Guest viewing limit reached
  • You have reached the maximum number of guest views allowed
  • Please register below to remove this limitation

M1P help!!

JudoJosh said:
So you suggestion was to stay away from M1P and instead go with Test suspension or anavar and to use over the counter products as his post cycle therapy?

Hmm...
Click to expand...

its the best case scenario............fast increase in strength and muscle to preserve any muscle wasting in boot camp, then use some OTC for a few weeks because neither suspension nor anavar shut you down that bad, short half life.............

then when he is back throw in the nolva/clomid mix and get blood work done
 
If your really in the military which i dont believe u are,steroids are not tested for in a random pee test, steroid tests are very expensive and u usually have to be commander selected with probable cause. Prohormones do nor show up...so the "random" test that u get everytime u take over two weeks of leave is a drug test and nothing more.

As for college, lol funny kid why would you lie about the scenario???
 
PVL said:
ur silly man...........im talking OCT PCT until back on leave...........
Click to expand...

Right back at ya buddy. You can not even take a multi vitamin in boot camp. So doing any steroids up to boot camp and then doing zero pct during the couple MONTHS that is basic training is a terribly stupid idea and simply isn't going to work
 
I feel like if you cannot commit the time and effort into a full cycle, with full and proper PCT you have no business running it. Waste of money, time and gains...
 
Srt4Muscle said:
If your really in the military which i dont believe u are,steroids are not tested for in a random pee test, steroid tests are very expensive and u usually have to be commander selected with probable cause. Prohormones do nor show up...so the "random" test that u get everytime u take over two weeks of leave is a drug test and nothing more.

As for college, lol funny kid why would you lie about the scenario???
Click to expand...

Correct, Airforce is same as the rest of the services on testing. They all follow a DOD standard
 
PVL said:
its the best case scenario............fast increase in strength and muscle to preserve any muscle wasting in boot camp, then use some OTC for a few weeks because neither suspension nor anavar shut you down that bad, short half life.............

then when he is back throw in the nolva/clomid mix and get blood work done
Click to expand...

So your stance is var isnt suppressive?

Or that test isnt suppressive?

All AAS will have an affect on your HPTA.
 
OP plane and simple your ****ed if you have been on for awhile 3-6 weeks, if its only been a couple days or a week drop the **** and just go be a solider, stop everything else you dont need it.
 
PVL said:
oh josh...........u silly billy..........one day u will grow up
Click to expand...

No. It is pretty obvious you have no clue and yet continue to try and give people advice.

lyfespan said:
yes all AAS will have a toll on the HPTA, but supression is based on doseages and lenght of tyme rum
Click to expand...

As little as 2.5mg of var can cause suppression. The dosage and lenth of time will determine HOW suppressed you become but there is no doubt that var is suppressive.

Effect of low dose oxandrolone and testosterone treatment on the pituitary-testicular and GH axes in boys with constitutional delay of growth and puberty.

AuthorsCrowne EC, et al. Show all Journal
Clin Endocrinol (Oxf). 1997 Feb;46(2):209-16.

Abstract
OBJECTIVE: To investigate the effect of low dose oxandrolone and testosterone on the pituitary-testicular and GH-IGF-I axes.

DESIGN: Prospective double-blind placebo-controlled trial.

PATIENTS: Sixteen boys with constitutional delay of growth and puberty (CDGP) with testicular volumes 4-6 ml were randomized to 3 months treatment: Group 1 (n = 5), daily placebo: Group 2 (n = 5), 2.5 mg oxandrolone daily or Group 3 (n = 6), 50 mg testosterone monthly intramuscular injections with assessment (growth, pubertal development and overnight hormone profiles) at 0, 3, 6 and 12 months.

MAIN OUTCOME MEASURES: LH and GH profiles (15-minute samples) were analysed by peak detection (Pulsar), Fourier transformation and autocorrelation. Testosterone levels were measured hourly and insulin, SHBG, IGF-I, and IGFBP-3 levels at 0800 h. Statistical analysis was by multivariate analysis of variance for repeated measures.

RESULTS: LH and testosterone parameters increased significantly with time in all 16 (LH AUC, P < 0.001; peak amplitude, P = 0.02; number of peaks, P = 0.02; testosterone AUC, P = 0.02; morning testosterone, P = 0.002). In Group 2, however, LH and testosterone parameters decreased at 3 months followed by a rebound increase at 6 and 12 months. SHBG levels were markedly reduced at 3 months (P = 0.006) and a wider range of dominant GH frequencies was present although GH AUC was not increased until 6 months, with an increase in GH pulse frequency but not amplitude. IGF-I levels were increased at both 3 and 12 months. In Group 3, pituitary-testicular suppression was not apparent, but GH levels increased with an increase in GH amplitude at 3 and 12 months.

CONCLUSION: Oxandrolone transiently suppressed the pituitary-testicular axis and altered GH pulsatility. Testosterone increased GH via amplitude modulation.

PMID 9135704
 
JudoJosh said:
No. It is pretty obvious you have no clue and yet continue to try and give people advice.



As little as 2.5mg of var can cause suppression. The dosage and lenth of time will determine HOW suppressed you become but there is no doubt that var is suppressive.

Effect of low dose oxandrolone and testosterone treatment on the pituitary-testicular and GH axes in boys with constitutional delay of growth and puberty.

AuthorsCrowne EC, et al. Show all Journal
Clin Endocrinol (Oxf). 1997 Feb;46(2):209-16.

Abstract
OBJECTIVE: To investigate the effect of low dose oxandrolone and testosterone on the pituitary-testicular and GH-IGF-I axes.

DESIGN: Prospective double-blind placebo-controlled trial.

PATIENTS: Sixteen boys with constitutional delay of growth and puberty (CDGP) with testicular volumes 4-6 ml were randomized to 3 months treatment: Group 1 (n = 5), daily placebo: Group 2 (n = 5), 2.5 mg oxandrolone daily or Group 3 (n = 6), 50 mg testosterone monthly intramuscular injections with assessment (growth, pubertal development and overnight hormone profiles) at 0, 3, 6 and 12 months.

MAIN OUTCOME MEASURES: LH and GH profiles (15-minute samples) were analysed by peak detection (Pulsar), Fourier transformation and autocorrelation. Testosterone levels were measured hourly and insulin, SHBG, IGF-I, and IGFBP-3 levels at 0800 h. Statistical analysis was by multivariate analysis of variance for repeated measures.

RESULTS: LH and testosterone parameters increased significantly with time in all 16 (LH AUC, P < 0.001; peak amplitude, P = 0.02; number of peaks, P = 0.02; testosterone AUC, P = 0.02; morning testosterone, P = 0.002). In Group 2, however, LH and testosterone parameters decreased at 3 months followed by a rebound increase at 6 and 12 months. SHBG levels were markedly reduced at 3 months (P = 0.006) and a wider range of dominant GH frequencies was present although GH AUC was not increased until 6 months, with an increase in GH pulse frequency but not amplitude. IGF-I levels were increased at both 3 and 12 months. In Group 3, pituitary-testicular suppression was not apparent, but GH levels increased with an increase in GH amplitude at 3 and 12 months.

CONCLUSION: Oxandrolone transiently suppressed the pituitary-testicular axis and altered GH pulsatility. Testosterone increased GH via amplitude modulation.

PMID 9135704
Click to expand...
ok so now we know you know howto go get abstracts on irrelivent subjects. DOOD come on man, really you come at me with this study of prepubescent boys that were either give TEST(probably undecanote, do to the lenght of study) or a small doseage of VAR.

I already told you that YES **** can be suppressive, but all depends on DOSAGE and LENGHT of tyme, and look they subject all had their HPTA rebound after 3 months, so the body fixed the supression didnt it? Not like they needed a extensive PCT?
 
PVL said:
thank you.........atleast someone isnt talking out of their ass............silly teenagers, trix are for kids
Click to expand...

Either way, your advice was neither good nor helpful. Suggesting someone to run AAS, with no PCT, leading into a training regimen that is counterproductive to gaining or sustaining muscle mass does not equal a good recipe. It's not like he is emaciated and has no strength at all. It is also not like he needs to be 220lbs and able to bench 300lbs in order to make it through boot camp. He simply needs to be able to do push ups, curl ups, pull ups, run, run, and run some more, and hang with calisthenics. His metabolic conditioning is going to play the more important factor. Running a cycle leading into this type of situation would only be a waste of time, money, and potentially lead to physical harm to his body for no gain or anything to show for it. We are of course for going the fact anyone may not actually be as old as the majority consensus feels is a good age to begin AAS use and all of that. Any muscle mass he may have now, will be more mature and he'll be able to gain it back fairly easily after boot camp, he can then look into running cycles when things can be done better.
 
lyfespan said:
ok so now we know you know howto go get abstracts on irrelivent subjects. DOOD come on man, really you come at me with this study of prepubescent boys that were either give TEST(probably undecanote, do to the lenght of study) or a small doseage of VAR.

I already told you that YES **** can be suppressive, but all depends on DOSAGE and LENGHT of tyme, and look they subject all had their HPTA rebound after 3 months, so the body fixed the supression didnt it? Not like they needed a extensive PCT?
Click to expand...

The study shows that even a small dose of var will cause suppression on the HPTA, which was the point of the post. Var is suppressive even in small dosages

Yes they recovered in 3 months, but does anyone really want to be shutdown for 3 months?
 
JudoJosh said:
The study shows that even a small dose of var will cause suppression on the HPTA, which was the point of the post. Var is suppressive even in small dosages

Yes they recovered in 3 months, but does anyone really want to be shutdown for 3 months?
Click to expand...
my point wuz being that these lil undeveloped kids are running 2.5mgof VAR ed for12 weeks, their HTPA already had trouble, yea the VAR helped but also taxed a underveloped system, Now go find a study with grown men under 55, and report those findings at the same doseage and tyme frame
 
He shouldnt be taking anything at 18. He will get big just by training and eating right.
 
lyfespan said:
my point wuz being that these lil undeveloped kids are running 2.5mgof VAR ed for12 weeks, their HTPA already had trouble, yea the VAR helped but also taxed a underveloped system,
Click to expand...

I have no clue how you cant grasp this. Androgens reduce luteinizing hormone which results in decreased testosterone production. How cant you follow this?

lyfespan said:
Now go find a study with grown men under 55, and report those findings at the same doseage and tyme frame
Click to expand...

PMID 10443664

Here they demonstrated that a dose of only 15 mg per day in healthy young men (for only five days) reduced total and free testosterone levels.

How anyone can think that ANY AAS wont be suppressive is just beyond me.
 
JudoJosh said:
I have no clue how you cant grasp this. Androgens reduce luteinizing hormone which results in decreased testosterone production. How cant you follow this?



PMID 10443664

Here they demonstrated that a dose of only 15 mg per day in healthy young men (for only five days) reduced total and free testosterone levels.

How anyone can think that ANY AAS wont be suppressive is just beyond me.
Click to expand...
Sounds like you got it all figured out then, cuz you have run VAR and been tested out. I barely expeience shutdown from test prop, at 300 a week while cruising, var didnt doo **** to me at 60 mgs for 8 weeks
 
Cause he's bi winning obviously haha
 
lyfespan said:
Sounds like you got it all figured out then, cuz you have run VAR and been tested out. I barely expeience shutdown from test prop, at 300 a week while cruising, var didnt doo **** to me at 60 mgs for 8 weeks
Click to expand...

Ahhh so here we see the root problem

The notion that in order to be shut down you have to "feel" something. This is false. Testicular size and libido are not definitive indicators of testosterone production.

And to answer you question, no I have not personally cycled var nor will I as it isnt very cost effective IMO but what difference does it matter what I have taken and what bloods I had drawn? The study I posted showed decreased test and free test levels after use, so are you saying you don't believe the study and need my blood work to confirm their results?
 
lyfespan said:
Sounds like you got it all figured out then, cuz you have run VAR and been tested out. I barely expeience shutdown from test prop, at 300 a week while cruising, var didnt doo **** to me at 60 mgs for 8 weeks
Click to expand...

Subjective data is not an absolute. More people need to realize this.
 
maybe we should adress what OP initially posted again. And id like to know if his junior in high-school brother stopped taking the M1P....
 
You must log in or register to reply here.
Back
Top