There is something just not right about all of this. Normal people dont piss blood after 5 days of M1T. Superdrol doesnt cause problems like this either unless abused in high doses for a long time. Nothing you have mentioned sounds like blatant abuse. If there is an underlying condition or previous abuse (you left out) it could be different. This is why i asked questions such as medication and so forth. Something like accutane could explain some of it. Antibiotics? HepC?
Either way, this is abnormal and there is more to the story. Either you arent being 100% honest or you are leaving out facts. Perhaps you have an undiagnosed condition. If everything you said is dead on accurate this is VERY abnormal. The only thing I can think of is that you have been pounding potent methyls (m1t, m5aa, superdrol) for a long time and you dont want to admit to really overdoing it. It's annoying when you contribute to a post in an effort to help and then slowly realize that none of it quite adds up. It's a waste of everyones time.
Im not trying to be a ****. If everything is 100% dead on accurate then there is defiantly an issue specific to you. Your liver is not handling anything very well at all. Then again, you have been checked out and they dont find any specific condition or abnormality. Again, makes no sense. There are a few cases of superdrol induced cholestasis attribted to SD alone. I still find it hard to believe. There is most likely something going on. Previous abuse not admitted to physician, undiagnosed liver condition, alcohol abuse, genetic condition predisposing patient to abnormal liver function and drug induced cholestasis (most likely). And if not, it IS abnormal for someone's liver to have this much trouble with normal methylated hormone use.
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Expert Opin Drug Saf. 2003 May;2(3):287-304.
Drug-induced cholestasis.
Velayudham LS, Farrell GC.
Storr Liver Unit, Westmead Millennium Institute, University of Sydney, NSW, Australia.
Drugs may cause several overlapping syndromes of cholestasis, the pathophysiological syndrome resulting from impaired bile flow. These reactions comprise approximately 17% of all hepatic adverse drug reactions (ADRs) and they may be severe. Causes of 'pure' (bland) cholestasis include oestrogens and anabolic steroids; rarer associations are with antimicrobials and NSAIDs. 'Cholestatic hepatitis' is a common drug reaction in which liver injury and inflammation cause significant elevation of serum alanine aminotransferase (ALT) as well as cholestasis. Chlorpromazine and ketoconazole are classic examples, but it is now exemplified by amoxycillin-clavulanate and other oxy-penicillins. Chronic cholestasis results from small bile duct injury leading to the vanishing bile duct syndrome (VBDS), a disorder mimicking primary biliary cirrhosis, or from injury to larger bile ducts causing secondary sclerosing cholangitis. Whilst there is increasing evidence of a genetic predisposition to cholestatic drug reactions, there are currently no pretreatment tests to predict drug safety. Prevention of severe reactions therefore relies on early detection of liver injury and prompt drug withdrawal. Symptomatic management includes relief of pruritus and correction of fat-soluble vitamin deficiency. In small cohort studies, ursodeoxycholic acid (UDCA) arrested progressive cholestasis in two-thirds of cases, but evidence for use of corticosteroids is anecdotal. This review considers diagnosis, pathogenesis, prevention and management of drug-induced cholestasis, with particular reference to frequently- and newly-described causes.
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Below is an attached case study (wildly popular) of a person who apprently had major issues from normal superdrol use. Again, the only way the doctor(s) knew his history of use (or abuse) is by what the patient told them. Assuming no other current/previous abuse and no other medication, condition, alcohol abuse, etc., this is very abnormal. If he truly did only consume the normal amount for this amount of time stated (2 weeks) its most likely a genetic predisposition to Drug-induced cholestasis. In this case, a number of drugs or medications could trigger this situation. Normal individuals should not be experiencing the things in this case study or in your post.
You could be prone to drug-induced cholestasis. In this case, i WOULD NOT test my luck with methylated hormones of any kind. You should be careful with anything that is metabolized heavily in the liver or puts stress on it. Other drugs besides these could trigger this for you.
. I used it recently and liked it because of low sides and easy recovery. If I were you I would never ever use anything methylated (steroid that is) again. It would never be worth it for a few lbs of muscle to risk what you went through. 
