If you had to use one or the other, which you would rather use for PCT for a cycle.
Yes this the steroid section and there is a PCT section but 97 viewing compared to 4 I hope to get a better response here.
this poll is designed just for these two over a discussion between the two
6-bromo can convert to an androgen, so its ATD for me
I'd doubtfully ever use an OTC AI for PCT but if I did the only one I'd likely choose is 6-OXO. If I HAD to choose between these 2 though it'd be 6-Bromo.
6-Bromo would likely only act as a PH if used at 3-4 times the recommended dose. Just like almost any steroidal AI - formestane, exemestane, all will act like a PH at doses much higher than recommended. But they'll all also act as a good AI at the lower recomended doses. As for ATD I'd never even consider using it. Blocking too much estrogen is just plain bad and ATD does just that.
Plus ATD being an anti-androgen, eliminates males libidos. Also the point of PCT is to restore natural test, by increase luteinizing hormone (LH) and follicle-stimulating hormone (FSH). In clinical tests ATD was shown to decrese testes size and decrease LH - the exact opposite thing you need to happen during PCT.
it would be really helpful if you could find those tests for me that show the declines
what I meant to say is that I don't fell comfortable on taking a compound (on pct)that can act like an androgen in the body , as a anabolic I believe its a good product but as a test booster I still have my doubts and I still haven't seen studies showing me that PA is wrong
I have a question regarding those studies, does lh and fsh decrease since the beginning of the test?
I ask cause if it only decreases in the end I believe it makes sense cause the hypothalamus will fell a major increase in the testosterone values sending signals to decrease lh and fsh, but in the first days I think they probably rise cause test increase a lot.
The big problem with atd is that it binds to ar`s in the brain that manage libido , when atd bind to them dht isn't capable to do the same sending libido to the floor.
I believe that lower atd doses will not bring this problem so with ai`s we always have the same problem , too much is not too good.
By the way I agree with the 6-oxo
I prefer 6-OXO. ATD is too rough on me...
ATD works as an AI to block estrogen (actually blocks too much in reality), but increasing LH, FSH, and natural production of test in the testes in crucial during PCT for proper recovery and future health.
In this study, 1,4,6-androstatriene-3,17-dione (ATD) was demonstrated to cause time-dependent loss of aromatase activity in rat ovarian microsomes in vitro. In vivo, an injection of ATD caused inhibition of ovarian aromatase and reduced estrogen secretion in pregnant mare's serum gonadotropin-primed rats for at least 24 hr after injection. In rats with 7,12-dimethylbenz[a]anthracene-induced, hormone-dependent, mammary tumors, marked regression occurred with ATD treatment. Although estrogen secretion was not reduced below the diestrus level of controls, the rats remained anestrus, indicating that the proestrus surge of estrogen was prevented. LH, FSH and prolactin levels were also basal and LH and FSH did not rise after ovariectomy. ATD had no detectable hormonal activity in bioassay. Consistent with this, the compound did not interact appreciably with either androgen or estrogen receptors, was not uterotrophic, and did not interfere with mammary tumor regression in ovariectomized rats. Thus, the major activities of the compound which cause mammary regression in the rat appear to be inhibition of estrogen synthesis, via aromatase and gonadotropin suppression.
i wouldn't touch either..
From my experience, a mild, tapered dose of ATD (50mg/day down to 25mg/week) is highly effective at eliminating gyno, while maintaining libido.