17-Methyl 1-Test

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  1. 17-Methyl 1-Test


    http://www.vialsandcrimpers.com/page20.html

    What are your guys thoughts on this? I saw curt post something about methylated 1-test and was wondering if this is what he was talking about?


  2. Probably a killer on the liver.
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  3. Originally posted by windwords7
    Probably a killer on the liver.

    Probably. But it will be orally active.

  4. im sold

  5. I may get some, not sure yet. It could be hard on the liver yes, but I'm thinking with some Milk Thistle, vitamin C, and r-ala....down the hatch!!
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  6. This is why ph's are probably going to be banned. Isnt that the same oral deliver as d-bol? If so why wouldnt you just go with transdermal?

  7. ????????? Why go with transdermal if the price is right? We are all about saving money, would be harder on the liver as most orals are but you'd only run it for 4 weeks or so anyways. Ph's have been on the brink of getting band for a long time, all these companies are coming out with different versions to milk it for what its worth for the time being. I don't think this would have any outcome on the banning of ph's IMO.

  8. its orally active and easier than rubbing a gel on urself 2x a day

  9. I would need to see the hepatic toxicity levels before even considering it. There are somethings that are just not worth the negatives that they can produce. Their is little reason to deviate from the bread and butter of either the PH or AAS sides of life ever. The basics will make you grow and keep you healty as long as you dont screw up!

  10. WHo said I didnt like saving money? I have a lifestyle to maintain here. Anyways if I were going to take a ph that damages my liver as much as d-bol......Id rather just take d-bol. WHy wouldnt you just up the grams of 1-test in a transdermal and get the same effects? I know rubbing on a trans twice a day gets old after a while, but its usually only for 4 weeks, and is way less stressful on your liver. But then again I dont have a BA in prohormoneology, so I could be wrong.

  11. Originally posted by windwords7
    I would need to see the hepatic toxicity levels before even considering it. There are somethings that are just not worth the negatives that they can produce. Their is little reason to deviate from the bread and butter of either the PH or AAS sides of life ever. The basics will make you grow and keep you healty as long as you dont screw up!
    What oral AAS has shown to be hepatoxic?
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  12. Originally posted by Jedi Master
    WHo said I didnt like saving money? I have a lifestyle to maintain here. Anyways if I were going to take a ph that damages my liver as much as d-bol......Id rather just take d-bol. WHy wouldnt you just up the grams of 1-test in a transdermal and get the same effects? I know rubbing on a trans twice a day gets old after a while, but its usually only for 4 weeks, and is way less stressful on your liver. But then again I dont have a BA in prohormoneology, so I could be wrong.
    Yes you are wrong. Show me one study showing D-bol to be hepatoxic when used properly? Actually better yet show me one study using D-Rol that shows it to be hepatoxic.

    You people need to stop spreading these bull**** myths about your liver failling out and how bad orals are on your liver. It has no basis whatsoever.
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  13. Originally posted by Bobo


    What oral AAS has shown to be hepatoxic?
    Some things are harder than others on the ole' liver. I would like to see where 17AA 1T falls in this spectrum.

  14. Originally posted by Jedi Master
    WHo said I didnt like saving money? I have a lifestyle to maintain here. Anyways if I were going to take a ph that damages my liver as much as d-bol......Id rather just take d-bol. WHy wouldnt you just up the grams of 1-test in a transdermal and get the same effects? I know rubbing on a trans twice a day gets old after a while, but its usually only for 4 weeks, and is way less stressful on your liver. But then again I dont have a BA in prohormoneology, so I could be wrong.
    Yeah, but many use ph's for the legal alternative. D-bol also is a different drug alltogether, 1-test is cleaner for gains etc.

  15. Originally posted by Bobo
    You people need to stop spreading these bull**** myths about your liver failling out and how bad orals are on your liver. It has no basis whatsoever.
    Agreed bobo.

  16. Originally posted by Bobo


    You people need to stop spreading these bull**** myths about your liver failling out and how bad orals are on your liver. It has no basis whatsoever.
    Define you people....

  17. Originally posted by windwords7


    Some things are harder than others on the ole' liver. I would like to see where 17AA 1T falls in this spectrum.
    Some things are harder on the liver because of the doses used. D-Rol is harder because you take 100-150mg/day. It depends on the molecular weight. THere isn't some magic property of D-rol that makes it more toxic.
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  18. Originally posted by windwords7


    Define you people....
    People that claim orals are hepatoxic and that orals cause liver damage. They stress the liver more just like alcohol and a million other things. Abuse cause liver damage, not some magic property of oral steroids.
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  19. All I'm saying is that some things are harder on the body than others and yes its dosage dependant. Drink in moderation and your fine, drink in excess and you can damage yourself. HOWEVER, drinking excess in beer is NOT as likely to cause damage than driking excess of Jack. That's what I mean by spectrum of ability to cause damage.

  20. They are harder on the body because of the amount. Compare the amount of Jack you do compared to the amount beer. So claiming 1-test is harder on the liver isn't accurate because the dosage needed is less. There isn't one case of hepatoxicity of orals steroids. The only remotely close is a 14yr old girl taking 100's of mg of D-rol for some disease she had. And she took if for years and had preexisting liver problems. AIDS patients use orals everyday (var) so if it was that bad, they wouldn't be taking it.


    ""here is our discusion with a gifted chemist :

    >do you think liver toxicity of orals has to do with number of >molcules or each
    oral has different toxicity rate regardless of >number of molcules ?

    the amount of liver stress is equal for any
    steroid, but not with respect to milligrams, but with respect to molecules. You
    know, every different steroid has a slightly different molecular weight, and
    this you will have to take into account. Furazabol, OT and stanozolol are MUCH
    heavier per molecule than methyltestosterone, for example, so 50mg of OT are
    less toxic than 50mg of Methyltest. See, the always-claimed extreme toxicity of
    oxymetholone is mainly due to the high doses that are commonly taken - most
    athletes use 100-150mg per day, whereas 25-30mg of DBol are a common dosage.

    >i mean is 50 mg of oxemtheolone less toxic than 50 mg of >methandienone ?

    Their molecular weight it quite similar, so you can compare liver toxicity of
    both.

    >also do you know anything on methlytrienbolone ? i heard dose is >only 2 mg ?

    No, effective dose is 5-15 mg a day. For mibolerone (cheque drops) it is much
    less as this is only used for mental effects (makes you extremely aggressive;
    Mike Tyson is said to have it used before the ear-bite).

    >how is it possible Oxandrolone is sythesised outside of liver >and less toxic
    as said by grundig in his book ? i think its not true because i can not find any
    info backing this and research on aids patients by Medline say its more toxic
    than oxymetholone on per mg basis ..

    What Grundig says is definitely wrong. I donīt know about liver toxicity of
    oxandrolone exactly, as oxandrolone has a modified steroid backbone and is
    therefore metabolized by a different pathway, I think."
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  21. Originally posted by Bobo


    People that claim orals are hepatoxic and that orals cause liver damage. They stress the liver more just like alcohol and a million other things. Abuse cause liver damage, not some magic property of oral steroids.
    Please get on some Clomid, go watch Beaches, have a good cry, and calm down!!

  22. I said its dosage dependant within reason.

  23. Originally posted by windwords7


    Please get on some Clomid, go watch Beaches, have a good cry, and calm down!!
    Don't yell at me because you didn't know the facts. Its people that don't understand these principle that perpetuate the myths. Don't be one of those people.
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  24. I understand it, but heres what it boils down to Winny, Dbol, I'm down for that. Methyl test, Methyl tren, no way, not ever.

    I never said you should not use orals or that they were more dangerous than any other steriod. FACT all steriods can be dangerous. Some more so than others because some it takes less to **** up with and/or a body that is already weakend by some deficency unknown to the user is likey to reap serious consequences using certain drugs over others.

    That's why Dr's don't prescribe you Serzone if your known to have a liver issue or have elevated enzymes. It's not that other AD's wont effect the liver but they wont effect the liver like Serzone can/will. All drugs that have to be broken down by liver enzymes (which is of course all of them) are potentialy hard on the liver but some are harder than others due to the amount of work the liver has to do.

  25. Here's that study. I got the number wrong on the dosage but she took it for years.

    Multiple hepatic adenomas caused by long-term administration of androgenic steroids for aplastic anemia in association with familial adenomatous polyposis.

    Nakao A, Sakagami K, Nakata Y, Komazawa K, Amimoto T, Nakashima K, Isozaki H, Takakura N, Tanaka N.

    Department of Surgery, Shobara Red Cross Hospital, Japan.

    We report a rare case of hepatic adenomas (HA), in a 20-year-old Japanese girl treated for 6 years with anabolic androgens for aplastic anemia. In a review of the world literature using computer MEDLINE search, we found only 17 cases of androgen-induced HA published between 1975 and 1998 in the English-language literature. The patient was referred to us because of liver lesions detected during a follow-up examination for familial adenomatous polyposis. After being diagnosed with aplastic anemia at 14 years of age, she had been treated with oxymetholone (30 mg/day) for 6 years. Laboratory evaluation revealed normal liver function. Ultrasonography (US) and computed tomography (CT) demonstrated multiple liver lesions. Histopathological examinations of biopsied specimens from the liver tumor showed HA. After the patient was diagnosed with HA, oxymetholone was tapered off. Patients taking androgenic-anabolic steroids should be carefully monitored with US and CT and tumor markers should be measured. This report may be helpful in identifying the population who is at risk of developing hepatic sex hormone-related tumors.
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