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Fixx Your Joints for $14.95 a Month

Royd The Noyd

Royd The Noyd

Board Sponsor
Advanced Muscle Science Joint Fixx is on sale now at the Planet...​

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Only $14.24 for 30 days worth of a very effective Joint Formula (that happens to stack well with other Joint Formulas with little over lap).

[h=2]A "Fix" For Your Joints During Aggressive Weight Training![/h]
  • Supports Heavy & Aggressive Training
  • Formulated for Easy Once-a-Day Dose
  • Twice as Fast Acting as Standard Joint Repair
  • Next Generation Joint Formula
[h=3]What is Joint Fixx?[/h]Joint Fixx(TM) is a potent cartilage repair supplement specifically formulated for aggressive weight training & competitive athletes.[h=3]Why is Joint Fixx™ Superior to Other Similar Products?[/h]Common relied upon joint repair formulas use the tired big 3; chondroitin, MSM & glucosamine and take between 4 & 6 weeks to start working. Further adding to the frustration – the big 3 need to be taken between 3 & 4 times daily. Joint Fixx works within 2 weeks of use and you don’t need to take a handful of capsules – just 2 caps, once per day. In addition, there are no other joint repair products in sports nutrition that use three (3) of the newest and most potent cartilage aids that are completely unknown to aggressive weight trainers & competitive athletes:
  • Turmeric Extract
  • Hyaluronic Acid
  • ASU (Avocado Soybean Unsaponifiable)
[h=3]How Does Joint Fixx™ Work?[/h]ASU, Turmeric and Hyaluronic Acid have been linked to positively affect epidermis, mucosa and collagen of the skin and bone through minimizing inflammation and cartilage damage from the day-to-day trauma of heavy lifting and athletic endeavors.[h=4]More specifically:[/h]Turmeric has been shown to promote Anti-catabolism, anti-inflammation, and joint protection.ASU Enhances collagen and proteoglycan synthesis in one daily dose compared to multiple doses that you would need for chondroitin sulfate, msm, and glucosamine.Hyaluronic Acid is a natural compound found in the fluid that surrounds and cushions your joints. Supplementing addiitional Hyaluronic Acid, improves skin and joint health and has been shown to efficiently reach both tissues.[h=3]What Makes Joint Fixx™ the Strongest on the Market?[/h]The synergistic effect of the three main (3) ingredients acts as an overall force field to protect your joints from heavy day to day trauma that can wear your body down over the course of aggressive, chronic weight training and athletic activities.[h=3]Why Should Someone Buy Joint Fixx?[/h]Simply put – There is no other joint support supplement on the market that can deliver bodybuilders and athletes safe, effective, efficient and REAL protection like Joint Fixx™ can.
 
Perhaps stack with green tea:

Expression of pro-inflammatory mediators is inhibited by an avocado/soybean unsaponifiables and epigallocatechin gallate combination
Ownby, SL; Fortuno, LV; Au, AY; Grzanna, MW; Rashmir-Raven, AM; Frondoza, CG
BACKGROUND: Osteoarthritis (OA) is characterized by inflammation, joint immobility, and pain. Non-pharmacologic agents modulating pro-inflammatory mediator expression offer considerable promise as safe and effective treatments for OA. We previously determined the anti-inflammatory effect of an avocado/soybean unsaponifiables (ASU) and epigallocatechin gallate (EGCG) combination on prostaglandin E2 (PGE2) production and nuclear factor-kappa B (NF-κB) translocation. The aim of this study was to evaluate the effects of ASU + EGCG on pro-inflammatory gene expression.
FINDINGS: Articular chondrocytes from carpal joints of mature horses were pre-incubated for 24 hours with control media alone or ASU (8.3 μg/mL) + EGCG (40 ng/mL), followed by one hour activation with interleukin-1 beta (IL-1β, 10 ng/mL) and tumor necrosis factor-alpha (TNF-α, 1 ng/mL). Total cellular RNA was isolated and real-time PCR performed to measure IL-1β, TNF-α, interleukin-6 (IL-6), cyclooxygenase-2 (COX-2), and interleukin-8 (IL-8) gene expression. Intracellular localization of NF-κB was analyzed by immunohistochemistry and Western blot. Pre-treatment with ASU + EGCG significantly (P < 0.001) decreased gene expression of IL-1β, TNF-α, IL-6, COX-2, and IL-8 in cytokine-activated chondrocytes. Western blot and immunostaining confirmed NF-κB translocation inhibition.

CONCLUSIONS: We demonstrate that ASU + EGCG inhibits cytokine-induced gene expression of IL-1β, TNF-α, IL-6, COX-2, and IL-8 through modulation of NF-κB. Our results indicate that the activity of ASU + EGCG affects a wide array of inflammatory molecules in addition to decreasing PGE2 synthesis in activated chondrocytes. The responsiveness of chondrocytes to this combination supports its potential utility for the inhibition of joint inflammation.
Type: Journal article
 
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