Still need a new name and a full edit, but I promised today.......
HUNG
We are delving into a deceptively complicated subject here. One that has eluded my mastery for going on 2 decades, and that is just now coalescing into a masterpiece. I’m sure that in the coming years our understanding will increase even more, allowing for updates.
Goal/Product Statement:
HUNG is designed as a correlary product to be used with exercise/stretching stimulus to amplify body response, healing, and ultimately increases in not only length and girth, but healthy function through vascular remodeling and restoration of youthful NO response and smooth vascular muscle dilation/contraction to optimize blood flow for ideal erectile response.
NOTE: This product will not be effective if you do not employ physical stimulus – stretching via jelqing and/or traction, pumping, heating, Infra-red, etc. HUNG simply activates/inhibits key pathways to allow for maximum recovery and protein/tissue synthesis.
OK, now to the good stuff. This is a rough draft meant to outlay the pathways and give a summary.
1. PROSTAGLANDINS
Prostaglandins are some of the most ancients stimulus/response pathways in existences, traversing almost all species – especially mammalian. They are named because they were first isolated in prostate glands. They cause rapid and powerful responses to injury/workouts, play key roles in estrous and fertility as well as labor induction. First though to be reproductive only, they are now understood to be a huge part of every tissue in the body. The ones we will focus on are the 2 series, mostly PGF2a and the 1 series (PGE1)
For all prostaglandins, incorporation of fatty acids and phospholipids in the cellular mebranes and strategic response release to various stimuli are key.
1a. PGF2a
Those familiar with my, and others’, work will already be familiar with this pathway. Arachidonic Acid (ArA)content, the primary precursor, can be increased in the cellular mebranes – especially in the different muscle types. Subsequently stimuli, such as lifting weights and other types of “injury”, stretching, etc – basically anything that causes sudden deformation and perturbation of the membrane- cause the ArA to be released from captivity in the membrane and interact with PhosphoLipase A2, and subsequently COX-2 to form the 2 series prostaglandins. The one we are interested in is PGF2a, an extremely potent and fast acting healing stimulus. PGF2a communicates with adjacent cells triggering a sort of outward communication and feed forward loop which then triggers inflammation, upregulation of pain (to discourage you from further injuring the area), and a rapid induction of the Insulinlike Growth Factor1 pathway resulting in tissue repair and growth. There are some correlary factors that optimize using this pathway.
Note: COX-2 is the enzyme inhibited by NSAIDs such as ibuprofen. BY preventing the formation of the 2 series prostaglandins, the pain/inflammatory response is blunted, preventing paid.
1a1. Beta-Escin
Beta-Escin is a fascinating compound. The primary active compound in Horse Chestnut, used for centuries to prevent vascular leakage, Beta-Escin, when it comes into contact with cells, causes tiny pore/inury to the membranes, thus releasing the ArA and setting into motion the process described above. By not relying exclusively on the mechanical (workout, etc) we get an amplified injury response.
1a2. TRPV (1 and 4)
The Transient Receptor Potentials are extremely complicated and far beyond the scope of this, but are generally environmental sensors. TRPV1 essentially being both a HIGH temperature sensor as well as noxious chemical sensor, it is most well know as the primary receptor activated by Capsaicin (Hot Peppers). The signal resembles a burn injury (which in our usage is only an illusion – no actual tissue damage being done) which then goes on to trigger the body to deploy their quick response for the “heal” the injury, resulting in an anabolic response. We are using a non-painful ingredient, Stearoyl Vannilylamide, to stimulate the TRPV1 receptor without pain. Pain down there sucks.
TRPV4, on the other hand, reacts strongly to stretch stimulus – exactly what we will be using to stimulate increases in size and girth. Not only does it amplify the signal, it also amplifies the repair response of protein synthesis. To stimulate TRPV4, I have developed some ultra pure Andrographolide-Nicotinamide cocrystals for better absorption.
2. PGE1
PGE1 is fascinating. Derived from not Ara, but from Gamma Linolenic Acid. The pathway is GLA-→Dihomo-GLA → PGE1
PGE1 (in its pure form) has been used for decades as a direct into penis injection that causes rapid and powerful erections. Turns out by using the GLA (and hopefully DHGLA eventually) to again saturate the membranes similar to ArA, we can cause amazing vasorelaxation, which allows more blood and nutrients into the tissue and keeps erectile response healthy.
2. Phosphatidic Acid
I’m going to this ingredient next because the process is similar to the above. PA activates a pathway called mTOR, which is THE main pathway involved in protein synthesis. In a serendipitous process crossover, the PGF2a release via Beta-Escin, we will also be triggering the localized release of PA, which will immediately cause protein synthesis in the penile tissue. Being a phospholipid, cellular mebranes LOVE it and will suck it up like...well, never mind (wink wink).
HUNG
We are delving into a deceptively complicated subject here. One that has eluded my mastery for going on 2 decades, and that is just now coalescing into a masterpiece. I’m sure that in the coming years our understanding will increase even more, allowing for updates.
Goal/Product Statement:
HUNG is designed as a correlary product to be used with exercise/stretching stimulus to amplify body response, healing, and ultimately increases in not only length and girth, but healthy function through vascular remodeling and restoration of youthful NO response and smooth vascular muscle dilation/contraction to optimize blood flow for ideal erectile response.
NOTE: This product will not be effective if you do not employ physical stimulus – stretching via jelqing and/or traction, pumping, heating, Infra-red, etc. HUNG simply activates/inhibits key pathways to allow for maximum recovery and protein/tissue synthesis.
OK, now to the good stuff. This is a rough draft meant to outlay the pathways and give a summary.
1. PROSTAGLANDINS
Prostaglandins are some of the most ancients stimulus/response pathways in existences, traversing almost all species – especially mammalian. They are named because they were first isolated in prostate glands. They cause rapid and powerful responses to injury/workouts, play key roles in estrous and fertility as well as labor induction. First though to be reproductive only, they are now understood to be a huge part of every tissue in the body. The ones we will focus on are the 2 series, mostly PGF2a and the 1 series (PGE1)
For all prostaglandins, incorporation of fatty acids and phospholipids in the cellular mebranes and strategic response release to various stimuli are key.
1a. PGF2a
Those familiar with my, and others’, work will already be familiar with this pathway. Arachidonic Acid (ArA)content, the primary precursor, can be increased in the cellular mebranes – especially in the different muscle types. Subsequently stimuli, such as lifting weights and other types of “injury”, stretching, etc – basically anything that causes sudden deformation and perturbation of the membrane- cause the ArA to be released from captivity in the membrane and interact with PhosphoLipase A2, and subsequently COX-2 to form the 2 series prostaglandins. The one we are interested in is PGF2a, an extremely potent and fast acting healing stimulus. PGF2a communicates with adjacent cells triggering a sort of outward communication and feed forward loop which then triggers inflammation, upregulation of pain (to discourage you from further injuring the area), and a rapid induction of the Insulinlike Growth Factor1 pathway resulting in tissue repair and growth. There are some correlary factors that optimize using this pathway.
Note: COX-2 is the enzyme inhibited by NSAIDs such as ibuprofen. BY preventing the formation of the 2 series prostaglandins, the pain/inflammatory response is blunted, preventing paid.
1a1. Beta-Escin
Beta-Escin is a fascinating compound. The primary active compound in Horse Chestnut, used for centuries to prevent vascular leakage, Beta-Escin, when it comes into contact with cells, causes tiny pore/inury to the membranes, thus releasing the ArA and setting into motion the process described above. By not relying exclusively on the mechanical (workout, etc) we get an amplified injury response.
1a2. TRPV (1 and 4)
The Transient Receptor Potentials are extremely complicated and far beyond the scope of this, but are generally environmental sensors. TRPV1 essentially being both a HIGH temperature sensor as well as noxious chemical sensor, it is most well know as the primary receptor activated by Capsaicin (Hot Peppers). The signal resembles a burn injury (which in our usage is only an illusion – no actual tissue damage being done) which then goes on to trigger the body to deploy their quick response for the “heal” the injury, resulting in an anabolic response. We are using a non-painful ingredient, Stearoyl Vannilylamide, to stimulate the TRPV1 receptor without pain. Pain down there sucks.
TRPV4, on the other hand, reacts strongly to stretch stimulus – exactly what we will be using to stimulate increases in size and girth. Not only does it amplify the signal, it also amplifies the repair response of protein synthesis. To stimulate TRPV4, I have developed some ultra pure Andrographolide-Nicotinamide cocrystals for better absorption.
2. PGE1
PGE1 is fascinating. Derived from not Ara, but from Gamma Linolenic Acid. The pathway is GLA-→Dihomo-GLA → PGE1
PGE1 (in its pure form) has been used for decades as a direct into penis injection that causes rapid and powerful erections. Turns out by using the GLA (and hopefully DHGLA eventually) to again saturate the membranes similar to ArA, we can cause amazing vasorelaxation, which allows more blood and nutrients into the tissue and keeps erectile response healthy.
2. Phosphatidic Acid
I’m going to this ingredient next because the process is similar to the above. PA activates a pathway called mTOR, which is THE main pathway involved in protein synthesis. In a serendipitous process crossover, the PGF2a release via Beta-Escin, we will also be triggering the localized release of PA, which will immediately cause protein synthesis in the penile tissue. Being a phospholipid, cellular mebranes LOVE it and will suck it up like...well, never mind (wink wink).