Thoughts about X-factor

350zTT

350zTT

Active member
I just recently acquired some X-factor that I was excited to use, however, I am now bummed because I found a study concluding that in highly trained individual using 1gm a day for 50 days, there was zero results obtained. What is everyones opinion on this supplement? Good or Bad?
 
I dont know what study you read but I loved this stuff. Produced incredible pumps and all day muscle hardness. Strength and body recomp was amazing. Ever since it hit the market I could count on one hand the number of people who didnt see results and in those cases you have to wonder what there diet and training was like. I am about to start another cycle on the 15th of November and cant wait. Check out MN website and you can read all about their independent research on it and results people have had on this stuff.
 
Thanks bigironkiller, you have restored my faith and now I am ready to kick it into high gear
 
its a good product for most people. if you dont eat and train right nothing works, but you got that down this is a very effective product.

be sure not to take anti-inflammatories while on this and keep fish oil a little low.
 
ArA is very effective for me...If you are training right and eating right it should do wonders
 
it works good but be carefull if your like me your joints will be p1ssed @ u. of corse i do alot of sprinting and jumping because of my sport.
 
does anyone know the reason it hurts the joints while on? i have bad knees already and they always hurt, just dont wana do any damage. but if its temporary pain.... worth the gains :p
 
So what are the new recomendations for x-factor use regarding fat intake? I noticed that the new version has omega's in it. So can I still take my fish oil and cocunut oil supps? Also, I am following a high protein, moderate fat, and low carb diet so is this going to be problematic with x-factor?
 
I have had great results, during all of the many runs of ArA (x-factor) It does make my joints a bit achy though, but nothing unbearable. I would eat like a horse and still loose a bit of fat while gaining weight. Remember it takes a few weeks to "load"
 
350zTT said:
So what are the new recomendations for x-factor use regarding fat intake? I noticed that the new version has omega's in it. So can I still take my fish oil and cocunut oil supps? Also, I am following a high protein, moderate fat, and low carb diet so is this going to be problematic with x-factor?
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Real simple, do not eat foods with omega3 fatty acids, or consume any omega 3 supps, no anti-inflammatory drugs NSAIDS, take on an EMPTY stomach, real simple.:cheers:
 
johnyq said:
I have serious issues considering a product that requires I avoid foods essential for life worthwhile.


Eat a lot of eggs and train hard.

http://anabolicminds.com/forum/supplement-articles/106901-high-levels-arachadonic.html
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Just to add to the discussion... in addition, you do not need to avoid any foods. In fact, please keep eating all the omega-3s/EFAs you want.


Arachidonic Acid in addition to DHA supplementation.....

Ara + DHA (Infancy)

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Issue: Mar 2008
Prenatal long-chain polyunsaturated fatty acid status: the importance of a balanced intake of docosahexaenoic acid and arachidonic acid
Mijna Hadders-Algra, 11Department of Paediatrics – Developmental Neurology, University Medical Center Groningen, Groningen, The Netherlands
Corresponding author: Mijna Hadders-Algra, MD, PhD Developmental Neurology University of Groningen Medical Centre Hanzeplein 1 9713 GZ Groningen The Netherlands Tel.: +31 50 361 9158 Fax: +31 50 363 6905 [email protected]
Citation Information. Journal of Perinatal Medicine. Volume 36, Issue 2, Pages 101–109, ISSN (Online) 16193997, ISSN (Print) 03005577, DOI: 10.1515/JPM.2008.029, Available online: 23 01 2008 , March 2008

Abstract
This review addresses the effect of prenatal long-chain polyunsaturated fatty acid (LCPUFA) status on neurodevelopmental outcome. It focuses on the major LPCUFA doxosahexaenoic acid (DHA; 22:6ω3) and arachidonic acid (AA; 20:4ω6). Due to enzymatic competition high DHA intake results in lower tissue levels of AA.
LCPUFA accumulation in the brain starts early and increases during the third trimester. Initially brain AA-accretion exceeds DHA-accretion; after term age DHA-accretion surpasses AA-accretion.
Animal studies indicated that early ω3-depletion results in poorer developmental outcome. They also showed that early ω3-supplementation had no effect on cognitive outcome, promotes visual development and impairs auditory and motor development. Only limited human data are available. Correlational studies suggest that neonatal AA status shows a positive relation with early neurodevelopmental outcome and that neonatal DHA status also might be correlated with improved outcome beyond infancy. Results of human intervention studies are equivocal: most studies were unable to demonstrate a positive effect of prenatal ω3-supplementation.
It is concluded that only limited evidence exists to support the notion that prenatal ω3-supplementation favours developmental outcome. Caution is warranted for an unbalanced high DHA intake during the first two trimesters of pregnancy, i.e., DHA without additional AA supplementation.

Keywords Arachidonic acid, brain development, doxosahexaenoic acid, LCPUFA, neurodevelopmental outcome, prenatal nutrition, pregnancy, review, trans-fatty acids
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Ara + DHA and ADHD

1: Am J Clin Nutr. 2000 Jan;71(1 Suppl):327S-30S. Links
Long-chain polyunsaturated fatty acids in children with attention-deficit hyperactivity disorder.
Burgess JR, Stevens L, Zhang W, Peck L.
Department of Foods and Nutrition, Purdue University, West Lafayette, IN 47907-1264, USA. [email protected]

Attention-deficit hyperactivity disorder (ADHD) is the diagnosis used to describe children who are inattentive, impulsive, and hyperactive. ADHD is a widespread condition that is of public health concern. In most children with ADHD the cause is unknown, but is thought to be biological and multifactorial. Several previous studies indicated that some physical symptoms reported in ADHD are similar to symptoms observed in essential fatty acid (EFA) deficiency in animals and humans deprived of EFAs. We reported previously that a subgroup of ADHD subjects reporting many symptoms indicative of EFA deficiency (L-ADHD) had significantly lower proportions of plasma arachidonic acid and docosahexaenoic acid than did ADHD subjects with few such symptoms or control subjects. In another study using contrast analysis of the plasma polar lipid data, subjects with lower compositions of total n-3 fatty acids had significantly more behavioral problems, temper tantrums, and learning, health, and sleep problems than did those with high proportions of n-3 fatty acids. The reasons for the lower proportions of long-chain polyunsaturated fatty acids (LCPUFAs) in these children are not clear; however, factors involving fatty acid intake, conversion of EFAs to LCPUFA products, and enhanced metabolism are discussed. The relation between LCPUFA status and the behavior problems that the children exhibited is also unclear. We are currently testing this relation in a double-blind, placebo-controlled intervention in a population of children with clinically diagnosed ADHD who exhibit symptoms of EFA deficiency.

PMID: 10617991 [PubMed - indexed for MEDLINE]
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1: J Nutr Biochem. 2004 Aug;15(8):467-72. Links
Dietary patterns and blood fatty acid composition in children with attention-deficit hyperactivity disorder in Taiwan.
Chen JR, Hsu SF, Hsu CD, Hwang LH, Yang SC.
Department of Nutrition and Health Sciences, Taipei Medical University, Taiwan.

Nutritional factors may be relative to attention-deficit hyperactive disorder (ADHD), although the pathogenic mechanism is still unknown. Based on the work of others, we hypothesized that children with ADHD have altered dietary patterns and fatty acid metabolism. Therefore, the aim of this study was to evaluate dietary patterns and the blood fatty acid composition in children with ADHD in the Taipei area of Taiwan. The present study found that 58 subjects with ADHD (average age 8.5 years) had significantly higher intakes of iron and vitamin C compared to those of 52 control subjects (average age 7.9 years) (P < 0.05). The blood total protein content in subjects with ADHD was significantly lower than that in control subjects (P < 0.05). On the other hand, children with ADHD had significantly higher blood iron levels compared to the control children (P < 0.05). Additionally, plasma gamma-linolenic acid (18:3 n-6) in children with ADHD was higher than that in control children (P < 0.05). Concerning the composition of other fatty acids in the phospholipid isolated from red blood cell (RBC) membranes, oleic acid (18:1n-9) was significantly higher, whereas nervonic acid (24:1n-9), linoleic acid (18:2n-6), arachidonic acid (20:4n-6), and docosahexaenoic acid (22:6n-3) were significantly lower in subjects with ADHD (P < 0.05). Our results suggest that there were no differences in dietary patterns of these children with ADHD except for the intake of iron and vitamin C; however, the fatty acid composition of phospholipid from RBC membranes in the ADHD children differed from that of the normal children.

PMID: 15302081 [PubMed - indexed for MEDLINE]
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