Guest viewing limit reached
  • You have reached the maximum number of guest views allowed
  • Please register below to remove this limitation

H-Drol and Nolvadex as PCT?

C Dub805

C Dub805

Member
The more and more research I do, the more and more questions come to mind as to why so many people advocate running Nolvadex as the most appropriate measure for a PCT to H-Drol.

Now, please excuse my often overly-analytical approach to most things in life. I have questions and concerns, but at the same time, I remain receptive to any and all advice given by you, my fellow Anabolic Minds members.

It is said that H-Drol DOES NOT aromatize or convert to estrogen and although one's estrogen levels MAY rise slightly as a result of an H-Drol cycle, they will CERTAINLY NOT rise to a point where gyno will start setting in.

That said, why is Nolvadex being advocated as the most appropriate and absolute necessary PCT for and H-Drol cycle when Nolvadex (a SERM) primary purpose is to prevent gyno by competing for the receptor site in breast tissue and binding to it? The effect of tamoxifen citrate (Nolvadex); therefore, is through estrogen receptor blockade of breast tissue. Nolvadex is essentially recommended for those who use steroids THAT CONVERT TO ESTROGEN. Again, H-Drol doesn't convert to estrogen, so why Nolvadex as PCT?

Also, per the science, Nolvadex is the most effective PCT for PH's, Designer Steroids and Steroids THAT Aromatize or CONVERT TO ESTROGEN. Again, H-Drol, whether it be labeled a PH or DS does not convert to estrogen.

I look forward to your feedback and justification for including Nolvadex as the bread and butter of your H-Drol PCT cycle.
 
Do some more research, the use of nolvadex in PCT has nothing to do with whether or not the steroid aromatizes.
 
Might as well handfeed you something. Taking a suppressive steroid like hdrol shuts down your normal test production. Estrogen also drops in response to this.

After stopping taking your steroid, your test levels start to go back up and your estrogen shoots up as well. Since there is a binding affinity to the breast tissue blocking that binding would be in your best interest.

However a SERMs main use is to restart your hpta , after it has been shut down by steroids.
 
Thanks deadaim, like I said, I'm not advocating that I know, I'm simply posing the question based on much of what I've read. I guess you can say I'm playing Devil's Advocate to incite a little more in depth discussion instead of hearing the typical bandwagon response from people. That is, many, if not most, recommend Nolvadex, but only because that's what they hear others say. I'd like to hear why people recommend it.

Perhaps you can tell me why you would advocate including Nolvadex in an H-Drol PCT? What are some of your reasons?
 
deadaim said:
Might as well handfeed you something. Taking a suppressive steroid like hdrol shuts down your normal test production. Estrogen also drops in response to this.

After stopping taking your steroid, your test levels start to go back up and your estrogen shoots up as well. Since there is a binding affinity to the breast tissue blocking that binding would be in your best interest.

However a SERMs main use is to restart your hpta , after it has been shut down by steroids.
Click to expand...

Very well said. And, I thank you for "handfeeding" me this. So, you advocate including Nolvadex in order to restart one's hpta. I can appreciate that. I am of the opinion, based on what I've read, that Nolvadex is very effective in increasing one's natural testosterone production post cycle. So, yes, it has many benefits beyond what is was initially intended for. Thanks for playing.
 
Even though, might I add, H-Drol IS NOT very suppressive at all. It's actually very slightly suppressive, so shut-down isn't really an issue. But, I guess any shut-down should be addressed.
 
C Dub805 said:
Even though, might I add, H-Drol IS NOT very suppressive at all. It's actually very slightly suppressive, so shut-down isn't really an issue. But, I guess any shut-down should be addressed.
Click to expand...

Incorrect, thanks for playing. There is plenty of bloodwork showing extremely low test levels coming off an hdrol cycle.

You may recover quicker than an SD cycle, but that doesnt make it "slightly suppressive"
 
C Dub805 said:
It is said that H-Drol DOES NOT aromatize or convert to estrogen and although one's estrogen levels MAY rise slightly as a result of an H-Drol cycle, they will CERTAINLY NOT rise to a point where gyno will start setting in.
Click to expand...
They can rise to the point where gyno will start setting in. I have seen people on this board who only ran h drol develop gyno during pct.

C Dub805 said:
That said, why is Nolvadex being advocated as the most appropriate and absolute necessary PCT for and H-Drol cycle when Nolvadex (a SERM) primary purpose is to prevent gyno by competing for the receptor site in breast tissue and binding to it? The effect of tamoxifen citrate (Nolvadex); therefore, is through estrogen receptor blockade of breast tissue. Nolvadex is essentially recommended for those who use steroids THAT CONVERT TO ESTROGEN. Again, H-Drol doesn't convert to estrogen, so why Nolvadex as PCT?
Click to expand...
Nolva is SERM and handles estrogen issues in all areas of the body, though strong binding to breast tissue has been observed.


C Dub805 said:
Also, per the science, Nolvadex is the most effective PCT for PH's, Designer Steroids and Steroids THAT Aromatize or CONVERT TO ESTROGEN. Again, H-Drol, whether it be labeled a PH or DS does not convert to estrogen.
Click to expand...
I would not say that "per the science, Nolvadex is the most effective PCT", I personally prefer clomid and I think a lot of others do too. Toremifene has made a huge impact on the market too. Also I think alot of ppl use OTC PTC.

C Dub805 said:
...so shut-down isn't really an issue...
Click to expand...

Yes it is.


Bottom line: People have successfully used OCT PTC for H Drol but others have had some serious estro issues. I recommend having a SERM on hand for ANY PH OR AAS CYCLE.
 
IMO H-drol is prefect for an OTC PCT (and pretty cheap with some of the prices right now). But, as always, a SERM will work wonders as well.
 
C Dub805 said:
Even though, might I add, H-Drol IS NOT very suppressive at all. It's actually very slightly suppressive, so shut-down isn't really an issue. But, I guess any shut-down should be addressed.
Click to expand...

I know this is what "everyone says," but as someone else pointed out, it's just not true. I saw a guy's bloodwork on another forum where his precycle test was normal (like around the upper 600s) and he got tested again on the last day of his cycle and his testosterone had dropped to 7. YES, SEVEN -- as in barely anything. He recovered just fine with a proper PCT, BTW.
 
greaser said:
I know this is what "everyone says," but as someone else pointed out, it's just not true. I saw a guy's bloodwork on another forum where his precycle test was normal (like around the upper 600s) and he got tested again on the last day of his cycle and his testosterone had dropped to 7. YES, SEVEN -- as in barely anything. He recovered just fine with a proper PCT, BTW.
Click to expand...

I never heard of a test that low. I wonder if maybe there was a mistake in the bloodwork - but your point is correct IMO. Suppression is suppression and needs a pct.
 
CrazyChemist said:
I never heard of a test that low. I wonder if maybe there was a mistake in the bloodwork - but your point is correct IMO. Suppression is suppression and needs a pct.
Click to expand...

I saw that particular test over on the Prohormone Forum -- it surprised the heck out of me!
 
i believe there's someone on this forum who towards the end of his cycle, his test was at 79, so obviously there's shutdown. there's also the issue of being gyno prone which would justify the use of a serm in pct with rebound.
 
When a steroid is brought exogeneously into the system, chemical balances shift around to attain a certain equilibrium. your body will increase production of estrogen, cortisol, and other hormones in response to heightened testosterone levels, while simultaneously slowering (or completely stopping) natural production of testosterone.

Ok, so while we're on the cycle, are natural test production is going down to compensate for the exogeneous test intake, and our production of other steroid hormones (i.e. Estrogen, Cortisol, etc.) is going up to compensate for the heightened test levels. When we come off a cycle, we cease intake of exogeneous testosterone. In other words, we have very low natural test levels now, and very high cortisol and estrogen levels: it's the EXACT OPPOSITE of what we had while starting our cycle.

When we have an excess of one hormone, the others will start shifting around, to attain a certain equilibrium. It is a very common misconception that we want to eradicate estrogen . High estrogen levels play an integral part in Post Cycle therapy. That's right, you want to welcome high estrogen with open freaking arms, but there's a trick to it. And that trick is the almighty SERM (Selective Estrogen Receptor Modulator).

The purpose of a SERM is to block the negative effects of estrogen, while your hormone levels go back to equilibrium.
 
I think OP might be a TROLL..
 
lol..just by being bored and sometimes something like this can be good info for somebody right now.. sorry to bring it back ..lol.
 
You must log in or register to reply here.
Back
Top