Chados
Well-known member
This is written by Mike Arnold first we have ratios.
When it comes to women and AAS, the first thing she needs to know is that all steroids can potentially cause androgenic (masculinizing) side effects in all tissues, as androgens are not selective in terms of AR (androgen receptor) binding…they simply possess differing degrees of potency in these tissues. In laymen’s terms, every steroid has the potential to cause every androgenic side effect. Not a single one is exempt. They simply vary in their ability to do so. One mistake many prospective steroid users make is using androgenic ratings as a measure of androgenic potency. They believe this tell them how likely a certain drug is to cause androgenic side effects throughout the entire body, without realizing that androgenic ratings were used to determine androgenic potency in only one tissue—the prostate. Being that women don’t have prostates, androgenic ratings are completely worthless in helping a women assess androgenic risk in tissues such as the vocal chords, scalp, skin, clitoris, etc.
Just because a steroid may have a high androgenic rating in prostate tissue, it does NOT necessarily mean that it has that same degree of potency in other areas of the body. Likewise, having a weak androgenic rating does not necessarily indicate reduced potency in non-prostate tissues. There are many steroids which demonstrate this type of imbalance. Masteron is a great example. With an androgenic rating of 40, it would be considered below average in the androgenic rating department. Yet, experience has shown it to be pretty harsh on the hair line, causing significant hair loss in a large percentage of genetically predisposed individuals. Clearly, having a low androgenic rating does not mean that a drug is a weak androgenic in general. The bottom line is that each steroid must be evaluated individually when attempting to asses side effect risk.
Second we have anavar vs superdrol
Since androgenic activity in non-prostate tissues was never scientifically measured, the best we can do is guess based on real-world experience. While this isn’t ideal, it’s all we’ve got and it is certainly a hell of a lot better than nothing at all. Fortunately, the combined experience of generations of bodybuilders has given us a pretty good idea of how each steroid effects in the body, particularly when it comes to external side effects like hair loss, voice changes, hair growth, etc. Although some may find the following recommendation to be somewhat controversial, extensive personal experience (in myself, clients, and many others I know) has proven it to be an effective and relatively safe course of action. I am talking about Superdrol. Yes, that Superdrol.
When evaluated purely from a masculinizing standpoint, I have found SD to provide the best balance of growth to masculinizing side effects. As most of you know by now, SD is an extremely powerful drug, with many bodybuilders claiming that no other drug ever allowed them to build as much muscle tissue as quickly as SD did. I will personally attest to this fact, as have many 1,000’s of other users all over the world. However, its incredible myotrophic potency has led many to assume that its androgenic character must be similar, when in reality it is extremely mild in comparison to almost all other steroids. In fact, SD’s has a Q ratio of 20:1 (the same as Anavar), which basically means that its anabolic effect is 20X greater than its androgenic effect. Even though Q Ratios are based partially on a steroid’s androgenic potency in the prostate, anabolic ratings can oftentimes give us a good idea of how a steroid might act in other tissues. In this case Superdrol displays very weak androgenic activity in many androgen responsive tissues, including the scalp, skin, and likely the vocal chords as well.
Overall, I have found it to be about as mild as Anavar (in terms of masculinization) when compared on a mg to mg basis. However, SD is a much more potent growth agent, with just 5 mg/day providing a growth response several times greater than an equivalent dose of Anavar. This increased myotrophic potency allows the drug to be used in one of two ways. She can either experience significantly greater growth with a side effect risk similar to Anavar, or she can experience roughly the same amount of growth with a side effect risk significantly below Anavar.
In other words, SD is actually less likely to cause masculinizing side effects than Anavar when used at doses that supply an equivalent muscle building effect. Again, this is due to its much more favorable androgenic to myotrophic ratio. Just to give you an idea of how strong this stuff is, most women will gain more muscle with just 10 mg of SD than they will with 50 mg of Anavar. Or, if a woman wishes to minimize the risk of side effects, she could use as little as 2-3 mg/day while still making gaining equivalent to 15-20 mg of Anavar. With such a low dose the possibility of experiencing meaningful adverse effects is almost non-existent.
When it comes to women and AAS, the first thing she needs to know is that all steroids can potentially cause androgenic (masculinizing) side effects in all tissues, as androgens are not selective in terms of AR (androgen receptor) binding…they simply possess differing degrees of potency in these tissues. In laymen’s terms, every steroid has the potential to cause every androgenic side effect. Not a single one is exempt. They simply vary in their ability to do so. One mistake many prospective steroid users make is using androgenic ratings as a measure of androgenic potency. They believe this tell them how likely a certain drug is to cause androgenic side effects throughout the entire body, without realizing that androgenic ratings were used to determine androgenic potency in only one tissue—the prostate. Being that women don’t have prostates, androgenic ratings are completely worthless in helping a women assess androgenic risk in tissues such as the vocal chords, scalp, skin, clitoris, etc.
Just because a steroid may have a high androgenic rating in prostate tissue, it does NOT necessarily mean that it has that same degree of potency in other areas of the body. Likewise, having a weak androgenic rating does not necessarily indicate reduced potency in non-prostate tissues. There are many steroids which demonstrate this type of imbalance. Masteron is a great example. With an androgenic rating of 40, it would be considered below average in the androgenic rating department. Yet, experience has shown it to be pretty harsh on the hair line, causing significant hair loss in a large percentage of genetically predisposed individuals. Clearly, having a low androgenic rating does not mean that a drug is a weak androgenic in general. The bottom line is that each steroid must be evaluated individually when attempting to asses side effect risk.
Second we have anavar vs superdrol
Since androgenic activity in non-prostate tissues was never scientifically measured, the best we can do is guess based on real-world experience. While this isn’t ideal, it’s all we’ve got and it is certainly a hell of a lot better than nothing at all. Fortunately, the combined experience of generations of bodybuilders has given us a pretty good idea of how each steroid effects in the body, particularly when it comes to external side effects like hair loss, voice changes, hair growth, etc. Although some may find the following recommendation to be somewhat controversial, extensive personal experience (in myself, clients, and many others I know) has proven it to be an effective and relatively safe course of action. I am talking about Superdrol. Yes, that Superdrol.
When evaluated purely from a masculinizing standpoint, I have found SD to provide the best balance of growth to masculinizing side effects. As most of you know by now, SD is an extremely powerful drug, with many bodybuilders claiming that no other drug ever allowed them to build as much muscle tissue as quickly as SD did. I will personally attest to this fact, as have many 1,000’s of other users all over the world. However, its incredible myotrophic potency has led many to assume that its androgenic character must be similar, when in reality it is extremely mild in comparison to almost all other steroids. In fact, SD’s has a Q ratio of 20:1 (the same as Anavar), which basically means that its anabolic effect is 20X greater than its androgenic effect. Even though Q Ratios are based partially on a steroid’s androgenic potency in the prostate, anabolic ratings can oftentimes give us a good idea of how a steroid might act in other tissues. In this case Superdrol displays very weak androgenic activity in many androgen responsive tissues, including the scalp, skin, and likely the vocal chords as well.
Overall, I have found it to be about as mild as Anavar (in terms of masculinization) when compared on a mg to mg basis. However, SD is a much more potent growth agent, with just 5 mg/day providing a growth response several times greater than an equivalent dose of Anavar. This increased myotrophic potency allows the drug to be used in one of two ways. She can either experience significantly greater growth with a side effect risk similar to Anavar, or she can experience roughly the same amount of growth with a side effect risk significantly below Anavar.
In other words, SD is actually less likely to cause masculinizing side effects than Anavar when used at doses that supply an equivalent muscle building effect. Again, this is due to its much more favorable androgenic to myotrophic ratio. Just to give you an idea of how strong this stuff is, most women will gain more muscle with just 10 mg of SD than they will with 50 mg of Anavar. Or, if a woman wishes to minimize the risk of side effects, she could use as little as 2-3 mg/day while still making gaining equivalent to 15-20 mg of Anavar. With such a low dose the possibility of experiencing meaningful adverse effects is almost non-existent.
