AP and DCP

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  1. Now from what I have read, TTA acts as a PPRAa activator in the liver making it a fat burning machine. The question is, how will fat be liberated to be fed into the furnace when DCP is used? I've looked at Omega Sports Burn3d which also looks solid because it adds in Green Tea and Forskolin to free up our fatty acids to be burnt, but I opted for DCP because of its other supporting goodies. So wouldn't something like cAMP, which is on sale, be more synergistic than just another PPRA activator like sesamin in a thermonuculear fat destoying stack?

    P.S. I have 2 DCP, 2 AP, and 3 Camp I'm planning to stack in the comming months. I will probably do a log, or at least a review. Is there a limit to the amount of time one can stay on cAMP?

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    Quote Originally Posted by saludable24
    Now from what I have read, TTA acts as a PPRAa activator in the liver making it a fat burning machine. The question is, how will fat be liberated to be fed into the furnace when DCP is used? I've looked at Omega Sports Burn3d which also looks solid because it adds in Green Tea and Forskolin to free up our fatty acids to be burnt, but I opted for DCP because of its other supporting goodies. So wouldn't something like cAMP, which is on sale, be more synergistic than just another PPRA activator like sesamin in a thermonuculear fat destoying stack?

    P.S. I have 2 DCP, 2 AP, and 3 Camp I'm planning to stack in the comming months. I will probably do a log, or at least a review. Is there a limit to the amount of time one can stay on cAMP?
    I agree and I would add about 200mg caffiene to my cAMP pre workout.
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  3. One thing that I have been pondering regarding the combination of DCP and AP is the PPAR effects.

    TTA is a strong activator of PPAR alpha, but also a mild agonist of ppar beta and a weak agonist of ppar gamma (in other words, it activates all the PPAR receptor subtypes but in the order of alpha > beta > gamm).

    AP is said to be antagonistic to PPAR gamma.

    I'm wondering if combining the two eliminates the PPAR gamma stimulation (that is, equal agonism and antagonism) or where exactly the happy medium between AP and DCP is regarding gamma stimulation (is AP a stronger antagonist than TTA is an agonist? Do we really know?).

    Not really asking for clarification unless this information is known, but something I was thinking about.
  

  
 

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