I essentially have 2 questions about use of Epiandro/Androsterone products.
First, I have read that the degree to which we become suppressed or shutdown during a prohormone/steroid cycle is largely dependent on how strongly the product/androgen we are using bonds to the AR. DHT is known to be the strongest endogenous hormone that binds to the AR, yet has very little anabolic activity.
So, when people run standalone Epiandro/Androsterone cycles to harden up and maybe lose some fat etc.. are we actually running a rather substantially suppressive cycle for very little actual benefit? I think we all consider a cycle of AndroHard for example to be a very light cycle, but how could this make sense if it binds to the AR stronger than test and we are taking it exogenously?
Secondly, if running a product like AndroHard during a prohormone/steroid cycle as a “test base”, are we actually lessening the effect of the actual anabolic substance we are taking because we are occupying our androgen receptors with DHT?
Thanks for any clarification
First, I have read that the degree to which we become suppressed or shutdown during a prohormone/steroid cycle is largely dependent on how strongly the product/androgen we are using bonds to the AR. DHT is known to be the strongest endogenous hormone that binds to the AR, yet has very little anabolic activity.
So, when people run standalone Epiandro/Androsterone cycles to harden up and maybe lose some fat etc.. are we actually running a rather substantially suppressive cycle for very little actual benefit? I think we all consider a cycle of AndroHard for example to be a very light cycle, but how could this make sense if it binds to the AR stronger than test and we are taking it exogenously?
Secondly, if running a product like AndroHard during a prohormone/steroid cycle as a “test base”, are we actually lessening the effect of the actual anabolic substance we are taking because we are occupying our androgen receptors with DHT?
Thanks for any clarification
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