- 10-19-2011, 05:24 PM
Im still waiting on studies from your end as well since I have done plenty of homework on the subject and info is all I need, Im not going to find it for you.
You CAN find advocates for just about anything, thats true, but I know what works for the general populace and has been shown effective through real world results time and time again, keeping BS levels constant will def boost metabolism and carb cycling will reduce pancreatic strain.
If your bulking then id def suggest using an insulin aid like anabolic pump as well to give your pancreas a break.... still insulin isnt everything when it comes to metabolism, the body uses tons of different mechanisms to increase your metabolic rate and having a constant source of fuel is imparitive.
- 10-19-2011, 05:26 PM
10-19-2011, 05:28 PM
10-19-2011, 05:40 PM
I snack on deli turkey a lot! It's a great filling snack! Or peanut butter and celery is really tasty!
10-19-2011, 05:44 PM
10-19-2011, 05:59 PM
10-19-2011, 06:24 PM
nice i like seeing that...except the one pubmed study being with obese individuals, not fit ones.... other then that very interesting.
10-19-2011, 06:37 PM
"this is in agreement with a previous report in which leptin response to carbohydrate intake is delayed for four to five hours after meal intake"
so wait 4-5 hours after you eat for leptin levels to return to baseline to prevent leptin resistance
10-19-2011, 06:51 PM
"This response was progressive and started to diverge from that of fat meal and of fast approximately 4–5 h after food ingestion. These results are in agreement with our previous study showing that the postprandial leptin response to a meal is delayed to 4 h after the meal"
The above just reiterating the top.
"Leptin response was significantly correlated to insulin response (r = 0.51,P < 0.0001). "
"In conclusion, a carbohydrate meal induces higher postprandial leptin levels than an isoenergetic fat meal."
"Its major findings are 1) that a carbohydrate meal induces a greater postprandial leptin response than an isoenergetic fat meal, "
so bulkin frequent meals are horrible for you and can lead to a greater chance of getting FAT.
Is it true that low carb diets this can be taken in differently , yes, but as this study above shows insulin responce to fat ingestions..
"In women, leptin levels were higher after the fat meal than while fasting."
heres something interesting...
"The relationship between leptin levels and carbohydrate intake suggests that leptin could contribute to the beneficial impact of carbohydrate-rich diets. Long-term studies are necessary to confirm this hypothesis."
high levels of leptin in the brain (ARC nucleous) is responsible for increased oxidation. However if there is a resistance (which we can see on a bulk diet) then theres less fatty acid oxidation. This study failed to mention it due to needing "long-term studies". Carb rich diets alotted in the 4-5 hours inbetween meal times would be more beneficial which would be still 2-3 meals. depending on how your caloric intake is.
10-19-2011, 06:53 PM
But you are right that lean individuals handle these things better, but lean individuals usually over eat to put on weight. and eat to frequently because they feel they need to.
10-19-2011, 07:07 PM
"In spite of our progress in understandingleptin resistance and obesity, critical questions remainunanswered. In particular, how does elevated leptin lead toleptin resistance? Although both leptin receptors and leptin
signaling are down regulated by chronically elevated leptin,the degree of down-regulation of either component isinsufficient to account for largely absent leptin responses.Could selective leptin resistance in the CNS explain thisdiscrepancy? Emerging evidence suggests that leptinsignaling is preferentially reduced in the arcuate nucleus ofthe hypothalamus and not in other regions such as theventromedial, dorsomedial and/or premammilary nucleusof the hypothalamus that also express leptin receptors (49).By what mechanism(s) is the leptin resistance prevented inthese other nuclei? If we can understand the nature ofleptin resistance and develop new ways to reverse thatresistance, we can unleash the potent fat reducing potentialof leptin, and leptin may once again referred to as the “antiobesityhormone”.
A study from 2007 kinda articulates difference between circulating vs ARC nucleus levels in modulation of adiposy.
These datademonstrate that chronic vanadium administrationenhances the weight-reducing effects of centrallyadministered leptin in lean animals. The underlying
mechanism appears to involve enhanced hypothalamicleptin signal transduction.
10-19-2011, 07:19 PM
10-19-2011, 07:44 PM
10-20-2011, 05:27 PM
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