Yeah man australian is right, you have to have a serm, clomid will do otherwise, i actualy prefer it with a dht compounds, but a serm isnt an option, its a must, if you do a 6 week bridge, you need lower dosages than normal.
some info for you...
While practically similar compounds in structure, few people ever really consider
Clomid and nolva to be similar. Its not just a common myth in steroid circles,
but even in the medical community. This misconception originates from their
completely different uses. Nolvadex is most commonly used for the treatment
of breast cancer in women, while Clomid is generally considered a fertility
aid. In bodybuilding circles, from day one, Clomid has generally been used as
post-cycle therapy and Nolvadex as an anti-estrogen.
But as I intend to demonstrate this is in essence the same. I believe the myth
to have originated because nolva is clearly a more powerful anti-estrogen, and
the people selling Clomid needed another angle to sell the stuff, so it was
mostly used as a post-cycle aid. But few users really understand how Clomid
(and also Nolvadex, logically) works to bring back natural testosterone in the
body after the conclusion of a cycle of androgenic anabolic steroids. After
a cycle is over, the level of androgens in the body drop drastically. The body
compensates with an overproduction of estrogen to keep steroid levels up. Estrogen
as well inhibits the production of natural testosterone, and in the period between
the return of natural testosterone and the end of a cycle, a lot of mass is
lost. So its in everybody's best interest to bring back natural test as soon
as humanly possible. Clomid and Nolvadex will reduce the post-cycle estrogen,
so that a steroid deficiency is constated and the hypothalamus is stimulated
to regenerate natural testosterone production in the body. That's basically
how the mechanism works, nothing more, nothing less.
Both compounds are structurally alike, classified as triphenylethylenes. Nolvadex
is clearly the stronger component of the two as it can achieve better results
in decreasing overall estrogen with 20-40 mg a day, than Clomid can in doses
of 100-150 mg a day. A noteworthy difference. Triphenylethylenes are very mild
estrogens that do not exert a lot, if any activity at the estrogen receptor,
but are still highly attracted to it. As such they will occupy the receptor
and keep it from binding estrogens. This means they do not actively work to
reduce estrogen in the body like Proviron, Viratase or arimidex would (by competing
for the aromatase enzyme), but that it blocks the receptor so that any estrogen
in the body is basically inert, because it has no receptor to bind to.