who can't spell? lol i made a simple mistype and can't edit for some reason. besides, why don't you mind your own business. if you cant see that dymethazine and superdrol are practically the same, maybe you lack intelligence.
here is the write up on it, but i doubt you will be able to understand it.
Roxilon (Dymethazine Write up)
From Anabolics 2009.
Roxilon (dimethazineJ
Androgenic 96
Anabolic 210
Standard Methyltestosterone (oraI)
Chemical Name 17beta-hydroxy-2alpha,17alphadimethyl-SaIpha-androstan-3-one azine mebolazine
Estrogenic Activity none
Progestational Activity none
Description:
Dimethazine, also known as mebolazine, is a potent oral anabolic steroid derived from dihydrotestosterone. The dimethazine molecule is fairly unique in structure, being made from two methyldrostanolone molecules bonded together with an azine bridge. The body breaks this bond, however, so that the drug actually provides free methyldrostanolone (Superdrol) to the user. Dimethazine is strongly anabolic, moderately androgenic, and not appreciably estrogenic or progestational. Although presently unavailable, this drug was once highly favored by athletes for its ability to promote solid gains in lean muscle tissue without excess water retention or fat gain. Qualitatively, the drug behaves in a very similar manner to drostanolone propionate (Masteron), although as an oral c17alpha alkylated .steroid it presents considerably more toxicity.
History:
Dimethazine was first described in 1962.714 It was
developed into a medicine by Ormonoterapia Richter in
Milan, Italy. The firm sold it under the Roxilon brand name
in Italy, and as Dostalon in Mexico. The Roxilon brand was
also reportedly sold under license by Lepetit. Dimethazine
has been evaluated clinically for a number of treatments,
often including use with women, the elderly, and children.
Such applications have included the promotion of growth
in underweight children and adolescents, the retention of
lean body mass with chronic pulmonary tuberculosis, the treatment of osteoporosis, and as a general anabolic in conditions necessitating the use of such an agent. In spite of a favorable record of efficacy and safety, dimethazine was ultimately a drug that saw only limited success as a prescription agent. It was discontinued many years ago, and has been unavailable worldwide for so long that few even recognize the active ingredient as a once-marketed commercial steroid.
Structural Characteristics:
Methyldrostanolone is a modified form of dihydrotestosterone. It differs by: 1) the addition of methyl group at carbon 17-alpha, which helps protect th hormone during oral administration, and 2) th\ introduction of a methyl group at carbon-2 (alpha), whicl considerably increases the anabolic strength of the steroi< by heightening its resistance to metabolism by the 3 hydroxysteroid dehydrogenase enzyme in skeletal muscl. tissue. Dimethazine consists of two methyldrostanolone molecules bonded together with an azine bridge. These molecules are metabolically separated, yielding free methyldrostanolone.
side Effects (Hepatotoxicity):
Dimethazine is a c17-alpha alkylated compound. This 31teration protects the drug from deactivation by the liver, 3110wing a very high percentage of the drug entry into the bloodstream following oral administration. e17-alpha 31kylated anabolic/androgenic steroids can be ~epatotoxic. Prolonged or high exposure may result in liver damage. In rare instances life-threatening ;jysfunction may develop. It is advisable to visit a :Jhysician periodically during each cycle to monitor liver runction and overall health. Intake of c17-alpha alkylated jsteroids is commonly limited to 6-8 weeks, in an effort to ~void escalating liver strain. Note that in studies administering 20 mg per day to patients for 45-95 days, dimethazine was shown to induce modest to moderate bilirubinemia (excess bilirubin in the blood, indicative of hepatic stress) in close to 50% of patients.?16
Approximately 25% of the patients noticed substantial increases in serum transaminases. These results suggest this steroid has a significant level of hepatotoxicity.
Side Effects (Testosterone Suppression):
All anabolic/androgenic steroids when taken in doses sufficient to promote muscle gain are expected to suppress endogenous testosterone production. Without the intervention of testosterone-stimulating substances, testosterone levels should return to normal within 1-4 months of drug secession. Note that prolonged hypogonadotrophic hypogonadism can develop secondary to steroid abuse, necessitating m'edical intervention.
The above side effects are not inclusive. For more detailed discussion ofpotential side effects, see the Steroid Side Effects section ofthis book.
Administration (Men):
An effective dosage of dimethazine for physique-or performance-enhancing purposes begins in the range of 10-20 mg per day, taken for no longer than 6 or 8 weeks. At this level it seems to impart a measurable musclebuilding effect, which is usually accompanied by fat loss and increased definition. Higher doses are usually avoided due to potential hepatotoxicity.
