FOLLIDRONE 2.0 USER RESULTS

[The_Old_Guy]

Im going to assume you read through everything I posted yes?

I was hoping to get this up to save us both some time, but my mobile app wasn't posting notifications. Oh well.

I was going to get into everything you posted, send out some emails, etc.. - but then I stopped, chuckled and realized I was getting drawn in, and that I have nothing to gain for the money I'd spend (my time). As you know, combing through study after study (the full FTs) is soooooo time consuming. And in the end, it would just be the usual, never ending merry-go'round of "Battle of the Cherry Picked Studies" - ad nauseum, ad infinitum.

So I'll just go the Occam's Razor route and direct anybody interested to read the following links (and this wasn't that exhaustive of a search):

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And then ask themselves if they really believe a boutique supplement company has done with swallowed extracts of Green Tea and Seaweed, what the Pharmaceutical Industry has yet failed to do - and that WADA (who bans everything) has failed take notice of: Putting muscle mass on bed-ridden children with Duchenne Muscular Dystrophy at 1500mg/Day for $50 a month, and it's not even patented! Do you not want to be a Bajillionaire? I'd look for plants that do Exon-Skipping, next!

Believe me, you show me R-DB-PC-C Study with sedentary humans on on an Iso or (better) Hypo-Caloric diet, orally administered your product, and they put on 10lbs - I'll buy the sh^t out of it. Cuz, I'd like to get bigger without taking potentially toxic substances (drugs) too!

Last word is yours mate, people have both sets of data if they want to investigate further. See ya on the next one!

 

[JakeAntaeus]

I was hoping to get this up to save us both some time, but my mobile app wasn't posting notifications. Oh well.

I was going to get into everything you posted, send out some emails, etc.. - but then I stopped, chuckled and realized I was getting drawn in, and that I have nothing to gain for the money I'd spend (my time). As you know, combing through study after study (the full FTs) is soooooo time consuming. And in the end, it would just be the usual, never ending merry-go'round of "Battle of the Cherry Picked Studies" - ad nauseum, ad infinitum.

So I'll just go the Occam's Razor route and direct anybody interested to read the following links (and this wasn't that exhaustive of a search)...

Well, the way I see it, whether or not Pfizer is working on a monoclonal antibody for myostatin is rather irrelevant.

In any case, there's no need for a battle of cherry-picked studies. I think that this is amenable to logical formalization:

1. Follistatin stimulates hypertrophy via several mechanisms.
Inhibition of:
-Myostatin
-Activin A
-Transforming growth factor-β1
-SMAD3 (PMC3157209)
-SMAD2 (PMID: 27787698)

And activation of:
-mTOR
-S6K
...Independently of myostatin inhibition. (PMC3384410)

2. Follistatin-induced muscle gains -- in mice, at least! -- appear to be functional.
Mice injected with follistatin gain strength as well as muscle, and to a similarly extreme degree. This is important to note, as the skeletal muscle gains elicited by myostatin inhibition, solely, appear to be less than perfectly functional.

3. (If I'm not mistaken) Epicatechin and e.cava have both been shown to increase circulating follistatin levels in humans and animals.

This is therefore consistent:
IF (i) follistatin is an anabolic hormone, which I think suggests itself quite clearly by now, AND (ii) epicatechin and e.cava increase circulating levels of follistatin in humans, at reasonable doses, THEN they should be effective as anabolic agents, at least to some extent.

The big questions then become (i) how effective are these substances at increasing follistatin levels in healthy humans, and (ii) whether or not this effect is sufficient to stimulate growth. Surely Brundel has more information than I do where all that is concerned.

But, anyway, my point is that you can't dismiss it out of hand. To say "Pfizer's spending billions on a myostatin blocking program, and drug candidates like MYO-029 are immensely complicated biologicals, therefore Black Lion's boutique product can't possibly be legit" is something of a non sequitur, isn't it?

I do certainly agree with you that there are lots of issues with myostatin inhibitors. But follistatin-boosting isn't exactly myostatin inhibition, and the product is an interesting one. To me, anyway!

Cheers!

 

[smith_69]

ok,

next up- Cherry flavoring} is it really that delicious?

 

[justhere4comm]

ok,

next up- Cherry flavoring} is it really that delicious?

Yes, and I have several anecdotal studies done by someone like myself that has quite a bit of knowledge and experience tasting things with cherry and cherry like flavoring.

 

[unitas27]

Well, the way I see it, whether or not Pfizer is working on a monoclonal antibody for myostatin is rather irrelevant.

In any case, there's no need for a battle of cherry-picked studies. I think that this is amenable to logical formalization:

1. Follistatin stimulates hypertrophy via several mechanisms.
Inhibition of:
-Myostatin
-Activin A
-Transforming growth factor-β1
-SMAD3 (PMC3157209)
-SMAD2 (PMID: 27787698)

And activation of:
-mTOR
-S6K
...Independently of myostatin inhibition. (PMC3384410)

2. Follistatin-induced muscle gains -- in mice, at least! -- appear to be functional.
Mice injected with follistatin gain strength as well as muscle, and to a similarly extreme degree. This is important to note, as the skeletal muscle gains elicited by myostatin inhibition, solely, appear to be less than perfectly functional.

3. (If I'm not mistaken) Epicatechin and e.cava have both been shown to increase circulating follistatin levels in humans and animals.

This is therefore consistent:
IF (i) follistatin is an anabolic hormone, which I think suggests itself quite clearly by now, AND (ii) epicatechin and e.cava increase circulating levels of follistatin in humans, at reasonable doses, THEN they should be effective as anabolic agents, at least to some extent.

The big questions then become (i) how effective are these substances at increasing follistatin levels in healthy humans, and (ii) whether or not this effect is sufficient to stimulate growth. Surely Brundel has more information than I do where all that is concerned.

But, anyway, my point is that you can't dismiss it out of hand. To say "Pfizer's spending billions on a myostatin blocking program, and drug candidates like MYO-029 are immensely complicated biologicals, therefore Black Lion's boutique product can't possibly be legit" is something of a non sequitur, isn't it?

I do certainly agree with you that there are lots of issues with myostatin inhibitors. But follistatin-boosting isn't exactly myostatin inhibition, and the product is an interesting one. To me, anyway!

Cheers!

Time to brew an e.cava-shilajit complex Jake!