So my lab rat started Ostarine MK-2866 recently and I put him on a graduating dose do to a “head rush” type side effect that was observed.
Started at 12.5/18.75/18.75/18.75/25/25 and noticed great results in hypertrophy and strength gains over that short period of time. From incline dumbbell press going from 45lbs 8 rep max to 70lbs for 8 reps in 5 weeks (that’s a pretty great difference imo).
My rat hasn’t experienced almost any suppression which is stellar but it has only been 6 weeks and 2 weeks on max dose so I guess we will see. What my question here is about how SARMS bind to the ARs. I have the option of running the Ostarine in my rat for 2-4 more weeks which is what other researchers believe is ideal, or I could run Ligandrol for 4 weeks in place of the Ostarine to try and maximize quick gains during the end of the cycle. So if I remove the Ostarine from the daily regimen and add LGD (I am not running both due to this being my rats first cycle) does the LGD work in the same manner and bind to the same receptors quickly as the Ostarine leaves my rats system or will it be like starting a new cycle and I’ll have to wait to see results in my rat over the next so many weeks? If continuing the Ostarine will work fine then that’s what I’ll do but if I could “upgrade” to LGD and have it continue where the Ostarine left off so to speak then that would be ideal. I figure most things in the same class will work in similar fashions (kind of like moving from one SSRI to another and feeling no withdrawal or having negative impacts from the last one, for example but reaping the benefits from the new one due to the build up of SSRI’s already in the body).
On another note I also wanted to possibly research Nutrobal for a PCT and see how that goes. Any advice or experience on that?
Started at 12.5/18.75/18.75/18.75/25/25 and noticed great results in hypertrophy and strength gains over that short period of time. From incline dumbbell press going from 45lbs 8 rep max to 70lbs for 8 reps in 5 weeks (that’s a pretty great difference imo).
My rat hasn’t experienced almost any suppression which is stellar but it has only been 6 weeks and 2 weeks on max dose so I guess we will see. What my question here is about how SARMS bind to the ARs. I have the option of running the Ostarine in my rat for 2-4 more weeks which is what other researchers believe is ideal, or I could run Ligandrol for 4 weeks in place of the Ostarine to try and maximize quick gains during the end of the cycle. So if I remove the Ostarine from the daily regimen and add LGD (I am not running both due to this being my rats first cycle) does the LGD work in the same manner and bind to the same receptors quickly as the Ostarine leaves my rats system or will it be like starting a new cycle and I’ll have to wait to see results in my rat over the next so many weeks? If continuing the Ostarine will work fine then that’s what I’ll do but if I could “upgrade” to LGD and have it continue where the Ostarine left off so to speak then that would be ideal. I figure most things in the same class will work in similar fashions (kind of like moving from one SSRI to another and feeling no withdrawal or having negative impacts from the last one, for example but reaping the benefits from the new one due to the build up of SSRI’s already in the body).
On another note I also wanted to possibly research Nutrobal for a PCT and see how that goes. Any advice or experience on that?