Any Waxy Maize Starch(WMS) data out there?

Guest

Guest
The reason for the use of WMS is faster digestion which should lead to a larger insulin response.

Any data out there?

WMS should signal a larger insulin release then dextrose.
 
ironman0370

ironman0370

New member
Awards
0
Here are a couple from Wilson6 over at mayhem. I'll check for more.

Title: The influence of starch structure on glycogen resynthesis and subsequent cycling performance.

Jozsi, A C; Trappe, T A; Starling, R D; Goodpaster, B; Trappe, S W; Fink, W J; Costill, D L

International journal of sports medicine. vol. 17, no. 5 (1996 Jul): 373-8.

Abstract: The present study was designed to evaluate the influence of starch structure on muscle glycogen resynthesis and cycling performance. Eight male cyclists (22 +/- 1 yr) completed an exercise protocol (DP) to decrease vastus lateralis glycogen concentration. This exercise consisted of 60 min cycling at 75% VO2max, followed by six 1-min sprints at approximately 125% VO2max with 1 min rest intervals. In the 12 hr after the exercise each subject consumed approximately 3000 kcal (65:20:15% carbohydrate, fat and protein). All of the carbohydrate (CHO) consumed was derived from one of four solutions; 1) glucose, 2) maltodextrin (glucose polymer), 3) waxy starch (100% amylopectin), or 4) resistant starch (100% amylose). Muscle biopsies were taken from the vastus lateralis muscle after DP and 24 hr later to determine glycogen concentrations. A 30 min cycling time trial (TT) was performed following the 24 hr post-DP muscle biopsy to examine the influence of the feeding regimen on total work output. The post-DP glycogen concentrations were similar among the four trials, ranging from 220.3 +/- 29.2 to 264 +/- 48.3 mmol.kg-1 dry weight (d.w.) muscle. Twenty-four hours after DP, muscle glycogen concentration had increased less (p < 0.05) in the resistant starch trial (+90.8 +/- 12.8 mmol.kg-1 d.w.) than in the glucose (+197.7 +/- 31.6 mmol.kg-1 d.w.), maltodextrin (+136.7 +/- 24.5 mmol.kg-1 d.w.) and waxy starch (+171.8 +/- 37.1 mmol.kg-1 d.w.) trials. There were no differences in total work output during the TT, or blood lactate concentration immediately following the TT in any of the CHO trials. In summary, glycogen resynthesis was attenuated following ingestion of starch with a high amylose content, relative to amylopectin or glucose; however, short duration time trial performance was unaffected.

The effects of pre-exercise starch ingestion on endurance performance.

Goodpaster, B H; Costill, D L; Fink, W J; Trappe, T A; Jozsi, A C; Starling, R D; Trappe, S W

International journal of sports medicine. vol. 17, no. 5 (1996 Jul): 366-72.

Abstract: This study compared the physiological responses and performance following the ingestion of a waxy starch (WS), resistant starch (RS), glucose (GL) and an artificially-sweetened placebo (PL) ingested prior to exercise. Ten college-age, male competitive cyclists completed four experimental protocols consisting of a 30 min isokinetic, self-paced performance ride preceded by 90 min of constant load cycling at 66% VO2max. Thirty min prior to exercise, they ingested 1 g.kg-1 body weight of GL, WS, RS, or PL At rest, GL elicited greater (p < 0.05) serum glucose and insulin responses than all other trials. During exercise, however, serum glucose, insulin, blood C-peptide and glucagon responses were similar among trials. The mean total carbohydrate oxidation rates (CHOox) were higher (p < 0.05) during the GL, WS, and RS trials (2.59 +/- 0.13, 2.49 +/- 0.10, and 2.71 +/- 0.15 g.min-1, respectively) compared to PL (2.35 +/- 0.12 g.min-1). Subjects were able to complete more work (p < 0.05) during the performance ride when they ingested GL (434 +/- 25.2 kj) or WS (428 +/- 22.5 kj) compared to PL (403 +/- 35.1 kj). They also tended to produce more work with RS ingestion (418 +/- 31.4 kj), although this did not reach statistical significance (p < 0.09). These results indicate that preexercise CHO ingestion in the form of starch or glucose maintained higher rates of total carbohydrate oxidation during exercise and provided an ergogenic benefit during self-paced cycling.
 
thesinner

thesinner

Recovering AXoholic
Awards
2
  • Legend!
  • Established
The reason for the use of WMS is faster digestion which should lead to a larger insulin response.

Any data out there?

WMS should signal a larger insulin release then dextrose.
Insulin release is controlled by the pancreas, not by the stomach or intestines. It is not how fast serum concentrations of the carbohydrate increase (through increased rate of digestion), but how much this concentration affects insulin release. You see what I'm getting at?
 
dsade

dsade

NutraPlanet Fanatic
Awards
4
  • RockStar
  • Legend!
  • Established
  • First Up Vote
Colby is running some glucometer testing on WMS. Not sure if he has his data compiled and analyzed yet.
 

Guest

Guest
Insulin release is controlled by the pancreas, not by the stomach or intestines. It is not how fast serum concentrations of the carbohydrate increase (through increased rate of digestion), but how much this concentration affects insulin release. You see what I'm getting at?
I'm fully aware that insulin is secreted by the pancreas.

You would think faster digestion equals higher levels of blood glucose which leads to insulin release from the pancreas. This is the basic principle of simple versus complex carbohydrates.

you get faster glycogen resynthesis through faster glucose Facilitation by translocating GLUT4 with insulin.
 
pmiller383

pmiller383

Well-known member
Awards
1
  • Established
Sounds like your up to something Jacob
 

Similar threads


Top