 19-nortestosterone. The compound has about three times stronger androgenic effect than testosterone propionate. Its index of dissociation between anabolic/androgenic activity is 2--3. This index is 3--10 for the other anabolic agents. As regards the virilizing potency, trenbolone acetate is also on the top of the list. It seems that androgenicity and degree of virilization run paralle. The antigonadotropic activity (inhibition of ovulation and testicular growth) of trenbolone acetate exceeds that of testosterone propionate by the factor 3. The compound is not estrogenic and seemingly not or only weakly progestationally active. In principle, the androgenic activity (symptoms of virilization) as well as the antigonadotropic effect (disturbances of the menstrual cycle in women, inhibition of spermiogenesis in men) of trenbolone acetate might be noted. This risk, however, can be excluded by mere calculation. In rats, 0.1 mg/kg trenbolone acetate have an antigonadotropic effect. This corresponds to a daily dose of 5--7 mg in humans. By the same extrapolation, a daily human dose of 100 mg can be calculated for androgenic activity. Such factors of conversion are, of course, not precise because rats are much less sensitive to androgens and anabolics than humans. Thus, testosterone propionate is active only in daily doses of 10--20 mg. If in humans trenbolone acetate also has three times the activity of testosterone propionate, effects in man had to be counted with not less than a daily intake of 3--5 mg trenbolone acetate. The dose which is recommended for livestock fattening is 300 mg. IT can, therefore, be excluded almost with certainty that the meat would contain such large amounts of hormone residues.
Does this mean I can't snort Fina or drink Winny?
I guess all I want to do is gain like 10 lbs. and look like Brad
Pitt....![]()
wow, yours and my goals are very similar!! lol