PCT for H-Drol Worthless?

yoss

New member
I am looking to set off something of a debate here, from the research I have done it appears that PCT for h-drol/turanibol LV has no value. SERM use assumes an unusually high amount of estrogens. They block their activity, specifically in breast tissue; they were designed to fight breast cancer by working as a antagonist to the estrogen receptors there.

If H-Drol does *not* aromatise, and all the steroid profiles of it I have read say that it does not, estrogens would be suppressed post-cycle because you have gone weeks with no substrate(natural testosterone.)

Why block the activity of something that isn't there? AI's don't make any sense to me either, preventing aromitization might increase T levels on the outset, but there would be an obvious dip when discontinued, until the HTPA axis recovered. I'm putting my money where my mouth is to some extent here, I ran toremifene after a 50mg 4-week cycle, and I've been going without(even though I have more tor) after a 6-week 90mg cycle for the past two weeks. My recovery is subjectively the same, has anyone gone 'naked' on this pro-steroid and had a negative experience? By the way, I'm open to being wrong, and I'm not an idiot, I have Tor on hand if I start getting itchy.
 
some guys get serious shutdown, just look around for some pre / post blood work.
 
Your research is inconclussive, I suggest you look for information on SERMs and the effects they have on the htpa, they are not just for estrogen control
 
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