1-testosteron question.

[marcink99]

if one was on Finasteride/Dutasteride or any other 5ar inhibitor would the prohormone still work? Since 1andro needs 5ar enzymes to convert to 1-testosteron?

I plan on doing a 4 week cut cycle at 175lbs do you think 220mg per day would work as good as 330?

Due to my schedule I dont want to go over 4 weeks. Previously I did 330 x 6 weeks with novedex xt as a pct and my test was at 7,9 ng/ml. with estrogen at 55,35 pg/ml dht 829,3 pg/ml during last week of pct.

Since its a mild cycle would your recommend I do clomid 25/25/25/25 or 50/50/25/25?

Thank in advance

 

[Robert11]

if one was on Finasteride/Dutasteride or any other 5ar inhibitor would the prohormone still work? Since 1andro needs 5ar enzymes to convert to 1-testosteron?

I plan on doing a 4 week cut cycle at 175lbs do you think 220mg per day would work as good as 330?

Due to my schedule I dont want to go over 4 weeks. Previously I did 330 x 6 weeks with novedex xt as a pct and my test was at 7,9 ng/ml. with estrogen at 55,35 pg/ml dht 829,3 pg/ml during last week of pct.

Since its a mild cycle would your recommend I do clomid 25/25/25/25 or 50/50/25/25?

Thank in advance

Where did you see 1-Andro is converted via the 5alpha r enzyme? It's actually converted by other enzymes to 1-AD metabolites which are then converted to 1-test (not sure by what in the secondary step). Anyway, if you're concerned, why not cycle off the fin/dut and replace with topical ru55941 which is probably a better name option anyway.

 

[marcink99]

I got it from here Invalid Link Removed

1-testosterone should actually be known as dihydroboldenone, which is what the steroid boldenone converts to when it interacts with the 5 alpha reductase (5AR) enzyme. Similar in the way that testosterone can convert to DHT (dihydrotestosterone), when 1-testosterone interacts with 5AR, the 1-testosterone is then modified in such a way that it can’t convert to estrogen.[3]

Because 1-testosterone is 5AR reduced, unlike regular testosterone, it doesn’t make conversion to DHT via 5AR in the scalp and the skin. Although there are still a significant amount of androgen receptors in both of these locations, 1-testosterone should be theoretically less apt to have an impact on those sites. Nonetheless, 1-testosterone is a highly androgenic (and anabolic) compound and the usual side effects such as acne and androgenic alopecia can still be a concern for those susceptible.

 

[Robert11]

I got it from here

1-testosterone should actually be known as dihydroboldenone, which is what the steroid boldenone converts to when it interacts with the 5 alpha reductase (5AR) enzyme. Similar in the way that testosterone can convert to DHT (dihydrotestosterone), when 1-testosterone interacts with 5AR, the 1-testosterone is then modified in such a way that it can’t convert to estrogen.[3]

Because 1-testosterone is 5AR reduced, unlike regular testosterone, it doesn’t make conversion to DHT via 5AR in the scalp and the skin. Although there are still a significant amount of androgen receptors in both of these locations, 1-testosterone should be theoretically less apt to have an impact on those sites. Nonetheless, 1-testosterone is a highly androgenic (and anabolic) compound and the usual side effects such as acne and androgenic alopecia can still be a concern for those susceptible.

That's not how I read that at all. What that is saying is that 1-test is also known as dihydroboldenone which is what the steroid boldenone converts to with 5ar. However, 1-andro is not boldenone nor does it convert to boldenone in the conversion so that does not apply. The conversion of 1-Andro is a two step process to 1-Test (DHB). Reduction of the 17-ketone functional group by 17b-HSD produces 1-Androstenediol while oxidation of the 3-hydroxyl group by 3a-HSD produces 1-Androstenedione. Neither of these conversions are with 5alpha. Then, both are further converted to 1-Testosterone. How, I'm not sure. But you can't assume it is the same way as boldenone to 1-Testosterone. So what you need to find out is what causes the conversion from 1-AD to 1-Testosterone.