wjats y
I've got a gyno issue that doesn't seem to want to resolve with even pretty heavy dosing of letro. The gyno flared up after a short little cycle of T-bol back in October (I know you aren't supposed to have much by way of sides from t-bol but for some reason that **** just kills me). I did standard PCT with Nolva at the end of the little 6 week cycle, and everything seemed cool but then after a month it kinda started to flare up. Tried Aromasin first, didn't improve, switched to Letro, bumped up to pretty serious dosing and still wasn't really helping so I'm thinking I need to get bloodwork to figure out what's going on.
So my question is this: should I stop taking anything for a few days (Letro @ 2.5 ED) to find out what the true bloodwork says?
How high is your body fat %. It may bot even be the TBOL , because it shouldn't... Unless its bunk, but raws aren't that expensive and if some small labs are still bunking, get it tested and whistle blow on them..
Any how People will possess different amounts of enzymes as we know, genetics and so on, so lets pass that winded rant.. Basically there's precursors throughout the body and these can be found in all sorts of tissue including adipose tissue..When a hormone simulates the effects of T it can awaken "storage like cells" or even receptors that will react as if the presence of a specific hormone is about..Now some agents like Drol doesn't aromatize nor can it activate estro metabolites but how does one get estro like sides?!?! In principle it's said that some AAS won't convert however it's been proven that in some instances "mostly rare" that this can in fact take place outside of what we know as "normal aromatization", there's a vast amount of transformations in the body..Some aas have been transformed within their positions to avoid this, and others by delivery method (attempting to avoid the stomach by way of enzymes, and gastro tracks or even directly with the liver)..
Why do some people always have sensitive nipples?
Endogenous estrogenic biosythesis and promoters of different transcription factors is vast with it's diversity of actions, some people such as your self may possess secondary messengers and promoters and this can be expressed through different skeletal tissue, and even by way of stomach enzymes and other non reproductive tissue with E2 synthesis... Genetics is a huge contributor!
Think of a forest fire and all hoses aimed down on one spot attempting to occupy and inhibit that fire from spreading, it just takes one random amber to fly out unbeknownst to anyone and create an entirely differ set of issues behind the backs of the fire-fighters, flanking them so to speak..
I see and recognize that most of us have a keen understanding of the biological actions in which E2 is mediated between with the basic formality of what we know as the "normal aromatization" by way of hormones and enzyme presence, but in all actuality with the manner in which E2 can be transcribed can be hosted by a variety of factors.. It's a complex interplay with multiple identities made up of various interconnected signaling and cross-talk!
Examining nipples to often:
Do yourself a favor and stop touching them, every-time you touch it you're stimulating the glandular duct tissue, the more stimulation you're giving greater the chance of you inducing further inflammation to the ducts.. it's very similar to the process with females when milking for babies, it's called supply and demand.. the more you touch it and the more you stimulate it the more potential you have of growth..
This is true...
In fact there was a study conducted on a unit in the German Army honor guard, they would drill repeatedly and their replica rifles would continuously hit a specific side of their pecs, I forget offhand what side it was but we're going to say the left side just for topic sake.. a majority of the soldiers in the honor guard unit were developing gynecomastia on the same exact side.. after a thorough investigation and a study was conducted it was found that the repetitiveness and continuous contact and stimulation to the nipple and the surrounding glandular duct tissue it was inducing gynecomastia.. only on that one side..
This is why I tell people do not continuously investigate, by habitually feeling around and playing with bumps/lumps.. if you can feel tenderness there's no need to investigate further take the appropriate protocols to Target the symptoms, Target first then Implement long-term treatment.. targeting would include tamoxifen as a first strike inhibitor, at the same time I would include or continue the same protocol that you have with your anti estrogen, that will slow down the overall circulation of estrogen as the tamoxifen binds aggressively to The receptors within the ducts blocking the present circulating estrogen from binding to The receptors..