Save your money use ECA Stack
- 05-12-2009, 03:27 AM
- 05-12-2009, 03:38 AM
05-12-2009, 03:59 AM
Good shout above!
Ive always taken green tea with the EC(Y) stack.
Being honest i only knew the surface of the science behind it but that nice little study has made me feel smug lol.
05-12-2009, 08:32 AM
05-12-2009, 10:59 AM
05-12-2009, 11:08 AM
05-12-2009, 11:12 AM
If you must use E and Y together I would use E/Y/G. You don't need C as Green Tea Extract has C.
GTE is a COMT inhibitor. This means it inhibits the downregulation of the stimulants. So it inversely increases the duration of their effects.
05-12-2009, 11:34 AM
05-12-2009, 12:10 PM
05-12-2009, 12:15 PM
I'm not saying throw out the Y. E targets beta receptors and Y targets alpha receptors. If you have adipose that is either, or, then use either, or, accordingly.
05-12-2009, 12:37 PM
I started Aspirin today (81mg) tomorrow I will increase the dose to 2x a day for a week or two, then 3x a day for a week, being that its baby Aspirin, and Im a big guy, how do you think I should dose it?
05-12-2009, 03:23 PM
05-12-2009, 03:25 PM
05-12-2009, 03:27 PM
05-12-2009, 03:30 PM
05-12-2009, 03:33 PM
The Historic PES Legend
05-12-2009, 03:39 PM
05-12-2009, 03:42 PM
05-12-2009, 04:18 PM
05-12-2009, 04:20 PM
05-12-2009, 04:33 PM
It may have been because of something I read, it could be because it was a convenient time to add into my shakes (not a good idea to use it too late because of the caffeine), it may be because I take it with dext/malto at that time so it might be absorbed faster.
05-12-2009, 04:35 PM
There are many studies done at countless universities that used aspirin in there testing.
it blocks Prostaglandin in the cycle for thermogenesis.
here is a picture from 1993 that was used in a study.ECA has been tested for up to 24 weeks and it showed that it kept on increasing ne while increasing the camp cycle.Where not taking about receptor downregulation,with eca you can get used to the stim affect after a week but there thermogenisis lasts for over 6 months.The aspirin plays a huge role in that.
An additional claim made by proponents of ECA-induced weight loss is that the thermogenic effects are
limited to fat catabolism, and that there is no protein catabolism, increased heart rate, or tremors which are
associated with other sympathetic stimulation. One hypothesis is that the main stimulation by ephedrine is
through beta2- and beta3-adrenergic receptor subtypes (2, 7), both of which are predominantly responsible
for lipolysis and protein synthesis, but are not associated with cardiovascular and central nervous system
effects mediated by beta1-receptor (2). Tolerance rapidly develops to the effects of ephedrine on heart rate,
but does not develop to the thermogenic effects (2, 8-12), suggesting that different mechanisms are
responsible for these different effects, and that ephedrine has longer-acting effects on thermogenesis.
The third component of the ECA combination, aspirin, enhances the peripheral actions of ephedrine and
caffeine by inhibiting prostaglandin (PG) synthesis. PGs, like adenosine, have been implicated in inhibiting
NE release from the post-synaptic nerve terminal, and in inhibiting the lipolytic actions of sympathetic
stimulants (2). However, these effects have been limited to only one study, and more experiments must be
performed before this effect can be conclusively linked to aspirin.
In summary, the effects of ephedrine, caffeine, and aspirin in rat brown adipose tissue involve a significant
central component of increased NE release, and a contributing peripheral part that has direct action on the
target tissue. These peripheral actions act to directly increase stimulation of beta-adrenergic receptors
(especially the beta2- and beta3-subtypes), to inhibit the regulatory mechanisms of negative feedback by
adenosine and PGs extracellularly, and to inhibit the destruction of cAMP by PDE intracellularly. This
overall scheme is depicted in Figure 1.
05-12-2009, 05:35 PM
05-12-2009, 05:51 PM
05-12-2009, 06:03 PM
05-12-2009, 06:18 PM
05-12-2009, 06:19 PM
05-12-2009, 06:32 PM
05-12-2009, 06:40 PM
05-12-2009, 06:53 PM
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