Ok I didn't read any responses nor questions but here's the truth bout test-tren ratio
U wan get cut af w least amount of sides?
High tren low test like 100-150mg tren and ED with 300mg test e/week
U wan feel great? Bump the test dose
Sex problems w high tren low test? Bump the test
No bber preps w low test n high tren. They all keep test at a g+ and pre contest 3-4weeks just drop it all.
I'd only agree with the first part, and a number of other people who have posted here have said the same thing:
Least sides, low test, if you can handle the sides more test. This is a case by case basis, but if you have sexual sides bumping test will make the problem worse, unless you control progestin. The estrogen progestin issue is a real nasty thing, and by taking steps to increase estrogen you'll only make it worse. That's why some can run low test without an dopamine agonist and not have issues- they're not sensitive to the progestin, as long as the estrogen is low.
I know a number of bodybuilders battleing gyno who basically weren't running Tren because of the issues associated with trying to increase there test dose to deal with sides.
So then what happens when you hit an AI? No estrogen problems
I've seen the problems continue until adding letro, which doesn't reverse the problem just stops it in place. Once the problem is in place you need to deal with the progestin.
Well the goal with an ai is to manage e2....which is also easier to accomplish on a lower dose of test. I like to keep my e2 at my off cycle number even when Im on cycle. Lets me get the benefits of e2 without the adverse effects. When I run tren or deca I tend to keep my e2 even a tad lower than it normally is when im off cycle. This works well for me personally to keep sides to a minimum.
What about the Tren upregulating the estrogen receptor? I've known a bunch of people with estrogen sides even when they're running an ai. I know it's a combination of e2 and progestin, but post blood work showing normal e2 and still seeing gyno flare ups until they add caber/prami shows a correlation.
Furthermore can you site information on receptor competition? I know a lot of this is bro science, but if you have something to show that it "doesn't work like this" I'd love to see it. Not questioning your credibility but the fact of the matter is I don't see a single person saying they have a better experience on high test/tren, and really it's the contrary- everyone has more sides with more test, estrogen control or not. I've personally done it with estrogen control and a dopamine agonist and still had sides I'd call unpleasant-unmanageable. Plain and simple I will not run Tren with anymore than 125-250mg of test ever again and I don't think I'm alone in saying this.
I mean if you want to believe estrogen is at the core of the problem you still have an issue cause ais lower estrogen by what, 30%? When you're talking super physiological levels of test, even double physiological levels it'd be prudent to expect double e2 and therefore 140% estrogen. That's with 250mg of test. 280% estrogen with 500mg of test is a lot of estrogen. Maybe the conversion isn't linear, but if estrogen control still causes you to have more than what you'd have at homeostasis that still poses a problem for a lot of people- and no one wants to run letro long term. Makes you feel like crap, bad for your joints and your immune system. So it seems the best way to control e2 with tren is... less test.