Exactly ya... you lowered through your sweet spot with estrogen. Ok, this will be hard to do, and take serious focus, but if you can do it, you will be OK IMHO. Let estrogen drift up and check how you feel as it goes up through the rebound of the letro(2 day half life, so by 4th day you should feel peak libido for your current state say 70%) -- your peak will feel less than 100% but better than now, say 70% for example. Ok, now you suicide inhibit down on day 5 or 6 likely (for maybe only a day or two or take the suicide inhibitor every other day for 1-3 days) until you feel about 70% or wherever you felt peak on the way up on the rebound... and you stop the suicide AI right there and wait, wait, wait, a week while your body fine tunes and you should improve while doing nothing more. Continue the caber a max of 4 weeks and this should raise your DHT which will increase libido and the range of your sweet spot with the estrogen -- I'll post an article to support DHT increase from reduced Prolactin because some "young bro science" will dispute it. You want to get your estrogen just right and your DHT just right and right now, likely both are off or moving toward right in light of the caber. (Me... I'm 43... with advanced degrees... lots of cycles and long cycles too... not a doctor, but I take this ****e seriously and I'm giving you my best bro so... at least you know).
Article:
The effect of prolactin on androgen response to human chorionic gonadotropin in normal men.
Lackritz RM, Bartke A.
Abstract
Testicular androgen responses to human chorionic gonadotropin (hCG) were compared in normal males before and after suppression of prolactin (PRL) secretion with bromocriptine. Baseline follicle-stimulating hormone, luteinizing hormone, and PRL levels were suppressed by bromocriptine, 2.5 mg daily (P < 0.05). Serum testosterone and dihydrotestosterone (DHT) levels were reliably increased by one intramuscular injection of hCG (P < 0.05). Although testosterone responses to hCG were not significantly different in normal PRL and suppressed PRL cycles (P > 0.05), the DHT response was significantly increased in the suppressed cycle (P < 0.05), suggesting a physiologic 5 alpha-reductase blockage by PRL in men.