dht is quickly de-activated in the muscle via enzymes into a worthless metabolite.
of all the steroids i've researched, oral turinabol had the lowest androgenic rating, coincidentally, cdma (halodrol) is a precursor to tbol, so most likely this also has a very low androgenic effect, possibly even lower than ot.
cdma being a diol does not need to convert to bind to the androgen recpetor, but this binding will not be as strong as it's parent compound.
this is where it's long half life comes into play.
also, though it can metabolize into 4-chloro methyl 1-test (after conversion into tbol) via the 5a-reductase enzyme, the 4-chloro helps lower the compounds androgenicity, also reducing the androgenic side effects noticed.
less strongly binding to the ar, along with reduced androgenicity in theory should equal an overall less androgenic compound, with the least worry for hair loss.
though as noted, everything that binds to the androgen receptor will be capable of androgenic side effects.
this is what makes the compounds androgens, from dht, all the way to methoxy gonadiene.
the make up of the steroid will be what causes the over all differences in androgen interaction, as well as person to person differences.