And so what if I have high E2 and low TT? The Clomid and Masteron will take care of the E2 and the Masteron will replace the TT. People forget that DHT is what makes testosterone "feel good" Guys with 5-AR deficiency have high TT and low or no DHT, average E2, and feel like absolute garbage. DHT...
I'm assuming it's active right now, since all signs point to having pulsatile LH (testicle size, weight, and firmness) and FSH (large volume of thick ejaculate). When I come off cycle, I'll be staying on Clomid at 25 mg/day and Naltrexone at 25 mg/day, and may run Mast E at 200 mg/week as well...
No. I specifically stated in other posts that 19-nors can damage Leydig cells. The method is malondialdehyde generation causing free-radical oxidation. 19-nors also increase the risk of testicular cancer.
Yeah, it's lacking in high-level cardiopulmonary knowledge, it's lacking in high-level vascular and endothilial knowledge, and my gastrointestinal knowledge is pretty thin.
Because AIs were new, patentable drugs, which never demonstrated efficacy greater than SERMs or Masteril (drostanolone).
Same as MAOIs ---> TCAs ---> SSRIs. Of these drugs, the MAOIs are the most efficious in the treatment of depression, followed by TCAs, and then SSRIs being the least...
You never reach a hyperthyroid state with T4. I already explained this. Keep up.
Taking high-dose T4 allows you to reach the limits of what your body considered high-but-not-hyperthyroid-T3, any excess T4 is converted to rT3, and a hyperthyroid state is prevented.
I gave a very detailed...
1. High-dose T4 allows one to maintain high levels of thyroid hormones without entering a hyperthyroid state. Once T3 levels get too high, T4 is converted to rT3.
2. T3 has a short half life and requires multiple doses per day. The half life of T4 is several days.
3. T4 is important in its own...
Because DHT works downstream to stop E2-evoked gene transcription which has already been triggered by E2. Lowering E2 at this point will accomplish very little, since the signal cascade has already started.
Treatment of Advanced Breast Carcinoma with Drostanolone Propionate
Bristol Medico-Chirurgical Journal. Vol. 85
"Virilization was not a noticeable side effect."
Can you all just stop making assumptions about things becuase you believe that they should be so, based upon your extremely limited...
Well, now you know the truth. I hate needles. Actually, I love them going into my muscles, but going into a vein is extremely painful and causes me to go into shock. So, as badly as I want to get blood-work, sometimes I just can't. Imagine facing your worst fear while being ridiculed by the...
I'm on Test, NPP, Mast, Clomid, T4, and Naltrexone. Going into my 7th week on AAS -- if something bad were going to happen it would have probably happened by now.
But I do get your point, and I can see the merits of your position.
Everybody knows someone who thinks they've seen a ghost.
What's your point?
I'm hardly a beginner at cycling and I'm hardly a bro-science pharmacologist. I have 10+ years of cycling and more than 18 years of post-grad pharmacology work.
If you want to discuss pharmacology in a scientific...
I'm not discounting the fact that some of us may be genetic anomalies when it comes to SERMs on-cycle, but I would still argue that a significant percentage of AAS users would see less suppression by running a SERM on-cycle.
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