#1: Daily cardioI thought it would be a good idea to start a thread about cardiovascular support supplements an individual can and should use while using anabolic steroids. I don't want to come off as a smart-ass, but I believe thread like this should be a sticky.
I am not saying that this thread is so well written that it should be a sticky, but surely, if someone more educated on the matter has the time and the will to do it, it would be very nice of him/her. Definitely the most dangerous deleterious effect of AAS is the damage they do to the cardiovascular system.
We often hear people talking about liver support supplements, but we don't hear too often the talk about supplements which could lessen the damage AAS use places on our cardiovascular system.
So here I will name a few and everyone else is invited to share their experience and knowledge when it comes those supplements and drugs.
Obviously, doing cardio, keeping your blood pressure under control, keeping your hemoglobin and hematocrit in reference range via blood donation/lowering of the doses is important. Doing an ECG or a whole day (even the 7 day) heart study where you wear that device all the time is something every prospecting user should look into.
Fish oil - may lower triglycerides and raise HDL (particularly in women, but there may be hope)
The effect of fish oil on blood pressure and high-density lipoprotein-cholesterol levels in phase I of the Trials of Hypertension Prevention. Trial... - PubMed - NCBI
Green Tea - shown to raise HDL, lowers fasting LDL
Coenzyme q10 - may be protective to those with congestive heart failure
Coenzyme Q10 in the treatment of heart failure: A systematic review of systematic reviews. - PubMed - NCBI
Cialis - lowers blood pressure, but not by much (but has other benefits )
Influence of a single dose of 20 mg tadalafil, a phosphodiesterase 5 inhibitor, on ambulatory blood pressure in subjects with hypertension
Cardarine - very favourable effects on both HDL and LDL
Lipid effects of peroxisome proliferator-activated receptor-δ agonist GW501516 in subjects with low high-density lipoprotein cholesterol: character... - PubMed - NCBI
So these are the ones I am currently aware of right away, feel free to add more and comment as I feel this is a very important topic, should be much more important than shut down, impotence and infertility issues we commonly discuss.
We know that AAS lead to the weakening of the heart muscle and consecutively to heart failure and sudden cardiac death. Maybe there is something that delays the thickening of the walls of our heart (other than keeping blood pressure under control of course)?
I think that something that would act like a SERM does, something like a SARM which could selectively attach itself to the heart and prevent androgens from attaching themselves instead and therefore inhibit the androgens from stimulating heart hypertrophy and undesirable changes would be the holy grail. This needs more research.
Can you give me a link to what you are discussing? Because I found 1 on Amazon that seems to list everything.What’s weird is they don’t list everything they put in there. That’s why FDA didn’t approve it. Why would they hide some of the stuff they put in carditone?
In my opinion it’s more of a temporary drug, for when blasting. If you have chronically high bp year round without it, that’s something that should be getting addressed with a doctor if possible.What’s weird is they don’t list everything they put in there. That’s why FDA didn’t approve it. Why would they hide some of the stuff they put in carditone?
I have no clue regarding that situation. I researched it extensively before deciding to pull the trigger. I already had BP meds on hand. I jumped in quite a few heart forums and asked questions. People were posting their numbers after taking it. I couldn't believe the numbers myself. But it was hundreds of people posting numbers. You'll see for yourself soon enough.I ordered some. I just find it sketchy that FDA refused it because they said a lot of stuff in it wasn’t mentioned on the the bottle.
Wonder what his autopsy revealed about his heart?I was actually contemplating about a substance which would behave like a SERM does, ie. attach itself to the receptor and instead of being there to deliberately exhibit the effecs that come with stimulating the receptor, it would merely attach itself to the androgen receptor in the cardiac muscle to prevent true androgens (testosterone etc.) from attaching themselves and exhibiting their deleterious effects. In a way tamoxifen prevents real estrogen from attaching itself to receptors in breast tissue.
Well, Franco Columbu seems to be lucky in that regard. He is 77 and still alive.
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