Higher ArA dosage, or ArA stack?

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  1. Quote Originally Posted by bolt10 View Post
    It'd be more beneficial to add the GMS and Carnitine IMO.
    This right here. I use Compete and Hemavol with my X-gels everytime I run ARA.


  2. Interested in tho as I was considering this on my next run
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  3. Quote Originally Posted by EBF Inc View Post
    Go with a 5-lpo inhibitor and hca for the next time you use it with GMS and carnitine.

    Muscular uptake and amount of ara In the stem would be high,
    How about Lodhra Bark (symplocos racemosa extract) as a lipoxygenase inhibitor?

    http://www.ncbi.nlm.nih.gov/pubmed/15938197

    If so ArA + Anabeta elite + metabolic powder might nake for a good stack
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  4. So your saying use AA and anebeta elite together with alcat
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  5. Alcar
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  6. Quote Originally Posted by laneanders View Post
    This right here. I use Compete and Hemavol with my X-gels everytime I run ARA.
    Great combo. Do you typically add any bulk GMS?

  7. Quote Originally Posted by cheftepesh1 View Post
    So your saying use AA and anebeta elite together with alcat
    Along with HCA which, as @EBF Inc had pointed out a couple post back, may help with cellular uptake
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  8. Quote Originally Posted by aaronuconn View Post
    Great combo. Do you typically add any bulk GMS?
    Hes an Iforce minion lol
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  9. Quote Originally Posted by cheftepesh1 View Post
    So your saying use AA and anebeta elite together with alcat
    They would stack very well
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  10. Quote Originally Posted by EBF Inc View Post
    Go with a 5-lpo inhibitor and hca for the next time you use it with GMS and carnitine.

    Muscular uptake and amount of ara In the stem would be high,
    Interested how you'd set this up?

  11. Quote Originally Posted by BPjohn123 View Post
    They would stack very well
    Yup. As pointed out by @EBF Inc

    Quote Originally Posted by EBF Inc View Post
    COX and LPO breakdown the ArA into the luekotrienes and thromboxanes

    Inhibiting them (5-LPO and COX-2 inhibitor) leaves more Arachidonic acid available for uptake into muscle cells. This would be a good thing.
    So when we take ArA some of it gets broken down into luekotrienes and thromboxanes via LPO and COX. Now if we could inhibit both of them it would result in more ArA to stay which meams there is more that could be used by our muscles

    So the proposed above stack would inhibit ArA being broken down into thromboxanes via l-carnitine ( http://ajpheart.physiology.org/content/284/1/H41) and inhibit ArA being broken down into luekotrienes via anabeta elite (http://www.ncbi.nlm.nih.gov/pubmed/15938197)

    So l-carnitine and anabeta elite would create the enviroment for optimal available ArA.

    Now also, as pointed out by @EBF Inc

    Quote Originally Posted by EBF Inc View Post
    This may also be where garcinia Cambogia (50% hca) can play into effect in the cellular uptake but via a different mechanism (CD36).
    So we add HCA to help all that now extra ArA to be able to get used by our muscles

    Hence the stack of ArA + Anabeta Elite + l-carnitine + metabolic powder
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  12. Quote Originally Posted by JudoJosh View Post

    Hence the stack of ArA + Anabeta Elite + l-carnitine + metabolic powder

    And so you'd reccomend this all pre-workout? Aren't ABE & MP meant to be taken with food?

  13. Quote Originally Posted by MultiVitamin View Post
    And so you'd reccomend this all pre-workout? Aren't ABE & MP meant to be taken with food?
    ABE is recommended pre-workout. MP is also recommended pre-workout. Both can be taken with or without food.
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  14. Quote Originally Posted by kbayne View Post
    ABE is recommended pre-workout. MP is also recommended pre-workout. Both can be taken with or without food.
    Okay thanks man

  15. ...
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  16. That's from this thread son!

  17. oops,my bad
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  18. Quote Originally Posted by aaronuconn View Post
    How about just the leucine then...
    Yes, that would be fine.

    Quote Originally Posted by EBF Inc View Post
    Can you give a partial or full explanation as I'm curious as well
    Short on time, but I will get into it later if I remember. I'm sure Cyrus or someone else can explain if they have time (assuming they're asked nicely ).

  19. Quote Originally Posted by Synapsin View Post
    Yes, that would be fine.



    Short on time, but I will get into it later if I remember. I'm sure Cyrus or someone else can explain if they have time (assuming they're asked nicely ).
    I'll be sending constant messages.

  20. Quote Originally Posted by Synapsin View Post

    Yes, that would be fine.

    Short on time, but I will get into it later if I remember. I'm sure Cyrus or someone else can explain if they have time (assuming they're asked nicely ).
    I actually just read a study while I was searching showing some sort of blunted effects in the presence of insulin maybe on the prostaglandin formation. **** I wish I remembered

  21. From what I can tell from reading information Neuron has posted:

    arachidonic acid --> triglyceride --> chylomicron --> interaction with enzyme Lipoprotein Lipase (LPL) --> "only isoform of LPL that is active with insulin is located on adipocytes".

  22. Quote Originally Posted by aaronuconn View Post
    From what I can tell from reading information Neuron has posted:

    arachidonic acid --> triglyceride --> chylomicron --> interaction with enzyme Lipoprotein Lipase (LPL) --> "only isoform of LPL that is active with insulin is located on adipocytes".
    Did he say that last statement? Both forms are active but the adipose form predominates with insulin

  23. Quote Originally Posted by mr.cooper69 View Post
    Did he say that last statement? Both forms are active but the adipose form predominates with insulin
    It's from his first post on the Maximizing AA to skeletal muscle on bb.com.

  24. Here's the entire first post from Neuron on the aforementioned thread:

    "I made a cursory glance into the X-Factor thread and I didn't see any suggestion on when to take X-Factor, or other Arachidonic acid products, so here is my suggestion.

    Background
    After ingestion, fatty acids like arachidonic acid are emulsifed and taken up into enterocytes where they are esterfied on a glycerol backbone to form a triglyceride. Next, the triglycerides and cholesterol esters are bound together to form a chylomicron. This entity can now diffuse into the lymphatics and then into the blood. Once in the blood, the chylomicrons are free to interact with various tissue receptors depending on energy homeostasis. When needed, they can interact with an enzyme called Lipoprotein Lipase (LPL) that will hydrolyze the triglycerides back into fatty acids to be used as fuel or for storage purposes.

    The point being...
    In which tissue the fatty acids are deposited is a function of the energetic state of the organism overall. In a post-prandial, high fed, state, the dominant hormone is insulin. Similarly, the only isoform of LPL that is active with insulin is located on adipocytes. It should be obvious now that consuming fats when eating will direct them to adipose. This isn't necessarily a bad thing in the context of fish oil, as adipose is now recognized as a great contributer to endogenous hormone production like the prostanoids.

    But...
    In the context of Arachidonic Acid, the final destination should be skeletal muscle. Luckily, there is an energetic state which favors fatty acid delievery to this tissue: Exercise (or acute starvation) [1]. In an exercise-induced environment, LPL on skeletal muscle becomes active which serves as an adjunct to glycolysis for the TCA cycle via beta-oxidation. Similarly, the enzyme DGAT also becomes active in skeletal muscle which functions to store excess fatty acids as triglycerides. Since both of these enzymes are heavily active during exercise, it is clear that an excess of fatty acids are being removed from blood plasma into skeletal muscle as a reservoir (this also serves to decrease insulin insensitivity [2]).

    Based on these observations
    To maximize ArA supplemention it is advisable to supplement 30-40 minutes prior to exercise in order to ensure plasma delievery of ArA and also to reduce exercises effects on gastric motility."

    [1] http://ajpendo.physiology.org/cgi/co...act/268/2/E229
    [2] http://ajpendo.physiology.org/cgi/co...act/294/5/E882

  25. Happy to hear that the stack I'm starting in a couple of weeks should be pretty kick ass.

    ArA + ABE + GMS + LCLT. (maybe Amino IV, too)

  26. Yeah, I make sure to take 1.5gms at least 45 minutes to an hour before I work out.

  27. Quote Originally Posted by doggy_dog View Post
    Happy to hear that the stack I'm starting in a couple of weeks should be pretty kick ass.

    ArA + ABE + GMS + LCLT. (maybe Amino IV, too)
    I'm doing the same stack, starting in mid Feb
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  28. Quote Originally Posted by doggy_dog View Post
    Happy to hear that the stack I'm starting in a couple of weeks should be pretty kick ass. ArA + ABE + GMS + LCLT. (maybe Amino IV, too)
    Throw some Agmatine in too
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  29. Quote Originally Posted by Misfit28 View Post
    Throw some Agmatine in too
    Pumps would hurt too much lol (kinda srs)

  30. Quote Originally Posted by kbayne View Post
    Pumps would hurt too much lol (kinda srs)
    Lol, may get counterproductive.
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