Rogue Drone said:In my experience, A superb Product for bodyfat reduction and increased muscle definition, with strength increases and a general increase in energy. I felt 10mgs of M4OHN (Designer, of course) for 29 days was comparable in effects and feel to a previous 30 mg Anavar cycle . I felt great from start to finish on it.
I'd estimate it reduced my body fat by 20% on not too clean a diet, saw a noticeable increase in muscular definition and a strength increase of about 15%. I wasn't fat or weak to start with and was pleasantly suprised at how apparant the effects were.
For PCT, I did just 6-oxo, a continuation of M4OHN's Aromatase Inhibition. I did'nt want to do a SERM (Toxo) from a position of Low E (the Aromatase inhibition of the 4-OH) and no apparant or previously reported test shutdown, my study would indicate that a SERM at the time of Low E with an expected quick rebound in HPTA could have an agonist effect on the E receptors.
Rogue Drone said:Yes, Alone. I tried both 5X2 and 10X1, felt considerably more from 10X1. I think Designer (F***LG and HM's untested high cost crap) M4OHN is truly great stuff, a best buy in Anabolics.
My next cycle will be M4OHN, a mass PH and M5AA pre-workout.
Rogue Drone said:For PCT, I did just 6-oxo, a continuation of M4OHN's Aromatase Inhibition. I did'nt want to do a SERM (Toxo) from a position of Low E (the Aromatase inhibition of the 4-OH) and no apparant or previously reported test shutdown. My study would indicate that a SERM at the time of Low E with an expected quick rebound in HPTA could have an agonist effect on the E receptors.
I had to read it twice also, but I'm pretty sure he meant he lost 20% of his body fat (ie he was 10% and now he's 8%)Kristopher said:is that a typo that you lost 20% BF in 29 days on M4OHN?
Rogue Drone said:Your PCT looks right to me. The 4-OH compounds of M4OHN and M4OHT appear to be unique Prohormones, in that you are taking an AI oncycle and probably having a minimal drop in HPTA. When you a run a combination,though, assume the 4-OH as AI is helping, but not sufficently well to not use Clomid or Nolva. I have nothing against Nolva, it's the way to go, except when E levels are low. Nolva is a Estrogen Receptor Modulator, an antagonist when E levels are high, an agonist when they are low.
Generally speaking for PCT I believe moderate (like a pattern of weeks 2,3,4,4 doseage levels instead of the standard 1,2,3,4) of both a SERM and an AI is the way to go. Attack from two angles,without overreliance on one.
An alternative method, that would work for the above combo and what I"ll be trying in the future, is to run a AI and a small daily dose of HCG oncycle. I'd rather deal with conversion and suppresion as I go, rather than play catch up afterwards.In theory, minimal conversion and test suppresion would better prevent E and DHT complications and more effectively keep the gains from an adequate level and quick rebound to full test levels. At that point, I'll be going to pre, half way through and post bloodwork as well, to more accurately quantify these protocols.
Rogue Drone said:Not a fan of PCT with 6-oxo or formadrol, huh? I'm not a fan of Formadrol either, but the concept of AI and SERM together has merit, IMO.I don't like Formadrol because 4-OHA has anabolic deratives and Daizen is a crappy Phytoestrogen, it has virtually zero RBA to the ER, it's just pissed away.
Here's my rationale, similar to the concept of Formadrol. An AI to inhibit conversion of T to E, and boosts Test through increase LH output. The AI I will use is a clinically proven test booster, all the AI's I've seen are, to one degree or another. The SERM is to insure that any T to E conversion that might be getting by the AI is blocked at the ER by the SERM, but not too much will be used, because there should be little E getting through and SERM's are not a good idea to introduce in the position of Low E. THe SERM (Nolva) will also increase LH output to boost test levels.
End result is, theoritically, no E effect and a double measure to boost T as fast as possible. I start with a moderate dose of each, enough to be effective but not excessive, and taper down to prevent any possible rebound effect.
The AI/HCG method is to inhibit and keep the test production up throughout the cycle in small doses of 250 to 500IU daily. It's an extension of the above, I'll probably run low dose Nolva anyway as an extra precaution.
I don't like the concept of test crash followed by recovery in PCT, I'd prefer to try and maintain levels and output as I go along.
Rogue Drone said:Jergo, I understand were you're coming from, I appreciate the input in how to build a better machine. If you look for studies on AI's ability to positively affect LH and test, you'll find them. I have them on Letrozole, Armidex, Fadrozole and Exemestane, but they're in .pdf form,I can't cut and paste excerpts here.
Manu20 said:Damn it Jergo I want that what a shot championship from you.
Rogue Drone said:Yeah, you're probably right, E is a scary thing for us old guys. I would'nt continue eliminating E, I know that's bad for the Cardio system, Brain, Bones and it's cancer preventative benefits.
Everytime I try to upload a .pdf here, I get a server failure notice. I"ll forward the one .pdf study I have on this machine to you at cyber-rights. It'll also be coming from Cyber-rights. It's a study that quantifies Letrozole's positive effects on LH and Test in Men . The profile on Fadrozole, as described in this study, is very similar.
It's the study entitled " Trunet PF, Trunet PF, Mueller P, Bhatnagar AS, Dickes I, Monnet G & White G 1993 Open dose-finding study of a new potent and selective nonsteroidal aromatase inhibitor, CGS 20 267, in healthy male subjects. Journal of Clinical Endocrinology and Metabolism 77 319–323
at Invalid Link Removed a collection of references on AI's effects.
Rogue Drone said:Is this the "explosive" growth the sellers talk about?